ライフサイエンスコーパス: jasmonate

1 ic acid and jasmonic acid (applied as methyl jasmonate).

2 as not modified by the application of methyl jasmonate.

3 ic biosynthetic pathways by the phytohormone jasmonate.

4 ) which was specifically activated by methyl jasmonate.

5 he gene can be induced in leaves with methyl jasmonate.

6 ns aphid resistance when treated with methyl jasmonate.

7 eatment of roots with F. oxysporum or methyl jasmonate.

8 y exogenous applications of the phytohormone jasmonate.

9 (1)O2-induced PCD, likely acting upstream of jasmonate.

10 investigate radial cell-to-cell transport of jasmonates.

11 scular cells, revealing a radial movement of jasmonates.

12 jasmonates, accumulates in opposite phase to jasmonates.

13 under normal conditions, addition of methyl jasmonate, a biotic stress hormone, induced expression i

14 ariants in Col-0 after treatment with methyl jasmonate, a condition known to "induce ESP", it was ind

15 Interestingly, methyl jasmonate, a repellent derived from the nonvolatile jasm

16 anceolata shoot cultures treated with methyl jasmonate, a well-known elicitor of plant specialized me

17 defense that often acts antagonistically to jasmonates, accumulates in opposite phase to jasmonates.

18 responsible and essential for stress-induced jasmonate accumulation in roots.

19 Circadian jasmonate accumulation occurs in a phase pattern consist

20 hat surprisingly only partly overlapped with jasmonate accumulation polymorphisms and deviated from c

21 ed the extent of the transport of endogenous jasmonates across the plant vegetative growth phase.

22 The findings indicate that jasmonates acting synergistically with ethylene are the

23 es involved in auxin synthesis/transport and jasmonate activity were differentially expressed, indica

24 erated senescence stimulated by darkness and jasmonate, although SUB1A significantly restrained dark-

25 nse to wounding followed by ethylene, methyl jasmonate and ABA treatment.

26 ids and lipophilic hormone precursors of the jasmonate and auxin biosynthetic pathways.

27 F-box proteins COI1 and TIR1, receptors for jasmonate and auxin, respectively.

28 induction of inositol pyrophosphate InsP8 by jasmonate and demonstrate that steady state and jasmonat

29 and Eui1-OX mutants combined with nutrient, jasmonate and gene expression analyses were used to test

30 ex and suggest that coincidence detection of jasmonate and InsP8 by COI1-JAZ is a critical component

31 ession was observed with the hormones methyl jasmonate and naphthalene acetic acid and diterpenes.

32 The systemic wound response was mediated by jasmonate and redox signaling.

33 Jasmonate and salicylic acid are positive regulators of

34 ng ethylene production and responsiveness to jasmonate and salicylic acid, thereby dampening the brea

35 The pathway is coordinately induced by jasmonate and the key enzyme, adenosine 5'-phosphosulfat

36 at foliar treatments carried out with methyl jasmonate and yeast extract achieved the best results, i

37 e results of this study indicate that methyl jasmonate and yeast extract applications could be a simp

38 wounding elicited transient accumulation of jasmonates and a decrease in exudation probably due to p

39 ncreasing CO(2) suppresses the production of jasmonates and ethylene and increases the production of

40 olution and collection of both major (methyl jasmonates) and minor (epi-methyl jasmonates) stereoisom

41 mones (auxins, cytokinins, abscisic acid and jasmonates), and in the nutrient composition of the leav

42 response to UV-B, dehydration, NaCl, methyl jasmonate, and abscisic acid treatments indicating its p

43 NT1 (BZR1)-that are key regulators in light, jasmonate, and brassinosteroid signaling pathways, respe

44 al defense hormone pathways (salicylic acid, jasmonate, and jasmonate/ethylene pathways) were up-regu

45 ry hub, integrating ethylene, abscisic acid, jasmonate, and redox signaling in the plant response to

46 and other plant hormones, including auxins, jasmonates, and gibberellins.

47 s and exogenous methyl salicylate and methyl jasmonate applications showed that plant defense against

48 th-defense tradeoffs mediated by the hormone jasmonate are uncoupled in an Arabidopsis mutant (jazQ p

49 Jasmonates are important phytohormones regulating reprod

50 Endogenous jasmonates are important regulators of plant defenses.

51 Exogenous jasmonates are known to trigger ISR by activating the pl

52 Jasmonates are oxygenated lipids (oxylipins) that contro

53 Jasmonates are oxylipin signals that play important role

54 hormone; thus, both the circadian clock and jasmonates are required.

55 Oxylipins including jasmonates are signaling compounds in plant growth, deve

56 d a higher level of jasmonic acid and methyl jasmonate, as well as the oxylipin-biosynthetic intermed

57 essors, and SA/JA cross talk did not involve JASMONATE ASSOCIATED MYC2-LIKEs, which are negative regu

58 GT1b impairs responses to the plant hormones jasmonate, auxin and gibberellic acid, but not brassinol

59 Plastid jasmonate biosynthesis enzymes were recruited to the k1

60 LOX2 encodes for a jasmonate biosynthesis gene, which is also targeted by c

61 Also jasmonate biosynthesis is induced, pointing to the need

62 Jasmonate biosynthesis was strongly induced in ch1 mutan

63 that mutant cgi-58 plants display changes in jasmonate biosynthesis, auxin signaling, and lipid metab

64 on of genes related to mechanical stress and jasmonate biosynthesis/signaling during wood formation i

65 s including organelle-specific regulation of jasmonate biosynthesis; simultaneous induction of synthe

66 Jasmonate biosynthetic genes were highly represented amo

67 lene precursor, and by treatment with methyl jasmonate, but disappeared upon treatment of seedlings w

68 Methyl jasmonate, chitosan, and a commercial yeast extract were

69 f active jasmonate, JAZ proteins function as jasmonate co-receptors by forming a hormone-dependent co

70 ibberellins, cytokinins, salicylic acid, and jasmonate compared with diploid individuals.

71 ecies other than Arabidopsis thaliana, and a jasmonate-containing galactolipid.

72 r and redefine the dynamic metabolic grid of jasmonate conversion in the wound response.

73 pyrophosphates to the F-box protein COI1-JAZ jasmonate coreceptor complex and suggest that coincidenc

74 All of the data demonstrate that the jasmonate defense signal pathway is a major defense sign

75 /AN1 were found to almost all be involved in jasmonate defense signaling pathways.

76 plants and by the increased tolerance of the jasmonate-deficient mutant delayed-dehiscence2.

77 Grafting experiments with a jasmonate-deficient mutant demonstrated that roots produ

78 We demonstrate that jasmonate-deficient Nicotiana attenuata plants suffer mo

79 but was not significantly affected by methyl jasmonate, dehydration or heat shock stress.

80 different types of herbivores in nature, and jasmonate-dependent defenses are important for plants to

81 ase CYP705A1 and is transiently induced in a jasmonate-dependent manner by infection with the root-ro

82 Stems are defended by jasmonate-dependent nicotine, and the native cottontail

83 and neither the salicylic acid-dependent nor jasmonate-dependent pathways were induced.

84 onclude that low ascorbate triggers ABA- and jasmonate-dependent signaling pathways that together reg

85 1, the levels of metabolites, ABA, auxin and jasmonate derivatives did not change significantly in de

86 Treatment with herbivore or jasmonate elicitors induces emission of (E)-alpha-bergam

87 Thus, endogenous jasmonates enable plants to resist different types of he

88 Several phytohormones, including methyl jasmonate, ethylene, and abscisic acid, regulated RIP2 p

89 four major sectors of the signaling network, jasmonate, ethylene, PAD4, and salicylate, are disabled,

90 containing four major signaling sectors, the jasmonate, ethylene, phytoalexin-deficient 4, and salicy

91 one pathways (salicylic acid, jasmonate, and jasmonate/ethylene pathways) were up-regulated in ago1 m

92 Here we describe a jasmonate family binding protein, cyclophilin 20-3 (CYP2

93 The jasmonate family of growth regulators includes the isole

94 The jasmonate family of phytohormones plays central roles in

95 acids was increased as consequence of methyl jasmonate foliar application, i.e., histidine, serine, t

96 Further, transcript profiling under methyl jasmonate, gibberellic acid, and yeast extract elicitati

97 The jasmonate hormonal pathway regulates important defensive

98 Jasmonate hormone (JA) plays critical roles in both plan

99 This system harnesses the plant auxin and jasmonate hormone-induced degradation pathways, and is d

100 lants with arrhythmic clocks or deficient in jasmonate hormone; thus, both the circadian clock and ja

101 In conclusion, foliar application of methyl jasmonate improved must nitrogen composition.

102 formation available on the multiple roles of jasmonates in plant development and defense, knowledge a

103 sjar1-3, to study the biological function of jasmonates in rice anthesis.

104 to understand the synthesis and function of jasmonates in roots.

105 , including a lower but more potent burst of jasmonates in several plant species.

106 Jasmonates, including jasmonic acid (JA) and methyl jasm

107 cient mutant demonstrated that roots produce jasmonates independently of leaves, despite low expressi

108 tes, including jasmonic acid (JA) and methyl jasmonate, induced the formation of tyloses, whereas tre

109 Because they are induced by JA we named them JASMONATE-INDUCED OXYGENASES (JOXs).

110 monate and demonstrate that steady state and jasmonate-induced pools of InsP8 in Arabidopsis seedling

111 Two closely related genes encoding the jasmonate-induced protein 60 (JIP60) were identified in

112 Interestingly, jasmonate induces intra- and extracellular UPI accumulat

113 Here, we report on two homologous jasmonate-inducible transcription factors of the basic h

114 lic acid and JASMONIC ACID RESISTANT1 (JAR1)/JASMONATE INSENSITIVE1 (JIN1)/MYC2 independent.

115 ion in a CORONATINE INSENSITIVE1 (COI1)- and JASMONATE INSENSITIVE1 (JIN1/MYC2)-dependent manner.

116 ine insensitive1 (coi1) and myc2 (allelic to jasmonate insensitive1) mutants, suggesting LBD20 may fu

117 iverse developmental and environmental cues, jasmonate is produced, conjugated to the amino acid isol

118 We found that the COR and jasmonate isoleucine (JA-Ile) co-receptor JAZ2 is consti

119 re-induced production of the stress hormones jasmonate-isoleucine conjugate and abscisic acid, which

120 The plant hormone jasmonate (JA) activates gene expression by promoting ub

121 TOR1 (ERF1) is an upstream component in both jasmonate (JA) and ethylene (ET) signaling and is involv

122 strate for the green leaf volatile (GLV) and jasmonate (JA) biosynthesis pathways, respectively.

123 5 min after induction, thereby preceding the jasmonate (JA) burst.

124 vity of the wound-inducible defence mediator jasmonate (JA) in undamaged tissues.

125 Jasmonate (JA) is a phytohormone that mediates various g

126 determine the effect of the defense hormone jasmonate (JA) on the growth, photosynthetic efficiency,

127 n of defence-related genes and activation of jasmonate (JA) pathway in the 'receiver' plant.

128 Here we show that jasmonate (JA) phytohormone both is required for and pro

129 The plant hormone jasmonate (JA) plays an important role in regulating gro

130 The plant hormone jasmonate (JA) plays an important role in regulating gro

131 Jasmonate (JA) promotes AAL toxin induced PCD in a COI1

132 The plant hormone jasmonate (JA) promotes the degradation of JASMONATE ZIM

133 nt studies have identified the plant hormone jasmonate (JA) receptor as one of the common targets of

134 affected in the organ-specific activation of jasmonate (JA) signaling in Arabidopsis thaliana seedlin

135 Jasmonate (JA) signaling in plants is mediated by the JA

136 Jasmonate (JA) signaling is essential for several enviro

137 Far-red (FR) light-coupled jasmonate (JA) signaling is necessary for plant defense

138 tric stimulation activates the touch hormone jasmonate (JA) signaling pathway, which initiates secret

139 1) mutants, suggesting LBD20 may function in jasmonate (JA) signaling.

140 indole and iridoid pathways that respond to jasmonate (JA) signaling.

141 ysis into acetate synthesis to stimulate the jasmonate (JA) signalling pathway to confer drought tole

142 * Here, we examined the impact of jasmonate (JA) treatment, branching and phloem girdling

143 activation of the intracellular signaling of jasmonate (JA), a well-characterized defense hormone.

144 ction of FAD7 also inhibits the synthesis of jasmonate (JA), the effects of this desaturase on aphid

145 :FR correlated with a reduced sensitivity to jasmonate (JA), thus resembling the antagonistic effects

146 ete neighbors but has a repressive effect on jasmonate (JA)-dependent defenses.

147 otic gut bacteria from CPB larvae suppressed jasmonate (JA)-induced defenses in tomato.

148 s varies greatly and leaves are protected by jasmonate (JA)-inducible defenses.

149 As a consequence, JAZ8 is stabilized against jasmonate (JA)-mediated degradation and, when ectopicall

150 3) protein is essential to fully enhance the jasmonate (JA)-mediated responses.

151 Mediator complex is a positive regulator of jasmonate (JA)-responsive gene expression in Arabidopsis

152 which structurally mimics the plant hormone jasmonate (JA).

153 ct on both basal and induced biosynthesis of jasmonates (JA).

154 g deficiencies were complemented with methyl jasmonate, JA-Ile, and its functional homolog, coronatin

155 Compromised jasmonate (jar1-1) and ethylene (ein2-1) signaling or ab

156 Jasmonates (JAs) are a class of signaling compounds that

157 Jasmonates (JAs) are signaling molecules that have been

158 ely to be caused by the over-accumulation of jasmonates (JAs) in the llb mutant including the JA prec

159 esponsible for wound-inducible production of jasmonates (JAs), and green leafy volatiles (GLVs) respe

160 However, only endogenous levels of jasmonates (JAs), but not salicylic acid (SA) and abscis

161 Jasmonic acid and its derivatives (jasmonates [JAs]) play central roles in floral developme

162 In the presence of active jasmonate, JAZ proteins function as jasmonate co-recepto

163 he oxi1 mutation was associated with reduced jasmonate levels and with the up-regulation of genes enc

164 pplications of phenylalanine (Phe) or methyl jasmonate (MeJ) could improve the synthesis of secondary

165 tudy the effect of the elicitation of methyl jasmonate (MeJ) supported by phenylalanine (Phe) as a pr

166 e, this work studied elicitation with methyl jasmonate (MeJ), supported by precursor feeding with phe

167 vest life, preharvest applications of methyl jasmonate (MeJA) and chitosan were evaluated during post

168 Exposure to methyl jasmonate (MeJA) and methyl salicylate (MeSA) vapours at

169 Methyl jasmonate (MeJA) elicits stomatal closure in many plant

170 Methyl jasmonate (MeJA) induced AtAO1 gene expression in vascul

171 Methyl jasmonate (MeJA) is commonly used to elicit plant stress

172 The effects of methyl jasmonate (MeJA) on leaf growth regulation were investig

173 Foliar application of methyl jasmonate (MeJA) or brassinazole (BRZ) resulted in a sig

174 with the well-known defense elicitor methyl jasmonate (MeJA) to young leaves of Arabidopsis (Arabido

175 Methyl jasmonate (MeJA) treatment increased betacyanin synthesi

176 pine needle transcriptomes, following methyl jasmonate (MeJA) treatment using RNA-seq.

177 iptional changes in sweet basil after methyl jasmonate (MeJA) treatment, which is considered an elici

178 Methyl jasmonate (MeJA) was applied in a vineyard on leaves and

179 in response to abscisic acid (ABA) or methyl jasmonate (MeJA) were identified by complementary proteo

180 the treatment doses of the elicitors: methyl jasmonate (MeJA), jasmonic acid (JA) and DL-methionine (

181 s of (3)C* and (1)O2* with five GLVs: methyl jasmonate (MeJa), methyl salicylate (MeSa), cis-3-hexeny

182 of phytohormone salicylic acid (SA), methyl jasmonate (MeJA), phytopathogenic bacteria, and flagelli

183 The effects of methyl jasmonate (MeJA), spermine (Spm), epibrassinolide (EBL)

184 onally characterized a leaf-specific, methyl jasmonate (MeJA)-responsive monoterpene synthase (Li3CAR

185 tebark pine needles demonstrated that methyl jasmonate (MeJA)-triggered transcriptome re-programming

186 es occur when the plant is exposed to methyl jasmonate (MeJA).

187 fferentially regulated in response to methyl jasmonate (MeJA).

188 strongly induced by the phytohormone methyl jasmonate (MeJA).

189 rant cultivars after elicitation with methyl jasmonate (MJ) were examined.

190 ers present in a commercial sample of methyl jasmonate (MJ) were isolated at semi-preparative scale b

191 he influence of foliar application of methyl jasmonate on must amino acid content.

192 L-carboxylic acid, salicylic acid and methyl jasmonate) on the phytochemical composition of broccoli

193 In seedlings, we found that jasmonate (or JA precursors) could translocate axially f

194 caterpillar herbivory, application of methyl jasmonate, or mechanical damage during vegetative growth

195 Jasmonates, oxylipin-type plant hormones, are implicated

196 of oxidative and hydrolytic branches in the jasmonate pathway highlight novel mechanisms of JA-Ile h

197 vary incrementally in the expression of the jasmonate pathway, which mediates induced resistance to

198 nd contrasting effects were observed for the jasmonate pathway.

199 vores induce plant resistance mainly via the jasmonate pathway.

200 Arabidopsis mutants that are deficient in jasmonate perception (coronatine insensitive1) or in the

201 ll discuss how several pathogens exploit the jasmonate perception and early signalling machinery to d

202 We identify a role of VIH2 in regulating jasmonate perception and plant defenses against herbivor

203 gulated in ago1 mutants, we demonstrate that jasmonate perception drives the lesion phenotype.

204 The plant hormone jasmonate plays crucial roles in regulating plant respon

205 ottontail rabbit Sylvilagus nuttallii avoids jasmonate-producing N. attenuata shoots because of their

206 by arthropod and vertebrate herbivores than jasmonate-producing plants in nature.

207 and biosynthesis of distinct MIAs following jasmonate production.

208 the first step of lipid oxidation leading to jasmonate production.

209 dation but not for the pathogen-induced free jasmonate production.

210 By contrast, treatment with methyl jasmonate promoted an increase of all analyzed AtPep-tri

211 up-regulation is negatively regulated by the jasmonate receptor Coronatine Insensitive1 (COI1), as lo

212 nts of ago1 and coronatine insensitive1, the jasmonate receptor, showed greatly decreased frequency o

213 vincristine and vinblastine, we identified a jasmonate-regulated basic helix-loop-helix (bHLH) transc

214 InsP8 by COI1-JAZ is a critical component in jasmonate-regulated defenses.

215 as associated with reduced expression of the jasmonate-regulated PFD1.2 gene, accelerated development

216 se involving crosstalk between the auxin and jasmonate regulatory pathways.

217 ylate-based immunity and the repression of a jasmonate-related branch.

218 factors that are known to additively control jasmonate-related defense responses, was shown to have a

219 ranscription factors that additively control jasmonate-related defense responses, we found that egg e

220 SA, JA and COR and co-operation between JAZ jasmonate repressor proteins during DC3000 infection.

221 d that NaJAZi functions as a flower-specific jasmonate repressor that regulates JAs, (E)-alpha-bergam

222 death phenotype was strongly reduced in the jasmonate resistant1 mutant background.

223 SA hydroxylase NahG transgene, but not in a jasmonate resistant1-1 mutant, after B. cinerea infectio

224 response and a K(+) -dependent Vm-activated jasmonate response associated with the release of VOCs.

225 fy ILL6 as a new regulatory component of the jasmonate response pathway.

226 nregulation of photosynthesis and attenuated jasmonate responses systemically within the plant.

227 Jasmonate responses were implicated in resistance to GLS

228 OI1-JAZ co-receptor to relieve repression of jasmonate responses.

229 ls and performs a key role in attenuation of jasmonate responses.

230 KEY MESSAGE: A methyl jasmonate responsive 3-carene synthase (Li3CARS) gene wa

231 Consequently, the expression of jasmonate-responsive genes was reduced as well.

232 s was associated with enhanced expression of jasmonate-responsive genes.

233 Last but not least, together with jasmonates, salicylate, and abscisic acid, ethylene is i

234 xpression of UPI is significantly induced by jasmonate, salicylic acid and abscisic acid, but is repr

235 more, the elicitations in the form of methyl jasmonate, salicylic acid, ultraviolet B light, and woun

236 egulator of downstream responses mediated by jasmonate-salicylic acid signaling cross talk, is involv

237 network robustness via its inhibition of the jasmonate sector.

238 s monocot crop against root nematodes, where jasmonate seems to play a key role.

239 sic acid sensing/signaling with ethylene and jasmonate sensing/signaling in RNAi compared to WT roots

240 In parallel to impaired jasmonate signaling and metabolism, irHER1 plants were m

241 ion of genes encoding negative regulators of jasmonate signaling and PCD.

242 ion has been shown previously to up-regulate jasmonate signaling and to increase plant resistance to

243 eus and, more generally, the cell biology of jasmonate signaling are not well understood.

244 illar feeding induces resistance through the jasmonate signaling pathway that is associated with the

245 and highlight a novel connection between the jasmonate signaling pathway, cell death, and sphingolipi

246 s in the control of leaf development via the jasmonate signaling pathway.

247 obal defense response is triggered involving jasmonate signaling, PR proteins and stilbenoid metaboli

248 on polymorphisms and deviated from canonical jasmonate signaling.

249 ling during herbivory, connecting Ca(2+) and jasmonate signaling.

250 stable protein JAZ10.1, which is involved in jasmonate signaling.

251 domonas syringae is modulated by SA, ET, and jasmonate signaling.

252 e growth and defense gene expression through jasmonate signaling.

253 Key mediators of jasmonate signalling include MYC transcription factors,

254 Loss of dormancy was dependent on an intact jasmonate signalling pathway and was associated with inc

255 In addition, jasmonate signalling plays a crucial role in the rhythmi

256 nvironment, illustrating the significance of jasmonate signalling, and of the proteins involved, for

257 thogens antagonize SA immunity by activating jasmonate signalling, for example Pseudomonas syringae p

258 wired to attenuate growth upon activation of jasmonate signalling.

259 The content of jasmonates, signalling molecules involved in glucosinola

260 of red raspberry fruits with the pure methyl jasmonate stereoisomers isolated proved that (-)-epi-MJ

261 or (methyl jasmonates) and minor (epi-methyl jasmonates) stereoisomers.

262 onsive to treatment with the defense hormone jasmonate, there are no significant changes in nucleosom

263 e tools with which to identify intercellular jasmonate transport routes.

264 reased tolerance to photooxidative damage of jasmonate-treated ch1 plants and by the increased tolera

265 highest in leaves, and was induced by methyl jasmonate treatment and wounding.

266 induction of MusaSAP1 by wounding and methyl jasmonate treatment indicated possible involvement of Mu

267 This finding suggests that methyl jasmonate treatment might be conducive to obtain wines o

268 In response to methyl jasmonate treatment, LIF2 was rapidly recruited to chrom

269 ced after F. oxysporum inoculation or methyl jasmonate treatment.

270 We found that upon wounding and methyl jasmonate treatments, MYB11 and ZML2 proteins are degrad

271 fection or after benzothiadiazole and methyl jasmonate treatments.

272 induced in leaves by alamethicin and methyl jasmonate treatments.

273 Hormonal elicitors, such as jasmonates, trigger a complex signaling circuit leading

274 ponse to the exogenous application of methyl jasmonate was associated with increased bark concentrati

275 opening induced by exogenous applied methyl jasmonate was impaired in osjar1 plants and was restored

276 significant reduction in the accumulation of jasmonates was observed, due to reduced expression of JA

277 Low R:FR reduced the stem levels of jasmonate, which is a known inducer of flavonoid synthes

278 .9%, and 0.18 mg for (-)- and (+)-epi-methyl jasmonates, with 98.6% and 91.6% respective purities.

279 amounts were 3.56 mg for (-) and (+)-methyl jasmonates, with respective purities of 96.1% and 99.9%,

280 The JASMONATE ZIM DOMAIN (JAZ) proteins function as negative

281 ncluding CORONATINE INSENSITIVE 1 (COI1) and JASMONATE ZIM domain (JAZ) proteins.

282 expression by stabilizing the interaction of JASMONATE ZIM domain (JAZ) repressors with the F-box pro

283 promoting ubiquitin-dependent degradation of jasmonate ZIM domain (JAZ) transcriptional repressor pro

284 We used a JASMONATE ZIM DOMAIN 10 (JAZ10) reporter to screen for m

285 the SA effect, requires a functional SCFCOI1-JASMONATE ZIM-DOMAIN (JAZ) JA receptor module.

286 JAsmonate ZIM-domain (JAZ) proteins repress the activity

287 (JA) signaling in plants is mediated by the JASMONATE ZIM-DOMAIN (JAZ) proteins that repress the act

288 e jasmonate (JA) promotes the degradation of JASMONATE ZIM-DOMAIN (JAZ) proteins to relieve repressio

289 As with other characterized JASMONATE ZIM-DOMAIN (JAZ) proteins, PtJAZ6 interacts wi

290 ense-related genes, including those encoding jasmonate ZIM-domain (JAZ) proteins, which play key role

291 ved by a co-receptor complex composed of the Jasmonate ZIM-domain (JAZ) repressor proteins and an E3

292 ranscription factors, which are repressed by jasmonate ZIM-domain (JAZ) transcriptional repressors in

293 ears to involve direct activation of several jasmonate ZIM-domain genes, encoding repressors of the J

294 8) that was induced by salicylic acid; and a jasmonate ZIM-domain protein 1 (DMG400002930) which was

295 ullin-F-box complex SCF(COI1), which targets JASMONATE ZIM-domain transcriptional repressor proteins

296 psis mutant (jazQ phyB) lacking a quintet of Jasmonate ZIM-domain transcriptional repressors and the

297 t did not enhance expression or stability of JASMONATE ZIM-domain transcriptional repressors, and SA/

298 n and removal of transcriptional repressors (JASMONATE-ZIM DOMAIN [JAZ] proteins) by an SCF receptor

299 D94N negatively affects the interaction with JASMONATE-ZIM DOMAIN protein, thereby resulting in const

300 HYTOCHROME-INTERACTING FACTOR3 (PIF3), PIF4, JASMONATE-ZIM DOMAIN1, and BRASSINAZOLE-RESISTANT1 (BZR1