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1 adykinin generation in plasma activated with kaolin.
2 % compared with equivalent concentrations of kaolin.
3 generates no bradykinin upon activation with kaolin.
4 heating a homogenised, pelletised mixture of kaolin (100 parts), sodium carbonate (100 parts), bitume
6 tion decreased to 0.51+/-0.09 units/mL, with kaolin ACT returning to 177+/-22 secs, celite ACT return
11 arterial pressure, central venous pressure, kaolin and celite activated clotting time (ACT), activat
13 nducted in a rat model of acute (carrageenan/kaolin) arthritis, with subsequent evaluation in a rat m
14 cified porcine whole blood, blood added with kaolin as an activator, and blood spiked with fibrinogen
15 ir high viscosity, while Natural, Valoal and Kaolin as emulsifiers for their good surface-active prop
17 h 2 measures sensitive to malaise, increased kaolin consumption (pica behavior) and failure to expres
18 resulted in increased food intake, increased kaolin consumption, and decreased need-induced sodium in
20 ed that charge neutralization contributed to kaolin flocculation, but was not involved in M. aerugino
21 duced by a single basal cistern injection of kaolin in 3-week-old rats, immediately followed by 3 or
24 Furthermore, TAFI deficiency did not alter kaolin-induced writhing response, implying that TAFI doe
25 nstrated the development of hydrocephalus in kaolin-injected rats but also revealed that continuous d
26 vels throughout the ventricular system after kaolin injection and also inhibited the deposition of th
28 activation of human platelet-poor plasma by kaolin led to cleavage of chem163S, which was undetectab
31 ng efficiencies were 93.34% within 2 min for kaolin suspension and 87.98% within 10 min for M. aerugi
34 an be expected that extracted olive oil from kaolin treated trees has a higher oxidative stability an
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