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1 ng cascade, the fibrinolytic system, and the kallikrein-kinin system.
2 important role in the assembly of the plasma kallikrein-kinin system.
3 at plays a central role in activation of the kallikrein-kinin system.
4 ole in the renin-angiotensin-aldosterone and kallikrein-kinin systems.
5 een GPCR that link the renin-angiotensin and kallikrein-kinin systems.
6 monoclonal antibody C11C1 attenuates plasma kallikrein-kinin system activation, local and systemic i
7 se the intriguing possibility that decreased kallikrein-kinin system activity may play an important r
13 ologic assembly and activation of the plasma kallikrein/kinin system and discusses its influence on v
14 s also been reported that alterations of the kallikrein-kinin system are associated with formation of
18 The relevance and significance of the plasma kallikrein/kinin system as a risk factor for the develop
20 vascular remodeling and the up-regulation of Kallikrein-kinin system contribute, at least in part, to
25 opposing roles of the renin-angiotensin and kallikrein-kinin systems in vivo, the distinct propertie
33 s studies in perfused limbs suggest that the kallikrein-kinin system may participate in the regulatio
35 r angiotensin II levels, suggesting that the kallikrein-kinin system partly mediates the effects of t
36 between MPO and the components of the plasma kallikrein-kinin system resulted in decreased bradykinin
37 or the assembly of the vasoregulatory plasma kallikrein-kinin system; thus we explored whether MPO an
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