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1 rom pUC21 (ampicillin resistance) and pUK21 (kanamycin resistance).
2 d replacing it with a gene cassette encoding kanamycin resistance.
3 is, which increase its expression and confer kanamycin resistance.
4 cation, and genes for rare transfer RNAs and kanamycin resistance.
5 utonomously replicating plasmid that confers kanamycin resistance.
6 hia coli-H. pylori shuttle vector conferring kanamycin resistance.
7 cells, and positive clones were selected by kanamycin resistance.
10 a bacteriophage T7 promoter, (ii) it confers kanamycin resistance, and (iii) it uses an R6K origin of
12 s to the stable phenotypes of tumorigenesis, kanamycin resistance, and stable beta-glucuronidase (GUS
14 D were each insertionally inactivated with a kanamycin resistance (aphA) cassette and allelic exchang
15 active hpyIIRM cassette [containing a 1.4 kb kanamycin resistance (aphA) marker], whether such acquis
16 he inactivation of sll0088 by insertion of a kanamycin resistance cartridge in the primary C14S(PsaC)
20 i, nixA was insertionally inactivated with a kanamycin resistance cassette (aphA) and this construct
22 ubunit sequence in E. coli by insertion of a kanamycin resistance cassette and sought to construct an
23 , and interruption of the cloned gene with a kanamycin resistance cassette eliminated the overexpress
24 ing sequence was removed and replaced with a kanamycin resistance cassette flanked by two res sites f
25 cells, and disruption of the oapA gene with kanamycin resistance cassette insertion resulted in a si
29 be required for macrophage killing, since a kanamycin resistance cassette introduced into icmS by ge
30 y disruption of the flaA or flaB gene with a kanamycin resistance cassette or by introduction of both
32 nC mutant generated by introducing an aphA-3 kanamycin resistance cassette produced CPS with no O-ace
34 rferi B31 by replacing the BBK32 gene with a kanamycin resistance cassette through homologous recombi
35 strain Ec1a (O1:K1:H7) were replaced with a kanamycin resistance cassette to produce an oxyRS mutant
36 hromosome by homologous recombination with a kanamycin resistance cassette to produce mutant J45-100.
37 ed in which the bca gene was replaced with a kanamycin resistance cassette via homologous recombinati
38 duce a GlnA-deficient mutant of H. pylori, a kanamycin resistance cassette was cloned into the Tth111
39 y of phiBB-1 to package and transduce DNA, a kanamycin resistance cassette was inserted into a cloned
41 of R. leguminosarum was created by placing a kanamycin resistance cassette within acpXL, the gene whi
46 nsion, was interrupted by the insertion of a kanamycin-resistance cassette between the ferrochelatase
47 phenicol-resistance cassette in place of the kanamycin-resistance cassette in pOX38-tra715 and a muta
49 sogenic H. pylori uvrB mutant by inserting a kanamycin-resistance cassette into uvrB and verified its
51 ent strain was constructed by insertion of a kanamycin resistance determinant within the ureC gene vi
52 (encoded by ssrA), coupled with a multicopy kanamycin resistance determinant, suppressed both lon ph
53 nstructed in this gene by the insertion of a kanamycin resistance DNA cassette into the chromosome.
55 ulting MS17 clone possessed erythromycin and kanamycin resistance, flat-wave morphology, and microsco
56 the corresponding gene was disrupted with a kanamycin resistance gene (aphA3) in H. pylori ATCC 4350
59 eliloti gltA gene was mutated by inserting a kanamycin resistance gene and then using homologous reco
61 train, E. coli O157:H7 W6-13, by inserting a kanamycin resistance gene cassette (kan) into wcaD and w
62 mutant, which was created by insertion of a kanamycin resistance gene cassette at the 5' region of i
63 e K8.1 open reading frame and insertion of a kanamycin resistance gene cassette within the K8.1 gene.
65 ch either fimA or orf365 was replaced with a kanamycin resistance gene did not participate in type 2
67 tions of the transposon Tn5 or Tn3-nice or a kanamycin resistance gene in each of these genes abolish
68 The MDV US3 orthologue was replaced with a kanamycin resistance gene in the infectious bacterial ar
69 nalysis revealed there was an insertion of a kanamycin resistance gene in the lgt2 gene of D4, which
71 ted DBB25, was constructed by insertion of a kanamycin resistance gene into alcA, one of the genes es
74 disrupted by insertion of the aphII neomycin-kanamycin resistance gene resulted in the accumulation o
75 disrupted in strain 81-176 by insertion of a kanamycin resistance gene through homologous recombinati
76 LEU2 genes and a transposon-derived neomycin/kanamycin resistance gene were successfully expressed fr
77 es a gC-negative deletion mutant harboring a kanamycin resistance gene, a markerless mutant with the
78 of pDMG21A, a pVT745 derivative containing a kanamycin resistance gene, displayed a structural rearra
79 pJGS84, a derivative of pAP1 containing a kanamycin resistance gene, was able to replicate in Esch
82 plants transformed with a vector containing kanamycin-resistance gene (npt) flanked by FRT sites, wh
83 gene product inhibited a transposon-derived kanamycin-resistance gene in both M. smegmatis and M. tu
84 lytic subdomains I-III by the insertion of a kanamycin-resistance gene resulted in slightly delayed,
85 nd negative selection markers, which are the Kanamycin-resistance gene, the sacB gene and temperature
86 plasmids containing mariner transposons and kanamycin resistance genes expressible in B. burgdorferi
87 segment required for psbL editing, chimeric kanamycin resistance genes were constructed containing p
88 ssette was designed so that the luxABCDE and kanamycin resistance genes were linked to form a single
90 mined in this study that exhibited low-level kanamycin resistance harbored eis promoter mutations.
91 antify the static and dynamic composition of kanamycin resistance in artificial microbiota to evaluat
92 plication origin, the nptIII gene conferring kanamycin resistance in bacteria, both the right and lef
96 rless chloramphenicol resistance (Cm(r)) and kanamycin resistance (Km(r)) cassettes, respectively, th
97 gI fragment within lob-2A was deleted, and a kanamycin resistance (Km(r)) gene was inserted into this
102 laced in a binary plasmid vectorcontaining a kanamycin resistance marker and a cauliflower mosaic vir
103 nstructs of fbpA and fbpB disrupted with the kanamycin resistance marker OmegaKm and containing varyi
108 These mutations restored high levels of kanamycin resistance not through an improvement in the p
111 with both approaches using attP vectors with kanamycin resistance or spectinomycin resistance as the
112 that suffers mutations during acquisition of kanamycin-resistance results in an overwhelming majority
113 cillin resistance), aphA1-Iab (which encodes kanamycin resistance), strA and strB (which encode strep
114 the ability of these heterodimers to confer kanamycin resistance to Escherichia coli cells was impai
115 hotransferase type II gene, which can confer kanamycin resistance to transgenic plants, represent an
116 The mutations R177S and V198E restored the kanamycin resistance to wild-type levels while maintaini
117 nce of AID and a genetic reversion assay for kanamycin-resistance to investigate the causes of multip
118 d a genetic selection for splicing-dependent kanamycin resistance with no significant bias when six a
119 model successfully predicted the dynamics of kanamycin resistance within artificial microbiota under
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