ライフサイエンスコーパス: kPa

1 solid stress is on the order of 1.3 to 13.0 kPa (10-100 mmHg)--high enough to cause compression of f

2 +/-5.5 kPa in the nonfailing to 134.4+/-35.0 kPa in the HF to 201.7+/-36.4kPa in the HF+LVAD groups (

3 A cutoff value of 51.0 kPa at 4 cm proximal to the medial malleolus revealed a

4 had the highest probabilities of LSM >/=8.0 kPa (overall probability: 17.2% [12.5-23.4]; this probab

5 LSM >8.0 kPa but not metabolic syndrome was an independent risk f

6 Patients with baseline LSM >8.0 kPa had higher cumulative probability of hepatic events

7 ed presence) were associated with LSM >/=8.0 kPa in multivariable analyses.

8 adjusted predicted probability of LSM >/=8.0 kPa increased per age decade, with probabilities ranging

9 = 7.3 kPa) and 63 less hypoxemic (Pao2 > 8.0 kPa) patients with COPD (39% vs. 52%; P = 0.27).

10 tion-based study of older adults, LSM >/=8.0 kPa, suggestive of clinically relevant fibrosis, was pre

11 LSM, 169 (5.6%) participants had LSM >/=8.0 kPa.

12 Patients with an increase (>0 kPa/year) in >/= 14 kPa bLSM had the worst prognosis.

13 a/year) in 7-14 kPa bLSM, or decrease (</= 0 kPa/year) in >/= 14 kPa bLSM (P = 0.949 between these tw

14 = 0.012; mean between-sex difference, 0.011 kPa s(-1); 95% CI, 0.003-0.019).

15 lytical determination (from 0.1627 to 0.0128 kPa at 0.5 kPa O2 and from 0.9393 to 0.1532 kPa at 20 kP

16 For girls, sRaw increased by 0.017 kPa s(-1) per year (95% CI, 0.011-0.023).

17 sor with a sensitivity ( approximately 0.018 kPa(-1) ), response time ( approximately 60 ms), and goo

18 ressive stiffness of O(1) kPa (0.49 +/- 0.04 kPa stress at 30% compressive strain) and therefore mimi

19 firm q-DC measurements of Ea = 0.53 +/- 0.04 kPa.

20 Average patient level variance was 0.042 kPa, with a coefficient of variation of 10.7%.

21 sorption, while the presence of CO2 at 0.045 kPa does not influence H2O adsorption.

22 c airway resistance (abeta coefficient, 0.06 kPa/s; 95% CI, 0.00-0.11 kPa/s; P = .05).

23 .47 kPa, 0.53 +/- 0.12 kPa and 7.06 +/- 4.07 kPa respectively.

24 s, leading to high sensitivities of a 0.0835 kPa(-1) change in output voltage.

25 Liver stiffness <12.1 kPa could rule out SPH with 50.0% specificity and >35 kP

26 us grade 1, 90.4 kPa +/- 9.3; grade 5, 120.1 kPa +/- 15.4), and there were weak correlations between

27 (20 +/- 7 kPa), PaCO2 40 +/- 5 torr (5 +/- 1 kPa), and core temperature 33 degrees C +/- 1 degrees C.

28 rrected PaCO2 (4.4 kPa [4.1-4.6 kPa] vs. 5.1 kPa [5.0-5.3 kPa], p = 0.0001), and temperature-uncorrec

29 </=0.30, or IQR/M >0.30 with LSE median <7.1 kPa), and "poorly reliable" (IQR/M >0.30 with LSE median

30 eliable" (IQR/M >0.30 with LSE median >/=7.1 kPa).

31 rain neurons (ECFNs; optimum approximately 1 kPa) and U251 glioma cells (optimum approximately 100 kP

32 s from 1% to 500%, and pressures as low as 1 kPa.

33 impaired in patients with an increase (>/= 1 kPa/year) in 7-14 kPa bLSM, or decrease (</= 0 kPa/year)

34 Pa(-1) is achieved in the pressure region <1 kPa with power generation of tens of muW cm(-2) from a g

35 ection and localized retention of a soft (<1 kPa) hydrogel.

36 having bLSM <7 kPa, SVR, or no increase (<1 kPa/year) in 7-14 kPa bLSM.

37 rix to collagen-1-coated 2D substrates of ~1 kPa results in self-organization of all three germ layer

38 e were 249.7 +/- 58.1 dB/m and 11.9 (6-18.1) kPa.

39 ith an average compressive stiffness of O(1) kPa (0.49 +/- 0.04 kPa stress at 30% compressive strain)

40 indicators is significantly different (0-10 kPa and 1-100 kPa CO2) that enables a broad variety of a

41 of hMSCs cultured on initially stiff (E ~ 10 kPa) and then soft (E ~ 2 kPa) phototunable PEG hydrogel

42 -supporting hydrogel with elastic moduli >10 kPa.

43 de substrates of physiological stiffness (10 kPa), and Matrigel micropatterns were used to generate p

44 significantly different (0-10 kPa and 1-100 kPa CO2) that enables a broad variety of applications.

45 th strains of 20-500% and stresses of 10-100 kPa, falling exactly into the dynamic range of a muscle.

46 de gels of three stiffnesses (1, 30, and 100 kPa) and treated with a range of epidermal growth factor

47 At 278 K and 100 kPa, the predicted selectivities for equimolar CO(2)/CH(

48 U251 glioma cells (optimum approximately 100 kPa).

49 c materials with a Young's modulus below 100 kPa conforming to biological applications, yet those gel

50 oft biological tissue (Young's modulus < 100 kPa), and the capability to undergo extreme deformations

51 creased by the highest stiffness tested (100 kPa).

52 278, 298, and 318 K and pressures up to 100 kPa, and correlated with the Langmuir model.

53 cisely controllable low moduli from 1 to 100 kPa, below the lower limit of traditional elastomers; mo

54 ical Young's modulus range of 0.5 kPa to 100 kPa.

55 ion and (TEM 2-22 degrees C and PRS 980-1025 kPa) for H5N1 infection.

56 2 partial pressures in the range of 0.02-105 kPa, where the ultralow partial pressure range is releva

57 airway resistance (abeta coefficient, -0.11 kPa/s; 95% CI, -0.21 to -0.02; P = .02), decreased bronc

58 a coefficient, 0.06 kPa/s; 95% CI, 0.00-0.11 kPa/s; P = .05).

59 uter boundaries of the vessel wall (from 11 kPa to zero).

60 ls with Young's moduli ranging from 6 to 110 kPa.

61 niform wall thickness models was 410 +/- 111 kPa.

62 those without atopy (mean difference, 0.116 kPa s(-1); 95% CI, 0.065-0.168; P < 0.001).

63 (day 14) of 1.04 +/- 0.47 kPa, 0.53 +/- 0.12 kPa and 7.06 +/- 4.07 kPa respectively.

64 Fibronectin-rich matrices with 3-12 kPa elastic modulus are sufficient to provide PLX4720 to

65 similar to cancerous breast tissue (E = 4-12 kPa) as compared to healthy tissue (E = 0.4-2 kpa).

66 n the final model (77 +/- 7 versus 67 +/- 12 kPa/cm, P=0.22).

67 , lower platelets, and liver stiffness >/=12 kPa at year 5 represent the main risk factors for late H

68 seline and year 5, and liver stiffness >/=12 kPa at year 5 were independently associated with more fr

69 ge of 67 kPa on the volcano surface and ~120 kPa in the spherical magma chamber.

70 le wall thickness increased PWS (481 +/- 126 kPa, P<0.001).

71 s not statistically significant (506 +/- 126 kPa, P=0.07), the addition of intraluminal thrombus to v

72 igh-frequency (2.5 MHz), high-intensity (>13 kPa) pressure transients.

73 anning a vapor pressure range of 0.027 to 13 kPa was generated with modulated peak fwhm (full width a

74 ts with an increase (>/= 1 kPa/year) in 7-14 kPa bLSM, or decrease (</= 0 kPa/year) in >/= 14 kPa bLS

75 a, SVR, or no increase (<1 kPa/year) in 7-14 kPa bLSM.

76 bLSM, or decrease (</= 0 kPa/year) in >/= 14 kPa bLSM (P = 0.949 between these two groups).

77 nts with an increase (>0 kPa/year) in >/= 14 kPa bLSM had the worst prognosis.

78 n using biogas as the only CO2 source at 140 kPa, the CO2 consumption rate corresponded to 2.59 L CO2

79 rmentation was increased from 101.325 to 140 kPa, higher CO2 solubility was achieved, thereby positiv

80 cifically investigated mild TBI range (0-145 kPa) using physiological (heart rate), pathological (lun

81 rats when the BOP is in the range of 85-145 kPa.

82 We determined a BOP of 145 kPa as upper mild TBI threshold (5% PMR).

83 -cortex detachment which increased from 0.15 kPa in stem cells to 0.71 kPa following chondrogenic dif

84 sing lengths of time under vacuum (i.e. 0.15 kPa).

85 or 10 min followed by vacuum treatment (0.15 kPa; i.e. conventional-vacuum roasting).

86 is (stage F3-F4, 46 [32.4%] of 142) was 4.15 kPa (AUROC = 0.954, sensitivity = 0.85, specificity = 0.

87 ices with stiffness measurements of 9 and 15 kPa, assessed by immunofluorescent imaging and quantitat

88 a moderate overpressure ( approximately 150 kPa) and stirring the sample.

89 kPa at 0.5 kPa O2 and from 0.9393 to 0.1532 kPa at 20 kPa O2) in comparison with a conventional phas

90 media and adventitia layers (from 80 to 160 kPa), dropped abruptly at the interface (from 160 to <5

91 rogels with an initial elastic modulus of 17 kPa.

92 as the difference between 5 and 20 Hz, 0.18 kPa.L(-1).s; Asthma Control Questionnaire score, 0.76; a

93 elastic modulus of the zebra finch ML is 18 kPa at 5% strain, which is comparable to elastic moduli

94 ll lines (Ea = 2.1 +/- 0.1 and 0.80 +/- 0.19 kPa for MCF-7 and MDA-MB-231 cells).

95 TE values changed by a mean of -0.2 kPa (P = .097), and declined significantly in subjects w

96 ticity, Et, increases daily for heart to 1-2 kPa by embryonic day 4 (E4), and although this is ~10-fo

97 , P = .0003) but not at 28 Hz (children, 1.2 kPa +/- 0.2; adolescents, 1.3 kPa +/- 0.3; adults, 1.2 k

98 2; adolescents, 1.3 kPa +/- 0.3; adults, 1.2 kPa +/- 0.2; analysis of variance, P = .40).

99 ated stump muscle (13.3+/-1.6 vs. 16.6+/-1.2 kPa).

100 P = .048) or TE values >7.6 kPa (mean, -1.2 kPa; P = .021).

101 hear wave elastography were 9.6 kPa and 10.2 kPa, and for cirrhosis 19.7 kPa and 16.4 kPa.

102 (children, 2.2 kPa +/- 0.3; adolescents, 2.2 kPa +/- 0.2; adults, 2.6 kPa +/- 0.3; analysis of varian

103 cents than in adults at 56 Hz (children, 2.2 kPa +/- 0.3; adolescents, 2.2 kPa +/- 0.2; adults, 2.6 k

104 ctuators that can generate up to 1.7 mN (3.2 kPa) of passive tension force and 300 muN (0.56 kPa) of

105 achieved HBsAg seroclearance (5.5 versus 5.2 kPa, respectively; P = 0.52).

106 sensitivity >/=90% were very close: 6.3/6.2 kPa for >/=F2, 8.3/8.2 kPa for >/=F3, and 10.5/9.5 kPa f

107 s were 5.7 kPa (interquartile range, 4.7-7.2 kPa) and independently associated with HBV DNA load, asp

108 e very close: 6.3/6.2 kPa for >/=F2, 8.3/8.2 kPa for >/=F3, and 10.5/9.5 kPa for F4.

109 radients of 0.5, 1.7, 2.9, 4.5, 6.8, and 8.2 kPa/mm, spanning the in vivo physiological and pathologi

110 ycol) (PEG) hydrogels (Young's modulus E ~ 2 kPa) depended on previous culture time on stiff tissue c

111 ally stiff (E ~ 10 kPa) and then soft (E ~ 2 kPa) phototunable PEG hydrogels resulted in either rever

112 sion with a bulk modulus, kappa = 140 +/- 20 kPa; applying negative pressures leads to volumetric exp

113 5 kPa O2 and from 0.9393 to 0.1532 kPa at 20 kPa O2) in comparison with a conventional phase-modulati

114 st, on stiff substrates (Young's modulus >20 kPa), traction stress plateaus at a limiting value and t

115 and a liver stiffness measurement (LSM) <20 kPa (Baveno VI criteria).

116 On soft ACA gels (Young's modulus <20 kPa), cell-exerted substrate deformation remains constan

117 s over an order of magnitude range of 10-200 kPa, which is in the region of many soft tissues.

118 er, the operating pressure was less than 200 kPa, which eliminated the need for an expensive high-pre

119 6, -2.37) compared with -0.77 (-1.32, -0.22) kPa; P < 0.001].

120 Also we determined 146-220 kPa and 221-290 kPa levels as moderate and severe TBI ba

121 strain, 1760+/-470 microstrain, and 119+/-23 kPa, respectively).

122 lant of our tissue-engineered construct (239 kPa versus normal=193, P=0.1; versus infarct=304 kPa, P=

123 Calibration experiments performed over 0-25 kPa O2 and 10-30 degrees C temperatures ranges reveal li

124 ints were chosen to predict the absence (<25 kPa) or presence (>/=40 kPa) of LREs, thus enabling corr

125 telet count >110 x 10(9) cells/L and LSM <25 kPa.

126 lnuts subjected to pressure treatment at 256 kPa, 138 degrees C, were able to diminish the IgE cross-

127 nd subsequent stiffening (from ca. 3.5 to 28 kPa) increased the cell area and nuclear localization of

128 7 (20-37) kPa before treatment to 18 (14-28) kPa after SVR (P < .05).

129 /- 0.12 in the steatohepatitis group vs 0.29 kPa +/- 0.01 in the control group; P = .02).

130 logical range of stiffness (E = 1.85 to 5.29 kPa), with and without RGD binding sites or collagen fib

131 Also we determined 146-220 kPa and 221-290 kPa levels as moderate and severe TBI based on 35%, and

132 d 70% PMR, respectively, while BOP above 290 kPa is lethal.

133 (children, 1.2 kPa +/- 0.2; adolescents, 1.3 kPa +/- 0.3; adults, 1.2 kPa +/- 0.2; analysis of varian

134 18%) had an abnormal result (range, 7.1-11.3 kPa).

135 ated cutoffs for moderate fibrosis (8.0-12.3 kPa) and severe fibrosis/cirrhosis (>12.3 kPa).

136 .3 kPa) and severe fibrosis/cirrhosis (>12.3 kPa).

137 at of the unaffected shoulder (median, 203.3 kPa; IQR, 144.1-242.7 kPa) in the shoulder-neutral posit

138 At a cutoff value of 22.3 kPa, sensitivity, specificity, positive predictive value

139 se without a bleed (60.4 kPa +/- 5.4 vs 30.3 kPa +/- 14.2, P = .01).

140 slinking) stiffened the hydrogel (41.4+/-4.3 kPa) after injection.

141 (4.4 kPa [4.1-4.6 kPa] vs. 5.1 kPa [5.0-5.3 kPa], p = 0.0001), and temperature-uncorrected PaCO2 (p

142 s 46 +/- 12 years, LSM value was 8.4 +/- 6.3 kPa, and 188 patients (12.8%) had metabolic syndrome.

143 ce was similar in 31 hypoxemic (Pao2 </= 7.3 kPa) and 63 less hypoxemic (Pao2 > 8.0 kPa) patients wit

144 no right-to-left shunt (7.7 vs. 8.6 vs. 9.3 kPa, respectively; P = 0.002).

145 brosis (magnetic resonance elastography >/=3 kPa).

146 Hypoxic incubation (3 kPa) caused (i) stabilization of HIF-2alpha and up-regul

147 have obtained can sustain up to 4.8 x 10(3) kPa pressure, a level comparable to cementitious materia

148 ns by a factor of approximately 360, from 30 kPa to 11 MPa along the first millimeter of the follicle

149 Cell adhesion to a 30-kPa Ecad-Fc PA gel required Cdc42- and formin-dependent

150 e were exposed to blast wave pressure of 300 kPa (43.5 psi) per day for 3 successive days, and euthan

151 re (100 degrees C) and applied pressure (300 kPa), ground TBEU thermoset powder can be remolded to bu

152 We found that 300-kPa blasts yielded no detectable cognitive or motor defi

153 versus normal=193, P=0.1; versus infarct=304 kPa, P=0.01).

154 2.4 years, median hepatic stiffness was 2.35 kPa.

155 93 kPa +/- 2.31 [standard deviation] vs 3.35 kPa +/- 1.44, P < .0001).

156 rule out SPH with 50.0% specificity and >35 kPa rule in SPH with 58.2% sensitivity.

157 acteristic curve for SWE with a cutoff of 35 kPa for differentiating benign from malignant lesions we

158 Use of a 35-kPa threshold at SWE may provide additional information

159 al (p > 0.08) over a large range (47 Pa - 36 kPa).

160 LSM decreased from 27 (20-37) kPa before treatment to 18 (14-28) kPa after SVR (P < .0

161 very narrow range of ECM stiffness (0.1-0.4 kPa), and such ILP formation is Src family kinase depend

162 gas-phase dehydration of 1-propanol (0.075-4 kPa) was studied on zeolite H-MFI (Si/Al = 26, containin

163 ng alternating traction forces, of up to 1.4 kPa, at each flank of the lamellipodium, ColXVII knockdo

164 Liver stiffness <11.4 kPa could rule out CSPH with 55.2% specificity and >21.9

165 sensor unit achieves high sensitivity (14.4 kPa(-1) ), low detection limit (2 Pa), fast response ( a

166 F4 were 7.4 kPa, 8.6 kPa, 9.6 kPa, and 14.4 kPa, respectively.

167 0.2 kPa, and for cirrhosis 19.7 kPa and 16.4 kPa.

168 t CSPH was 88.7% for liver stiffness of 17.4 kPa and 77.7% for spleen stiffness of 47.6 kPa.

169 and the mean post-meal delta increase of 2.4 kPa.

170 p = 0.006), temperature-corrected PaCO2 (4.4 kPa [4.1-4.6 kPa] vs. 5.1 kPa [5.0-5.3 kPa], p = 0.0001)

171 er median liver stiffness values (7.1 vs 4.4 kPa; P < .001); HIV positive/HCV negative and HIV negati

172 had similar liver stiffness values (both 4.4 kPa; P = .94).

173 gher SS than did those without a bleed (60.4 kPa +/- 5.4 vs 30.3 kPa +/- 14.2, P = .01).

174 stages >/=F1, >/=F2, >/=F3, and F4 were 7.4 kPa, 8.6 kPa, 9.6 kPa, and 14.4 kPa, respectively.

175 ransistors with a maximum sensitivity of 8.4 kPa(-1), a fast response time of <10 ms, high stability

176 % CI for death by LSM were as follows: 6-9.4 kPa, 1.7 (0.63-4.79), P = 0.288; 9.5-14.5 kPa, 3.38 (1.2

177 5% CI by LSM category were as follows: 6-9.4 kPa, 1.89 (0.18-20.3), P = 0.599; 9.5-14.5 kPa, 6.59 (0.

178 06 (12.3%) subjects had TE measurements >9.4 kPa.

179 kPa +/- 2.1; annulus fibrosus grade 1, 90.4 kPa +/- 9.3; grade 5, 120.1 kPa +/- 15.4), and there wer

180 ict the absence (<25 kPa) or presence (>/=40 kPa) of LREs, thus enabling correct classification of 82

181 ions, under an oxygen partial pressure of 40 kPa.

182 f oxygen, whilst they were degraded under 40 kPa of oxygen.

183 0, 130, 160, 190, 230, 250, 290, 350 and 420 kPa) and determines the mortality rate as a non-linear f

184 ed a compressive modulus of 499.18 +/- 86.45 kPa.

185 monstrates 24 hour survival of rats in 0-450 kPa (0-800 Pas impulse) range at 10 discrete levels (60,

186 d cells and nuclei (day 14) of 1.04 +/- 0.47 kPa, 0.53 +/- 0.12 kPa and 7.06 +/- 4.07 kPa respectivel

187 mal fouling of the MnO2 coated membrane (0.5 kPa for 70 days), while the uncoated membrane experience

188 ermination (from 0.1627 to 0.0128 kPa at 0.5 kPa O2 and from 0.9393 to 0.1532 kPa at 20 kPa O2) in co

189 e physiological Young's modulus range of 0.5 kPa to 100 kPa.

190 tile stress (from 1.8 +/- 0.7 to 4.7 +/- 0.5 kPa), and the average strain energy (0.4 +/- 0.2 to 2.1

191 ration grade (nucleus pulposus grade 1, 12.5 kPa +/- 1.3; grade 5, 16.5 kPa +/- 2.1; annulus fibrosus

192 .4 kPa, 1.7 (0.63-4.79), P = 0.288; 9.5-14.5 kPa, 3.38 (1.2-9.5), P = 0.021; >/=14.6 kPa, 12.7 (4.9-3

193 4 kPa, 1.89 (0.18-20.3), P = 0.599; 9.5-14.5 kPa, 6.59 (0.73-59.2), P = 0.092; >/=14.6 kPa, 59.5 (8.3

194 sus grade 1, 12.5 kPa +/- 1.3; grade 5, 16.5 kPa +/- 2.1; annulus fibrosus grade 1, 90.4 kPa +/- 9.3;

195 dry/wet scribed tape peel adhesion and 17.5 kPa Taber linear abraser tests.

196 absence and presence of co-fed water (0-2.5 kPa) at 413-443 K.

197 situ hydrogel softening (from ca. 14 to 3.5 kPa) led to a decrease in the cell area and nuclear loca

198 orta progressively stiffened from 74.3+/-5.5 kPa in the nonfailing to 134.4+/-35.0 kPa in the HF to 2

199 ly higher hepatic stiffness (15.6 versus 5.5 kPa, P < 0.0001), higher celiac RI (0.78 versus 0.73, P

200 (children, 5.6 kPa +/- 0.8; adolescents, 6.5 kPa +/- 1.2; adults, 7.8 kPa +/- 1.2; analysis of varian

201 Samples were fried under vacuum (6.5 kPa, Twater-boiling-point=38 degrees C) or atmospheric c

202 h cell pre-stress with culture on stiff (7.5 kPa) polyacrylamide gels (with or without transforming g

203 r >/=F2, 8.3/8.2 kPa for >/=F3, and 10.5/9.5 kPa for F4.

204 d abruptly at the interface (from 160 to <5 kPa), and increased slightly towards the outer boundary

205 drogels mimicking the stiffness of normal (5 kPa) and glaucomatous (75 kPa) HTM.

206 ss values mimicking those found in normal (5 kPa) and glaucomatous meshworks (75 kPa), or tissue cult

207 s display controllable stiffness in the 5-50 kPa range.

208 ge of stiffness from approximately 0.5 to 50 kPa, stemness maintenance did not correlate with initial

209 stiffness at 80 Hz and 48-week feeding, 0.51 kPa +/- 0.12 in the steatohepatitis group vs 0.29 kPa +/

210 air reveal that the presence of H2O at 2.55 kPa enhances CO2 adsorption, while the presence of CO2 a

211 tor that can generate up to 300 microN (0.56 kPa) of active tension force in response to a noninvasiv

212 ) of passive tension force and 300 muN (0.56 kPa) of active tension force in response to external sti

213 ife scratch and sandpaper abrasion under 570 kPa.

214 th blebbistatin or culture on soft gels (0.6 kPa).

215 fness was around 150 Pa and increased to 1-6 kPa in areas near fibrillar collagen deposition in fibro

216 tients with a reduction in LSM to below 13.6 kPa after SVR still had CSPH.

217 14.5 kPa, 3.38 (1.2-9.5), P = 0.021; >/=14.6 kPa, 12.7 (4.9-33.6), P < 0.0001.

218 .5 kPa, 6.59 (0.73-59.2), P = 0.092; >/=14.6 kPa, 59.5 (8.3-427), P < 0.0001.

219 ls (kPa), and cirrhosis as an LSM of >/=14.6 kPa.

220 3; adolescents, 2.2 kPa +/- 0.2; adults, 2.6 kPa +/- 0.3; analysis of variance, P = .009) and 84 Hz (

221 ct SPH was 83.1% for liver stiffness of 20.6 kPa and 77.7 % for spleen stiffness of 50.7 kPa.

222 LS of more than 24.6 kPa with SWE had a sensitivity, specificity, and accurac

223 emperature-corrected PaCO2 (4.4 kPa [4.1-4.6 kPa] vs. 5.1 kPa [5.0-5.3 kPa], p = 0.0001), and tempera

224 4 kPa and 77.7% for spleen stiffness of 47.6 kPa.

225 ntaining physiological PaCO2 values: 4.8-5.6 kPa (36-42 torr).

226 variance, P = .009) and 84 Hz (children, 5.6 kPa +/- 0.8; adolescents, 6.5 kPa +/- 1.2; adults, 7.8 k

227 (mean, -0.8 kPa; P = .048) or TE values >7.6 kPa (mean, -1.2 kPa; P = .021).

228 /=F1, >/=F2, >/=F3, and F4 were 7.4 kPa, 8.6 kPa, 9.6 kPa, and 14.4 kPa, respectively.

229 dimensional shear wave elastography were 9.6 kPa and 10.2 kPa, and for cirrhosis 19.7 kPa and 16.4 kP

230 F2, >/=F3, and F4 were 7.4 kPa, 8.6 kPa, 9.6 kPa, and 14.4 kPa, respectively.

231 ves were approximately 3 and approximately 6 kPa, respectively.

232 categories were fibrosis stage 0 and LSM <6 kPa.

233 eneration, with optimal bone formation at 60 kPa.

234 mechanical behavior (elastic modulus: ca. 60 kPa; fatigue life: >5000 cycles; mechanically stable at

235 ilopodia formation, whereas adhesion to a 60-kPa Ecad-Fc PA gel induced Arp2/3-dependent lamellipodia

236 gths, generate stresses of approximately 600 kPa, and produce peak power densities over 2 kW/kg-all e

237 rences between the flow channels of over 600 kPa.

238 A threshold of >3.63 kPa had a sensitivity of 0.86 (95% confidence interval [

239 agnosed by liver stiffness greater than 3.63 kPa using magnetic resonance elastography (MRE).

240 with higher pressure (777 kPa +/- 308 vs 630 kPa +/- 252, respectively; P < .001).

241 of the microvalve increase linearly up to 65 kPa as the closing pressure increases.

242 es C) and vapor pressure deficit (VPD, +0.66 kPa), annual percent deviation in precipitation (-38%),

243 o, inducing peak dynamic stress change of 67 kPa on the volcano surface and ~120 kPa in the spherical

244 - 0.8% vs 90.1% +/- 0.8%, for a 500 Torr (67 kPa) Xe cell loading-corresponding to nuclear magnetic r

245 A pressure sensitivity of 0.7 kPa(-1) is achieved in the pressure region <1 kPa with p

246 ve 600 mm and below warm season VPD of c.1.7 kPa.

247 9.6 kPa and 10.2 kPa, and for cirrhosis 19.7 kPa and 16.4 kPa.

248 han 50 mm Hg, PaO2 150 +/- 50 torr (20 +/- 7 kPa), PaCO2 40 +/- 5 torr (5 +/- 1 kPa), and core temper

249 houlder (median, 203.3 kPa; IQR, 144.1-242.7 kPa) in the shoulder-neutral position (P = .004) but not

250 kPa; interquartile range [IQR], 174.4-256.7 kPa) was significantly greater than that of the unaffect

251 of inspired oxygen (FiO) of 200 mm Hg (26.7 kPa) or less and an expected duration of ventilation of

252 Median TE values were 5.7 kPa (interquartile range, 4.7-7.2 kPa) and independently

253 kPa and 77.7 % for spleen stiffness of 50.7 kPa.

254 sis was excellent in patients having bLSM <7 kPa, SVR, or no increase (<1 kPa/year) in 7-14 kPa bLSM.

255 The median LSM change was -0.70 kPa (95% confidence interval, -3.0 to +1.7) and was simi

256 or compensated cirrhosis (FibroScan, 9.6-70 kPa; cirrhosis, n = 9); median baseline HCV-RNA level wa

257 ncreased from 0.15 kPa in stem cells to 0.71 kPa following chondrogenic differentiation.

258 rfaces of varying substrate compliance (4-71 kPa) and tissue culture plastic (TCP) (> 1 gigapascal [G

259 fness of normal (5 kPa) and glaucomatous (75 kPa) HTM.

260 ormal (5 kPa) and glaucomatous meshworks (75 kPa), or tissue culture polystyrene (TCP; >1 GPa).

261 .29 measured at GRE MR elastography and 2.76 kPa +/- 1.39 at SE-EPI MR elastography (n = 290; P = .15

262 ate was associated with higher pressure (777 kPa +/- 308 vs 630 kPa +/- 252, respectively; P < .001).

263 A cellular elastic modulus of 0.8 kPa was determined using the attached colloid as indente

264 fovir HBV DNA levels >2000 IU/mL (mean, -0.8 kPa; P = .048) or TE values >7.6 kPa (mean, -1.2 kPa; P

265 l offering a maximum adhesive stress of ~2.8 kPa.

266 s of the symptomatic shoulder (median, 234.8 kPa; interquartile range [IQR], 174.4-256.7 kPa) was sig

267 Subjects with a FibroScan >6.8 kPa had higher alanine aminotransferase (42 versus 28U/L

268 8; adolescents, 6.5 kPa +/- 1.2; adults, 7.8 kPa +/- 1.2; analysis of variance, P = .0003) but not at

269 cartilage, with the Young's modulus of >800 kPa and equilibrium friction coeffcient of <0.3.

270 ombus to variable wall thickness (359 +/- 86 kPa, P </= 0.001) reduced PWS.

271 ng all features also reduced PWS (368 +/- 88 kPa, P<0.001).

272 /= F3, and =F4 were 7.1, 8.8, 10.7, and 16.9 kPa, respectively.

273 le out CSPH with 55.2% specificity and >21.9 kPa rule in CSPH with 74.4% sensitivity.

274 With a cutoff of 5.9 kPa for LS, sensitivity and specificity for detection of

275 when tested at an applied gas pressure of 90 kPa and a temperature of (20+/-1) degrees C.

276 Mean liver stiffness was 2.92 kPa +/- 1.29 measured at GRE MR elastography and 2.76 kP

277 moderate fibrosis (stages F0, F1, F2) (5.93 kPa +/- 2.31 [standard deviation] vs 3.35 kPa +/- 1.44,

278 was used (mean +/- standard deviation, 6.94 kPa +/- 1.42 vs 5.96 kPa +/- 1.31; P = .006).

279 tandard deviation, 6.94 kPa +/- 1.42 vs 5.96 kPa +/- 1.31; P = .006).

280 oxia was defined as PaO2 >/=300 mm Hg (39.99 kPa), hypoxia as any PaO2 <60 mm Hg (7.99 kPa) or PaO2/F

281 PaO2 more than or equal to 300 mm Hg (39.99 kPa), hypoxia as any PaO2 less than 60 mm Hg (7.99 kPa)

282 xia was defined as PaO2 >/= 300 mm Hg (39.99 kPa), hypoxia as any PaO2<60 mm Hg (7.99 kPa) or PaO2/Fi

283 .99 kPa), hypoxia as any PaO2<60 mm Hg (7.99 kPa) or PaO2/FiO2 ratio </= 300, and normoxia, not defin

284 99 kPa), hypoxia as any PaO2 <60 mm Hg (7.99 kPa) or PaO2/FiO2 ratio </=300 and normoxia, not defined

285 hypoxia as any PaO2 less than 60 mm Hg (7.99 kPa) or PaO2/FIO2 ratio less than or equal to 300, and n

286 With spherical tips, E (kPa) was 0.71 +/- 0.16 in HUVEC and 0.94 +/- 0.06 in SC

287 resolving small pressure changes in the few kPa range, making them unsuitable for applications such

288 ly to 2.3 g mm/m(2)h kPa from 1.7 g mm/m(2)h kPa for SPI alone whereas rutin decreased water vapour p

289 permeability significantly to 2.3 g mm/m(2)h kPa from 1.7 g mm/m(2)h kPa for SPI alone whereas rutin

290 water vapour permeability to 1.2 g mm/m(2)h kPa.

291 ffness measurement (LSM) >/=8.0 kilopascals (kPa) was used as a cutoff suggesting clinically relevant

292 s defined as an LSM of 9.5-14.6 kilopascals (kPa), and cirrhosis as an LSM of >/=14.6 kPa.

293 er stiffness measurements (LSM; kiloPascals [kPa]) at ART initiation were categorized as no or minima

294 er vapor permeability (WVP, from 3 to 9 g.mm.kPa(-1).h(-1).m(-2)), and decreased insoluble matter (IM

295 2) ) and an ultrahigh sensitivity (of 3.1 nF kPa(-1) ), which is a thousand times greater than that o

296 pressure-to-capacitance sensitivity (114 nF kPa(-1) ), which is at least 1000 times higher than any

297 ting in an extraordinary sensitivity (217 nF kPa(-1) or 24 muC N(-1) at a bias of 1.0 V) without usin

298 tiffness, from hundreds of Pa to hundreds of kPa, T cell metabolic properties and cell cycle progress

299 preclinical study, we used MRE to quantify (kPa) the elasticity modulus Gd and viscosity modulus Gl

300 ic duplex US with SWE for hepatic stiffness (kPa).