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1 of myoepithelial cells and cells expressing keratin 6.
2 ed from its presumed polymerization partner, keratin 6.
3 als independently regulate the expression of keratin 6.
4 ed hyperkeratosis and aberrant expression of keratin 6.
5 al surface; the expression of involucrin and keratins 6, 13, 14, and 19; and the absence of keratin 1
6 al cells expressing progenitor cell markers, keratin 6 and Sca-1; subsequent tumors express these mar
9 maturation is delayed, and wound-associated keratins 6 and 16 are induced, in both involved and clin
10 rmis is hyperproliferative and overexpresses keratins 6 and 16, indicating abnormal differentiation.
11 2 and 3: EGF, FGF-2, IFNalpha2, IL-1RA, HSA, keratin-6, and involucrin; cortisol was significantly hi
14 maintained periderm-like cells that express keratin 6, but we observed abnormal expression of GRHL3.
15 ority of CD34+ cells (98%) were positive for keratin 6, establishing this population as basal keratin
18 ilaggrin/filaggrin along with focal areas of keratin 6 expression in the interfollicular epidermis.
20 the promoter/regulatory region of the bovine keratin 6 gene was used to target ODC transgene expressi
23 n culture treated with interleukin-1 express keratin 6 in all suprabasal layers of the epidermis, thr
24 t also by inducing directly the synthesis of keratin 6 in epidermal keratinocytes, and thus changing
25 in 16 along with its type II keratin partner keratin 6 in the companion layer of the outer root sheat
27 yzed the molecular mechanisms regulating the keratin 6 induction by interleukin-1, and found that the
34 nogenesis, we used bovine keratin 5 (K5) and keratin 6 (K6) promoter elements to direct the expressio
35 sed ODC in the skin of Ptch1+/- mice using a keratin 6 (K6) promoter that directs constitutive ODC ex
37 ified epithelia causes a strong induction of keratins 6 (K6) and 16 (K16) in post-mitotic keratinocyt
38 known as K6hf) is one of the isoforms of the keratin 6 (KRT6) family located within the type II cytok
39 I keratin 16 (Krt16) and its partner type II keratin 6 (Krt6a, Krt6b) cause pachyonychia congenita (P
40 ssion of differentiation markers, keratin 1, keratin 6, loricrin, and filaggrin in ML.myc2 transgenic
41 ncreased expression of interleukin-1beta and keratin 6, markers of keratinocyte activation seen in wo
42 in genes on mouse chromosome 15, between the keratin 6 (mK6alpha/mK6beta) and hair keratin genes.
43 were p63 negative, keratin 17 positive, and keratin 6 positive and present at sites of adhesion, alt
44 interleukin-1 responsive DNA element in the keratin 6 promoter, and determined that it contains a co
46 follicular differentiation markers including keratin 6, transglutaminase, and the hair keratins mHa2
47 keratinocyte differentiation, mouse-specific keratin 6 was overexpressed in the suprabasilar, hyperpl
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