ライフサイエンスコーパス: kanamycin A

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1 ives of neomycin B, neamine, tobramycin, and kanamycin A.

2 n the nanomolar range for all substrates but kanamycin A.

3 formers were determined for the enzyme-bound kanamycin A.

4 es or disappears as compared to the original kanamycin A.

5 ucture of the enzyme with bound MgAMPCPP and kanamycin A.

6 anamycin A and 4'-(m-nitrobenzyl phosphoryl)-kanamycin A.

7 eotide hairpin library that bind 6'-acylated kanamycin A (1) and 6'-acylated neamine (2) identified b

8 For three of the aminoglycosides, 6''-azido-kanamycin A, 5-O-(2-azidoethyl)-neamine, and 6''-azido-t

9 ucts formed are 4'-(adenosine-5'-phosphoryl)-kanamycin A and 4'-(m-nitrobenzyl phosphoryl)-kanamycin

10 ining 2'-amino substituents, also acetylated kanamycin A and amikacin that contain a 2'-hydroxyl subs

11 t multiamine-containing glycosides including kanamycin A and B, tobramycin, paromomycin, and neomycin

12 Conformations of kanamycin A and ribostamycin were compared to those of o

13 Bound conformations of kanamycin A and ribostamycin, in the active site of the

14 t acetylation takes place on the 6'-amine of kanamycin A and the adenylation on 3''- and 9-hydroxyl g

15 uation of derivatives based upon neomycin B, kanamycin A, and tobramycin conjugates of 9-aminoacridin

16 owing unique trends: 5'UNNNC3' loops for the kanamycin A derivative (where N is any nucleotide); 5'UN

17 erivative, and pyrimidine-rich loops for the kanamycin A derivative.

18 the divergent synthesis of three classes of kanamycin A derivatives.

19 a concentration range from 100pM to 1muM and kanamycin A from 10nM to 1mM.

20 Aminoglycoside antibiotics kanamycin A, gentamycin, and G418 stimulated PI-PLC clea

21 B, the derivatives based upon tobramycin and kanamycin A have slightly lower RRE affinities, but bett

22 ion by modularly assembling 6'-N-5-hexyonate kanamycin A (K) onto a peptoid backbone.

23 stance to the aminoglycoside (AG) antibiotic kanamycin A (KAN) in Mycobacterium tuberculosis.

24 Antibiotic substrates for AAC include kanamycin A, kanamycin B, tobramycin, sisomicin, neomyci

25 Gram-negative APH(3') types Ia and IIa with kanamycin A, neamine, and their respective difluorinated

26 o, binary, and ternary complexes of APH with kanamycin A, neomycin B, and metal-nucleotide.

27 agnetic resonance analysis of the product of kanamycin A phosphorylation revealed that modification o

28 ons as the acetyltransferase responsible for kanamycin A resistance, a hallmark of extensively drug-r

29 ptamer probes with affinity to ampicillin or kanamycin A, respectively.

30 '' and/or O-6'' positions on the ring III of kanamycin A, show very strong activity as antifungal age

31 unambiguously assigned to the 4' hydroxyl of kanamycin A; thus, the products formed are 4'-(adenosine

32 Surprisingly, binding of gentamicin and kanamycin A to the chemically synthesized terminal hairp

33 f RNA hairpin loops that bind derivatives of kanamycin A, tobramycin, neamine, and neomycin B via two

34 ied products of both reactions revealed that kanamycin A was modified only at one position.

35 Two conformers of kanamycin A were matched well with the two conformers of

36 However, kanamycin A, which has a 2'-OH, shows a much smaller dec

37 lyzed by kanamycin nucleotidyltransferase of kanamycin A with either ATP or m-nitrobenzyl triphosphat

38 nucleotidyltransferase catalyzed reaction of kanamycin A with m-nitrobenzyl triphosphate is 2 orders

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