ライフサイエンスコーパス: kaempferol

1 ator (LTTR), was identified as responsive to kaempferol.

2 ring the relative abundance of quercetin and kaempferol.

3 all the clones were increased in response to kaempferol.

4 in the ratio of the flavonols, quercetin and kaempferol.

5 , mainly glycosylated forms of quercetin and kaempferol.

6 gic acid>quercetin>protocatechuic acid>rutin>kaempferol.

7 ncluding apigenin, naringenin, luteolin, and kaempferol.

8 rogenic acid, rutin, quercetin, luteolin and kaempferol.

9 0.09mg/d, of p-coumaric acid 0.0068mg/d, of kaempferol 0.0034mg/d, of luteolin 0.0525mug/d, of querc

10 eractions were stabilized by the addition of kaempferol (20 microM).

11 nt flavonol and phenolic acid compounds were kaempferol (23.62mg/g) and 3-hydroxy-4-metoxy cinnamic a

12 O-sophoroside, kaempferol 3,7-O-diglucoside, kaempferol 3,7,4'-O-triglucoside, kaempferol 3-O-sophoro

13 way that is associated with the induction of kaempferol 3,7-dirhamnoside, whereas aphid feeding induc

14 l 3-O-glucoside, kaempferol 3-O-sophoroside, kaempferol 3,7-O-diglucoside, kaempferol 3,7,4'-O-triglu

15 sociations for the flavonoids gallocatechin, kaempferol 3-glucoside and quercetin 3-glucoside and the

16 products were obtained namely quercetin and kaempferol 3-O-(5''-O-malonyl)-alpha-l-arabinofuranoside

17 a result of a metabolomic study of saffron (kaempferol 3-O-glucoside, kaempferol 3-O-sophoroside, ka

18 study of saffron (kaempferol 3-O-glucoside, kaempferol 3-O-sophoroside, kaempferol 3,7-O-diglucoside

19 glucoside, kaempferol 3,7,4'-O-triglucoside, kaempferol 3-O-sophoroside-7-O-glucoside).

20 W1 was the richest in kaempferol 3-sophoroside (30.34 mg/g dry FB) and delphin

21 ified, including 4 quercetin, 5 myricetin, 4 kaempferol, 3 isorhamnetin, 2 laricitrin, 3 syringetin a

22 crog/g), isoquercetin (3.19-155.58microg/g), kaempferol-3-glucoside (2.31-2462.25microg/g) and myrice

23 ide (Q-Glu), kaempferol-3-rutinoside (K-Ru), kaempferol-3-glucoside (K-Glu) and derivative of quercet

24 techniques and chemometric tools proved that kaempferol-3-glucoside is one of the strongest markers f

25 picatechin-3-gallate], flavones (kaempferol, kaempferol-3-glucoside, quercetin, naringenin) and hydro

26 The most abundant flavonol was kaempferol-3-glucoside.

27 e most abundant flavonols were identified as kaempferol-3-O-(2-hexosyl)hexoside-7-O-rhamnosides.

28 Just kaempferol-3-O-(2-rhamnosyl)rutinoside was found in all

29 ed the highest amount of phenolic compounds, kaempferol-3-O-(6-rhamnosyl)hexoside plus kaempferol-3-O

30 ed as kaempferol glycosidic conjugates, with kaempferol-3-O-[glucopyranosyl-(1-->2)-galactopyranosyl-

31 of two major flavonoid compounds in saffron: kaempferol-3-O-beta-D-glucopyranosyl-(1-2)-beta-D-glucop

32 oxidants to be quercetin, kaempferol, rutin, kaempferol-3-O-beta-rutinoside and N(1),N(5),N(10)-triph

33 de, isoquercitrin, kaempferol-3-O-rutinoise, kaempferol-3-O-glucoside, quercetin-3-O-(6''-malonyl)-gl

34 uercetin-3-O-galactosyl-rhamnosyl-glucoside, kaempferol-3-O-glucosyl-rhamnosyl-glucoside, theaflavin,

35 s, kaempferol-3-O-(6-rhamnosyl)hexoside plus kaempferol-3-O-hexoside being the main compounds.

36 Rutin, hyperoside, isoquercitrin, kaempferol-3-O-rutinoise, kaempferol-3-O-glucoside, quer

37 ives the most abundant compounds, especially kaempferol-3-O-rutinoside.

38 etected in all the tissues and identified as kaempferol-3-O-sophoroside-7-O-rhamnoside.

39 oside (Q-Ru), quercetin-3-glucoside (Q-Glu), kaempferol-3-rutinoside (K-Ru), kaempferol-3-glucoside (

40 , (+)-catechin (60%), (-)-epicatechin (60%), kaempferol (33%) and quercetin-3-rutinoside (29%) decrea

41 -O-tetradecanoylphorbol-13-acetate, and that kaempferol, a natural compound found in edible plants, s

42 of these inhibitors is a naturally occurring kaempferol-alpha-L-diacetylrhamnoside, SL0101.

43 Kaempferol also inhibited the proliferation and migratio

44 Supplementation of roots with the flavonol kaempferol (an inhibitor of auxin transport), in combina

45 as found among women with a higher intake of kaempferol, an individual flavonol found primarily in br

46 In addition, kaempferol, an inhibitor of RSK2, suppressed EGF-induced

47 Except for kaempferol and daidzein, there were no significant assoc

48 egrative computational framework to identify kaempferol and esculetin as putatively novel therapies f

49 onfirmed significantly abrogated fibrosis by kaempferol and esculetin in vivo.

50 tally validated the anti-fibrosis effects of kaempferol and esculetin using renal tubular cells in vi

51 cute rejection in renal transplantation, and kaempferol and esculetin, two drugs not previously descr

52 a UDP-glucose-derived donor and the acceptor kaempferol and in complex with UDP and quercetin.

53 quercetin-3-glucoside, myricetin, quercetin, kaempferol and isorhamnetin, were found in the range of

54 The concentration of kaempferol and myricetin started decreasing at 150 degre

55 arious (iso)flavonoids such as the flavonols kaempferol and myricetin, the isoflavone formononetin, a

56 the k(cat)/K(m) values of rF3GalTase, using kaempferol and quercetin as substrates, approaches that

57 lic acids along with cyanidin-3-O-glucoside, kaempferol and quercetin.

58 nic acid, cryptochlorogenic acid, quercetin, kaempferol and their glycosides were identified together

59 reased throughout plant development, whereas kaempferol and total flavonoid glycosides showed higher

60 in and adenosine diphosphate glucose (ADPG), kaempferol and UDPG, quercetin and UDP-galactose, isoliq

61 the simultaneous separation of glycosylated kaempferols and geniposide consisted of the use of a C18

62 evels of flavonoids (anthocyanin, quercetin, kaempferol) and selected isoprenoid derivatives (chlorop

63 of three flavonols (myricetin, quercetin and kaempferol) and total phenolic content (TPC) in Moringa

64 Daidzein, genistein, kaempferol, and coumestrol (group 2) activated both ERal

65 Intake of total catechin, epicatechin, kaempferol, and myricetin and consumption of black tea w

66 Intake of individual flavonols (quercetin, kaempferol, and myricetin) and flavones (apigenin and lu

67 nd chrysin, and flavonols, such as galangin, kaempferol, and quercetin, were able to inhibit endothel

68 ites three were identified as flavonols (one kaempferol- and two quercetin-derivatives) and two as ot

69 ancer cell apoptosis, but on the other hand, kaempferol appears to preserve normal cell viability, in

70 s led to the isolation and identification of kaempferol as a pollen germination-inducing constituent

71 ize information concerning the extraction of kaempferol, as well as to provide insights into the mole

72 2) and Lys(100) are critical amino acids for kaempferol binding and RSK2 activity.

73 y role in substrate phosphorylation and that kaempferol binds with the NTD but not the CTD in both th

74 of the related biosynthetic pathways (e.g., kaempferol biosynthesis) are ascertained from the detect

75 ich makes no flavonols, and tt7, which makes kaempferol but not quercetin, showed that quercetin deri

76 ing the predominant flavonols, quercetin and kaempferol, by collisionally activated dissociation (CAD

77 s evaluated by HPLC and ESI-MS/MS, detecting kaempferol, catechin, quercetin and procyanidins B1 and

78 ounds such as: hesperetin (in citrus honey); kaempferol, chrysin, pinocembrin, caffeic acid and narin

79 Both quercetin and kaempferol competed with NPA for AtGSTF2 binding, indica

80 in 3-O-arabinoside, glycosides of quercetin, kaempferol, cyanidin, pelargonidin, peonidin, ellagic ac

81 (n) metabolic profiles showed high levels of kaempferol derivatives and anthocyanins.

82 f saffron through the analysis of a group of kaempferol derivatives recently proposed as novel authen

83 ealed interesting bioactive properties being kaempferol derivatives the most abundant compounds, espe

84 ruinosa (Vogel) Fortunato & Wunderlin, being kaempferol derivatives the most representative ones.

85 Kaempferol down-regulated ERK phosphorelation as well as

86 e sequences showing the greatest response to kaempferol encode proteins that have regulatory or signa

87 Flavonols as rutin and kaempferol, flavonoids as naringin, phenolic acids as ga

88 = microg of quercetin/g of kale or microg of kaempferol/g of kale by fresh weight, 5-15% relative sta

89 highest in June, while quercetin-glucoside, kaempferol-glucoside and total phenols, increased toward

90 side isomer, quercetin-malonyl-glucoside and kaempferol-glucoside at the end of the season.

91 different flavonol glycosides (quercetin and kaempferol glucosides).

92 quercetin glucosides and Eruca accumulating kaempferol glucosides.

93 lso involved in abiotic stress responses, as kaempferol glycosides were down-regulated in cml42, and

94 trilignols, altered accumulation patterns of kaempferol glycosides, and changes in minor conjugates o

95 iscrimination among species were assigned as kaempferol glycosidic conjugates, with kaempferol-3-O-[g

96 At the molecular level, kaempferol has been reported to modulate a number of key

97 Kaempferol has been reported to reduce the risk of ovari

98 All flavonols except kaempferol have absorption maxima above 440nm and so rea

99 described the beneficial effects of dietary kaempferol in reducing the risk of chronic diseases, esp

100 evelopmental and tissue-specific manner with kaempferol in the epidermis and quercetin in the cortex.

101 , was investigated to quantify quercetin and kaempferol in those samples.

102 include the synthesis of a protected form of kaempferol in which all four hydroxy groups are differen

103 e production of the flavonols, quercetin and kaempferol, in a tissue-specific and inducible manner.

104 and kaeR are upregulated in the presence of kaempferol, indicating the role of KaeR as a transcripti

105 In addition, kaempferol inhibited proliferation of malignant human ca

106 Significantly, kaempferol inhibits cancer cell growth and angiogenesis

107 s have shown an inverse relationship between kaempferol intake and cancer.

108 Women in the highest quintile of kaempferol intake relative to those in the lowest had a

109 The lower risk associated with kaempferol intake was probably attributable to broccoli

110 Kaempferol is a polyphenol antioxidant found in fruits a

111 technology to improve the bioavailability of kaempferol is also discussed.

112 avonols (glycosides of quercetin, myricetin, kaempferol, isorhamnetin, syringetin and laricitrin) hav

113 echin, (-)-epicatechin-3-gallate], flavones (kaempferol, kaempferol-3-glucoside, quercetin, naringeni

114 activated by the small polyphenolic molecule kaempferol (KPF).

115 ouse was found to enhance both quercetin and kaempferol levels in Vates kale.

116 a membrane, and endomembrane system, whereas kaempferol localized in the nuclear region and plasma me

117 The flavonoids tested were: quercetin, kaempferol, luteolin, fisetin, chrysin, galangin, flavon

118 oids, i.e., apigenin, genistein, hesperetin, kaempferol, luteolin, rhamnetin, rutin, tricetin and que

119 Lactucopicrin and kaempferol malonyl glucoside were consistently related t

120 The contents of quercetin-malonyl-glucoside, kaempferol-malonyl-glucoside isomer and kaempferol-malon

121 ide, kaempferol-malonyl-glucoside isomer and kaempferol-malonyl-glucoside were higher than that of th

122 quercetin-3-O-(6''-malonyl)-glucoside and a kaempferol-malonylhexoside were the most abundant flavon

123 Kaempferol may help by augmenting the body's antioxidant

124 rgeting RSK2 with natural compounds, such as kaempferol, might be a good strategy for chemopreventive

125 For kaempferol, most of the individual ORs were statisticall

126 be partly due to the presence of quercetin, kaempferol, naringenin and naringenin chalcone.

127 apic acid-O-hexoside, catechin-O-dihexoside, kaempferol-O-hexoside, and apigenin-C-hexoside-pentoside

128 ro-oxidant effect of flavonols quercetin and kaempferol on iron-based Fenton reaction were documented

129 lavonoids naringenin, isoliquiritigenin, and kaempferol, or with the synthetic auxin transport inhibi

130 id pentoside and deoxyhexose, quercitrin and kaempferol pentoside.

131 e 77 or 244 ppm quercetin and 235 or 347 ppm kaempferol (ppm = microg of quercetin/g of kale or micro

132 ed by Chang et al., works well for flavonols kaempferol, quercetin and myricetin, but not for gossype

133 The intakes of kaempferol, quercetin, luteolin, matairesinol and lignan

134 Flavonols kaempferol, quercetin, myricetin and gossypetin, and fla

135 and lower levels of the downstream products kaempferol, quercetin, myricetin, and anthocyanins, than

136 produced more flavonols but the quercetin-to-kaempferol ratio was also higher than the UV-A-supplemen

137 , we tend to expand our understanding on how kaempferol regulates VEGF expression and angiogenesis in

138 most important antioxidants to be quercetin, kaempferol, rutin, kaempferol-3-O-beta-rutinoside and N(

139 n O-di-hexoside, isorhamnetin rutinoside and kaempferol rutinoside were found in grape for the first

140 aB-cMyc-p21-VEGF pathway, which accounts for kaempferol's angioprevention effects in ovarian cancer c

141 s our comprehension of the mechanisms behind kaempferol's biological influence in ovarian cancer cell

142 Kaempferol significantly attenuated TGF-beta1-mediated p

143 Furthermore, we found that kaempferol suppressed angiogenesis through inhibiting VE

144 t being p-hydroxybenzoic acid, baicalein and kaempferol (T. aestivum), epicatechin and catechin (T. m

145 ived natural products including the flavonol kaempferol, the isoflavone biochanin A, and the chalcone

146 It was found that kaempferol time-dependently inhibited VEGF secretion, an

147 In addition, exposure of apigenin and kaempferol to cultured hepatocytes, mimicking first pass

148 Adding kaempferol to Fl-deficient pollen causes rapid and synch

149 reated K562 cell lines with nicotinamide and kaempferol to inhibit deacetylase activity of SIRT3 and

150 We show that adding micromolar quantities of kaempferol to the germination medium or to the stigma at

151 arian cancer cells, and better characterized kaempferol toward chemoprevention.

152 retion, and studied in-vitro angiogenesis by kaempferol treatment.

153 tion of DYVE-D3 indicates that the flavonoid kaempferol was the active substance.

154 acids were the principal phenolic acids and kaempferol was the predominant flavonoid found in raw mi

155 Three compounds: silymarin, quercetin and kaempferol were evaluated for their in vitro antiviral a

156 nd day length, and contents of quercetin and kaempferol were lower in phytotron than under semi-field

157 predominant phenolic acids, and luteolin and kaempferol were major flavonoids in the soluble fraction

158 Glycosides of kaempferol were the main flavonoids found (10 non-acylat

159 Glycosides of quercetin and kaempferol were the major phenolics.

160 genin, quercetin, apigenin-7-O-glucoside and kaempferol) were quantified using calibration curves.

161 avonols, including quercetin, myricetin, and kaempferol, were also not related to a lower risk of col

162 ar conjugates of the flavonols quercetin and kaempferol, which could be increased by threefold on int

163 g to the group of isorhamnetins (50-62%) and kaempferols, which represent the major part of flavonols