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1 ectively, among nine patients with localized juvenile periodontitis.
2 contribute to the pathogenesis of localized juvenile periodontitis.
3 the FPR have been associated with localized juvenile periodontitis.
4 -onset periodontitis, 11 with post-localized juvenile periodontitis, 41 with adult periodontitis, and
5 ts were prompted by our studies of localized juvenile periodontitis, a disease associated with high I
6 ed to be the causative organism of localized juvenile periodontitis, a disease in which there is rapi
7 omitans, is the putative agent for localized juvenile periodontitis, a particularly destructive form
8 mcomitans, the etiologic agent for localized juvenile periodontitis and certain other human infection
10 negative bacterium responsible for localized juvenile periodontitis and other infections such as endo
11 leukocyte syndrome associated with localized juvenile periodontitis, cyclic neutropenia, and congenit
12 thogen responsible for an aggressive form of juvenile periodontitis, form tenaciously adherent biofil
13 Those with adult periodontitis, localized juvenile periodontitis, generalized early-onset periodon
14 ed juvenile periodontitis (LJP), generalized juvenile periodontitis (GJP), and incidental attachment
15 ed juvenile periodontitis (LJP), generalized juvenile periodontitis (GJP), incidental attachment loss
16 ssified into localized (LJP) and generalized juvenile periodontitis (GJP), incidental attachment loss
18 erations in neutrophil function in localized juvenile periodontitis is presented as a model system fo
19 by comparing proximal caries prevalence in a juvenile periodontitis (JP) group to a matched non-perio
20 adult periodontitis, their relationship with juvenile periodontitis (JP) has not been established.
21 y-nine subjects had localized or generalized juvenile periodontitis (JP), 218 had incidental attachme
23 hat the monocytes of subjects with localized juvenile periodontitis (LJP) are functionally distinct f
24 Neutrophils from patients with localized juvenile periodontitis (LJP) exhibit decreased binding a
31 trains isolated from patients with localized juvenile periodontitis (LJP) was investigated by using p
32 rom 146 subjects including 71 with localized juvenile periodontitis (LJP), 4 with early-onset periodo
34 e individuals were classified into localized juvenile periodontitis (LJP), generalized juvenile perio
35 ere identified and classified into localized juvenile periodontitis (LJP), generalized juvenile perio
36 ve forms of periodontitis, such as localized juvenile periodontitis (LJP), respond to Aa challenge wi
41 emcomitans, the etiologic agent of localized juvenile periodontitis, produces a potent leukotoxin tha
42 ly progressive periodontitis and generalized juvenile periodontitis), were analyzed both separately a
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