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1 ocnemius and blended with the posterolateral joint capsule.
2 covering the distal anterior part of the hip joint capsule.
3 four of five perforations and delineated the joint capsule.
4 uction of MMP-9 in the resident cells of the joint capsule.
5 e lower-extremity arteries and hand and foot joint capsules: all five siblings in one family, three s
6 yperplasia, and increased cellularity in the joint capsule and extracapsular ligaments.
7 DAMTS-4 and ADAMTS-5 activity is detected in joint capsule and synovium in addition to cartilage, and
8 mation at sites where ligaments, tendons, or joint capsules are attached to bone).
9 sues, including bone, muscle, ligaments, and joint capsule, as well as cartilage, are increasingly re
10  synovial fibroplasia, and distension of the joint capsule into the surrounding tissue.
11 containing nociceptors include primarily the joint capsule, ligaments, synovium, bone, and in the kne
12 ytes and synoviocytes, the resident cells of joint capsule, markedly increase transcription of MMP-9
13 epiction of ligaments with distention of the joint capsule, may be the procedure of choice, barring o
14              The physiological properties of joint capsule mechanical nociceptors of monoarthritic ch
15          These changes in sensitivity of the joint capsule mechanical nociceptors provides peripheral
16 ction process especially in the cells of the joint capsule remains elusive.
17 cartilage and adjacent structures (meniscus, joint capsule, synovial fluid, muscle) was calculated in
18  at the insertions of ligaments, tendons, or joint capsules to bone, which is termed enthesitis, is a
19 X(-/-)) mice received a puncture of the knee joint capsule with a needle to induce hemarthrosis; huma
20 rtilage, meniscus, subchondral bone, and the joint capsule with synovium.

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