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   1 ative SDK channels, and nitrergic inhibitory junction potentials.                                    
     2 eased the amplitude of purinergic excitatory junction potentials.                                    
     3 cation of longitudinal stretch inhibited all junction potentials.                                    
     4 he amplitude of slow waves and of inhibitory junction potentials.                                    
     5 microM) affected the amplitude of excitatory junction potentials (2 +/- 5 % and -3 +/- 10 %) or NCTs 
  
     7 e, and cyclopiazonic acid, reduce inhibitory junction potentials and responses to sodium nitroprussid
  
  
    10 junction, and the current-voltage curves and junction potentials are strongly and self-consistently m
    11 servations cannot be explained by the liquid junction potential between two mutually miscible electro
    12 rfacial potential with an additive diffusion/junction potential due to the increase in ionization fro
    13 represents a discreet cholinergic excitatory junction potential (EJP) that involves the synaptic acti
  
  
    16  microm), an ongoing discharge of excitatory junction potentials (EJPs) and inhibitory junction poten
    17 ns evoked an ongoing discharge of excitatory junction potentials (EJPs) at the oral recording site (r
    18 T potently facilitated B38-evoked excitatory junction potentials (EJPs) but had only a small effect o
    19 CgTX GVIA) irreversibly abolished excitatory junction potentials (EJPs) evoked by trains of < or = fi
    20 ormed intracellular recordings of excitatory junction potentials (EJPs) in the muscle fibers to deter
  
    22 ding electrodes elicited compound excitatory junction potentials (EJPs) synchronously in both muscles
  
    24  time-locked with the onset of an inhibitory junction potential (IJP) at an anal recording electrode,
  
    26 ls showed an ongoing discharge of inhibitory junction potentials (IJPs) anally, or excitatory junctio
  
    28 bitory nerve stimulation elicited inhibitory junction potentials (IJPs) and relaxations in wild-type 
    29 ssion resulting in a reduction in inhibitory junction potentials (IJPs) in colonic circular muscle.  
  
  
  
    33 in PDGFRalpha(+) cells by EFS and inhibitory junction potentials (IJPs) recorded with intracellular m
  
    35 ry junction potentials (EJPs) and inhibitory junction potentials (IJPs) were recorded simultaneously 
    36 sed in relation to the idea that spontaneous junction potentials in colonic CM are not monoquantal ev
    37 y of membrane potential following inhibitory junction potentials in gastrointestinal smooth muscle or
  
    39 al field stimulation yielded fast excitatory junction potentials in the smooth muscle that were block
    40 ngs from CM cells revealed either no ongoing junction potentials, or alternatively, small potentials 
    41 The amplitude of NO-mediated slow inhibitory junction potentials (S-IJPs) evoked by electric field st
  
    43 y junction potentials (sIJPs) and excitatory junction potentials (sEJPs) were recorded from all anima
  
  
    46 ime with those in CM, spontaneous inhibitory junction potentials (sIJPs) were found to occur synchron
    47 region of smooth muscle at which spontaneous junction potentials (sJPs) were coordinated in both spac
  
    49 nt for salinity-induced variations in liquid junction potentials that, if not taken into account, wou
    50  light resulted in excitatory and inhibitory junction potentials, the electrical events underlying co
  
    52 Slow wave amplitude and nitrergic inhibitory junction potentials were reduced while solid emptying wa
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