ライフサイエンスコーパス: itch sensation

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1 he body (interoception) (e.g., visceral-cold-itch sensations).

2 he molecules, cells, and circuits underlying itch sensation.

3 h a focus on the role of Mrgprs in mediating itch sensation.

4 to pruritic chemicals and playing a role in itch sensation.

5 erties to initiate and maintain long-lasting itch sensation.

6 or exploring the mechanism underlying spinal itch sensation.

7 receptor-4 (PAR-4) was recently suggested in itch sensation.

8 nisms underlying the central transmission of itch sensation.

9 s required selectively for histamine-induced itch sensation.

10 g normal skin near a site of itch can elicit itch sensation, a phenomenon known as alloknesis.

11 rin-releasing peptide, a peptide involved in itch sensation, and MrgprC11.

12 ortance, the underlying neural mechanisms of itch sensation are poorly understood.

13 fiber-dominated itch, (2) the time course of itch sensation differs between subjects with A-fiber- ve

14 nd unmyelinated fibers, (5) the time of peak itch sensation for subjects with A-fiber-dominated itch

15 myelinated fibers, and (6) the time for peak itch sensation for subjects with C-fiber-dominated itch

16 n of either BRAF or GRP signaling attenuated itch sensation in chronic itch mouse models.

17 molecule that is dedicated to mediating the itch sensation in the dorsal horn of the spinal cord, an

18 (GRPR) plays an important part in mediating itch sensation in the dorsal spinal cord.

19 ns constitute a long-sought labeled line for itch sensation in the spinal cord.

20 nt of local neuronal circuits for processing itch sensation in the spinal cord.

21 strate that PLCbeta3 is required to mediate "itch" sensation in response to histamine acting on the h

22 This itch sensation is normally suppressed by inputs from mec

23 Despite the clinical importance of the itch sensation, its mechanism remains elusive.

24 Scratching significantly attenuated the itch sensation (P<0.001) and evoked an associated pleasu

25 rate that GRPR is required for mediating the itch sensation rather than pain, at the spinal level.

26 The itch sensation results from the excitation of primary se

27 s of this class of neurons parallel the pure itching sensation this stimulus elicits in humans, and m

28 tone via administration of 5-HT potentiates itch sensation, whereas mice lacking 5-HT or serotonergi

29 es, cells, and circuits known to mediate the itch sensation, which, coupled with advances in understa

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