ライフサイエンスコーパス: jaundice

1 newborn children and leads to physiological jaundice.

2 jugated bilirubin in the blood, resulting in jaundice.

3 mporary relief for patients with obstructive jaundice.

4 onset of nausea, vomiting, malaise and deep jaundice.

5 ition to modulating the severity of neonatal jaundice.

6 6-week-old boy who presented with prolonged jaundice.

7 for investigation of infants with persistent jaundice.

8 atient with complaints of abdominal pain and jaundice.

9 by high levels of aminotransferases and mild jaundice.

10 n effective method of palliating obstructive jaundice.

11 design a simple clinical diagnostic tool for jaundice.

12 e obstructive jaundice than in those without jaundice.

13 talloporphyrins in the treatment of neonatal jaundice.

14 omplications including bile leak, biloma, or jaundice.

15 therapeutic target for clinical treatment of jaundice.

16 humans as epigastric pain, weight loss, and jaundice.

17 ology, prognostic score, dyspnea, fever, and jaundice.

18 th encephalopathy, bleeding, and cholestatic jaundice.

19 hyrin is useful in the treatment of neonatal jaundice.

20 roved pharmaceutical treatments for neonatal jaundice.

21 reat neonatal, genetic, or acquired forms of jaundice.

22 y used in Asia to prevent and treat neonatal jaundice.

23 t of CAR activity may contribute to neonatal jaundice.

24 No patients had recurrent jaundice.

25 rase (ALT) elevations, and then symptoms and jaundice.

26 tients with clinically suspected obstructive jaundice.

27 to accurately predict clinically significant jaundice.

28 inversin gene results in situs inversus and jaundice.

29 ent and only one patient developed temporary jaundice.

30 and 89% and 94% in patients who did not have jaundice.

31 HCV]-coinfected) developed light-to-moderate jaundice.

32 naemia sometimes presents as an intermittent jaundice.

33 HCV seroconversion illness, including 2 with jaundice.

34 13), which rose to 45.5% in the presence of jaundice.

35 rval, 1.2-6.1) in pregnancies complicated by jaundice.

36 ed UGT1 (hUGT1) mice physiologically develop jaundice.

37 severe bacterial infections, or pathological jaundice.

38 ears; mean age, 66.9 years) with obstructive jaundice.

39 rd clamping groups required phototherapy for jaundice.

40 ion- and drug-induced hemolysis and neonatal jaundice.

41 these infants, 6 of 15 (40%) presented with jaundice, 1 of whom also had petechiae.

42 reatitis (48% vs. 24%, P < 0.05) rather than jaundice (11% vs. 30%, P < 0.05) or cholangitis (0% vs.

43 learance (42.9%) compared with those without jaundice (13.7%).

44 The prevalence of abdominal pain was 75%, jaundice 25%, weight loss 42%, steatorrhea 37.5%, diabet

45 levels > 15 mg/dL; P < .001), more prolonged jaundice (25% vs. 0% with peak bilirubin >5 weeks after

46 cases typically presented with both clinical jaundice (254/318 [80%]) and severe anemia (hemoglobin l

47 20 days (range, 8-77 days); 26 patients had jaundice (27%) and 22 patients were hospitalized (23%) f

48 .001) and children were more likely to have jaundice (31.9% vs 11.6%; P < .001).

49 tal DILI (23% versus 4%, P = 0.001), but not jaundice (46% versus 35%, P = 0.2) or liver transplantat

50 equent symptoms predicted pancreatic cancer: jaundice (51 [49%] of 105 patients with pancreatic cance

51 28B rs12979860 C/C more frequently developed jaundice (53.2% vs 27.6%; P = .022) than carriers of the

52 istress, (4) initial low blood pressure, (5) jaundice, (6) rupture of liver abscess, (7) endophthalmi

53 learance (56.3%) compared with those without jaundice (60.6%).

54 included pain (67%), weight loss (65%), and jaundice (62%).

55 ase by our service; 36 of these patients had jaundice (7 percent).

56 ic symptoms (23%), pain (12%), dyspnea (9%), jaundice (7%) or other symptoms (15%).

57 dominal pain (25%), fullness/mass (10%), and jaundice (7%); 47% were asymptomatic.

58 in the blood [15,19], as occurs in neonatal jaundice [7].

59 = 50) had significantly higher frequency of jaundice (74% versus 40%, P = 0.0001).

60 62 y) men (85%), presenting with obstructive jaundice (77%) associated with autoimmune pancreatitis (

61 The majority had ascites (91%), jaundice (88%), elevated prothrombin time (18 +/- 3 seco

62 in whom surgery was unsuccessful in clearing jaundice 9 (22%) died and 30 (73%) underwent liver trans

63 Newborns are at increased risk of jaundice, a condition in which excess bilirubin accumula

64 Early clearance of jaundice after portoenterostomy was achieved in 81 of 14

65 When patients develop jaundice after transplant, the time to search for treata

66 y advanced disease) and later development of jaundice after transplantation predicted inferior surviv

67 y of diagnosis, multiple causes of symptoms, jaundice, an initial therapy algorithm, secondary therap

68 ed liver disease (REILD) has been defined as jaundice and ascites appearing 1 to 2 months after RE in

69 ologically confirmed VOD who had progressive jaundice and ascites.

70 alcohol (age, 18-75 y) with recent onset of jaundice and biopsy-proven severe AH in our study, perfo

71 pe in particular is associated with neonatal jaundice and circulation of bilirubin in blood at high c

72 is characterized by an abrupt development of jaundice and complications related to liver insufficienc

73 vestigated the etiology and risk factors for jaundice and death.

74 nd encephalopathy 6 weeks after the onset of jaundice and fatigue.

75 iated cholangitis presented with obstructive jaundice and had increased serum IgG4 levels and IgG4-po

76 pigment responsible for the yellow color of jaundice and healing bruises.

77 variate analysis disclosed that preoperative jaundice and intraoperative blood transfusion were posit

78 Bilirubin, a key biomarker for the jaundice and its clinical diagnosis needs a better analy

79 , a well-known formulae for the treatment of jaundice and liver disorders, against the cholestasis us

80 Alcoholic hepatitis (AH) is a syndrome of jaundice and liver failure that occurs in a minority of

81 itis is a clinical syndrome characterized by jaundice and liver impairment that occurs in patients wi

82 All had jaundice and markedly abnormal results on liver function

83 iasis, often in association with obstructive jaundice and pancreatitis.

84 th C3H/HeJ mouse strains was associated with jaundice and pulmonary hemorrhage, similar to the patien

85 ystitis subsequently developed waxing waning jaundice and recurrent episodes of upper gastrointestina

86 ded a higher level of monitoring for newborn jaundice and treatment of hyperbilirubinemia in an effor

87 She remained jaundiced and a liver biopsy revealed cirrhosis with reg

88 All were jaundiced and had low albumin levels, and most had coagu

89 efinition, were less likely (p < 0.01) to be jaundiced and more likely (p < 0.05) to present with abd

90 The adult jaundiced and spherocytic mice present with greatly decr

91 Jaundiced and spherocytic mice were analyzed histopathol

92 one-half of bilirubin, the yellow pigment of jaundice) and its homologues with hexanoic and longer ac

93 many (34.3%) had concurrent dehydration and jaundice, and 29% were admitted through emergency depart

94 nts, 85% and 81% in those who presented with jaundice, and 89% and 94% in patients who did not have j

95 normalization of pruritus, disappearance of jaundice, and alanine aminotransferase (ALT) levels <1.5

96 ediatric clinician-- immunizations, neonatal jaundice, and animal-induced injuries-are concisely revi

97 clinical pediatrics: immunizations, neonatal jaundice, and animal-induced injuries.

98 ticing pediatrician: immunizations, neonatal jaundice, and animal-induced injuries.

99 inues to do well with resolution of ascites, jaundice, and coagulopathy as of her last outpatient vis

100 r damage, TG2(-/-) mice had more gallstones, jaundice, and ductal proliferation than wild-type mice.

101 nage a wide range of ailments such as edema, jaundice, and gonorrhea.

102 toxicity, thrombocytopenia, nausea, fatigue, jaundice, and muscle aches.

103 On multivariate analysis, hyperparasitemia, jaundice, and shock were all associated independently wi

104 examination revealed the absence of fever or jaundice, and the laboratory tests, including that for p

105 ophysiology of breastfeeding and breast milk jaundice, and the realization that Gilbert's syndrome ma

106 enterostomy, may restore bile flow and clear jaundice, and, if successful, achieve a 10-year survival

107 evere distress, with unstable vital signs, a jaundiced appearance, and substantial pain in her chest

108 trics treatment recommendations for neonatal jaundice are based on age-specific total serum bilirubin

109 The most common causes of cholestatic jaundice are biliary atresia and idiopathic neonatal hep

110 itis, biphasic fever, flaccid paralysis, and jaundice are typical manifestations of diseases in human

111 r treatable causes is early in the course of jaundice, as the risk of mortality rises steeply with sm

112 Hepatitis E virus (HEV) causes outbreaks of jaundice associated with maternal mortality.

113 sure total bilirubin in neonates at risk for jaundice at Queen Elizabeth Central Hospital in Blantyre

114 The duration of jaundice before onset of encephalopathy ranged from 4 to

115 We assessed clearance of jaundice (bilirubin <20 micromol/L) as an early outcome

116 with exaggerated physiologic and pathologic jaundice but adapts it to the microfluidic level for the

117 clamping is not apparently a risk factor for jaundice but warrants more study.

118 loss of IFN-gamma did not alter the onset of jaundice, but it remarkably suppressed the tissue-specif

119 A Chinese herbal remedy for jaundice called Yin Zhi Huang is now shown to activate a

120 Left untreated, jaundice can lead to neurological impairment and death.

121 performed to minimize risks of pancreatitis, jaundice, cholangitis, and stenosis.

122 transplantation, all four patients developed jaundice, cholestatic elevation of liver enzymes, and hi

123 r appearance at KPE, and early postoperative jaundice clearance are significant predictors of transpl

124 o thrive and had progressive cholestasis and jaundice, coagulation disorders, bilateral ureterostomie

125 Preoperative obstructive jaundice considerably increases perioperative risk.

126 The management strategy for neonatal jaundice continues to focus on screening and prevention.

127 as admitted in the emergency department with jaundice, dark urine and pale stools.

128 ed with an increased risk of readmission for jaundice, dehydration, and sepsis.

129 patients with right upper quadrant pain and jaundice.Detailed imaging by MRI/MRCP should be done.

130 Preoperative jaundice did not protect against biliary stricture forma

131 Rates of gastrointestinal disorders, jaundice, dry skin, and photosensitivity were increased

132 ral days, the knockout mice developed severe jaundice due to an increase in unconjugated serum biliru

133 Although rare, obstructive jaundice due to external bile duct compression or ruptur

134 Jaundice during acute infection was more common among pa

135 In C/C patients, jaundice during acute infection was not associated with

136 risk of perinatal mortality associated with jaundice during pregnancy.

137 with HH were screened for the development of jaundice during the course of HH.

138 rated an increase in hepatic enzymes without jaundice during the pregnancy.

139 that ajmaline may induce severe cholestatic jaundice even after a single dose administration.

140 ed the observation that acute hepatocellular jaundice from a drug is associated with death or the nee

141 Patients who developed jaundice (group 1) needed vasopressor treatment (P < 0.0

142 ine (P < 0.05), compared to patients without jaundice (group 2).

143 is tissue specific, resulting in progressive jaundice, growth failure, and greater than 90% mortality

144 Double heterozygous mice exhibit jaundice, growth retardation, impaired differentiation o

145 Jaundiced Gunn rat pups (jjs) exhibit similar BAEP abnor

146 In summary, biliverdin administration in jaundiced Gunn rat pups produces BAEP abnormalities cons

147 oked potential (BAEP) abnormalities occur in jaundiced Gunn rats given sulfadimethoxine to displace b

148 UDP-glucuronosyltransferase (BUGT)-deficient jaundiced Gunn rats with a recombinant adenovirus (5 x 1

149 tal vein catheter in bilirubin-UGT-deficient jaundiced Gunn rats, mean serum bilirubin concentrations

150 cted into the main portal vein of deficient, jaundiced Gunn rats.

151 -glucuronosyltransferase-1 (BUGT1)-deficient jaundiced Gunn rats.

152 the C/T or T/T genotype who did not develop jaundice had a lower chance of spontaneous clearance of

153 Although obstructive jaundice has been associated with a predisposition towar

154 Bilirubin, an abundant pigment that causes jaundice, has long lacked any clear physiologic role.

155 Symptomatic patients with jaundice have a higher likelihood of spontaneous viral c

156 Total diagnostic intervals were shorter when jaundice (hazard ratio [HR] 1.38, 95% CI 1.07-1.78; p=0.

157 surgery, liver transplantation, obstructive jaundice, hepatitis C antiviral treatment) does not impr

158 e of severe nonspherocytic hemolytic anemia, jaundice, hepatosplenomegaly, and marked erythroblastosi

159 Patients exhibit prolonged neonatal jaundice, hepatosplenomegaly, and progressive neurodegen

160 high death rate, particularly in those with jaundice; however, children and human immunodeficiency v

161 measuring unbound unconjugated bilirubin in jaundiced human newborns or animal models of kernicterus

162 Both patients presented with jaundice, hyperbilirubinemia, and mild-to-moderate eleva

163 le duct destruction and effect resolution of jaundice if given early.

164 or neoplasms were identified as the cause of jaundice in 11 patients (30 percent), with 4 having intr

165 was manifested as weight gain, ascites, and jaundice in 7 patients.

166 st common clinical condition associated with jaundice in adults is Gilbert's syndrome, which is chara

167 valuate the incidence and clinical effect of jaundice in critically ill patients with HH.

168 resia is the commonest cause of pathological jaundice in infants and the leading indication for liver

169 the association between breast feeding with jaundice in mice.

170 ase to identify studies on the management of jaundice in patients undergoing PD or liver resection.

171 No clinical data exist about new onset of jaundice in patients with HH.

172 ts (CDC) are important causes of obstructive jaundice in pediatric patients.

173 this study, we examined the pathogenesis of jaundice in the inv mouse, a transgenic mouse in which a

174 The management of jaundice in the newborn infant is an area of clinical pr

175 c hepatitis, as indicated by recent onset of jaundice in the prior 3 months and a Maddrey score of at

176 IDS), the causes, evaluation, and outcome of jaundice in these patients have not been systematically

177 ppropriate solutions to diagnose and monitor jaundice in these settings.

178 utside hospital with symptoms of obstructive jaundice, including abdominal pain and yellowing of the

179 erations and functional impairment caused by jaundice increase the risk of surgery; therefore, preope

180 llular drug-induced liver injury (DILI) with jaundice indicates a serious reaction, is used widely to

181 3) ITx and was characterized by intermittent jaundice, intractable pruritus, increased serum bile aci

182 Jaundice is a common finding during the course of HH.

183 Obstructive jaundice is associated with immunologic derangements and

184 Although neonatal jaundice is mostly benign, excessively high levels of se

185 The extreme jaundice is present as a phenotype in skin color after 8

186 only help confirm that the story of neonatal jaundice is still unfolding.

187 In conclusion, jaundice is uncommon in AIDS and may result from a varie

188 h a one-month history of epigastric pain and jaundice, itching, flushing, cough and wheezing.

189 m birth, hypoglycemia, respiratory distress, jaundice, large for gestational age, and hospitalization

190 nsferase >3 times the upper limit of normal, jaundice, liver failure, liver transplantation, or fatal

191 nonresponders and exhibited earlier onset of jaundice (<9 months), neonatal cholestasis, and higher A

192 sent with biliary strictures and obstructive jaundice, making cholangiocarcinoma (CCA) an important d

193 bilirubin concentration (Bf), would improve jaundice management as it better predicts bilirubin neur

194 have found that phototherapy and/or neonatal jaundice may be associated with asthma.

195 The resulting jaundice may be managed with phototherapy to isomerize t

196 Although transcutaneous quantification of jaundice may help discern which patients warrant further

197 ng Bf and BT into the management of neonatal jaundice may help move clinical practice from its tradit

198 d that the immunosuppression associated with jaundice may result from the functional impairment of li

199 Bodybuilding HDS caused prolonged jaundice (median, 91 days) in young men, but did not res

200 were observed in cardiac tissue from 4 of 13 jaundiced mice and 15 of 15 spherocytic mice, and thromb

201 icular valves or within the atria of 2 of 13 jaundiced mice and 15 of 15 spherocytic mice.

202 mplete absence of erythroid beta-spectrin in jaundiced mice leads to no detectable structural defects

203 Jaundiced mice, ja/ja, suffer from a severe hemolytic an

204 Blood parameters and histopathology of the jaundiced mouse were compared with that from spherocytic

205 toimmune hepatitis (n = 10), and obstructive jaundice (n = 12).

206 cluded parasitemia >100 000/microL (n = 18), jaundice (n = 20), respiratory distress (n = 14), hypote

207 gy Clinic for urgent evaluation of new onset jaundice, nausea and fatigue associated with a >40-fold

208 ist, gastroenterologist, and radiologist) of jaundiced neoplastic patients should be performed before

209 ion by the combined method in plasma from 20 jaundiced newborns was significantly greater than and po

210 In summary, significant jaundice occurred in 26% of patients and was predominant

211 New onset of jaundice occurred in 63 of 175 patients with HH (36%).

212 Four deaths among pregnant women with jaundice occurred in an urban community near Dhaka, Bang

213 tres with an early success rate for clearing jaundice of 55% overall.

214 port the possibility that the phenomenon of "jaundice of sepsis" represents an adaptive physiological

215 syndrome may play a greater role in neonatal jaundice, only help confirm that the story of neonatal j

216 Patency was defined as absence of jaundice or cholangitis that necessitated hospitalizatio

217 (95% CI 51,000-380,000) cases with fever and jaundice or haemorrhage including 78,000 (95% CI 19,000-

218 by hepatic complications such as cholestatic jaundice or hepatomegaly.

219 n-NICCD, infants with idiopathic cholestatic jaundice or INH were enrolled.

220 Individuals with preexisting jaundice or liver cirrhosis at the time of admission (n

221 R = 2.167, 95% CI: 2.104-13.150, p = 0.003), jaundice (OR = 1.9, 95% CI: 1.246-3.297, p = 0.008), rup

222 6.661 [95% CI, 2.126-20.876], P = .001) and jaundice (OR, 5.701 [95% CI, 1.776-18.306], P = .003) we

223 cant differences with gender, apneic spells, jaundice, or phototherapy.

224 ilatation of more than 5 mm (P < 0.001), and jaundice (P < 0.001) were statistically significant vari

225 (p =0.007), low blood pressure (p = 0.024), jaundice (p = < 0.001), rupture of liver abscess (p < 0.

226 T) > 3 x upper limit of normal (P = 0.10) or jaundice (P = 0.16).

227 were more likely to be white (P =.004), have jaundice (P =.03), and have lower peak viral titer (P =.

228 e confirmed Gilbert's patient, two recurrent jaundice patients (with suspected Gilbert's syndrome), a

229 can quantify GCDC acid serum on obstructive jaundice patients and can be used to support its pharmac

230 ntify the serum level of GCDC in obstructive jaundice patients.

231 ne sequences, obtained from serum samples of jaundiced patients from Laos.

232 Treatment-naive, jaundiced patients presenting to our tertiary unit betwe

233 Jaundiced patients represent a major challenge for surge

234 Papers considering palliative drainage in jaundiced patients were excluded.

235 the indications for preoperative drainage in jaundiced patients who are candidates for pancreaticoduo

236 e and to report on the current management of jaundiced patients with periampullary or proximal bile d

237 ultrasonography in the investigation of non-jaundiced patients with unexplained abdominal pain or we

238 bilirubin level of more than 7 mg/dL or, in jaundiced patients, an increasing bilirubin level on day

239 dominant disorder characterized by neonatal jaundice, paucity of intrahepatic bile ducts, and abnorm

240 She decompensated with jaundice, peripheral edema, ascites, encephalopathy, coa

241 ternal smoking during pregnancy, or neonatal jaundice predict islet autoimmunity in children at genet

242 article, we review recent research regarding jaundice predischarge risk assessment, current expert re

243 Clinical jaundice, present in 49 of 111 patients, was resolved un

244 cluding antifibrinolytic agents, obstructive jaundice, prostaglandin inhibitors, cyclosporine A, radi

245 there were no clinical signs of cholestatic jaundice, pruritis, or liver dysfunction.

246 mary sclerosing cholangitis include fatigue, jaundice, pruritus, or steatorrhoea.

247 First, clinical assessment of jaundice remains critically important as "early discharg

248 Jaundice resolved in all NICCD and in 87.5% of non-NICCD

249 As a consequence, jaundice resolved, and long-term survival improved to gr

250 Mild gastrointestinal disorders, jaundice resulting from isolated unconjugated hyperbilir

251 Jaundice resulting from unconjugated hyperbilirubinemia

252 erm birth (RR = 1.06; 95% CI, 0.93-1.21), or jaundice (RR = 0.96; 95% CI, 0.48-1.91).

253 6%) who underwent PD developed postoperative jaundice secondary to a stricture of the biliary-enteric

254 e role of preoperative ERCP in patients with jaundice secondary to pancreatic cancer was raised in a

255 Thus, obstructive jaundice selectively expands liver myeloid DCs that are

256 N: A 50 year male presented with acute onset jaundice, significant weight loss and elevated liver enz

257 d abrupt onset of fever, abdominal pain, and jaundice, sometimes with the presence of ascites even in

258 al recent series with up to 20% mortality in jaundiced subjects.

259 Among patients who were deeply jaundiced, survival was related to the absence of multio

260 auma, eight vs three; sepsis, six vs 13; and jaundice, ten vs 12 after vaginal delivery and caesarean

261 ly in patients with preoperative obstructive jaundice than in those without jaundice.

262 ms could contribute to pruritus and painless jaundice that occur during cholestatic liver diseases.

263 transferase level, 198 U per liter; none had jaundice); the levels of alanine aminotransferase and HG

264 ale) had other documented causes of neonatal jaundice; the mean ages at US assessment were 48.5 and 5

265 age <24 months (GRAFT), and time from onset jaundice to encephalopathy <7 days (PATIENT).

266 cCrCl <60 mL/min/1.73m, and time from onset jaundice to encephalopathy <7 days.

267 testing the hypothesis that the duration of jaundice up to a given point in time provides more progn

268 ian time to stricture formation resulting in jaundice was 13 months (range, 1-106 months) and was sim

269 ed the acute event of HH, median duration of jaundice was 6 days (interquartile range, 3-8).

270 In contrast, in non-C/C patients, jaundice was associated with a higher likelihood of spon

271 Occurrence of jaundice was associated with an increased frequency of c

272 (adjusted HR, 2.43; 2.21-2.66), and neonatal jaundice was associated with more than a 50% increased r

273 The major cause for obstructive jaundice was choledocholithiasis.

274 Jaundice was defined as a serum bilirubin concentration

275 The etiology of jaundice was determined by the pattern of liver biochemi

276 Jaundice was diagnosed in patients with plasma total bil

277 Jaundice was more frequent in patients with cancer (12.5

278 bin, the yellow-orange neurotoxic pigment of jaundice, was synthesized following Friedel-Crafts acyla

279 s of end-stage liver disease can appear with jaundice, weakness, wasting, and gastrointestinal bleedi

280 The most common causes of jaundice were drug-induced hepatitis, occurring in 11 pa

281 symptoms such as nausea, fever, fatigue, and jaundice were palliated in 35% of group 1 and 90% of gro

282 low pH, hyperparasitemia, severe anemia, and jaundice were statistically significant indicators of de

283 acute BCS had a significantly higher rate of jaundice whereas a lower rate of abdominal and chest var

284 ater, he presented with painless cholestatic jaundice which peaked in severity at eleven weeks after

285 nusual clinical manifestation of obstructive jaundice (which has not been reported so far) along with

286 on has been suggested as a cause of neonatal jaundice, which can further cause permanent brain damage

287 pparently healthy and persons suffering from jaundice, which correlated well with a standard colorime

288 Jaundice, which is caused by accumulation of bilirubin,

289 Defects in this process result in jaundice, which is particularly common in neonates.

290 nient and efficient method to treat neonatal jaundice while allowing continuous breastfeeding.

291 resented with encephalopathy and cholestatic jaundice with a Hemoglobin S (HbS) level of 69.6%.

292 The patient was found to have obstructive jaundice with multiple mass lesions in the liver, spleen

293 nt accuracy in the evaluation of obstructive jaundice with regards to the level and cause of obstruct

294 ole of MDCT in the evaluation of obstructive jaundice with respect to the cause and level of the obst

295 ks of gestation by cesarean section, and was jaundiced, with low birth weight and height.

296 th hypoalbuminemia, cholangitis or long-term jaundice; with an FLR < 30% or 40%) given the high risk