ライフサイエンスコーパス: keratinize

1 anatomy of the area, including the width of keratinized and attached gingiva, the facial-to-lingual

2 The vaginal epithelium, in contrast, is not keratinized and can allow absorption of other molecules.

3 Both keratinized and nonkeratinized surfaces of esophagi were

4 The protein is expressed in keratinizing and nonkeratinizing stratified squamous epi

5 sociated with HPV, however, only a subset of keratinizing and verrucous penile carcinomas is positive

6 In Mode 3B, keratinized apoptosed epithelial cells became permanent

7 ha protein, and expression was restricted to keratinized areas.

8 se to at least six different cell types that keratinize as they move up the hair shaft and inner root

9 and mandibular symphyses that probably bore keratinized beaks.

10 condition and finally becomes a part of the keratinized cells of the cornified epidermal layer.

11 ermal cells to a fully inactive state in the keratinized cells of the cornified layer.

12 inactive condition and becomes a part of the keratinized cells of the cornified layer.

13 Ac-T antibody was observed in the late-stage keratinized conjunctival epithelia of patients with OCP.

14 eir distribution with that in pathologically keratinized conjunctival epithelia.

15 epithelia, including the stratified but non-keratinized corneal, limbal and conjunctival epithelium,

16 The lower telomerase activity in the keratinized (differentiated) vagina was probably due to

17 Olmsted syndrome (OS) is a rare keratinizing disorder characterized by excessive epiderm

18 Pachyonychia congenita (PC) is a keratinizing disorder predominantly caused by mutations

19 EPPK) is one of >30 autosomal-dominant human keratinizing disorders that could benefit from RNA inter

20 y pattern was observed in samples from other keratinizing disorders, demonstrating that loss of LEKTI

21 on detection strategy for ongoing studies of keratinizing disorders.

22 differentiation switch to express markers of keratinized epidermis); cornea stroma changes including

23 a major component of cornified envelopes in keratinized epidermis, were substantially up-regulated i

24 Cytokeratins typical of keratinized epithelia (K1, K2, and K10) and the keratini

25 The mice developed severe hypertrophy of all keratinized epithelia, but no malignancies were observed

26 ce that expressed the E6 and E7 oncogenes in keratinized epithelia.

27 opriate development of stratified, squamous, keratinizing epithelia, such as the epidermis and oral e

28 nown as 14-3-3sigma) show similar defects of keratinizing epithelia.

29 olite, produced in large amounts in squamous keratinizing epithelia.

30 limb abnormalities as a result of defects in keratinizing epithelia.

31 the cornea consists of a unique type of non-keratinized epithelial cells arranged in an orderly fash

32 creased incidence of squamous cell tumors of keratinized epithelial cells of the skin and esophagus.

33 zed in transgenic mice overexpressing NGF in keratinized epithelium (e.g. skin, tongue and oral cavit

34 Tumors occurred in regions that had keratinized epithelium and were subjected to repeated me

35 lobal change in the tumorigenic potential of keratinized epithelium in Atp2a2+/- mice.

36 rging functions in the genesis of stratified keratinized epithelium, hair follicles, and squamous cel

37 for maintenance of colonization at sites of keratinized epithelium, such as the vagina, or for adher

38 ified squamous epithelium to a nonsecretory, keratinized epithelium.

39 ne expression during malignant conversion of keratinized epithelium.

40 physiological and pathological remodeling of keratinized epithelium.

41 psies appeared as normal mucoperiosteum with keratinized epithelium.

42 g epithelium of attached gingiva and the non-keratinizing epithelium lining the upper two-thirds of t

43 These results demonstrate that both the keratinizing epithelium of attached gingiva and the non-

44 al epithelium, in concert with the epidermal keratinized eyelid epithelium, function together to main

45 eks was compared to the mean width of buccal keratinized gingiva (KG) of adjacent teeth.

46 Width of keratinized gingiva (KG) was determined at baseline and

47 ingival tissue thickness (GTT), and width of keratinized gingiva (KG) were assessed at baseline, and

48 cluded: percent root coverage, the amount of keratinized gingiva (KG), and probing depth (PD).

49 nt level (CAL), probing depth (PD), width of keratinized gingiva (KG), attached gingiva (AG), and rec

50 ession depth, recession width (RW), width of keratinized gingiva (KW), clinical attachment level (CAL

51 ), papilla index score (WDPIS), and width of keratinized gingiva (WDKG) between initial and last meas

52 level (CAL), recession height (RH), width of keratinized gingiva (WKG) and assessment of gingival bio

53 Clinical parameters (width of keratinized gingiva [WKG], facial soft tissue level [FST

54 The standard of care for increasing keratinized gingiva adjacent to teeth that do not requir

55 ican female was referred for augmentation of keratinized gingiva around implants at the right and lef

56 ture as the adjacent tissue, a 1-mm width of keratinized gingiva at 6 months, patient treatment prefe

57 the ability of the LCC to regenerate >/=2 mm keratinized gingiva at 6 months.

58 Spectral reflection of keratinized gingiva at upper central incisors was measur

59 CDH and improve patient quality of life, by keratinized gingiva augmentation and impact on physical

60 tment; 4) gingival recession; and 5) band of keratinized gingiva for each of the six anterior mandibu

61 After removing the mucosa and keratinized gingiva from the test site, either an LCC or

62 recession depth (RD), recession width (RW), keratinized gingiva height measured apico-coronally (KG)

63 At 6 months, the LCC regenerated >/=2 mm of keratinized gingiva in 95.3% of patients (81 of 85 patie

64 preferentially located in plaque, others in keratinized gingiva or buccal mucosa, and some oligotype

65 Width of keratinized gingiva significantly increased after IMITG

66 As expected, the FGG generated more keratinized gingiva than the LCC (4.57 +/- 1.0 mm versus

67 Results confirm the keratinized gingiva to have increased tensile strength (

68 At that time, 112 sites of inadequate keratinized gingiva were found.

69 terally lacking sufficient zones of attached keratinized gingiva were randomly assigned to soft tissu

70 ness, gingival ridge dimension, and width of keratinized gingiva were the esthetic outcomes reviewed.

71 All patients achieved >/=1 mm keratinized gingiva with the LCC treatment by 6 months,

72 inical attachment level, GR height, width of keratinized gingiva, and assessment of gingival biotype.

73 GR dimensions, amount of keratinized gingiva, and clinical attachment level were

74 statistically better root coverage, width of keratinized gingiva, and complete root coverage.

75 teeth being partially or totally engulfed by keratinized gingiva, causing aesthetic and functional pr

76 the output variables -- changes in width of keratinized gingiva, changes in bucco-lingual width, and

77 milar community compositions: buccal mucosa, keratinized gingiva, hard palate; saliva, tongue, tonsil

78 led a thick biotype with an adequate band of keratinized gingiva, Miller Class I mucogingival defects

79 sthesia, incisions were initiated within the keratinized gingiva.

80 All patients had a minimum of 4 mm of keratinized gingiva.

81 This flap design is carried out within keratinized gingiva.

82 rous enlargement of maxillary and mandibular keratinized gingiva.

83 ign, localized or generalized enlargement of keratinized gingiva.

84 effective therapy for augmenting the zone of keratinized gingiva.

85 Clinical parameters including the keratinized gingival (KG) width, KG thickness, horizonta

86 e aim of the present study is to compare the keratinized gingival tissue (KT) height labial to the ma

87 depth, clinical attachment level (CAL), and keratinized gingival width (KGW) was recorded at baselin

88 Moreover, the increased value of the keratinized gingival width attested to the positive outc

89 Clinical measurements of keratinized gingival width, probing depth, and recession

90 readily, removal of upper skin layer exposed keratinized hair shafts at the skin surface.

91 ypes: well keratinized (K-SCC), intermediate keratinized (I-SCC), and poorly keratinized (P-SCC).

92 SCC was classified into three subtypes: well keratinized (K-SCC), intermediate keratinized (I-SCC), a

93 The columnar epithelium undergoes keratinizing metaplasia, which is reversed when R is sup

94 need for keratinized mucosa (KM) or immobile keratinized mucosa (i.e., attached mucosa [AM]) for the

95 Whether a minimal width of keratinized mucosa (KM) is required to maintain peri-imp

96 The need for keratinized mucosa (KM) or immobile keratinized mucosa (

97 ied plaque index (mPI), and the width of the keratinized mucosa (WKM) were performed at baseline (at

98 nd between induced defect depth and width of keratinized mucosa at baseline (P = 0.03).

99 P), mucosal redness (MR), suppuration (SUP), keratinized mucosa dimension, and marginal bone loss.

100 The mean width of keratinized mucosa measured at the mid-buccal location p

101 tached gingiva of the molars and the lingual keratinized mucosa of the incisor region.

102 Width of keratinized mucosa was measured in the region of each im

103 The keratinized mucosa width (KMW) gain was significantly gr

104 romising alternative for the regeneration of keratinized mucosa without tissue harvesting.

105 e active and proliferative state than native keratinized mucosa.

106 ized epithelial layer similar to native oral keratinized mucosa.

107 gold standard for augmenting small areas of keratinized mucosa.

108 similar healing, with increased peri-implant keratinized mucosa.

109 dulus of 19.75 +/- 6.20 MPa) relative to non-keratinized mucosal regions, where densely arranged elas

110 ced, nonkeratinized (n = 6), and late-stage, keratinized (n = 8).

111 to the specialized epithelia surrounding the keratinized nail plate.

112 The consequences of Notch1 activation in keratinizing nail cells were investigated in a transgeni

113 30 eyes of 30 patients with SJS-induced dry keratinized ocular surfaces; the patients underwent vari

114 Matriptase was also expressed in keratinizing oral epithelium, where it was required for

115 tively, revealed extensive detachment of the keratinized outer layer and distention of spinous and ba

116 hological changes included detachment of the keratinized outer layer, distention of spinous and basal

117 intermediate keratinized (I-SCC), and poorly keratinized (P-SCC).

118 to compare the CM versus FGGs for augmenting keratinized peri-implant mucosa based on clinical and hi

119 amine green (LG; 0.15 mul) was placed on the keratinized portion of the central lower lid.

120 osteo-odonto keratoprosthesis surgery in dry keratinized post-SJS eyes.

121 ossly, the CXCR3(-/-) mice presented a thick keratinized scar compared with the wild-type mice in whi

122 Over a human's life span, keratinized skin cells, immune cells, and microbes all i

123 l-fate switch from transparent epithelium to keratinized skin-like epidermis, which led to corneal bl

124 --which turns cornea into a non-transparent, keratinized skin-like epithelium--causes corneal surface

125 Although keratinizing squamous cell carcinoma and verrucous carci

126 ly these tumors consisted largely of focally keratinizing squamous cell carcinoma with high-grade mal

127 avicular lesion and also detected concurrent keratinizing squamous cell metastasis in the right axill

128 EN deficiency accelerated the development of keratinizing squamous metaplasia (KSM).

129 elial hyperplasia and female mice developing keratinizing squamous metaplasia.

130 All biopsies showed a multilayered, keratinized, squamous epithelium.

131 ole in establishing the boundary between the keratinized, squamous esophagus/forestomach and glandula

132 and cytokeratin 10/13, suggested a premature keratinized state.

133 the morphologically different layers of the keratinized stratified epithelium of the dorsal layer of

134 d a chronically inflamed cyst lined by a non-keratinized stratified squamous epithelium.

135 rneal epithelial progenitor cells into a non-keratinizing stratified epithelium.

136 In the keratinizing stratified squamous epidermis of skin, howe

137 areas of squamous metaplasia in the form of keratinizing stratified squamous epithelium, similar to

138 m spinosum and stratum granulosum, and a non-keratinizing stratum corneum.

139 Rather, they develop a specialized keratinized structure, called the rhamphotheca, that cov

140 +/- 1.7 mm and 2.2 +/- 1.6 mm), increase in keratinized tissue (0.7 +/- 0.8 mm and 1.2 +/- 1.0 mm),

141 linical attachment level (CAL), and width of keratinized tissue (KG) were evaluated at 12 months.

142 The standard of care for increasing keratinized tissue (KT) and vestibular area is an autoge

143 The role of keratinized tissue (KT) for maintenance of periodontal h

144 Although the need for "adequate" amount of keratinized tissue (KT) for periodontal health is questi

145 for the purposes of increasing the width of keratinized tissue (KT) is an important aspect of period

146 /- 1.1 mm (74.1%) (both P<0.01), increase in keratinized tissue (KT) of 1.2 +/- 1.3 and 1.6 +/- 1.9 (

147 coverage, recession reduction, and amount of keratinized tissue (KT) were analyzed using descriptive

148 GR depth, keratinized tissue (KT) width, and probing depth were me

149 lts for root coverage and greater amounts of keratinized tissue (KT) with the acellular dermal matrix

150 ding vertical recession (VR), probing depth, keratinized tissue (KT), and attachment level were recor

151 t circumstances require an increased zone of keratinized tissue (KT), or is KT important?

152 h, clinical attachment level (CAL) gain, and keratinized tissue (KT).

153 ing depth (PD; 2.3 +/- 0.2 mm), and width of keratinized tissue (KT; 2.4 +/- 0.3 mm); measurements we

154 ), clinical probing depth (PD), and width of keratinized tissue (KTW) were measured preoperatively an

155 ondary efficacy parameters included width of keratinized tissue (KTw), probing depth (PD), clinical a

156 was a statistically significant increase in keratinized tissue (mean 0.88 mm) and tissue thickness (

157 dimension was correlated with the amount of keratinized tissue (P = 0.010) and also with defect cove

158 , clinical attachment level (CAL), height of keratinized tissue (wKT), and percentage of root coverag

159 idth (RW), gingival thickness (GT), width of keratinized tissue (WKT), clinical attachment level (CAL

160 idth (RW), gingival thickness (GT), width of keratinized tissue (WKT), clinical attachment level (CAL

161 evel (CAL), probing depth (PD), and width of keratinized tissue (WKT), were recorded presurgery (at b

162 (3) clinical attachment level; (4) width of keratinized tissue (wKT); (5) percentage of root coverag

163 was accompanied by a significant increase in keratinized tissue 6 months after surgery.

164 along with a 0.5-mm decrease in the width of keratinized tissue after healing.

165 l parameters with the exception of amount of keratinized tissue and percent shrinkage of keratinized

166 The GA generated more keratinized tissue and shrank less than the HF-DDS graft

167 n terms of root coverage, increased width of keratinized tissue and thickness of the gingival biotype

168 elanosis of the buccal gingiva and a lack of keratinized tissue around implants at sites #4 and #13.

169 procedure designed to increase the amount of keratinized tissue around teeth that do not require root

170 owed a significant increase in the amount of keratinized tissue at 9 and 12 months compared to baseli

171 The mean width of keratinized tissue at the beginning of the study was 1.7

172 h good color match and an increased width of keratinized tissue at the surgical site.

173 complete RC (CRC), attachment gain (AG), and keratinized tissue change (KTC) were also calculated.

174 a connective tissue graft tended to increase keratinized tissue compared to ADM (0.52-mm difference;

175 pression and function of matriptase in mouse keratinized tissue development, homeostasis, and maligna

176 tissue graft demonstrated greater amount of keratinized tissue during the 12-month evaluation period

177 Limiting the flap design to keratinized tissue facilitates flap closure and avoids w

178 The mean increase in keratinized tissue for both treatments was 0.80 mm.

179 elimination and augmentation of the zone of keratinized tissue for prosthetic reasons.

180 f the outcomes measured (recession coverage, keratinized tissue formation, probing depths, and clinic

181 cluded recession, clinical attachment level, keratinized tissue height, and plaque index.

182 e amount of root coverage, probing depth, or keratinized tissue in coronally advanced flaps for root

183 There was greater increase in the width of keratinized tissue in the E group (1.5+/-1.1 mm) than th

184 Only keratinized tissue in the test group demonstrated a stat

185 probing attachment level, and the amount of keratinized tissue in the treatment of localized recessi

186 ted in a statistically significant effect on keratinized tissue increase, but no significant effects

187 ary benefit, it is believed that the zone of keratinized tissue is additionally increased after gingi

188 al inflammation, areas with small amounts of keratinized tissue may remain stable over long periods o

189 self-reported discomfort, and the amount of keratinized tissue obtained.

190 There was a mean increase in keratinized tissue of 0.60 mm for test sites and a mean

191 t (FGG), evaluating their ability to augment keratinized tissue or gingiva.

192 p exhibited an average of 1.0 to 1.2 mm more keratinized tissue over time than the test group (P <0.0

193 V infection was greater (31.0%) across the 3 keratinized tissue sites (genital skin, eyebrow hairs, f

194 root coverage, keratinized tissue width, or keratinized tissue thickness (P >0.05).

195 Keratinized tissue thickness gain was significant only i

196 Keratinized tissue was increased for the ADM group by 0.

197 t coverage of 5 mm along with a 2-mm band of keratinized tissue was obtained at 24 weeks.

198 -1.19 versus 3.50 +/-0.73), and the width of keratinized tissue was significantly increased (2.47+/-1

199 root coverage (CRC), width, and thickness of keratinized tissue were assessed by the same masked exam

200 differences in recession depth and width of keratinized tissue were seen at 5 months between the GP+

201 a mean root coverage of 87.4% and increased keratinized tissue width (1.2 mm) when compared to their

202 h (PD), clinical attachment level (CAL), and keratinized tissue width (KT).

203 ded the following: 1) probing depth (PD); 2) keratinized tissue width (KT); 3) tissue biotype (TB); a

204 Mean keratinized tissue width (KTW) increased at a similar ra

205 bing depth, clinical attachment level (CAL), keratinized tissue width (KTW), horizontal recession (HR

206 Clinical measurements (GR length [REC], keratinized tissue width [KT], complete root coverage [C

207 robing depth, clinical attachment level, and keratinized tissue width and thickness were measured at

208 leeding on probing, plaque index scores, and keratinized tissue width then recorded.

209 Clinical measurement of recession and keratinized tissue width was standardized using customiz

210 there was improvement in gingival thickness, keratinized tissue width, and root coverage.

211 orrelation with percentage of root coverage, keratinized tissue width, or keratinized tissue thicknes

212 lt in additional (or "rebound") increases in keratinized tissue width.

213 DDS graft was safe and capable of generating keratinized tissue without the morbidity and potential c

214 ad significantly more tissue thickness gain (keratinized tissue) than the crestal group compared to b

215 ed that 19 sites showed a slight increase in keratinized tissue, 35 were unchanged (for a total of 54

216 ft exposed resulted in a greater increase of keratinized tissue, and complete coverage of the graft r

217 t the widest point, probing depth, amount of keratinized tissue, and marginal tissue thickness.

218 me with increased gingival tissue thickness, keratinized tissue, and root coverage.

219 ), marginal tissue levels, biotype, width of keratinized tissue, and soft tissue thickness.

220 n terms of root coverage, increased width of keratinized tissue, and thickness of the gingival biotyp

221 : recession depth, recession width, width of keratinized tissue, clinical attachment level, and probi

222 r recession depth, recession width, width of keratinized tissue, clinical attachment level, and probi

223 ificant differences in root defect coverage, keratinized tissue, clinical attachment level, or clinic

224 istically equivalent, including increases in keratinized tissue, esthetic results, and subject satisf

225 n depth, clinical attachment level, width of keratinized tissue, mobility, and plaque score.

226 l defect, width (WKT) and thickness (TKT) of keratinized tissue, probing depth (PD), and clinical att

227 Clinical measurements of vertical recession, keratinized tissue, probing depths, and attachment level

228 and slight, but significant improvements in keratinized tissue, tissue thickness, and bone level.

229 o the gingival margin, and with 2 to 3 mm of keratinized tissue.

230 ot coverage with an increase in the width of keratinized tissue.

231 ge, probing attachment levels, and increased keratinized tissue.

232 DM to free gingival graft in augmentation of keratinized tissue.

233 keratinized tissue and percent shrinkage of keratinized tissue.

234 robing depth, recession width, and amount of keratinized tissue.

235 es), and 7 sites showed a slight decrease in keratinized tissue.

236 gingival index, probing depth, and width of keratinized tissue.

237 re common in females, and often found on the keratinized tissue.

238 eans to treat gingival recession and augment keratinized tissue.

239 3) determine the effect of the procedure on keratinized tissue; and 4) evaluate the amount of creepi

240 onal extent of the gingival defect; width of keratinized tissue; probing depth; clinical attachment l

241 root coverage (RC) and ADM-based increase in keratinized tissues to other commonly used mucogingival

242 rsor of the cornified envelopes of mammalian keratinizing tissues.

243 Well keratinized tonsillar SCCs lack HPV DNA and are associat

244 almost exclusively associated with a poorly keratinized tumor histology.

245 Interestingly, a majority of the keratinized tumors expressed high levels of miR-21.

246 These results demonstrate that the non-keratinized vaginal epithelium permits a rapid absorptio

247 s of aire-deficient mice were pathologically keratinized with significant epithelial damage and alter