1 soactive mediator within the confines of the juxtaglomerular apparatus.

2 s confirmed that BNZ affected TGF within the juxtaglomerular apparatus.

3 tive renin in afferent arterioles and in the juxtaglomerular apparatus.

4 ad fewer or no renin-expressing cells in the juxtaglomerular apparatus.

5  of renal glomeruli producing renin in their juxtaglomerular apparatus and by four times wild-type nu

6 in the renin-secreting granular cells of the juxtaglomerular apparatus and the collecting duct.

7 ecretion is important for the maintenance of juxtaglomerular apparatus architecture.

8 o signal the release of renin from the renal juxtaglomerular apparatus, especially during volume depl

9  and provide evidence for a new role for the juxtaglomerular apparatus in the maintenance of the mesa

10               Macula densa (MD) cells of the juxtaglomerular apparatus (JGA) are salt sensors and gen

11 t 20-28 h, while Thy 1-positive cells in the juxtaglomerular apparatus (JGA) were sequestered from th

12 erized by hypertrophy and hyperplasia of the juxtaglomerular apparatus of the kidneys, aldosteronism

13 duction was similar in the isolated perfused juxtaglomerular apparatus of wild-type (WT) and nitric o

14 buloglomerular feedback in isolated perfused juxtaglomerular apparatus preparations, although minor d

15                      TGF operates within the juxtaglomerular apparatus, sensing changes in tubular fl

16 t generate paracrine chemical signals in the juxtaglomerular apparatus to control vital kidney functi

17                                          The juxtaglomerular apparatus was still able to respond to t

18             Olfr78 is expressed in the renal juxtaglomerular apparatus, where it mediates renin secre