ライフサイエンスコーパス: juvenile rat

1 he microcircuitry of somatosensory cortex of juvenile rat.

2 in layers 2-3 of visual cortical slices from juvenile rats.

3 a about these cells and their development in juvenile rats.

4 neuron function in the prefrontal cortex of juvenile rats.

5 afferent neurotransmission in NTS neurons in juvenile rats.

6 ney, and liver during growth deceleration in juvenile rats.

7 ess toward a predatory odor were assessed in juvenile rats.

8 t contribute to pacemaking in DCN neurons of juvenile rats.

9 of predatory odors on play were assessed in juvenile rats.

10 emia impaired neurobehavioral performance in juvenile rats.

11 midal cells in acute hippocampal slices from juvenile rats.

12 rical synapses mediate rhythmic synchrony in juvenile rats.

13 ect the immaturity of these brain regions in juvenile rats.

14 ay index an appetitive motivation to play in juvenile rats.

15 ajor role in the control of play behavior in juvenile rats.

16 By contrast, in juvenile rats, 100 units of IL-1 beta injected into the

17 n the hippocampus (CA1) and dentate gyrus of juvenile rats acutely and chronically exposed to antidep

18 dissected out, with their nerve supply, from juvenile rats aged 8-15 days (a period corresponding to

19 the dorsal-ventral axis previously shown in juvenile rats also appears in control adult mice, indica

20 -positive hair cells in the utricle peaks in juvenile rats and declines in early adulthood.

21 for LTP induction in the adult as opposed to juvenile rats and we hypothesize that the basis for this

22 ne receptors on ventral horn neurones in the juvenile rat are heteromers and have fast gating, simila

23 t layer 4 (L4) to L2/3 (L4-L2/3) synapses in juvenile rat barrel cortex.

24 re, we used a fear conditioning procedure in juvenile rats before maturation of the neural systems su

25 esponsible for driving pacemaker activity in juvenile rat brain slices were probed by replacing nativ

26 ake, and spectrin degradation in slices from juvenile rats but not adult rats.

27 revented the development of hydrocephalus in juvenile rats by blocking transforming growth factor-bet

28 Juvenile rats can exhibit maternal behavior after being

29 performed on DCN neurones in acute slices of juvenile rat cerebellum.

30 projections from the BSTp of male and female juvenile rats combined with immunohistochemical labeling

31 olved in the maternal behavior of 27-day-old juvenile rats compared with 60-day-old adults.

32 ntricle significantly reduced body weight in juvenile rats, compared with saline-injected controls.

33 han during a control condition; captive-born juvenile rats did not behave differently during snake an

34 e authors provide initial documentation that juvenile rats emit short, high-frequency ultrasonic voca

35 Maternally lead (Pb)-exposed, juvenile rats exhibit significant deficits in spatial re

36 Purified elastin implanted subdermally in juvenile rats exhibited progressive calcification in a t

37 0.04, respectively; P=.030), indicating that juvenile rats experienced more extensive remodeling than

38 ed reorganization in cuneate nucleus (CN) in juvenile rat following forelimb amputation (n=34) and in

39 natural responsiveness to odors, we exposed juvenile rats for 1 h daily to a dynamic series of inter

40 izures (ictal-like activity) was recorded in juvenile rat hippocampal-entorhinal cortex slices and hi

41 cerebral blood flow (CBF) in three groups of juvenile rats (hyperglycemic, ketotic, and normal contro

42 esponses to serotonin (5-HT) of neonatal and juvenile rat hypoglossal motoneurons (HMs) by using intr

43 sceptibility' to an inflammatory stimulus in juvenile rats, if paralleled in humans, may be a major f

44 ation with fresh controls in a subcutaneous, juvenile rat implant model of calcification.

45 ro, and in the more intact nervous system of juvenile rats in arterially perfused brainstem-spinal co

46 D1 dopamine receptor expression compared to juvenile rats in both the OFC and piriform cortex.

47 To address these questions, we reared juvenile rats in complex multitone environments that dif

48 ) and central vagus nerves from neonatal and juvenile rats in situ.

49 3 mg/kg) also produced depressive effects in juvenile rats, in a linear dose-dependent manner.

50 Juvenile rats infused with picrotoxin into the prelimbic

51 cts and pinning to quantify play behavior in juvenile rats, it was found that the D2 agonist, quinpir

52 age clamp in molecular layer interneurons of juvenile rats or mice reveals an exponential component w

53 ask, we sought to extend alcohol findings to juvenile rats (PD30).

54 In juvenile rats (PN26), the mPFC receives information on p

55 In dentate granule cells from juvenile rats polysynaptic feedback IPSCs, but not monos

56 sing hyperpolarization-activated currents in juvenile rat prefrontal cortex, while exerting excitator

57 oup (VRG) in the in situ arterially perfused juvenile rat preparation.

58 s (n = 31) in brainstem slices prepared from juvenile rat pups.

59 eurons or cultured slices to use prenatal or juvenile rats seriously limit the advantages of these sy

60 n in the GABAergic neurons in slices through juvenile rat SNr.

61 of pyramidal cells from layers II-VI of the juvenile rat somatosensory cortex suggest common design

62 mechanism of electrically evoked LTP in the juvenile rat sSC.

63 The effects of CCK were more evident in juvenile rats, suggesting that they are developmentally

64 The results suggest that in juvenile rats, the role of the MPOA neurons in MB is onl

65 regulation in the young organism, we exposed juvenile rats to a threatening or nonthreatening stimulu

66 causes a 3-day advancement in the ability of juvenile rats to use relational cues in a water maze tas

67 stratum radiatum of hippocampal slices from juvenile rats, using extracellular, concentric pH- and C

68 y perfused preparations of both neonatal and juvenile rats, we provide the first description of the l

69 Juvenile rats were either treated for 1 week with magnes

70 onflicts with observations we report here in juvenile rats, where by administration of fentanyl, a se

71 RESEARCH DESIGN AND We evaluated 50 juvenile rats with DKA and 21 normal control rats using

72 Treatment of ovariectomized juvenile rats with estradiol induced expression of GluR1

73 Treatment of estrogen-primed ovariectomized juvenile rats with progesterone caused an initial increa

74 Juvenile rats with streptozotocin-induced DKA were treat