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1 lity at 18 months among infants with hypoxic-ischemic encephalopathy.
2 died had a cause of death other than hypoxic-ischemic encephalopathy.
3 ng the critical period for perinatal hypoxic-ischemic encephalopathy.
4 g children who did not have neonatal hypoxic-ischemic encephalopathy.
5 rapeutic target in infants suffering hypoxic-ischemic encephalopathy.
6 brain injury, and eight patients had hypoxic ischemic encephalopathy.
7 y in infants with moderate or severe hypoxic-ischemic encephalopathy.
8 ow of therapeutic opportunity during hypoxic-ischemic encephalopathy.
9 and received a clinical diagnosis of hypoxic-ischemic encephalopathy.
10 ge (IPH), extraaxial hemorrhage, and hypoxic-ischemic encephalopathy.
11 ity at age 18 months in infants with hypoxic-ischemic encephalopathy.
12 m recovery potential in patients with anoxic-ischemic encephalopathy.
13 nce interval) were calculated: adult hypoxic-ischemic encephalopathy: absent 0% (0%-1%), abnormal 22%
14 justed HR, 46.4; 95% CI, 42.2-51.0), hypoxic ischemic encephalopathy (adjusted HR, 23.6; 95% CI, 20.6
15 were compared with severity of acute hypoxic-ischemic encephalopathy and long-term clinical outcome.
16 ebral sinovenous thrombosis, 11 with hypoxic ischemic encephalopathy, and 5 with neonatal arterial is
17 onates (>/=36 weeks' gestation) with hypoxic-ischemic encephalopathy at 18 US centers in the Eunice K
18 death or disability for infants with hypoxic-ischemic encephalopathy at 36 weeks' or later gestation.
19 erm neonates with moderate or severe hypoxic-ischemic encephalopathy, cooling for longer than 72 hour
20 er gestation with moderate or severe hypoxic-ischemic encephalopathy enrolled at 6 to 24 hours after
24 xpression patterns in human neonatal hypoxic-ischemic encephalopathy (HIE) and MS as well as developm
33 n or equal to 17 mug/L argue against hypoxic-ischemic encephalopathy incompatible with reawakening.
34 re full-term with moderate or severe hypoxic ischemic encephalopathy, longer cooling, deeper cooling,
36 ely analyze the hypoxic component of hypoxic-ischemic encephalopathy, rats were prepared such that th
37 degrees C for 72 hours for neonatal hypoxic ischemic encephalopathy reduces death or disability to 4
38 hours at 33.5 degrees C for neonatal hypoxic-ischemic encephalopathy reduces death or disability, but
39 tic barbiturate therapy for neonatal hypoxic-ischemic encephalopathy showed no significant effect on
40 whole-body hypothermia for neonatal hypoxic-ischemic encephalopathy showing a significant reduction
41 s; age range, 2-12 days) with severe hypoxic-ischemic encephalopathy were examined during the first 1
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