1 syndrome), but the mechanism underlying these
associations is unknown.
2 The etiology of isolated
BA is unknown, with evidence for infectious, environmental, and
3 d and integrated into the brain to drive migratory
behavior is unknown.
4 fter chromosome condensation and nuclear envelope
breakdown is unknown.
5 udies have been published and the true PeV infection
burden is unknown.
6 he knowledge of the authors the association with tau
burden is unknown.
7 Hitherto, their role in
cancer is unknown.
8 havior, but how this signal is received by epithelial
cells is unknown.
9 the functional consequence of LEC priming of CD8(+) T
cells is unknown.
10 Whether premature menopause is associated with
CHIP is unknown.
11 ation and function of this mark within the infectious
cycle is unknown.
12 associative brain regions is established during
development is unknown.
13 ting RV hypertrophy and failure in congenital heart
disease is unknown.
14 ceptive placebos represent genuine psychobiological
effects is unknown.
15 -TP exposure, and therefore compromise prevention
efficacy,
is unknown.
16 acetylation, but how SIRT6 is able to accomplish this
feat is unknown.
17 The prevalence of
FH is unknown in 90% of countries in the world.
18 unction in chronic liver injury sequelae, such as
fibrosis,
is unknown.
19 eptor; however, its impact on pancreatic beta-cell
function is unknown.
20 ism for this tolerance-inducing effect of alcohol,
however,
is unknown.
21 ion depends on bacteria-specific or tumor-specific
immunity is unknown.
22 nduced pulmonary congestion carries prognostic
implications is unknown.
23 the consequences of plant species composition for
infection is unknown.
24 lying IOP elevation associated with intravitreal
injections is unknown.
25 and UL48, but the functional relevance of this
interaction is unknown.
26 It is unknown how coexisting cells differ in their response to n
27 It is unknown whether concomitant AS-CA has worse outcomes or re
28 It is unknown whether cone beam computed tomography (CBCT) image
29 It is unknown, however, how manganese accumulates in dopaminergi
30 cells (cDC1s and cDC2s, respectively) is well accepted;
it is unknown how robust this dichotomy is under inflammatory co
31 resulting burns remain sensitive to touch for weeks, but
it is unknown whether calves experience ongoing, non-evoked pain
32 However,
it is unknown which, if any, of these NET-affiliated proteins is
33 the hands is associated with rheumatoid arthritis (RA),
it is unknown whether tenosynovitis of the forefoot is associate
34 alue of mucosal rejection assessment for patient
management is unknown.
35 The role of TET2 in
PAH is unknown.
36 infants, but whether IGF-1 treatment can prevent BPD or
PH is unknown.Objectives: To evaluate whether postnatal treatmen
37 ts role in placentally related disorders of human
pregnancy is unknown(3).
38 d an inflammatory phenotype associated with CAD
progression is unknown.
39 ing site (here named EACBE) in dynamic chromatin
regulation is unknown.
40 ist within kidney transplants or play any role in
rejection is unknown, however, in part because of limited techniques fo
41 ce on the antigen-specific T cell receptor (TCR)
repertoire is unknown.
42 ntial role driving or regulating active avoidance
responses is unknown.
43 RC contributes to transcription regulation in Ewing
sarcoma is unknown.
44 (CNS) innate immune cells contributing to oxidative
stress is unknown, and therapies to target their neurotoxic function
45 e a meaningful impact on cardiovascular health and
survival is unknown.
46 s of COPD, including increased exacerbation
susceptibility,
is unknown.
47 ng, with relaxed eligibility criteria and bridging
therapy,
is unknown.
48 ure-while reducing the need for red blood cell
transfusions-
is unknown.
49 ructurally defined; however, the efficacy of h5B3.1 in
vivo is unknown.
50 Why some AIV lineages but not others evolve in this
way is unknown.