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1 get in each individual (i.e. the critical AT isthmus).
2 med at the pulmonary vein and cavo-tricuspid isthmus.
3 al of most midbrain/hindbrain cells near the isthmus.
4 71%) had an identifiable endocardial circuit isthmus.
5 t mapping overestimates the true size of the isthmus.
6 tified to attempt to trace the course of the isthmus.
7 ding projections beyond the diencephalon and isthmus.
8 lbumin+ labeling, is observed in the ventral isthmus.
9 ed in the tricuspid annulus-eustachian ridge isthmus.
10 stal Plain and the tropical Central American Isthmus.
11  neurons project anterodorsally to enter the isthmus.
12 n guiding ablation within the cavo-tricuspid isthmus.
13 was consistent with entrainment criteria for isthmus.
14 ent by mapping to identify a reentry circuit isthmus.
15 s quiescent stem cells in the gastric corpus isthmus.
16  specificity for identifying the clinical VT isthmus.
17 mic isthmuses containing VT re-entry circuit isthmuses.
18 +/-0.3 m/s) and nearly normal at the central isthmus (0.62+/-0.2 m/s).
19 s action potential duration alternans at the isthmus (11 of 20 patients) and 2:1 isthmus conduction b
20                                              Isthmus 1A had the greatest dimensions (mean length, 3.9
21                                              Isthmus 1A is associated with the largest morphological
22             For subjects>/=5 years at death, isthmuses 1A and 1B were present in 88%, isthmus 2 in 25
23 ength, 3.9+/-1.08; thickness, 1.5+/-0.3 cm), isthmus 1B intermediate dimensions (mean length, 2.4+/-0
24 mensions, whereas the nearby newly described isthmus 1B is significantly smaller.
25 th, isthmuses 1A and 1B were present in 88%, isthmus 2 in 25%, isthmus 3 in 94%, and isthmus 4 in 13%
26 th, 2.4+/-0.8; thickness, 1.1+/-0.4 cm), and isthmuses 2, 3, and 4 the smallest dimensions (mean leng
27 ) was significantly shorter than the central isthmus (24 mm +/- 4.3; range, 12-43 mm) and the central
28 l isthmus was shorter than the inferolateral isthmus (27 mm +/- 4.8; range, 13-45 mm) (P < .001).
29 nd 1B were present in 88%, isthmus 2 in 25%, isthmus 3 in 94%, and isthmus 4 in 13%.
30   Of isthmuses with the smallest dimensions, isthmus 3 is the most common.
31 88%, isthmus 2 in 25%, isthmus 3 in 94%, and isthmus 4 in 13%.
32  function and isthmus-dependent re-entry, VT isthmus ablation can be curative.
33  cava isolation in 6 patients, cavotricuspid isthmus ablation in 5 patients, and ablation of sites of
34 ractionation and may explain the benefits of isthmus ablation in preventing AF recurrence.
35                    Therefore, cavo-tricuspid isthmus ablation is appropriate during pulmonary vein is
36 block were thoroughly reviewed in 271 mitral isthmus ablation procedures undertaken among 236 patient
37                               Cavo-tricuspid isthmus ablation was performed in 28 of the 133 patients
38   Among the 105 patients who did not undergo isthmus ablation, 25 patients (24%) were documented to h
39 on was performed followed by roof and mitral isthmus ablation, before CFAE ablation in the CFAE arm.
40 ibility of pulmonary vein and cavo-tricuspid isthmus ablation.
41 0) with symptomatic atrial flutter underwent isthmus ablation.
42 timulation maneuvers and occasional complete isthmus ablation.
43              TTE cells were localized to the isthmus, above and distinct from TFF2 protein-expressing
44              TTE cells were localized in the isthmus adjacent to doublecortin CaM kinase-like-1(+) pu
45                      FGF8 signaling from the isthmus alters Hoxa2 expression and consequently branchi
46            The low CF obtained at the mitral isthmus and anterior segments of the left pulmonary vein
47 e (skeletonized) geometric parameters of the isthmus and border zone were measured from the maps.
48 e show that Gli3 regulates patterning of the isthmus and cerebellar anlage by confining Fgf8 expressi
49 and E11.0, Gli3 continues to be required for isthmus and cerebellum development, but primarily for de
50  Otx2 in the hindbrain, thereby allowing the isthmus and cerebellum to form.
51 or establishing a distinct posterior tectum, isthmus and cerebellum, but does not play a role in the
52 deling studies recapitulated DEEPs at the VT isthmus and demonstrated their role in VT initiation wit
53 on (CI) was performed incorporating putative isthmus and early exit site(s) based on standard criteri
54 ction potentials (MAPs) were recorded at the isthmus and either high or low right atrium (HRA, LRA) d
55 ontractions of the muscles in the pharyngeal isthmus and function systemically to regulate an enhance
56  proliferation is reduced in both the normal isthmus and in the mutant anterior r1.
57  a linear lesion-successfully eliminated the isthmus and local abnormal voltage activities.
58  procedural success (transection of anatomic isthmus and noninducibility) and freedom of VT recurrenc
59 lation lines also were created in the mitral isthmus and posterior left atrium.
60 rtic sites terminated VT related to a septal isthmus and prevented reinduction.
61 rmation that forms ventromedially within the isthmus and r1.
62 overing that the IP develops strictly within isthmus and rhombomere 1.
63 ppeared to be differentiating neurons in the isthmus and the rostral hindbrain region, including the
64 he projection of trochlear axons towards the isthmus and their subsequent growth within that tissue m
65 s this, we conditionally ablated Fgf8 in the isthmus and uncovered that prolonged expression of Fgf8
66 ty of any VT and transection of the anatomic isthmus and was achieved in 25 (74%) patients.
67 fter PVI and linear lesions (roof and mitral isthmus), and biatrially after LA CFE ablation.
68 r anlage by confining Fgf8 expression to the isthmus, and attenuates growth of dorsal r1 (before E11.
69 enitors results in deletion of the midbrain, isthmus, and cerebellum.
70  secondary gustatory/visceral nucleus in the isthmus, and for distinguishing territories in the prima
71 ng for the loss of brain structures near the isthmus, and instead demonstrate that tissue transformat
72 nderestimates the anatomic complexity of the isthmus, and likely, its functional correlates.
73  regions such as the midbrain tectum, dorsal isthmus, and motor nuclei, ASP and GABA immunoreactivity
74 m body, anterior midbody, posterior midbody, isthmus, and splenium) and for overall CC size, with lef
75 pital fasciculus, internal capsule, callosal isthmus, and the corona radiata (p=0.04 for FIQ and p=0.
76  from the floor plate (DV) and Fgf8 from the isthmus (AP).
77 es that target both scar-related and classic isthmuses appear necessary to prevent long-term recurren
78  targeted deletion of PPARgamma in the bulge/isthmus area of the hair follicle epithelium generates a
79 iculum of the isthmus (PaSi), area 29 of the isthmus (area 29i) and area prostriata (Pro), which has
80 6 of the 19 patients, a total of 41 distinct isthmus areas of 41 distinct VTs were identified and suc
81 in comparison with controls, although aortic isthmus/arterial duct diameter ratio was lower in fetuse
82 on of complex re-entry circuits and critical isthmuses as targets for ablation.
83 8 signaling from the mid/hindbrain boundary (isthmus) as being responsible for induction of different
84 part of the cingulum and the cingulate gyrus isthmus, as well as the precuneal GM, may be distinctive
85                              Once within the isthmus, axons form large fascicles that extend to a dor
86 ngeal muscles and marginal cells forming the isthmus between the anterior and posterior pharyngeal bu
87 accharomyces cerevisiae they localize at the isthmus between the mother and the daughter cells, where
88        Linear radiofrequency ablation in the isthmus between the TA and IVC (TI isthmus) terminated t
89 asured the conduction time, CTi, through the isthmus between the tricuspid annulus and eustachian rid
90 sal r1 (before E11.0) and the dorsal mes and isthmus (beyond E11.0) through regulation of cell prolif
91     Short-term end points were bidirectional isthmus block and no inducible AFL.
92  radiofrequency catheter ablation similar to isthmus block for atrial flutter.
93 is study evaluates the long-term efficacy of isthmus block for treatment of re-entry VT in adults wit
94 dered a reliable criterion of cavo-tricuspid isthmus block in patients undergoing radiofrequency abla
95 ed RA maze procedure is superior to anatomic isthmus block in treating reentrant AT in postoperative
96                              Complete mitral isthmus block predicted greater CFE reduction (P=0.02).
97                                Bidirectional isthmus block resulted in elimination of LLR.
98      All the available assessments of mitral isthmus block were thoroughly reviewed in 271 mitral ist
99 uch-up lesions was necessary to complete the isthmus block with conventional fluoroscopy (median, thr
100 ictors for unsuccessful bidirectional mitral isthmus blockade were the need for epicardial ablation f
101 cardiomyopathy, obliteration of a conductive isthmus both anatomically and functionally and abolition
102 ependent mechanism (ie, LLR or subeustachian isthmus breaks).
103 l in which Wnt regulates FGF activity at the isthmus by driving both FGF and Sprouty gene expression.
104  infarct regions will locate reentry circuit isthmuses by defining their borders.
105                 Ventricular tachycardia (VT) isthmuses can be defined by fixed or functional block.
106                                           VT isthmuses can be identified and part of their course del
107                                        These isthmus characteristics may enable the spontaneous initi
108 frequency ablation by targeting the critical isthmus (CI).
109      Atrophy in mesial and lateral temporal, isthmus cingulate, and orbitofrontal areas aided discrim
110 eral orbitofrontal cortex, and the bilateral isthmus cingulate.
111 , across Beringea, and across the Panamanian isthmus coincide in timing and location with multiple we
112 00% of patients when the RF line included an isthmus compared with 53% when RF had to be guided by pa
113 s at the isthmus (11 of 20 patients) and 2:1 isthmus conduction block immediately preceding AF (4 of
114 y AFL was ablated by achieving bidirectional isthmus conduction block.
115          Ablation aimed to transect anatomic isthmuses containing VT re-entry circuit isthmuses.
116                   Ablation targeted anatomic isthmuses containing VT re-entry circuits, which were id
117                                              Isthmus contraction occurred primarily in the posterior
118 ena cava, crista terminalis, tricuspid valve isthmus, coronary sinus orifice, membranous fossa ovalis
119 ow-up, WM volume in the left cingulate gyrus isthmus correlated with clinical scores of anxiety (Spea
120 m of the study was to determine whether a VT isthmus could be identified and followed by pace mapping
121 AJ disconnection combined with cavotricuspid isthmus (CTI) ablation (group 1, n=49) or PV-LAJ disconn
122        Achievement of complete cavotricuspid isthmus (CTI) conduction block reduces typical atrial fl
123 or scar-dependent (n = 15) and cavotricuspid isthmus (CTI)-dependent (n = 14) flutter were studied.
124 al fibrillation may experience cavotricuspid isthmus (CTI)-dependent atrial flutter during follow-up.
125  the surface expression of non-cavotricuspid isthmus (CTI)-dependent right atrial (RA) or left atrial
126  atrial flutter (AFL) from the cavotricuspid isthmus (CTI).
127                   However, dimensions of the isthmus defined by entrainment exceeded dimensions of th
128 sthmus-dependent (CWID) and counterclockwise isthmus-dependent (CCWID) right atrial flutter (AFL) and
129 ve) morphology among patients with clockwise isthmus-dependent (CWID) and counterclockwise isthmus-de
130 y ablation was performed in 30 patients with isthmus-dependent AFL.
131 tra-atrial re-entrant tachycardia (IART) and isthmus-dependent atrial flutter (IDAF) in patients pres
132        The aim of this study was to separate isthmus-dependent atrial flutter (IDAFL) from non-isthmu
133 t atrial tachycardias included cavotricuspid isthmus-dependent atrial flutter (n=7), non-isthmus-depe
134 us-dependent atrial flutter (IDAFL) from non-isthmus-dependent atrial flutter (NIDAFL) from the elect
135 rial tachycardia was seen in 7 patients, and isthmus-dependent atrial flutter occurred in 14 patients
136 ring 66 ATs in 62 patients: 20 cavotricuspid isthmus-dependent ATs, 20 perimitral ATs, 13 focal ATs w
137 to delineate the precise mechanism of the TI isthmus-dependent clockwise right atrial flutters.
138  from the scar to another anatomic boundary (isthmus-dependent group).
139                                       In the isthmus-dependent group, three of seven (42%) patients w
140 ight AFL is most commonly associated with an isthmus-dependent mechanism (ie, LLR or subeustachian is
141 ease with preserved ventricular function and isthmus-dependent re-entry, VT isthmus ablation can be c
142  isthmus-dependent atrial flutter (n=7), non-isthmus-dependent right atrial reentry (n=7), and 1 foca
143 ardias were macroreentrant (noncavotricuspid isthmus-dependent), and 35 ATs were focal (either trigge
144 -macro-re-entrant (perimitral, cavotricuspid isthmus-dependent, and roof-dependent circuits) versus c
145 t to determine whether the atrial flutter is isthmus-dependent, non-isthmus-dependent, or atypical; (
146 the atrial flutter is isthmus-dependent, non-isthmus-dependent, or atypical; (2) interrupting the atr
147                                      Central isthmus depth was classified as straight (3 mm), concave
148                                  Mean aortic isthmus diameter z scores measured either in sagittal (P
149                             Consistencies in isthmus dimensions and histology are found among patient
150 est: the optic tectum, torus semicircularis, isthmus, dorsal and medial nuclei of the octavolateral a
151 size that HSPGs are necessary for pharyngeal isthmus elongation, and pyr-1 functions upstream of prot
152 t pyr-1(cu8) exhibiting defective pharyngeal isthmus elongation, cytoskeletal organization defects, a
153 ansion and the repolarizing cells within the isthmus enabled retrograde flow of depolarizing electrot
154 ere slowest at the inward curvature into the isthmus entrance (0.28+/-0.2 m/s), slightly faster at th
155 h gastric mucus neck cells located below the isthmus express trefoil factor family 2 (TFF2) protein,
156 blation has been used to target the critical isthmuses for re-entrant monomorphic ventricular tachyca
157 amber of origin in all 10, and distinguished isthmus from nonisthmus dependent atrial flutter.
158 rom initial experiments, correctly predicted isthmus geometry (mean estimated/actual isthmus overlap
159 y in this canine model and are predictive of isthmus geometry changes.
160 th pacing threshold (r=0.64, P<0.0001), many isthmuses had very low-amplitude electrograms, and EUS c
161 ing and the relationship to the protected VT isthmus identified by entrainment mapping is unknown.
162 sful ablation sites were localized within an isthmus identified by pace mapping in all of these 10 pa
163 ed in the MI region either through a circuit isthmus identified from entrainment mapping or a target
164 ed by pace mapping (P=0.0002); those with an isthmus identified received shorter ablation lines (4.9+
165                            All 20 VT circuit isthmuses identified were adjacent to EUS.
166                         Of the 19 VT circuit isthmuses identified, 12 were associated with an endocar
167 ed RFCA for VTs dependent on septal anatomic isthmuses improves ablation outcome in repaired Tetralog
168                                 The critical isthmus in 115 of the 120 LA re-entrant ATs (96%) traver
169 lopment and becomes restricted mainly to the isthmus in adult glands, akin to its known localization
170 ured at 90% repolarization was longer at the isthmus in all patients, and failed to shorten with rate
171                                The diastolic isthmus in ventricular tachycardia was mapped in 3 patie
172 ombined mapping approach identified a narrow isthmuses in the lateral atrium, where the first RF lesi
173 ) in repaired Tetralogy of Fallot focuses on isthmuses in the right ventricle but may be hampered by
174                         Anatomically defined isthmuses included (1A) ventriculotomy-to-tricuspid annu
175                                   The mitral isthmus is a critical part of perimitral reentrant tachy
176               These findings reveal that the isthmus is a key cortical zone connecting both the cingu
177 mical results presented here reveal that the isthmus is composed of four cortical areas.
178 ss ablation is required if a reentry circuit isthmus is identified even when multiple and unstable VT
179 timing of dispersal or vicariance across the Isthmus is not explained by the ecological factors teste
180 whereas the absence of Gly-ir neurons in the isthmus is shared by all these species, except for lampr
181                                          The Isthmus is traditionally understood to have fully closed
182 hin the tricuspid annulus-inferior vena cave isthmus (IS) and either side of the crista terminalis (C
183                          Larger decreases in isthmus length corresponded to higher rates of linear cy
184                                         When isthmus length decreased, isthmus width decreased at its
185 nd tachycardia cycle length increased, while isthmus length decreased.
186 the anterior region of r1 is converted to an isthmus-like tissue.
187                         The effect of mitral isthmus line (MIL) ablation on outcomes after SRI has no
188          Deployment of an endocardial mitral isthmus line (MIL) with the end point of bidirectional b
189 ary vein isolation in 50 (100%), left atrial isthmus line in 47 (94%), anterior line in 45 (90%), com
190 cing techniques in patients with left mitral isthmus linear ablation.
191 essment of bidirectional block across mitral isthmus linear lesion using differential coronary sinus
192 round the left septum primum with a critical isthmus located between the pulmonary veins posteriorly
193         For all 36 experiments with reentry, isthmus location and shape were then estimated on the ba
194 n for prediction of reentry inducibility and isthmus location and shape.
195 uld be quantified to predict reentry and the isthmus location.
196 progressive pacing first caused alternans of isthmus MAP duration and amplitude at mean cycle length
197 potential duration rate maladaptation at the isthmus may lead to action potential duration alternans
198               At middiastole, the paraseptal isthmus (mean length, 20 mm +/- 3.5; range, 11-34 mm) wa
199 ed by entrainment exceeded dimensions of the isthmus measured by activation mapping by 32+/-18%.
200                  All tachycardias crossed an isthmus (median, 0.52 mV, 13 mm) bordered by nonconducti
201 tion, the completion of the Central American Isthmus more extensively than any other.
202 n strategies, ablations of the cavotricuspid isthmus (n=4), fossa ovalis (n=4), and pulmonary veins (
203 had a sensitivity and specificity for the VT isthmus of 29+/-10% and 95+/-1%, respectively.
204                        Egypt, located on the isthmus of Africa, is an ideal region to study historica
205 d BZ channels identified 74% of the critical isthmus of clinical VTs and 50% of all the conducting ch
206 gene Axin2, is limited to the base and lower isthmus of gastric glands, where the stem cells reside.
207                         The stem cell in the isthmus of gastric units continually replenishes the epi
208 gh the activity of stem cells located in the isthmus of individual gland units.
209 h America starting with the emergence of the Isthmus of Panama 3-4 million years ago (Ma).
210 des occurred at 4.90 Ma, indicating that the Isthmus of Panama allowed genetic exchange until the Pli
211 he linking of North and South America by the Isthmus of Panama had major impacts on global climate, o
212 South America and subsequent bridging of the Isthmus of Panama.
213  that are protected and part of the critical isthmus of post-infarction VT.
214 reater connectivity in the posterior midbody/isthmus of the corpus callosum and that fractional aniso
215 ed that diffusion anisotropy in the body and isthmus of the corpus callosum was negatively correlated
216 on, and diffusion anisotropy in the body and isthmus of the corpus callosum was shown to mediate this
217        Gastric stem cells are located in the isthmus of the gastric glands and give rise to epithelia
218                                 The critical isthmus of the re-entry circuit was identified by entrai
219 f the reentrant ventricular tachycardia (VT) isthmus or diastolic pathway.
220 a before the first humans crossed the Bering Isthmus or the onset of climate changes during the termi
221 ck in the tricuspid annulus-eustachian ridge isthmus or was associated with development of transient
222 cy results in reduced Fgf8 expression in the isthmus organiser (IsO), an embryonic signalling centre
223 cted isthmus geometry (mean estimated/actual isthmus overlap 70.5%).
224 and after (0.80+/-1.59 mV; 1+/-0.73 m/s) the isthmus (P<0.001 for all).
225                In 11 of 13 of the identified isthmus parts, the QRS morphology of the pace map matche
226  of the isthmus (PrSi), parasubiculum of the isthmus (PaSi), area 29 of the isthmus (area 29i) and ar
227 n at the isthmus requires the function of no isthmus/pax2.1, as well as Fgf signaling.
228 ria, activates overall feeding by activating isthmus peristalsis as well as pharyngeal pumping.
229 ing have no defects in pharyngeal pumping or isthmus peristalsis rates, but their growth defect depen
230 t of the two motions were distinct, but each isthmus peristalsis was coupled to the preceding pump.
231                               For activating isthmus peristalsis, SER-7 in the M4 (and possibly M2) m
232 inct feeding motions, pharyngeal pumping and isthmus peristalsis.
233 rly, and the PoS and PaS reach the cingulate isthmus posteriorly.
234        These include the presubiculum of the isthmus (PrSi), parasubiculum of the isthmus (PaSi), are
235 rily in the posterior segment of the central isthmus (RCA to inferior vena cava distance).
236 phageal squamous epithelium, and in the neck/isthmus region of healthy stomach.
237                                          The isthmus region to be targeted was chosen based solely on
238              This suggests that although the isthmus regulates the size of the cerebellar anlage, the
239 n=7) revealed increased fibrosis in anatomic isthmuses relative to nonisthmus controls.
240 nce of lmx1b.1 and lmx1b.2 expression at the isthmus requires the function of no isthmus/pax2.1, as w
241 atrial (LA) ablation targets were the mitral isthmus, roof, and septum.
242 cond ablation was performed either along the isthmus (scar-dependent group) or from the scar to anoth
243 nus rhythm, pace mapping near the exit of an isthmus should produce a QRS similar to that of VT.
244 encephalon, diencephalon, mesencephalon, and isthmus showed some deviation from the main scheme.
245 pping data were analyzed from the identified isthmus site and from sites at progressively increasing
246                                         A VT isthmus site was contained within a channel in only 11 o
247                                           An isthmus site was defined by entrainment and/or VT termin
248 ively increasing distances from this initial isthmus site.
249     Radiofrequency ablation was performed at isthmus sites as defined by pace-mapping (perfect pace-m
250 was to identify ventricular tachycardia (VT) isthmus sites by pace-mapping within scar tissue and to
251                        Overdrive pacing from isthmus sites determined by activation mapping was consi
252          Among all endocardial sites, the VT isthmus sites displayed the most delay and broadest EGMs
253 teristics that are helpful in identifying VT isthmus sites during sinus rhythm (SR).
254                This method could help target isthmus sites for ablation during stable sinus rhythm.
255                                              Isthmus sites of the VT re-entry circuits were identifie
256 se channels in predicting the location of VT isthmus sites was only 30%.
257 ting of two wide regions connected by a thin isthmus.Structural heterogeneities provided a substrate
258  at inferior levels and the midbrain-pontine isthmus suggests a vulnerable region of passage for comp
259 on standard characterization of suspected VT isthmus surrogates thus limiting ablation target size.
260 preoptic area, ventral hypothalamus, nucleus isthmus, tectum mesencephali, inferior colliculus, and h
261 on in the isthmus between the TA and IVC (TI isthmus) terminated the tachycardia in all patients.
262        The substrate often includes anatomic isthmuses that can be transected by radiofrequency cathe
263 halamus, the habenula, the optic tectum, the isthmus, the cranial motor nuclei, and the spinal motor
264                     Thus, in addition to the isthmus, the roof plate represents an important signalin
265 i at approximately the middle portion of the isthmus; the latter lacking the CB+ neuropil.
266 tionship of the ventricular tachycardia (VT) isthmus to channels of preserved voltage on an electroan
267 structures found progressively closer to the isthmus to form.
268 nt, this CD44(+) population expands from the isthmus toward the base of the unit.
269 ft side, and (3) left-sided RFCA resulted in isthmus transection and prevention of VT induction.
270 d rostrocaudal restriction in their origins (isthmus versus anterior or posterior r1 regions).
271                                  The reentry isthmus was characterized by a relatively small volume o
272 epted date of 3 million years ago (Ma), the Isthmus was effectively complete by the middle Miocene,
273              The 16 patients in whom > or =1 isthmus was identified and ablated were free of arrhythm
274 hermore, 22 of the 81 VTs (27%) for which no isthmus was identified became noninducible after ablatio
275           The monkey cingulo-parahippocampal isthmus was identified recently in the depths and latera
276                                       The VT isthmus was identified using entrainment mapping.
277 mm +/- 4.3; range, 12-43 mm) and the central isthmus was shorter than the inferolateral isthmus (27 m
278                  At middiastole, the central isthmus was straight in 8% of patients, concave in 47% o
279 e posterior left atrium and along the mitral isthmus, was performed under the guidance of an electroa
280            Median CF at the ridge and mitral isthmus were 5.1g and 6.9g, respectively.
281      Conduction velocities within the shared isthmus were dependent on the activation vector, consist
282 hologies with opposite axes sharing the same isthmus were mapped.
283                                              Isthmuses were 16.4+/-7.2 mm long and 7.4+/-2.8 mm wide.
284 liminated, and if VT was tolerated, critical isthmuses were also approached.
285                              Reentry circuit isthmuses were identified by entrainment mapping or pace
286            In all these regions, parts of VT isthmuses were identified by pace mapping.
287 ber passage occurred at the midbrain-pontine isthmus where all of the fiber bundles overlapped.
288 Fgf8 expression normally correlates with the isthmus where cells undergo low proliferation and that i
289 , undifferentiated cells in the gastric unit isthmus where stem cells are known to reside.
290  in positioning the mid/hindbrain organizer (isthmus), which regulates midbrain and cerebellar develo
291 nd (2) targeted illumination of the critical isthmus, which was identified via analysis of simulated
292 dorsal projection, which circumnavigates the isthmus, while those of more posterior trochlear neurons
293 hibchan speakers on both sides of the Panama isthmus, who have ancestry from both North and South Ame
294               When isthmus length decreased, isthmus width decreased at its narrowed portion.
295 age channels with ILPs harbored the clinical isthmus with a sensitivity and specificity of 78% and 85
296                                           Of isthmuses with the smallest dimensions, isthmus 3 is the
297 ines in the posterior left atrium and mitral isthmus, with an 8-mm-tip catheter.
298 ally complex structures, particularly at the isthmus, with substrate for multiple VT morphologies aft
299 2 minutes) were formed at the cavo-tricuspid isthmus, with the end point of bidirectional block.
300  WM bilaterally and the left cingulate gyrus isthmus WM, as well as the right precuneal GM, showed si

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