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1 atures, and treatment options for prosthetic joint infection.
2 e positivity for the diagnosis of prosthetic joint infection.
3 n aseptic prosthesis loosening and low-grade joint infection.
4 rimination of aseptic loosening vs low-grade joint infection.
5 rimination of aseptic loosening vs low-grade joint infection.
6 the formation of bacterial aggregates during joint infection.
7 more virulent in a nonhuman primate model of joint infection.
8 specificity for the diagnosis of prosthetic joint infection.
9 blished case of Coxiella burnetii prosthetic joint infection.
10 ignificant impact on incidence of prosthetic joint infection.
11 icant predictors of postoperative prosthetic joint infection.
12 ision arthroplasty and a previous prosthetic joint infection.
13 is an independent risk factor for prosthetic joint infection.
14 s had aseptic failure, and 79 had prosthetic-joint infection.
15 ve the microbiologic diagnosis of prosthetic-joint infection.
16 ined in 108 staphylococci causing prosthetic joint infection.
17 photopenia in the setting of acute bone and joint infection.
18 ward a potential novel approach for treating joint infections.
19 es and promising candidates for treatment of joint infections.
20 t Listeria monocytogenes-associated bone and joint infections.
21 sly reported cases of Actinomyces prosthetic joint infections.
22 ritis and an increasing number of prosthetic joint infections.
23 or the microbiologic diagnosis of prosthetic-joint infection among patients undergoing hip or knee re
24 lococcal isolates associated with prosthetic joint infection and 23 coagulase-negative staphylococci
25 dimentation rate in the diagnosis of bone or joint infection and in monitoring a patient's clinical r
26 ight into the pathogenesis of staphylococcal joint infection and the mechanisms underlying resistance
27 valuate potential risk factors of prosthetic joint infection and to clarify if RA is an independent p
28 detection system, for diagnosing prosthetic joint infection and to compare it with combined (111)In-
29 rgical debridement is typically required for joint infections and chronic osteomyelitis, whereas acut
30 luding native valve endocarditis, prosthetic joint infection, and intravascular catheter-related infe
31 n aseptic prosthesis loosening and low-grade joint infection, and which biomarker combinations are mo
33 ate-onset chronic (low-grade) periprosthetic joint infections are often accompanied by unspecific sym
35 two-step model of staphylococcal prosthetic joint infection: As we previously reported, interaction
36 3) of arthroplasty-associated non-prosthetic joint infection-associated coagulase-negative staphyloco
37 ither arthroplasty-associated non-prosthetic joint infection-associated isolates (e.g., Staphylococcu
38 mplications for the management of prosthetic joint infections, because treatment strategies depend on
39 or the microbiologic diagnosis of prosthetic-joint infection, but this method is neither sensitive no
40 approach, we show that individual as well as joint infection by RSV and PIV can be specifically preve
46 tant Staphylococcus aureus, a major cause of joint infections, forms exceptionally strong biofilmlike
47 s identified an increased risk of prosthetic joint infections (HR 4.08, 95% CI 1.35-12.33) in patient
48 However, the RecA mutant was attenuated for joint infection in competitive-infection assays with the
50 97 included two large, multi-year surveys of joint infection in patients from defined European health
55 coagulase-negative staphylococcal prosthetic joint infection isolates were icaA positive, and 30% (7
56 diabetic pedal osteomyelitis and prosthetic joint infection, it is not useful for spondylodiskitis.
61 95% CI] 1.02-8.75) and a previous prosthetic joint infection of the replaced joint (HR 5.49, 95% CI 1
62 te infections in general, and periprosthetic joint infections particularly, has prompted implementati
63 amples were classified as showing prosthetic joint infection (PJI) and aseptic failure (AF), respecti
64 f these, 152 and 279 subjects had prosthetic joint infection (PJI) and aseptic failure, respectively.
67 onstrated improved sensitivity of prosthetic joint infection (PJI) diagnosis using an automated blood
68 onstrated improved sensitivity of prosthetic joint infection (PJI) diagnosis using an automated blood
70 R has been previously studied for prosthetic joint infection (PJI) diagnosis; however, few studies ha
73 Accurate and rapid diagnosis of prosthetic joint infection (PJI) is vital for rational and effectiv
74 g the prognosis of staphylococcal prosthetic joint infection (PJI) managed with debridement, antibiot
76 species are occasional causes of prosthetic joint infection (PJI), but few data are available on the
77 des for bone infection, including prosthetic joint infection (PJI), often in combination with rifampi
83 ureus is a leading cause of human prosthetic joint infections (PJIs) typified by biofilm formation.
86 additional cases of Campylobacter prosthetic joint infection reported in the literature are reviewed.
87 anisms involved in bone loss during bone and joint infection, suggesting that osteoclasts could be a
88 nmicrobiologic criteria to define prosthetic-joint infection, the sensitivities of periprosthetic-tis
89 ation for the reported extreme resistance of joint infection to antibiotic treatment, lend support to
90 l Tract, Intraabdominal Infections, Bone and Joint Infections, Urinary Tract Infections, Genital Infe
94 uid cultures for the diagnosis of prosthetic joint infection were 54% and 75%, whereas the specificit
96 lacement are at increased risk of prosthetic joint infection, which is further increased in the setti
97 ccus aureus is a leading cause of prosthetic joint infections, which, as we recently showed, proceed
99 ia retains a nearly unique susceptibility to joint infection with mycoplasmas, which can cause consid
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