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1                                                 This action is probably due to disengagement of Smc1-Smc3 heads prompted
2  progression toward autoimmune insulin deficiency in adults is probably due to a lower genetic load overall combined with
3 vative, XAC-X-BY630, revealed that the decrease in affinity is probably due to a significant increase in dissociation rat
4 re, the emergence of bone marrow failure in Fanconi anaemia is probably due to aldehyde-mediated genotoxicity restricted
5        This effect is sex- and oestrous cycle-dependent and is probably due to an oestrous cycle-dependent change in the
6 th predicted in vitro tropism of SIVrcm for macrophages and is probably due to expansion of coreceptor usage.
7 g a variety of blockers we determined that the augmentation is probably due to insertion of GABA(A)Rs into the synapse by
8         A synthetic experiment indicates that this behavior is probably due to dimerization.
9 tidis pfs and luxS mutants is not due to quorum sensing but is probably due to metabolic imbalance and, in the case of pf
10    The smaller amount of the FMO protein in the mutant cell is probably due to the degradation of the apo-FMO protein at
11 fold selectivity of PAP-1 for Kv1.3 over other Kv1 channels is probably due to its preferential affinity to the C-type in
12 epancy between observed and expected nitrite concentrations is probably due to a combination of factors, including an inc
13                                          Their conservation is probably due to the necessity for interaction of proximall
14                                                   This cost is probably due to the adverse effects of autoimmunity and cu
15                                             This difference is probably due to the fruiting of mycorrhizal fungi partly d
16                                 The insignificant RH effect is probably due to the weak interaction between water and squ
17                                                  This event is probably due to the tidal disruption of a star falling int
18 d H4K12 acetylation that occurs following acrolein exposure is probably due to the formation of acrolein-histone lysine a
19 yed temporal response between RLC phosphorylation and force is probably due to mechanical effects associated with elastic
20 di-GMP to form aggregates (as compared to that of c-di-GMP) is probably due to a higher activation barrier to convert fro
21 erence in Lisch nodule burden between light and dark irides is probably due to the photoprotective effects of pigmentatio
22 retains its native globular structure, so its activity loss is probably due to loss of specific toxin-receptor residue pa
23  This discrepancy with the earlier studies using microsomes is probably due to the known accessibility of microsomal carb
24                               Overall, MSMD in this patient is probably due to autosomal dominant (AD) IFN-gammaR2 defici
25   The increased severity of the Lama1 null mutant phenotype is probably due to the complete loss of the inner limiting me
26                                           This reactivation is probably due to the increased driving force for Ca(2+) ent
27                                     The latter relationship is probably due to immunosenescence, because heart failure wa
28                                  Increased Nodal signalling is probably due to diminished expression of the Lefty1 and Le
29                               The reduction in nuclear size is probably due to a change in DNA heterochromatinization, as
30 association between high transcription and genome stability is probably due to a variety of factors including an enhanced
31  reduced in the nuclei of transfected cells, an effect that is probably due to a general defect in 3' RNA processing.
32 s markedly enhanced upon acetylation of K56, an effect that is probably due to increased conformational entropy of the al
33 re is large unexplained variability in cytokine levels that is probably due to genetic-environmental associations.
34                                                        This is probably due to a number of confounding factors, particula
35                                                        This is probably due to a range of factors, including contradictor
36                                                        This is probably due to better accessibility of gaseous mutagenic
37                                                        This is probably due to cluster and hydrogen bonds formation betwe
38                                                        This is probably due to deformation-induced alignment of MgSiO(3)-
39                                                        This is probably due to the fact that SML exchange between bilayer
40                                                        This is probably due to the greater number of scavenging reactions
41                                                        This is probably due to the poor ionization efficiency of the drug
42                                                        This is probably due to the superior movements of the wristed inst
43                                                        This is probably due to their high rigidity, which hinders some of
44 ha-Smooth Muscle Actin) upon treatment with TGF-beta1; this is probably due to intrinsic variability of cytokine concentr
45 ther than restricted developmental potential, however, this is probably due to a matter of timing.
46 antly reduced the N2 O production from these pathways; this is probably due to the observed reduction in the expression o
47                               Our results suggest that this is probably due to redundancy with Pax8 and that the function
48 gulator, and the recalcitrancy to transformation in At vip2 is probably due to the combination of muted gene expression r
49 ron and manganese remain mainly between +II and +III, which is probably due to short residence time of these particles in
50 tead of isolated pathways, may overcome this problem, which is probably due to the extreme heterogeneity of human tumors.

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