ライフサイエンスコーパス: jejuni

1 Aeromonas, Vibrio cholerae O1, Campylobacter jejuni).

2 rapid and sensitive detection method for C. jejuni.

3 required to regulate flagellar number in C. jejuni.

4 or to the capnophilic growth phenotype of C. jejuni.

5 in vivo of a highly pathogenic strain of C. jejuni.

6 dentical structure purified directly from C. jejuni.

7 y metabolism and microaerobic survival in C. jejuni.

8 er intestinal inflammation in response to C. jejuni.

9 the detection of food pathogen Campylobacter jejuni.

10 athy most frequently caused by Campylobacter jejuni.

11 evelopment of GBS after an infection with C. jejuni.

12 tein while CtsP is membrane associated in C. jejuni.

13 l for their function in transformation of C. jejuni.

14 lasmic binding protein CeuE of Campylobacter jejuni.

15 ficile A/B toxins; and 90% for Campylobacter jejuni.

16 ain differences in protective immunity to C. jejuni.

17 e Ent trilactone esterase Cee (Cj1376) in C. jejuni.

18 iosynthesis of UDP-diNAcBac in Campylobacter jejuni.

19 ity of the pmf for motility and growth of C. jejuni.

20 lopment of vaccines against hypervirulent C. jejuni.

21 in dissemination of FQ(R) C. coli but not C. jejuni.

22 effectively control infections caused by C. jejuni.

23 to study determinants of pathogenicity of C. jejuni.

24 10(-/-);Rag2(-/-) mice were infected with C. jejuni (10(9) CFU/mouse).

25 0(-/-); Nod2(-/-) mice were infected with C. jejuni (10(9) colony-forming units/mouse) 24 hours after

26 Subsequently, we showed that C. jejuni 81-176 (wildtype) exhibited enhanced chemoattract

27 ence plasmid named pVir was identified in C. jejuni 81-176 and IA3902, but determining the role of pV

28 The capsule of C. jejuni 81-176 has been shown to be required for serum re

29 age on calcofluor white (CFW) resulted in C. jejuni 81-176 isolates with morphology changes: either a

30 C. jejuni 81-176 naturally lacks the fuc locus and is unabl

31 Campylobacter jejuni, a leading bacterial cause of foodborne illness,

32 Here we show that Campylobacter jejuni, a leading bacterial cause of human diarrheal ill

33 Campylobacter jejuni, a leading cause of bacterial gastroenteritis, is

34 In contrast to NLRP3 activation by ATP, C. jejuni activation did not require priming of these macro

35 A C. jejuni addAB mutant demonstrated enhanced sensitivity to

36 C. jejuni also activated the NLRP3 inflammasome in human ma

37 coli, one additional E. faecium, and one C. jejuni also developed resistance when exposed to 0.25 mi

38 the antimicrobial susceptibilities of 320 C. jejuni and 115 C. coli isolates obtained from feedlot ca

39 oquinolone resistance reached to 35.4% in C. jejuni and 74.4% in C. coli, which are significantly hig

40 th a couple of exceptions, the ecology of C. jejuni and C. coli differed, with the latter forming a m

41 sequences from 1,713 genetically diverse C. jejuni and C. coli genomes, supported by RT-PCR testing,

42 -characterised isolates, we sequenced 600 C. jejuni and C. coli isolates from various stages of poult

43 hape and extended this to a wide range of C. jejuni and C. coli isolates.

44 ecies conservation of the target genes in C. jejuni and C. coli Rare instances of a lack of specifici

45 yzed for Campylobacter species other than C. jejuni and C. coli using a filtration method and microae

46 0% for Shigella spp., 97.5% and 99.0% for C. jejuni and C. coli, and 100% and 99.7% for Shiga toxins,

47 7% for Shigella spp., 97.2% and 98.4% for C. jejuni and C. coli, and 97.4% and 99.3% for Shiga toxins

48 campylobacteriosis, caused by Campylobacter jejuni and C. coli, remains a leading cause of bacterial

49 t receptors (CfrA and CfrB) in Campylobacter jejuni and C. coli, the enteric human pathogens that do

50 Campylobacter jejuni and Campylobacter coli are zoonotic pathogens onc

51 ed laboratory, clinical and environmental C. jejuni and Campylobacter coli contained genetic changes

52 iated with diarrhoea-including Campylobacter jejuni and Campylobacter coli, Cryptosporidium spp, ente

53 and ceuE for the detection of Campylobacter jejuni and Campylobacter coli, leading global causes of

54 OTases, such as the PglBs from Campylobacter jejuni and Campylobacter lari, catalyze transfer of glyc

55 se elicited by the helix-shaped wild-type C. jejuni and complemented strains.

56 the increased pathogenicity of sialylated C. jejuni and may be key to the initiation of B cell-mediat

57 gher sequence diversity in the Campylobacter jejuni and Neisseria meningitidis genomes encoded hypoth

58 We identified the ability of both viable C. jejuni and purified flagellum to bind to Siglec-10, an i

59 leads to secretion of an alpha-dextran by C. jejuni and that a secreted protease, Cj0511, is required

60 uggest that the capsule polysaccharide of C. jejuni and the MeOPN modification modulate the host immu

61 te that LpxJ and homologues in Campylobacter jejuni and Wolinella succinogenes can act before the 2'

62 almonella spp., Shigella spp., Campylobacter jejuni, and Campylobacter coli and an EIA for Shiga toxi

63 ibitors of Bacillus anthracis, Campylobacter jejuni, and Clostridium perfringens IMPDHs.

64 into the molecular mechanism utilized by C. jejuni, and possibly other intestinal pathogens, to surv

65 glycosylation (Pgl) pathway of Campylobacter jejuni are evaluated for their tolerance for azide-modif

66 In this study, C. jejuni arsP was expressed in Escherichia coli and shown

67 Despite the importance of Campylobacter jejuni as a pathogen, little is known about the fundamen

68 abundant periplasmic c-type cytochrome in C. jejuni, as a novel and unexpected protein required for c

69 The central enzyme in the Campylobacter jejuni asparagine-linked glycosylation pathway is the ol

70 ate probability of illness for Campylobacter jejuni at the study beaches, especially where recreation

71 cells in vitro The importance of Ape1 in C. jejuni biology makes it a good candidate as an antimicro

72 adenovirus A, Salmonella spp., Campylobacter jejuni, bovine polyomavirus, and bovine rotavirus A were

73 d for in vitro competition with wild-type C. jejuni but is dispensable for growth in monoculture.

74 lodytis (33%), C. upsaliensis (7.7%), and C. jejuni/C. coli (2.6%).

75 and duplex PCR to distinguish Campylobacter jejuni/C. coli and non-jejuni/coli Campylobacter on 432

76 According to PCR, Campylobacter jejuni/C. coli infections represented less than half of

77 positive samples were positive by PCR for C. jejuni/C. coli, but 27.6% were positive for non-jejuni/c

78 We compared culture for C. jejuni/C. coli, EIA (ProSpecT), and duplex PCR to distin

79 ely, compared with the results of PCR for C. jejuni/C. coli.

80 es from Clostridium difficile, Campylobacter jejuni, Campylobacter concisus, and Salmonella enterica

81 cted 80/89 (89.9% sensitivity) Campylobacter jejuni/Campylobacter coli-positive cases.

82 Although C. jejuni can be transformed by C. jejuni-derived DNA, it i

83 he leading foodborne pathogen, Campylobacter jejuni, can carry multiple plasmids associated with anti

84 The Campylobacter jejuni capsular polysaccharide is important for virulenc

85 Most C. jejuni capsules are known to be decorated nonstoichiomet

86 port the crystal structures of Campylobacter jejuni Cas9 (CjCas9), one of the smallest Cas9 orthologs

87 To investigate how Campylobacter jejuni causes the clinical symptoms of diarrhoeal diseas

88 able tools for investigating the roles of C. jejuni cell surface glycoconjugates in host pathogen int

89 jejuni received an initial challenge with C. jejuni CG8421 with rechallenge 3 months later.

90 bjects underwent a primary infection with C. jejuni CG8421; 14 (93.3%) experienced campylobacteriosis

91 highly pathogenic, tetracycline-resistant C. jejuni clone (named SA) has become the predominant cause

92 and functional dynamics of the Campylobacter jejuni CmeB multidrug efflux pump.

93 thors present the structure of Campylobacter jejuni CmeB pump combined with functional FRET assays to

94 inguish Campylobacter jejuni/C. coli and non-jejuni/coli Campylobacter on 432 diarrheal and matched c

95 Sequencing of 16S rRNA from 53 of these non-jejuni/coli Campylobacter samples showed that it most cl

96 uni/C. coli, but 27.6% were positive for non-jejuni/coli Campylobacter species.

97 enteric pathogens tested, only Campylobacter jejuni/coli detection was significantly reduced in the O

98 evelopment of novel strategies to prevent C. jejuni colonization of food-producing animals or to trea

99 em was found to be required for efficient C. jejuni colonization of the chicken intestine.

100 ts with superoxide, rescued the growth of C. jejuni cultured in the presence of deoxycholate.

101 that in the gut of warm-blooded animals, C. jejuni depends on at least formate or hydrogen as donor

102 Although C. jejuni can be transformed by C. jejuni-derived DNA, it is poorly transformed by the same

103 ess than 1 in 1,000 persons infected with C. jejuni develop GBS, and the factors that determine GBS s

104 s a unique insight into the mechanisms of C. jejuni disease in terms of host physiology and contribut

105 e we report the complete transcriptome of C. jejuni during colonization of the chicken cecum and in t

106 sumption of meat that was contaminated by C. jejuni during harvest.

107 treatment (1 mM) did not reduce C. coli and jejuni during pure culture but did during co-culture wit

108 One phase-variable gene of Campylobacter jejuni encodes a homologue of an unusual Type IIG restri

109 (Bacteroides thetaiotaomicron, Campylobacter jejuni, Enterococcus faecalis, Escherichia coli K12, E.

110 igated dairy manure containing Campylobacter jejuni, enterohemorrhagic Escherichia coli (EHEC), or Sa

111 m-negative bacteria, including Campylobacter jejuni, Escherichia coli O157:H7, and multidrug resistan

112 The dispersion of pathogens (Campylobacter jejuni, Escherichia coli O157:H7, non-O157 E. coli, List

113 We report novel C. jejuni factors essential throughout its life cycle.

114 saccharyltransferase, PglB, of Campylobacter jejuni favors acceptor proteins with consensus sequences

115 Here, we report a detailed analysis of C. jejuni fitness across models reflecting stages in its li

116 expression, an intriguing observation as C. jejuni FlaA is not a TLR5 agonist.

117 agella-TLR5 driven pro-inflammatory axis, C. jejuni flagella instead promote an anti-inflammatory axi

118 The Campylobacter jejuni flagellum exports both proteins that form the fla

119 We explored how the C. jejuni flagellum is a versatile secretory organelle by e

120 Additionally, we found that C. jejuni FlhG influences FlhF GTPase activity, which may m

121 We discovered that Campylobacter jejuni FlhG is at the center of a multipartite mechanism

122 Instead, two regions of C. jejuni FlhG that are absent or significantly altered in

123 t exposure to pancreatic amylase protects C. jejuni from stress conditions in vitro, suggesting that

124 shows antibody is ineffective in clearing C. jejuni from the ceca within the production lifetime of c

125 aight morphology, representing the second C. jejuni gene affecting cell shape.

126 Further, we assessed the C. jejuni genes required for infection of the porcine gastr

127 We have searched C. jejuni genome for homologues and found one candidate tha

128 e essential for DNA replication) in other C. jejuni genomes.

129 apatA and DeltapatB had minimal impact on C. jejuni growth and fitness under the conditions tested.

130 ck of insight into the mechanisms driving C. jejuni growth and survival within hosts and the environm

131 Campylobacter jejuni harbors a branched electron transport chain, enab

132 of relevant disease models for Campylobacter jejuni has long been an obstacle to research into this c

133 hich has two major porins, OmpC and OmpF, C. jejuni has one, termed major outer membrane protein (MOM

134 Campylobacter jejuni helical shape is important for colonization and h

135 ptidase Pgp1 essential for maintenance of C. jejuni helical shape was recently identified.

136 e major contributors to microaerophily in C. jejuni; hemerythrins help prevent enzyme damage microaer

137 elated to the capsular polysaccharides of C. jejuni HS:4 is very remarkable, owing to the unique, mul

138 Recent work in C. jejuni identified a gene encoding a novel phosphoethanol

139 is first study of functional immunity to C. jejuni in chicken and shows antibody is ineffective in c

140 neumoniae in mice, and against Campylobacter jejuni in chicken.

141 More specifically, continuous growth of C. jejuni in deoxycholate was found to: 1) induce the produ

142 NPase) facilitates survival of Campylobacter jejuni in low temperatures and favors swimming, chick co

143 We recently found that growth of C. jejuni in medium with deoxycholate, a component of bile,

144 of 150 colony forming unit (CFU)mL(-1) of C. jejuni in solution.

145 onal role of B lymphocytes in response to C. jejuni in the chicken through depletion of the B lymphoc

146 required for efficient transformation in C. jejuni include those similar to components of type II se

147 In response to a low oxygen tension, C. jejuni increases the transcription and activity of the d

148 Lipo-oligosaccharides (LOS), expressed by C. jejuni induce antibodies that cross-react with self-glyc

149 C. jejuni-induced bactericidal NO production was reduced in

150 ortantly, sodium nitroprusside attenuated C. jejuni-induced colitis in Il10(-/-); Nod2(-/-) mice.

151 thermore, neutrophil depletion attenuated C. jejuni-induced crypt abscesses and intestinal inflammati

152 containing protein 2 (NOD2) in Campylobacter jejuni-induced intestinal inflammation.

153 some activation and likely contributes to C. jejuni-induced intestinal inflammation.

154 Our findings indicate that C. jejuni-induced PI3K-gamma signaling mediates neutrophil

155 nts, who previously developed GBS after a C. jejuni infection (n = 27) and controls (n = 26).

156 In this study, we report that C. jejuni infection of mouse macrophages induces upregulati

157 Genes preferentially expressed during C. jejuni infection were screened, and acs, cj1385, cj0259

158 severe intestinal inflammation following C. jejuni infection, compared with Nod2(-/-) and Il10(-/-)

159 Here we used an infant rabbit to study C. jejuni infection, which enables us to define several pre

160 al inflammation in response to intestinal C. jejuni infection.

161 oor understanding of the immunobiology of C. jejuni infection.

162 whereas pigs were a negligible source for C. jejuni infections.

163 Formate, a primary energy source for C. jejuni, inhibits oxidase activity in other bacteria.

164 ization and culture assay showed enhanced C. jejuni invasion into the colon and mesenteric lymph node

165 The C. jejuni invasion-related activation of the NLRP3 inflamma

166 cj1385, cj0259 seem to be responsible for C. jejuni invasion.

167 Campylobacter jejuni is a commensal bacterium in the intestines of ani

168 Campylobacter jejuni is a common cause of diarrhea and is associated w

169 Campylobacter jejuni is a helix-shaped enteric bacterial pathogen and

170 Campylobacter jejuni is a leading cause of bacterial gastroenteritis i

171 Campylobacter jejuni is a leading cause of bacterial gastroenteritis w

172 Campylobacter jejuni is a leading cause of gastrointestinal infections

173 Campylobacter jejuni is a major cause of bacterial diarrheal disease w

174 Campylobacter jejuni is a major cause of bacterial gastroenteritis wor

175 Campylobacter jejuni is a major zoonotic pathogen, and its resistance

176 Campylobacter jejuni is a natural commensal of the avian intestinal tr

177 Control of C. jejuni is a priority for the poultry industry but no vac

178 Campylobacter jejuni is a zoonotic pathogen, and a hypervirulent clone

179 Campylobacter jejuni is an important zoonotic pathogen transmitted to

180 C. jejuni is capable of systemic invasion in the rabbit, an

181 This mechanism of DNA discrimination in C. jejuni is distinct from the DNA discrimination described

182 C. jejuni is known to produce capsular polysaccharide (CPS)

183 The human pathogen Campylobacter jejuni is naturally competent for transformation with it

184 Campylobacter jejuni is one of the leading infectious causes of food-b

185 n the microaerophilic pathogen Campylobacter jejuni is potentially vulnerable, as it employs pyruvate

186 Campylobacter jejuni is the leading cause of foodborne bacterial gastr

187 The Gram-negative organism Campylobacter jejuni is the major cause of food poisoning.

188 Campylobacter jejuni is the most common bacterial cause of human gastr

189 The Gram-negative pathogen Campylobacter jejuni is the most common cause of bacterial foodborne d

190 the phosphoramidate moiety in the CPS of C. jejuni is unknown.

191 Utilizing a series of C. jejuni isogenic mutants we found the major flagellin pro

192 sourceR is demonstrated using Campylobacter jejuni isolate data collected in New Zealand between 200

193 he reannotation of the genome sequence of C. jejuni isolate NCTC 11168, chosen as being present in >9

194 riosis isolates, comprising 2,207 (89.3%) C. jejuni isolates and 265 (10.7%) C. coli isolates.

195 nt time periods and compared them with 42 C. jejuni isolates associated with sheep abortion during 20

196 Here, we analyzed 54 C. jejuni isolates collected from U.S. sheep abortions at d

197 In contrast, C. jejuni isolates from Great Britain were genetically dive

198 Campylobacter jejuni isolates from human (n = 65), bovine (n = 28), an

199 single Thr-86-Ile mutation in GyrA, FQ(R) C. jejuni isolates had other mutations in GyrA in addition

200 2 clades of Campylobacter jejuni subspecies jejuni isolates resulted in a prolonged outbreak among m

201 The mean probability of human C. jejuni isolates to originate from chickens was highest (

202 and time of isolation, while the analyzed C. jejuni isolates were genetically diverse, suggesting tha

203 is of biofilm formation in 102 Campylobacter jejuni isolates.

204 in both the OS and LA among 15 different C. jejuni isolates.

205 Campylobacter jejuni, known for being a major cause of bacterial gastr

206 antimicrobial activity against Campylobacter jejuni, L. monocytogenes, and Pseudomonas fluorescens.

207 ulations and provides evidence that major C. jejuni lineages have distinct genotypes associated with

208 C. jejuni lineages vary in host range and prevalence in hum

209 tion of the carbohydrate sialic acid into C. jejuni lipooligosaccharides (LOS) is associated with inc

210 Furthermore, analysis of C. jejuni localization within the ceca of infected mice det

211 High responsiveness to C. jejuni LOS by former GBS patients was evidenced by incre

212 hese results indicate that sialylation of C. jejuni LOS increases DC activation and promotes subseque

213 intrinsic dendritic cell (DC) response to C. jejuni LOS through Toll-like receptor 4 (TLR4) activatio

214 The DC response to C. jejuni LOS was highly variable between, but not within,

215 Intrinsic DC responsiveness to C. jejuni LOS was investigated first in 20 healthy controls

216 oxin producing E. coli (stx2), Campylobacter jejuni (mapA), Shigella spp. (ipaH), and a Salmonella en

217 Herein, we aimed to investigate C. jejuni-mediated effects on dendritic cell (DC) immunity.

218 We report the 2.1-A crystal structure of C. jejuni MOMP expressed in E. coli and a lower resolution

219 In certain environments, changes in C. jejuni morphology due to genetic heterogeneity may promo

220 We hypothesized that C. jejuni must repair DNA damage caused by reactive oxygen

221 Following deep sequencing of C. jejuni mutants in the cecal outputs, several novel facto

222 Analysis of a vast array of C. jejuni mutants with defects in capsule formation, LPS bi

223 Compared with C. jejuni NCTC 11168 and 81-176, a clone SA isolate (IA3902

224 opyranose residue found in the Campylobacter jejuni NCTC11168 (HS:2) capsular polysaccharide is repor

225 C. jejuni NCTC11168 forms less biofilms in the presence of

226 ve Escherichia coli (EIEC) and Campylobactor jejuni o C coli (around two times), and heat-stable ente

227 eca, the main site of colonisation, where C. jejuni persist to beyond commercial slaughter age, but r

228 ese analyses enhance our understanding of C. jejuni PG maturation and help to clarify how PG structur

229 While C. jejuni PglB (CjPglB) can transfer many diverse glycan st

230 een peptide and glycan substrates and the C. jejuni PglB offer new experimental information on substr

231 ity of the protein N-glycosylation enzyme C. jejuni PglB was tested using a logical, synthetic array

232 Here, oxygen-dependent changes in C. jejuni physiology were studied at constant growth rate u

233 orne microaerophilic pathogen, Campylobacter jejuni, possesses a periplasmic formate dehydrogenase an

234 an was diagnosed with probable Campylobacter jejuni prosthetic knee infection after a diarrheal illne

235 CeuE within the Fe(III) uptake system of C. jejuni, provide a molecular-level understanding of the u

236 unologic evidence of prior infection with C. jejuni received an initial challenge with C. jejuni CG84

237 ervirulent and rapidly expanding clone of C. jejuni recently emerged, which is able to translocate ac

238 A comparison of the C. jejuni requirements to colonize the mouse intestine with

239 Like many competent organisms, C. jejuni restricts the DNA that can be used for transforma

240 Inactivation of arsB in C. jejuni resulted in 8- and 4-fold reduction in the MICs o

241 t Escherichia coli and the native system, C. jejuni, revealed that efficient glycosylation of glycopr

242 elated with possession of the pathway for C. jejuni RM1221 (fuc+) and 81116 (fuc-).

243 , formate might play a role in optimizing C. jejuni's adaptation to the oxygen-limited gastrointestin

244 is mouse model was used to define whether C. jejuni's characteristic helical shape plays a role in it

245 Formate also significantly increased C. jejuni's growth, motility, and biofilm formation under m

246 A synthesis via RNR which is required for C. jejuni's growth.

247 these results support a critical role for C. jejuni's helical morphology in enabling it to traverse a

248 ve Microbial Risk Assessment was done for C. jejuni, Salmonella spp., and enteroviruses to estimate r

249 x 10(-6), and 3 x 10(-7) [corrected] for C. jejuni, Salmonella spp., and enteroviruses, respectively

250 T], Cj0202c from Campylobacter jejuni subsp. jejuni serotype O:2 (strain NCTC 11168) [PDB:3K7C], rumg

251 lue with E. coli K12 and rose bengal with C. jejuni showed an enhancing effect.

252 DNA derived from a ctsM mutant transforms C. jejuni significantly less well than DNA derived from cts

253 A genome-wide association study (GWAS) in C. jejuni ST-21 and ST-45 complexes identified genetic elem

254 h fecal specimens positive for Campylobacter jejuni (ST45) intermittently during a 10-year period.

255 drases (CAs) are encoded in the genome of C. jejuni strain NCTC 11168 (Cj0229 and Cj0237).

256 o-heptose synthesis pathway of Campylobacter jejuni strain NCTC 11168.

257 and phase-variants, the cj0031c allele in C. jejuni strain NCTC11168 was demonstrated to specifically

258 ivation libraries were generated in three C. jejuni strains and the impact on fitness during chicken

259 iverse Campylobacter fetus and Campylobacter jejuni strains have been implicated in such infections,

260 r fitness during in vitro growth in three C. jejuni strains, revealing that a large part of its genom

261 For C. jejuni, strains with the fuc locus possess a competitive

262 5482) [PDB:3KZT], Cj0202c from Campylobacter jejuni subsp. jejuni serotype O:2 (strain NCTC 11168) [P

263 al transmission of 2 clades of Campylobacter jejuni subspecies jejuni isolates resulted in a prolonge

264 In this model, C. jejuni successfully established infection and piglets de

265 due to genetic heterogeneity may promote C. jejuni survival.

266 In Campylobacter jejuni the periplasmic binding protein CeuE, an integral

267 Campylobacter jejuni, the leading cause of human bacterial gastroenter

268 Campylobacter jejuni, the most common cause of bacterial diarrhoeal di

269 Campylobacter jejuni, the most frequent cause of food-borne bacterial

270 ntified in invasive strains of Campylobacter jejuni, the most prevalent cause of bacterial gastroente

271 zation, and host-pathogen interactions in C. jejuni Therefore, changes in PG greatly impact the physi

272 y models of the N-linked glycoproteome of C. jejuni This evaluation highlights the potential diversit

273 All rod-shaped C. jejuni Tn mutants and all rod-shaped laboratory, clinica

274 We found C. jejuni to be a potent inducer of human and murine DC int

275 n understanding the mechanisms that allow C. jejuni to colonize the chicken gastrointestinal tract.

276 n N-glycosylation pathway from Campylobacter jejuni to Escherichia coli in 2002 can be considered as

277 f infection, the adhesion and invasion of C. jejuni to eukaryotic cells.

278 tead, secretion of CiaI was essential for C. jejuni to facilitate commensal colonization of the natur

279 required for the chemotactic response of C. jejuni to galactose, as shown using wild type, allelic i

280 Exposure of C. jejuni to pancreatic amylase promotes biofilm formation

281 A C. jejuni transposon (Tn) mutant library was screened for n

282 To address this, we generated a C. jejuni transposon mutant library that is amenable to ins

283 C. jejuni typically colonizes the gut, but a hypervirulent

284 croaerophilic mucosal pathogen Campylobacter jejuni under oxygen-limited conditions was stimulated by

285 aerophilic food-borne pathogen Campylobacter jejuni uses complex cytochrome-rich respiratory chains f

286 gh-level resistance to chloramphenicol in C. jejuni, using integrated genomic and proteomic analyses.

287 se of enterobactin hydrolysis products by C. jejuni, Vibrio cholerae, and other bacteria with homolog

288 gene with the highest p-distance value in C. jejuni was annotated in the reference genome as a putati

289 Altogether, C. jejuni was isolated from 7 of 15 (46.7%) bovine fecal, 1

290 bacterial permease, ArsP, from Campylobacter jejuni, was recently shown to confer resistance to roxar

291 gic development of protective immunity to C. jejuni, we assessed the ability of an initial infection

292 ecombination with it, while in Campylobacter jejuni, we find a minority population we predict will co

293 lable mouse monoclonal antibodies against C. jejuni were investigated to construct direct, sandwich a

294 in dairy manure, while risk estimates for C. jejuni were not sensitive to any single variable.

295 ted with 1 mM thymol, Campylobacter coli and jejuni were reduced during pure or co-culture with a bet

296 parameters of PglC, a PGT from Campylobacter jejuni, were quickly established using this assay.

297 PCR-derived DNA can efficiently transform C. jejuni when only a subset of the CtsM sites are methylat

298 recombination is sufficient to transform C. jejuni, whereas otherwise identical unmethylated DNA is

299 lasmic binding protein CeuE of Campylobacter jejuni, which was previously thought to bind the Fe(III)

300 ds within this Tn library, and in various C. jejuni wild type strains, were compared and correlated t