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1 any fully expressed, progressive infections (kala-azar).
2 ses that includes visceral leishmaniasis, or Kala Azar.
3 prevalence and mortality reported levels of Kala-azar.
4 l in treating T cell-deficient patients with kala-azar.
5 have committed to reducing the incidence of kala-azar, a clinical manifestation of visceral leishman
6 ate, could have a key role in the control of kala-azar, and prevent its resurgence when paired with t
7 nosing patients before the onset of clinical kala-azar (before 14 days fever), and show that this cou
11 obtained from healthy, healed VL (HVL), post kala-azar dermal leishmaniasis(PKDL) and VL subjects.
13 (2003) and five (2005) of the most affected Kala-azar districts had been classified as low-risk when
15 responses do not appear to develop in Indian kala-azar; instead, there is an initial mixed Th1-Th2 ce
17 gh frequency of treatment failures in Indian kala-azar might be due to infection with antimony-resist
19 tor in the failure of metalloid treatment in kala-azar patients infected with antimony-resistant Leis
20 ve expressions for, and compute estimates of Kala-azar's reproduction numbers, an indirect measure of
21 patients with cutaneous, visceral, and post-kala azar visceral leishmaniasis indicated that a majori
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