ライフサイエンスコーパス: is sufficient

1 in the strongly nonlinear turbulent regime, provided there is sufficient drag at the bottom of the atmosphere and ocean.

2 good school enrolment, we find that school-based treatment is sufficient for achieving EPHP.

3 ors are required to reduce flees from females and either AR is sufficient for attacking males.

4 memory and the criterion for deciding what memory strength is sufficient for identification.

5 ing delivery by virus-like particles (VLPs), that Vpr alone is sufficient for REAF degradation in primary macrophages.

6 ingly, the expression of the viral nucleoprotein (NP) alone is sufficient for the generation of IBs, indicating that it p

7 Considering that the N-terminal half of TIMP-1 is sufficient for TIMP-1's MMP-inhibitory activity, we propos

8 quires O(2) as the final electron acceptor, but O(2) supply is sufficient in most situations, including exercise and ofte

9 ever, further consideration is needed to determine if there is sufficient patient load to maintain such a service.

10 rates a uniform magnetic field gradient within a space that is sufficient to accommodate an organ-stage mouse embryo unde

11 IL-1alpha is sufficient to activate cultured fibroblasts and primary he

12 tion leads to reduced levels of the encoded proteins, which is sufficient to bring about disorganized mitochondrial netwo

13 voked release, suggesting that aberrant spontaneous release is sufficient to cause disease in humans.

14 errant expression of a testis-specific transcription factor is sufficient to co-opt somatic transcriptional machinery to

15 the catalytic domain of PDE3A in cancer cells lacking PDE3A is sufficient to confer sensitivity to DNMDP, and substitutio

16 d stably expressing oncogenic RasV12 in untransformed cells is sufficient to decrease pHlys.

17 Expressing the Epe1 C-terminus is sufficient to disrupt heterochromatin by outcompeting the

18 ling of the fusogenic ectodomain to branched actin assembly is sufficient to drive cell-cell fusion.

19 BFD1 accumulates during stress and its synthetic expression is sufficient to drive differentiation.

20 show that either flavin reduction or the binding of NADP(H) is sufficient to drive the FAD to the in conformation.

21 hit of the screen is the cytosolic Malic Enzyme (ME1), that is sufficient to enable survival and proliferation of CI muta

22 amin and colleagues demonstrates that mutant YAP expression is sufficient to enhance tumor cell dissemination in zebrafis

23 cy has been improved more than twofold and this improvement is sufficient to establish the dynamics of cell growth and ad

24 parsimonious mechanism that involves differential motility is sufficient to explain the spontaneous patterning of the ce

25 al mathematical model demonstrating growth factor signaling is sufficient to guarantee this robustness and which anticipa

26 Nevertheless, DNA damage is sufficient to induce activation of canonical p53 target ge

27 ds in both the plasma membrane and synthetic membranes, and is sufficient to induce extensive membrane tubulations.

28 These findings reveal that chronic p16(INK4a) expression is sufficient to induce hyperplasia through Wnt-mediated para

29 of the replication-competent Orsay virus RNA1 segment alone is sufficient to induce most of the IPR genes in a manner dep

30 antly, either systemic or neutrophil-specific Akt1 deletion is sufficient to inhibit metastasis of Akt-proficient tumors.

31 letion of the hyaluronic acid precursor UDP-glucuronic acid is sufficient to inhibit several mesenchymal-like properties

32 eurons that were activated during a previous bout of torpor is sufficient to initiate the key features of torpor, even in

33 ch beams can generate fluences exceeding 10 mJ/cm(2), which is sufficient to irreversibly inactivate most common pathogen

34 t not HDAC1, inhibition in the ventral tegmental area (VTA) is sufficient to normalize behavior.

35 odel replicative DNA polymerase to variously structured DNA is sufficient to predict the complex genomic behaviour of STR

36 xynojirimycin; MON-DNJ) capable of inhibiting Glu I in vivo is sufficient to prevent death in mice infected with lethal v

37 nctional homology, whereby CTIP2 or SATB2 of either species is sufficient to promote a subcerebral or commissural fate, r

38 that disruption of the normal bone marrow microenvironment is sufficient to promote leukemic transformation and survival

39 that selective engagement and activation of FZD7 signaling is sufficient to promote mesendodermal differentiation of hPS

40 show in mice that loss of C9orf72 from myeloid cells alone is sufficient to recapitulate the age-dependent lymphoid hype

41 ical Gli-dependent Hedgehog signaling by Gli1 gene transfer is sufficient to recover salivary function impaired by irradi

42 hat experimentally altering the timing of Hgf/Met signaling is sufficient to redirect axon targeting and disrupt the topo

43 tained long term, suggesting that latent epitope expression is sufficient to retain gB-CD8s.

44 Moreover, a subset panel of the 12 most powerful SNPs is sufficient to separate the two pure species, even when tis

45 PLB inhibition of SERCA, and binding of a single Ca(2+) ion is sufficient to shift the protein population toward a struct

46 ion of the paternally inherited copy of Dot1l in the embryo is sufficient to support development.

47 The repair is sufficient to support fertility and maintain health and ge

48 o pattern the cortex during the process of mitotic rounding is sufficient to translate interphase shape into a cortical p

49 central SNAREpins associated with Synaptotagmin-1 by Ca(2+) is sufficient to trigger rapid (<100 msec) and synchronous fu

50 ly, the fraction of total RNA that phase-separates in vitro is sufficient to trigger SG formation.