ライフサイエンスコーパス: karyokinesis

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1 /B-C- cardiac myocytes show major defects in karyokinesis.

2 e states without carrying out cytokinesis or karyokinesis.

3 hase-anaphase transition leading to abnormal karyokinesis.

4 dent mitotic delay and also cause defects in karyokinesis.

5 ton, increased cell spreading, and increased karyokinesis.

6 suggesting that byr4 is required for proper karyokinesis.

7 M II in maintaining mitotic stability during karyokinesis.

8 nd of daughter buds toward the completion of karyokinesis.

9 ntractility and in processes such as mitotic karyokinesis.

10 2 did not rescue the Wee1 inhibition-induced karyokinesis and cytokinesis defects.

11 hology and to highly enlarged cells in which karyokinesis and cytokinesis frequently are uncoupled.

12 ndle assembly as well as the coordination of karyokinesis and cytokinesis in mouse oocytes.

13 eate parasite can establish a state in which karyokinesis and cytokinesis occur in phase with the hos

14 centrosome that segregates the functions of karyokinesis and cytokinesis provides an explanation for

15 otypes consistent with spatial uncoupling of karyokinesis and cytokinesis suggesting that GEMINI POLL

16 were complemented with the demonstration of karyokinesis and cytokinesis to provide structural evide

17 of growth of adjacent cells, and defects in karyokinesis and cytokinesis.

18 progression of all mitotic phases leading to karyokinesis and cytokinesis.

19 did not result in any inhibitory effects on karyokinesis and early stages of cytokinesis.

20 bitor of glucosylceramide synthesis, blocked karyokinesis and reduced cyst production in culture.

21 nd chromosome segregation defects, defective karyokinesis, and a failure to complete cytokinesis.

22 ssociated with rampant aneuploidy, defective karyokinesis, and consequently, a failure of cytokinesis

23 ence of mitotic spindles, contractile rings, karyokinesis, and cytokinesis was also recorded.

24 pindles, the formation of contractile rings, karyokinesis, and cytokinesis--were identified; these fe

25 furrow initiation, mitotic spindle function, karyokinesis, and partitioning of intrinsic components a

26 i composed of cells in which cytokinesis and karyokinesis are uncoupled.

27 Thus, CHO1 may not be required for karyokinesis, but it is essential for the proper midzone

28 CHO1 was originally implicated in karyokinesis, but the invertebrate homologues of CHO1 we

29 II is involved in regulating cardiac myocyte karyokinesis by affecting microtubule dynamics.

30 , in spite of the inhibition of cytokinesis, karyokinesis continued, with the result that cells conta

31 with donut-shaped nuclei exhibit defects in karyokinesis, develop aneuploidy, and are often binuclea

32 pression of a non-cleavable SCC1 resulted in karyokinesis failure.

33 ation at E12.5, reflecting the occurrence of karyokinesis in the absence of cytokinesis.

34 This requirement for NM II in karyokinesis is further demonstrated in the HL-1 cell li

35 e rigid matrix facilitated nuclear division (karyokinesis) leading to binucleation, while compliant m

36 , and its depletion causes severe defects in karyokinesis, loss of individual chromosomes, and gross

37 dicating that inaccurate endoreplication and karyokinesis occur during MK polyploidization.

38 onse to mutants that perturb cytokinesis and karyokinesis, suggesting interactions between byr4 and t

39 f root-knot nematodes are formed by repeated karyokinesis uncoupled from cytokinesis, whereas the syn

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