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1 depression, smoking, alcohol use, and other joint pain).
2 s can be altered significantly by changes in joint pain.
3 in elasticity, tissue fragility, and chronic joint pain.
4 p emphasizing nonpharmacologic management of joint pain.
5 erized by an acute febrile phase and chronic joint pain.
6 gue without exacerbating disease activity or joint pain.
7 tes robust oral activity in rodent models of joint pain.
8 made between subjects with and those without joint pain.
9 al and central sensitization contributing to joint pain.
10 ritis are accompanied by significant chronic joint pain.
11 cules that mediate osteoarthritis-associated joint pain.
12 ature about obesity, knee osteoarthritis and joint pain.
13 f articular cartilage accompanied by chronic joint pain.
14 0.74-0.81) for the 267 participants with no joint pain.
15 P-13 inhibitors on cartilage degradation and joint pain.
16 subjectively better control of the rash and joint pain.
17 t pathology and attenuation of the attendant joint pain.
18 with objective measures of skin clearance or joint pain.
19 enes for the management of temporomandibular joint pain.
20 emoved from the study because of intolerable joint pain.
21 the 300 participants with 2 or more sites of joint pain, 0.90 (95% CI, 0.87-0.92) for the 181 partici
23 39% (23-57), and 86% (56-93), respectively; joint pain 14% (11-18), 42% (26-60), and not available,
24 %), dermatitis (25.3%), oral ulcers (24.2%), joint pain (23.0%), pleural effusion (16.5%) and increas
25 e seronegative population; P<.001), bone and joint pain (27% vs. 9%; P<.001), headache (27% vs. 12.3%
26 41 (5%) had fatigue, 41 (5%) had muscle and joint pains, 37 (5%) had nausea, 36 (4%) had vomiting, 3
27 [corrected] [13%] vs 85 [corrected] [12%]), joint pain (41 [corrected] [6%] vs 38 [5%]), infection (
29 llows: pruritus (95 %), skin burning (81 %), joint pain (69%), arthritis (51%), and psoriatic arthrit
30 loss of appetite (87.0%), headache (77.9%), joint pain (73.7%), vomiting (71.2%), and diarrhea (70.6
31 ycycline did not reduce the mean severity of joint pain, although pain scores in both treatment group
32 survey, 94 (47%) reported having AI-related joint pain and 88 (44%) reported AI-related joint stiffn
34 tanezumab was associated with a reduction in joint pain and improvement in function, with mild and mo
37 or acute neuroborreliosis more often now had joint pain and sleep difficulty and lower scores on the
38 ikely than other patients to have AI-related joint pain and stiffness (odds ratio [OR] = 4.08, 95% CI
39 eated with TA had significant improvement of joint pain and stiffness, which was not seen with SA.
41 on adverse events were a short-term flare in joint pain and swelling following treatment, a side effe
44 nal study to describe the characteristics of joint pain and to examine the relationship between QST m
45 ivity measures (daily diary reports of rash, joint pain and/or swelling, and fevers), health quality
46 tective effects and can potentially modulate joint pain, and are, therefore, uniquely suited as poten
47 presentations such as severe back and small joint pain, and debilitating arthritis associated with c
48 ue, nausea, liver pain, anorexia, muscle and joint pain, and general health remained significantly be
55 lationship between occupational hand use and joint pain; and the extent of occupational hand use amon
57 tion, it is essential that the management of joint pain be considered in light of the impact of multi
58 s the duration and severity of abdominal and joint pain, but corticosteroids do not prevent the devel
59 s of education on walking knee pain, overall joint pain (by HAQ), and general health status (by QWB)
62 ), osteoarthritis (7.1 million), nonspecific joint pain/effusion (7.0 million), and rheumatoid arthri
68 orted higher rates of physical symptoms (eg, joint pains, headaches, and hot flashes) than healthy wo
70 administration of GW3965 potently alleviated joint pain in a rat meniscal tear model of osteoarthriti
71 ) had more joint pain (odds ratio for having joint pain in any joint, 2.1 [CI, 1.2 to 3.5]; P = 0.007
75 described and discussed their experience of joint pain in the context of standard self-assessment qu
76 r three months before admission, followed by joint pains in her knees, elbows and several proximal in
80 Similar patterns were observed for worst joint pain, joint stiffness, pain interference, and func
81 ded injection-site AEs from days 1 to 5, and joint pain, joint swelling, vesicular lesions (blisters)
82 interest of Latinos in various arthritis and joint pain management programs could prove to be an impo
84 erized by gastrointestinal symptoms, chills, joint pain, myalgia, thrombocytopenia, leukocytopenia, a
85 with American Indians who experience chronic joint pain (n = 56), to elicit descriptions and self-rep
86 ion to study evaluation, 6.0 years) had more joint pain (odds ratio for having joint pain in any join
88 mined by a rheumatologist because of chronic joint pain or evidence of small-vessel disease (0.7%).
89 nt or physician assessment), joint swelling, joint pain or tenderness, erythrocyte sedimentation rate
90 (OR 1.27; 95% CI 0.08, 19.63) or prevent new joint pains (OR 0.72; 95% CI 0.11, 4.68) in RA participa
91 n EM was present, during initial episodes of joint pain, or during the maximal period of arthritis.
92 ere in the body influences the experience of joint pain, pain is inextricable from function, and adap
95 at emphasized nonpharmacologic management of joint pain, preservation of function by problem-solving,
98 isted after adjustment for CVD risk factors, joint pain, rheumatoid factor positivity, and inflammato
99 arly-stage breast cancer and who had average joint pain score of >/= 4 out of 10 that developed or wo
105 n routine clinical settings and across other joint pain sites, our findings suggest that focal charac
106 reast cancer receiving an AI who had a worst joint pain/stiffness score >/= 5 of 10 using the Brief P
108 n, elbow, knee and metacarpophalangeal (MCP) joint pain, swelling, and/or deformity, and radiographic
109 codes 715, 716, or 719, and if they reported joint pain, swelling, or stiffness during the previous 1
110 typically induced by cold (rash, fever, and joint pain/swelling) improved within days of rilonacept
111 rns and calluses, fungal signs, edema, ankle joint pain, tenderness to palpation, and sensory loss.
113 efinition of treatment response was based on joint pain/tenderness and swelling scores and physician
114 n in patients with axial manifestations, and joint pain/tenderness scores and joint swelling scores i
115 ional hand use were more likely to have hand joint pain than those with moderate hand use (66% versus
116 g disease in humans characterized by intense joint pain that can persist for weeks, months, or even y
117 fected with CHIKV suffer from incapacitating joint pain that severely affects their daily functioning
119 ed to calculate 3-dimensional moments at the joints; pain, using a separate visual analog scale for e
124 s in fatigue were correlated with decreasing joint pain, whereas improvements in symptoms of depressi
125 phavirus, causes febrile disease, muscle and joint pain, which can become chronic in some individuals
127 tal of 66.2% of the subjects reported recent joint pain, with a median average pain severity of 5 of
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