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1 ciated with uveitis also are idiopathic (eg, juvenile idiopathic arthritis).
2 tones risk; and between immunoglobulin A and juvenile idiopathic arthritis).
3 been made in the measurement of outcomes in juvenile idiopathic arthritis.
4 t rheumatoid arthritis might have utility in juvenile idiopathic arthritis.
5 idity and mortality seen with systemic onset juvenile idiopathic arthritis.
6 redominantly in children with systemic onset juvenile idiopathic arthritis.
7 ry outcome measures in therapeutic trials in juvenile idiopathic arthritis.
8 ic arthritis (SoJIA) represents up to 20% of juvenile idiopathic arthritis.
9 diopathic uveitis or uveitis associated with juvenile idiopathic arthritis.
10 ophage activation syndrome in systemic onset juvenile idiopathic arthritis.
11 isk of lower respiratory tract infection and juvenile idiopathic arthritis.
12 omandibular joint arthritis in children with juvenile idiopathic arthritis.
13 11 years, range 4-16), of whom 37 (74%) had juvenile idiopathic arthritis.
14 edict temporomandibular joint involvement in juvenile idiopathic arthritis.
15 f S100A12 correlate with disease activity in juvenile idiopathic arthritis.
16 2001on the matter of uveitis associated with juvenile idiopathic arthritis.
17 as been used to treat children with systemic juvenile idiopathic arthritis.
18 he interleukin-1 signature in systemic onset juvenile idiopathic arthritis.
19 compromise the resolution of inflammation in juvenile idiopathic arthritis.
20 tionally is classified under the umbrella of juvenile idiopathic arthritis.
21 anti-citrullinated protein antibody-positive juvenile idiopathic arthritis.
22 anti-citrullinated protein antibody-positive juvenile idiopathic arthritis.
23 ocular morbidity of uveitis associated with juvenile idiopathic arthritis, aggressive therapies can
24 ackground rate of malignancy associated with juvenile idiopathic arthritis, although the impact of me
25 losing spondylitis, psoriatic arthritis, and juvenile idiopathic arthritis among the inflammatory joi
26 ed with biologic agents for the treatment of juvenile idiopathic arthritis and childhood-onset system
28 recognized in children and young adults with juvenile idiopathic arthritis and is multifactorial in o
29 rsity before and after HSCT in patients with juvenile idiopathic arthritis and juvenile dermatomyosit
30 aired in a large proportion of children with juvenile idiopathic arthritis and other rheumatic diseas
31 de has been utilized to treat systemic onset juvenile idiopathic arthritis and related autoinflammato
32 tive diseases, significantly most similar to juvenile idiopathic arthritis and significantly least si
34 itions, and preliminary quality measures for juvenile idiopathic arthritis, and cites examples of qua
35 hildhood-onset systemic lupus erythematosus, juvenile idiopathic arthritis, and juvenile dermatomyosi
36 hildhood-onset systemic lupus erythematosus, juvenile idiopathic arthritis, and juvenile dermatomyosi
38 ty of life in large cohorts of children with juvenile idiopathic arthritis are important in guiding i
39 tic reports of the use of bisphosphonates in juvenile idiopathic arthritis are welcome additions to t
41 ase pathogenesis of lupus and systemic onset juvenile idiopathic arthritis as well as related conditi
42 ctors, and course of ocular hypotony (OH) in juvenile idiopathic arthritis-associated uveitis (JIAU).
43 ses were birdshot chorioretinopathy (n = 3), juvenile idiopathic arthritis-associated uveitis (n = 3)
44 -Hispanic African-American children with non-juvenile idiopathic arthritis-associated uveitis may hav
46 setting of certain forms of uveitis, such as juvenile idiopathic arthritis-associated uveitis, remain
48 the incidence of malignancy associated with juvenile idiopathic arthritis, both with and without tre
49 related protein-14 (S100A9) in children with juvenile idiopathic arthritis can indicate clinically oc
52 lity of new medications for the treatment of juvenile idiopathic arthritis has made the accurate asse
53 ith rheumatoid factor positive polyarticular juvenile idiopathic arthritis, have the greatest deficit
55 with seronegative spondyloarthropathies and juvenile idiopathic arthritis; however, etanercept has f
57 on electrode, aiming at the diagnosis of the juvenile idiopathic arthritis in real serum samples.
60 tivity in the development of osteoporosis in juvenile idiopathic arthritis is the focus of several st
61 ), fulfillment of criteria for >1 subtype of juvenile idiopathic arthritis (JIA) (5%), and HLA-B27 in
62 about exercise therapy in the management of juvenile idiopathic arthritis (JIA) along with activity
63 dministered to children and adolescents with juvenile idiopathic arthritis (JIA) and pediatric inflam
64 ination on disease activity in patients with juvenile idiopathic arthritis (JIA) are matters of conce
68 olyarticular rheumatoid factor (RF)-negative juvenile idiopathic arthritis (JIA) due to the presence
69 loci in oligoarthritis in UK Caucasians with juvenile idiopathic arthritis (JIA) has not been describ
72 ciations for Rheumatology criteria parse out juvenile idiopathic arthritis (JIA) into seven groups, w
78 predict response to therapy in polyarticular juvenile idiopathic arthritis (JIA) is an important issu
88 concentrations in a cohort of patients with juvenile idiopathic arthritis (JIA) to determine the pre
89 aluations of the well-being of children with juvenile idiopathic arthritis (JIA) typically rely on pa
90 amily in which 9 members were diagnosed with juvenile idiopathic arthritis (JIA) was ascertained.
91 n this work, a novel biosensor for detecting juvenile idiopathic arthritis (JIA) was developed based
92 y reported to be a characteristic feature of juvenile idiopathic arthritis (JIA), but the relevance o
93 chip array to analyze 2,816 individuals with juvenile idiopathic arthritis (JIA), comprising the most
94 in patients with the most common subtypes of juvenile idiopathic arthritis (JIA), IgM rheumatoid fact
95 diseases in the UK population, including RA, juvenile idiopathic arthritis (JIA), psoriasis, psoriati
96 ultifactorial diseases, including autoimmune juvenile idiopathic arthritis (JIA), result from a compl
98 S on children with the polyarticular form of juvenile idiopathic arthritis (JIA), using 2 independent
99 association study of Caucasian patients with juvenile idiopathic arthritis (JIA), we have previously
100 t evidence from the literature pertaining to juvenile idiopathic arthritis (JIA)-associated uveitis.
122 associated with increased risk for systemic juvenile idiopathic arthritis, leprosy and Crohn's disea
123 in weight-bearing cartilage in patients with juvenile idiopathic arthritis may help with early detect
124 approved dosage for rheumatoid arthritis and juvenile idiopathic arthritis, may be an effective treat
125 tients with uveitic glaucoma associated with juvenile idiopathic arthritis (n = 20), idiopathic uveit
126 NK cells isolated from the synovial fluid of juvenile idiopathic arthritis patients failed to inhibit
130 (SF) from patients with osteoarthritis (OA), juvenile idiopathic arthritis, psoriatic arthritis (PsA)
132 re was achieved in the eyes of patients with juvenile idiopathic arthritis-related uveitis glaucoma w
134 e far more costly than that of adult SLE and juvenile idiopathic arthritis reported in the literature
135 eitis in children are highlighted, including juvenile idiopathic arthritis, sarcoidosis and Behcet's
136 ned from the synovial fluid of patients with juvenile idiopathic arthritis show marked downregulation
137 rs most frequently in patients with systemic juvenile idiopathic arthritis (SJIA) and systemic lupus
140 escent from the United Kingdom with systemic juvenile idiopathic arthritis (sJIA), with the GG genoty
146 pivotal trial of infliximab in polyarticular juvenile idiopathic arthritis suggested efficacy, but th
147 osed rheumatic diseases in children, such as juvenile idiopathic arthritis, systemic lupus erythemato
150 more frequent manifestation in patients with juvenile idiopathic arthritis than previously believed,
152 charged with developing quality measures for juvenile idiopathic arthritis; thus, there is a commitme
153 iments, the addition of NGF to LPS-activated juvenile idiopathic arthritis to both mononuclear cells
154 ortant; no child should be labeled as having juvenile idiopathic arthritis unless there is a clear hi
155 ciated at the inflamed site in patients with juvenile idiopathic arthritis, which led us to question
157 ic illness and rheumatic conditions, such as juvenile idiopathic arthritis, who demonstrated no socia
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