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1 arides, but the molecular basis for this increased activity is not known.
2                                     The pathogenesis of ASD is not known, but it involves activation of microglia.
3 tionally repair damaged neural circuitry in the adult brain is not known.
4 lling VEGF A and FGF 2 signaling in the CL of water buffalo is not known.
5  therapeutic target in cancer, yet its role in immune cells is not known.
6  of locomotion-related VIP neuron activity to visual coding is not known.
7 between the HPC and mPFC for memory transfer/consolidation, is not known.
8 esemble common fragile sites (CFSs)-but how they are formed is not known.
9 ications of ACTN4 on podocyte integrity and kidney function is not known.
10 gh its role in the regulation of their physiologic function is not known.
11 itable for obtaining molecules with any type of heteroatoms is not known so far.
12  generation of invariant gammadelta T cells occur in humans is not known.
13                                                          It is not known how activating ESR1 mutations may alter the pred
14                                                          It is not known if murine leukemia virus (MLV) encodes a Vif-lik
15                                                          It is not known if they are present before the onset of psychosi
16                                                          It is not known if use of colloid solutions containing hydroxyet
17                                                          It is not known what proportion of United States patients with g
18                                                          It is not known whether final adult height will be increased, or
19                                                          It is not known whether selectins induce similar signaling event
20                                                          It is not known whether such neuroadaptations are affected by nu
21 n succinoglycan intervenes in the symbiotic process, and it is not known whether the plant lysin-motif receptor-like kina
22  chronicity of pruritus are not yet fully understood and it is not known whether these mechanisms diverge between differe
23 ng waves have been reported during anaesthesia(2-7), but it is not known whether they have a role during waking perceptio
24                                             Furthermore, it is not known which components of psychological interventions
25                                                 However, it is not known what controls the formation and templated seedin
26                                                 However, it is not known when succinoglycan intervenes in the symbiotic p
27  to a greater risk of developing schizophrenia; however, it is not known whether C4 plays a causative role in this brain
28                                                 However, it is not known whether IAP induces autophagy.
29                                                 However, it is not known whether physical properties such as cellular sti
30                                                 However, it is not known whether there is a causal relationship between t
31 has been reported to allosterically activate the lipase, it is not known whether Rap1A has the same ability or what its m
32 but whether they can accurately predict health and lifespan is not known.
33  ready (AdoCbl) or not [cob(II)alamin] for transfer to MCM, is not known.
34 in's transcriptional function, but the underlying mechanism is not known.
35 GP2 domain-mediated fusion of viral and endosomal membranes is not known.
36 eir influence on hyperfine quenching in a reactant molecule is not known.
37 ges, the stage at which this cortical feedback first occurs is not known.
38 ter medullary collecting duct from the inner stripe (OMCDi) is not known.
39 siologic significance of low levels of detectable C-peptide is not known.METHODSWe studied 63 adults with type 1 diabetes
40 owever, the underlying mechanism for the class II phenotype is not known.
41 n (PA) for OPAT, yet the impact of the inpatient PA process is not known.
42 f so how these N-glycans contribute to monocyte recruitment is not known.
43 , and whether they contribute to atherosclerosis regression is not known.
44 is is regulated to fulfill these physiological requirements is not known.
45 t al., 2019); however, the mechanism of substrate selection is not known.
46 ever, the mechanism for stimulating beta-arrestin signaling is not known, making it difficult to design optimum biased li
47        However, the basis of DGAT2's acyl-donor specificity is not known because of the inherent challenges of predicting
48 l and acentrosomal microtubule formation, yet its structure is not known.
49 , but how microcephaly is linked to dysfunctional telomeres is not known.
50  a safer profile; however, its neuroprotective role in vivo is not known.The goal of this study was to examine the effect