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1 age: 68 +/- 12 years; 53.2% had a history of ischemic heart disease).
2 d cardiomyopathy and 21 patients (21.6%) had ischemic heart disease).
3 bral microbleeds, hypertension, diabetes and ischemic heart disease.
4 y for the management of patients with stable ischemic heart disease.
5 se affects almost one-third of patients with ischemic heart disease.
6 poptosis is a significant problem underlying ischemic heart disease.
7 s with acute myocardial infarction or stable ischemic heart disease.
8 Equal numbers in each group died of ischemic heart disease.
9 saved for each exposed worker who died from ischemic heart disease.
10 ing fluid was recently linked to deaths from ischemic heart disease.
11 -effectiveness of CABG versus PCI for stable ischemic heart disease.
12 0, 5,807) life-years among those who died of ischemic heart disease.
13 edical therapy alone in patients with stable ischemic heart disease.
14 of cardiac stem/progenitor cell therapy for ischemic heart disease.
15 tion of PM2.5 with circulatory mortality and ischemic heart disease.
16 ents who had both type 2 diabetes and stable ischemic heart disease.
17 and ameliorates remodeling in patients with ischemic heart disease.
18 ysis of preclinical data of cell therapy for ischemic heart disease.
19 also be confirmed in patients suffering from ischemic heart disease.
20 ticularly high rates of premature death from ischemic heart disease.
21 scular disease but one only half as great on ischemic heart disease.
22 maging immune activity in organs involved in ischemic heart disease.
23 tion report of no association between SU and ischemic heart disease.
24 duced risks of ischemic vascular disease and ischemic heart disease.
25 ve coronary angiography for suspected stable ischemic heart disease.
26 nce on mitral valve geometry and function in ischemic heart disease.
27 d rural communities for patients with stable ischemic heart disease.
28 f care and outcomes for patients with stable ischemic heart disease.
29 ancer therapy, particularly in patients with ischemic heart disease.
30 duced risks of ischemic vascular disease and ischemic heart disease.
31 greater proportionate mortality burden from ischemic heart disease.
32 nary syndromes and 2062 patients with stable ischemic heart disease.
33 tion of the myocardial injury occurring with ischemic heart disease.
34 y age, sex, time of symptom onset, and known ischemic heart disease.
35 ied more frequently of other causes, such as ischemic heart disease.
36 se hearts subjected to conditions that mimic ischemic heart disease.
37 tions, particularly in those with underlying ischemic heart disease.
38 d complementary information in patients with ischemic heart disease.
39 es to the severity of myocardial damage from ischemic heart disease.
40 iglyceride-rich lipoprotein particles causes ischemic heart disease.
41 novel diagnostic and therapeutic targets for ischemic heart disease.
42 lated with QTc prolongation in patients with ischemic heart disease.
43 reducing myocardial injury in subjects with ischemic heart disease.
44 2 could be a potential therapeutic target in ischemic heart disease.
45 ejection fraction not caused by valvular or ischemic heart disease.
46 rophy, cardio-oncology, aortic stenosis, and ischemic heart disease.
47 y and the proportion of adults with baseline ischemic heart disease.
48 larizing damaged myocardium in patients with ischemic heart disease.
49 ding those presenting early or known to have ischemic heart disease.
50 1941-1994) for mortality from all causes and ischemic heart disease.
51 iding important diagnostic information about ischemic heart diseases.
52 nterval [CI], 0.35 to 0.54; hazard ratio for ischemic heart disease, 0.40; 95% CI, 0.31 to 0.52).
53 1.05) for overall CVD, 0.97 (0.86, 1.09) for ischemic heart disease, 0.93 (0.81, 1.07) for stroke, 0.
54 9-1.6), 1.5 (1.2-2.0), and 2.0 (1.4-2.9) for ischemic heart disease; 1.4 (0.9-2.1), 1.2 (0.8-1.9), an
55 analyses revealed a significant increase in ischemic heart disease (10-year hazard ratio, 1.14; 95%
56 ation was observed in 30.5% of patients with ischemic heart disease: 11.3% had mild airflow limitatio
57 at the 90th and 10th percentile was 2.0 for ischemic heart disease (119.1 vs 235.7 deaths per 100000
58 nal class III (1 point) or IV (3 points); no ischemic heart disease (2 points); renal dialysis (3 poi
59 gnosed previously with these conditions (for ischemic heart disease, 26.3% compared with 43.7%; for c
60 riable odds ratio estimates were as follows: ischemic heart disease 3.30 (95% confidence interval, 2.
61 fold for myocardial infarction, 3.2-fold for ischemic heart disease, 3.2-fold for ischemic stroke, an
62 was responsible for 31% of all deaths, with ischemic heart disease (31%) and cerebrovascular disease
63 mated loss in life expectancy was related to ischemic heart disease (36% in men, 31% in women) but de
64 D as the underlying cause, including chronic ischemic heart disease (42% of CVD deaths), hypertensive
65 77%) participants were men, had a history of ischemic heart disease (56%), and were prescribed beta-b
68 24.6%-25.4%] for those aged 80-84 years) and ischemic heart disease (8.6% [8.3%-8.9%] versus 19.0% [1
69 tion fraction who have a lower prevalence of ischemic heart disease, a less severe hemodynamic, bioma
72 l PM2.5 was associated with a higher risk of ischemic heart disease among aluminum manufacturing work
73 nd mortality (all-cause, cardiovascular, and ischemic heart disease) among 835 white men in the Norma
74 d concordance statistic of 0.70 and 0.70 for ischemic heart disease and 0.63 and 0.66 for stroke, res
76 were 0.87 (95% CI: 0.78, 0.97; P = 0.01) for ischemic heart disease and 0.80 (95% CI: 0.73, 0.88; P <
77 was 0.90 (95% CI: 0.75, 1.08; P = 0.27) for ischemic heart disease and 0.88 (0.72, 1.08; P = 0.22) f
78 he equation for prognostic information about ischemic heart disease and all-cause death was evaluated
82 ature mortality, and the leading causes were ischemic heart disease and cancer, which appeared to be
83 d diabetes were independent risk factors for ischemic heart disease and cardiomyopathy, and smoking w
84 were 2.5 times and 5.9 times more at risk of ischemic heart disease and cardiomyopathy/heart failure
86 diomyopathy, and smoking was associated with ischemic heart disease and diabetes (odds ratios [ORs],
88 We applied Cox MSMs in a study of incident ischemic heart disease and exposure to particulate matte
91 e matrix metalloproteinases in patients with ischemic heart disease and HF with a reduced EF can prov
93 declines in the incidence and mortality from ischemic heart disease and ischemic stroke since the mid
94 combination-associate with increased risk of ischemic heart disease and myocardial infarction indepen
95 al population, mainly because of deaths from ischemic heart disease and other smoking-related disease
96 al protective role in the pathophysiology of ischemic heart disease and pressure-overload heart failu
98 risk of adverse outcome after adjustment for ischemic heart disease and QRS width (hazard ratio [HR]:
99 tment in improving survival in patients with ischemic heart disease and reduced ejection fraction.
101 rpose We aimed to predict individual risk of ischemic heart disease and stroke in 5-year survivors of
105 y can predict individual risk for subsequent ischemic heart disease and stroke with reasonable accura
107 lence of airflow limitation in patients with ischemic heart disease and the effects on quality of lif
108 ents who had both type 2 diabetes and stable ischemic heart disease and were enrolled in the Bypass A
109 f noninvasive modalities in the detection of ischemic heart disease, and discuss nonischemic cardiomy
111 996 through 2013; with spending on diabetes, ischemic heart disease, and low back and neck pain accou
113 such chronic conditions as type 2 diabetes, ischemic heart disease, and some types of cancer, but th
114 ts with atrial fibrillation, cardiomyopathy, ischemic heart disease, and valvular heart disease.
115 ects, except for a subgroup of patients with ischemic heart disease as a result of severe coronary ar
116 king fluids and mortality, particularly from ischemic heart disease, as well as an instructive exampl
118 d our ability to understand the pathology of ischemic heart disease, atherosclerosis, and heart failu
119 ized with a principal discharge diagnosis of ischemic heart disease before (November 1, 2004, to July
122 PCI) relieves angina in patients with stable ischemic heart disease, but clinical trials have not sho
123 iated with better prognosis in patients with ischemic heart disease, but the underlying mechanisms re
124 from all cardiovascular diseases, including ischemic heart disease, cerebrovascular disease, ischemi
125 s, hypertension, dyslipidemia) and diseases (ischemic heart disease, cerebrovascular, and peripheral
127 compared these results to those for cancer, ischemic heart disease, chronic obstructive pulmonary di
128 ent comorbidities, including cardiomyopathy, ischemic heart disease; chronic renal failure, with and
129 zations, whereas 172 suffered from a chronic ischemic heart disease (CIHD), 126 of whom underwent cha
131 likely to have causes of death of cancer or ischemic heart disease, compared with younger elderly pa
132 ch included all Ontario patients with stable ischemic heart disease confirmed on cardiac catheterizat
133 isease developed in 10,797 participants, and ischemic heart disease developed in 7557 of these 10,797
134 n patients with hypertrophic cardiomyopathy, ischemic heart disease, diabetes mellitus, and more.
135 0% and comorbid cardiac risk factors such as ischemic heart disease, diabetes mellitus, or hypertensi
136 openhagen were genotyped, of whom 11,984 had ischemic heart disease diagnosed between 1976 and 2010.
137 ased, including liver disease, hypertension, ischemic heart disease, disorders of lipid metabolism, a
140 United States who were followed for incident ischemic heart disease from 1998 to 2012, and we address
142 ry angiography for possible suspected stable ischemic heart disease, from October 1, 2008, to Septemb
145 ic resonance strain imaging in patients with ischemic heart disease have been limited by sample size
146 last few decades, but mortality trends from ischemic heart disease have been more varied, with some
147 cardiac comorbidities in patients with COPD: ischemic heart disease, heart failure, and atrial fibril
148 Among patients with diabetes and stable ischemic heart disease, higher SYNTAX scores predict hig
149 larization is an effective means of treating ischemic heart disease; however, current therapeutic rev
150 favorable prognosis in patients with native ischemic heart disease; however, the impact of collatera
151 lung cancer (HR = 1.08, 95% CI: 1.02, 1.14), ischemic heart disease (HR = 1.09, 95% CI: 1.02, 1.16),
152 ension was associated with increased risk of ischemic heart disease (HR, 1.44 [95% CI, 1.24-1.68]), m
153 0.87; 95% CI: 0.76-0.99), a 26% reduction in ischemic heart disease (IHD) (RR: 0.74; 95% CI: 0.63-0.8
154 terval: 0.011, 0.393) (n = 5,818 deaths) for ischemic heart disease (IHD) after adjustment for dose f
155 vement in clinical outcomes of patients with ischemic heart disease (IHD) after ventricular tachycard
157 Although emotional stress is associated with ischemic heart disease (IHD) and related clinical events
159 tion between carbohydrate intake and risk of ischemic heart disease (IHD) has not been fully explored
160 ous studies on dietary magnesium and risk of ischemic heart disease (IHD) have yielded inconsistent r
165 n to cardiovascular disease (CVD) mortality, ischemic heart disease (IHD) mortality, and all-cause mo
166 iations between long-term PM2.5 exposure and ischemic heart disease (IHD) mortality, as established i
168 s in morbidity and mortality attributable to ischemic heart disease (IHD) require an understanding of
169 tudies have examined differences in incident ischemic heart disease (IHD) risk between vegetarians an
170 aise the impact of risk factor confluence on ischemic heart disease (IHD) risk by testing whether gen
174 To clarify the role of thyroid function in ischemic heart disease (IHD) we assessed IHD risk and ri
175 he pollutants and all-cause, cardiovascular, ischemic heart disease (IHD), and respiratory mortality.
176 cular disease (total cardiovascular disease, ischemic heart disease (IHD), and stroke) in adult women
177 including myocardial infarction (MI), other ischemic heart disease (IHD), congestive heart failure (
179 ble age-related signs associate with risk of ischemic heart disease (IHD), myocardial infarction (MI)
180 consumption of nuts and legumes and risk of ischemic heart disease (IHD), stroke, and diabetes have
181 cardiovascular outcomes were CVD [including ischemic heart disease (IHD), stroke, and vascular inter
189 ques in coronary arteries from patients with ischemic heart disease implying a role in human arterial
193 ations have been associated with low risk of ischemic heart disease in prospective studies, but resul
194 M2.5 exposure increases the risk of incident ischemic heart disease in workers in both aluminum smelt
195 n, coronary revascularization, or death from ischemic heart disease) in 2168 women who underwent radi
196 nant cholesterol is a causal risk factor for ischemic heart disease independent of reduced high-densi
197 s associated with a 2.8-fold causal risk for ischemic heart disease, independent of reduced HDL chole
200 l incidence rate (IR) for each condition are ischemic heart disease, IR=1518.7; myocardial infarction
202 ure with reduced ejection fraction caused by ischemic heart disease is associated with increased morb
204 Risk stratification in patients with stable ischemic heart disease is essential to guide treatment d
205 the prevalence of angina and mortality from ischemic heart disease is higher for women than men.
208 eart failure due to cardiomyocyte loss after ischemic heart disease is the leading cause of death in
212 risk of developing cardiovascular diseases (ischemic heart disease, ischemic stroke), metabolic dise
213 OPD) exacerbations, pneumonias, lung cancer, ischemic heart disease, ischemic stroke, and all-cause m
214 Its major clinical manifestations include ischemic heart disease, ischemic stroke, and peripheral
215 e risk of first fatal or nonfatal CVD event (ischemic heart disease, ischemic stroke, heart failure,
217 dalities for the diagnosis and management of ischemic heart disease, it is important for radiologists
220 to be younger, men, have diabetes mellitus, ischemic heart disease, lower left ventricular ejection
221 1.1%-3.6%) and 6.9% (95% CI, 4.0%-9.9%) for ischemic heart disease mortality among adults 65 years a
225 .81, 1.07) for stroke, 0.98 (0.77, 1.24) for ischemic heart disease mortality, 0.92 (0.56, 1.50) for
229 Mortality rates (MRs) for each condition are ischemic heart disease, MR=105.5; ischemic stroke, MR=42
230 rs at exacerbation were higher in those with ischemic heart disease (n = 12) than those without (n =
232 s between ingestion of inorganic arsenic and ischemic heart disease, nonmalignant respiratory disease
236 r filtration rate, age, history of diabetes, ischemic heart disease or hypertension, Killip class, le
238 altered abundance in septic cardiomyopathy, ischemic heart disease, or dilated cardiomyopathy, in co
239 did not differ according to age, race, prior ischemic heart disease, or ejection fraction (all intera
240 and the coexistence of atrial fibrillation, ischemic heart disease, or hypertensive cardiopathy.
241 defined as a history of atrial fibrillation, ischemic heart diseases, or congestive heart failure.
242 cancer (HR = 1.14; 95% CI: 1.03, 1.27), and ischemic heart disease (overall HR = 1.61; 95% CI: 1.14,
244 ilar to those for risk of cancer (p = .002), ischemic heart disease (p = 4 x 10(-99)), chronic obstru
247 1 year, cardiac-related death, hypertension, ischemic heart disease, pulmonary circulation problems,
248 n (COURAGE) trial, some patients with stable ischemic heart disease randomized to optimal medical the
250 y syndrome (ACS), the acute manifestation of ischemic heart disease, remains a major cause of morbidi
252 (rate ratio [RR], 1.14; 95% CI, 1.13-1.14), ischemic heart disease (RR, 1.11; 95% CI, 1.10-1.11), ma
253 (rate ratio [RR], 1.62; 95% CI, 1.20-21.8), ischemic heart disease (RR, 4.31; 95% CI, 3.38-5.49), an
254 ), asthma (RR=1.12, 95% CI: 1.03, 1.22), and ischemic heart disease (RR=1.19, 95% CI: 1.03, 1.38).
255 Autologous and allogeneic cell therapy for ischemic heart disease show a similar improvement in lef
256 prediction model including QRS duration and ischemic heart disease significantly improved the net re
257 -world population among subjects with stable ischemic heart disease (SIHD) and acute coronary syndrom
258 tor (FGF)-23 identifies patients with stable ischemic heart disease (SIHD) at high risk of cardiovasc
259 neous coronary intervention (PCI) for stable ischemic heart disease (SIHD) declined after publication
261 of common clinical presentations for stable ischemic heart disease (SIHD) to consider use of stress
263 atures such as age, history of arrhythmia or ischemic heart disease, size of goiter, and severity of
264 Cardiovascular diseases (CVDs), including ischemic heart disease, stroke, and heart failure, are w
265 rtension, diabetes mellitus, hyperlipidemia, ischemic heart disease, stroke, total cholesterol level,
268 in patients with congestive heart failure or ischemic heart disease than in those without (P = 0.021
269 in patients with congestive heart failure or ischemic heart disease than in those without (P = 0.021
270 the existence of differentiated patterns of ischemic heart disease that combine focal and diffuse co
271 ts with systolic heart failure not caused by ischemic heart disease, the association between the ICD
272 between urban and rural patients with stable ischemic heart disease, there were no outcome difference
273 TSD) is associated with an increased risk of ischemic heart disease, though the pathophysiologic mech
274 ogenic precursors link coronary anomalies to ischemic heart disease.Though coronary arteries are cruc
275 uggest that soy foods can potentially reduce ischemic heart disease through multiple mechanisms.
276 randomly assigned 2287 patients with stable ischemic heart disease to an initial management strategy
277 phases of the disease, potentially allowing ischemic heart disease to be tracked during a patient's
278 t they might be used in patients with stable ischemic heart disease to identify those at high risk fo
280 elevation acute coronary syndromes or stable ischemic heart disease undergoing percutaneous coronary
281 ty of mitral regurgitation, 67 patients with ischemic heart disease underwent cardiac magnetic resona
282 rtality ratios and absolute excess risks for ischemic heart disease, valvular heart disease, and card
283 ar disease such as congestive heart failure, ischemic heart disease, valvular heart disease, pulmonar
285 ntative sample of 50 patients with suspected ischemic heart disease was retrospectively selected from
286 Among the four health outcomes examined, ischemic heart disease was the greatest cause of death.
287 cells in immunosuppressed animals with acute ischemic heart disease, we previously showed that these
288 y, peripheral neuropathy, diabetic foot, and ischemic heart disease were 21.9%, 17.6%, 28.0%, 6.2%, a
289 creatinine level, black race, older age, and ischemic heart disease were associated with troponin ele
290 in acute coronary syndromes (ACS) and stable ischemic heart disease were combined into 1 document.
291 incidences of ischemic vascular disease and ischemic heart disease were reduced in heterozygotes as
292 tissue samples from patients suffering from ischemic heart disease were used to validate our finding
293 d exertional E/e' >13), excluding those with ischemic heart disease, were recruited in a tertiary car
294 gly recognized as an elementary component of ischemic heart disease, which can be accurately assessed
295 examining the proportion of patients without ischemic heart disease who were on a high-dose statin.
297 elevation acute coronary syndromes or stable ischemic heart disease, who underwent percutaneous coron
298 significant in the pathophysiology of human ischemic heart disease with a preservative role in maint
299 ted myocardium, even in patients with stable ischemic heart disease with preserved LV ejection fracti
300 dox is an observation of a low prevalence of ischemic heart disease, with high intakes of saturated f
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