ライフサイエンスコーパス: keratinocyte

1 coincident with enhanced cell death in human keratinocytes.

2 mis through SCF production by LTA-stimulated keratinocytes.

3 s the neutrophil chemoattractant capacity of keratinocytes.

4 and activator of transcription 3 pathway in keratinocytes.

5 displayed a greater capacity for invasion of keratinocytes.

6 ition enhanced, IL-17-driven inflammation in keratinocytes.

7 a dose- and time-dependent manner in normal keratinocytes.

8 0A12 was knocked down by RNA interference in keratinocytes.

9 poptosis both in vivo epidermis and in vitro keratinocytes.

10 ll-length SERPINB7 protein synthesis in NPPK keratinocytes.

11 (ECM), and stimulates reepithelialization by keratinocytes.

12 ntiation and hyperproliferation of epidermal keratinocytes.

13 IFN-kappa decreased IL-6 production by lupus keratinocytes.

14 promotes glycolytic metabolism in epidermal keratinocytes.

15 heterochromatin protein 1-alpha in p63-null keratinocytes.

16 ashion with predominant effects on epidermal keratinocytes.

17 nder irradiation on nontumorigenic NCTC-2544 keratinocytes.

18 vesicle release from squamous cell carcinoma keratinocytes.

19 ntiation and promotes proliferation of human keratinocytes.

20 n of melanin and hyperproliferation of basal keratinocytes.

21 uced OCRL protein as compared to the control keratinocytes.

22 P cytoprotection in response to UVB in human keratinocytes.

23 nd limiting innate immune responses in human keratinocytes.

24 ion and impaired terminal differentiation of keratinocytes.

25 hat these genes are direct VDR targets in WT keratinocytes.

26 d adherence of S. epidermidis to keratin and keratinocytes.

27 apsid formation take place in differentiated keratinocytes.

28 chromosomal DNA synthesis in UVB-irradiated keratinocytes.

29 n-activated protein kinases in primary human keratinocytes.

30 mmatory cytokines and chemokines by infected keratinocytes.

31 ollagen at similar levels compared to normal keratinocytes.

32 enhancer element that is active in epidermal keratinocytes.

33 and epigenomic landscape of mouse and human keratinocytes.

34 took advantage of a unique finding in human keratinocytes.

35 asal/stem cell-associated phenotype in human keratinocytes.

36 lectin (KACL) selectively expressed by human keratinocytes.

37 es a close interplay between fibroblasts and keratinocytes.

38 eficiency promotes inflammatory responses in keratinocytes.

39 in the development of malignant neoplasms of keratinocytes.

40 mutants results in impaired KF formation in keratinocytes.

41 N blockade decreased IL-6 secretion by lupus keratinocytes.

42 with these sequences in VDR(-/-) but not WT keratinocytes.

43 ause extensive protein damage in HaCaT human keratinocytes.

44 silencing of ROCK1 was sufficient to rescue keratinocyte adhesion and biochemical differentiation in

45 cterized role in pro-inflammatory responses, keratinocytes also directly support immune-suppressive r

46 Furthermore, gammac-deficient keratinocytes also exhibit defective induction of T-cell

47 suggest that persistent gammac-deficiency in keratinocytes alters immune cell recruitment to the skin

48 pear to play regulatory roles in a number of keratinocyte and endothelial cellular traits associated

49 ugh in the epidermis Blimp1 is important for keratinocyte and sebocyte differentiation, its role in d

50 nd are associated with genes involved in key keratinocyte and skin-centric biological processes.

51 oblasts in collagen rafts seeded with turtle keratinocytes and (ii) keratinocyte maturation induced b

52 ation of DNA during cornification of lingual keratinocytes and aberrant DNA retention is tolerated in

53 t mevastatin inhibited cortisol synthesis in keratinocytes and biopsies from patients with DFU.

54 ments in the cytoplasm of cornifying lingual keratinocytes and co-deletion of DNase1L2 and Trex2 caus

55 bound human keratins 1 and 10 and adhered to keratinocytes and epithelial cells.

56 Cultured keratinocytes and fibroblasts from an affected individua

57 At baseline, both resident cells, such as keratinocytes and fibroblasts, and infiltrating immune c

58 also noted in remaining melanocytes and some keratinocytes and fibroblasts.

59 We measured OCRL protein levels in patient keratinocytes and found that Lowe 1 patient cells had si

60 LR1, TLR6, IL-25, and IL-33 in human primary keratinocytes and HaCaT cells.

61 ammation in lipopolysaccharide (LPS)-induced keratinocytes and imiquimod (IMQ)-induced psoriasiform d

62 46a inhibits inflammatory responses in human keratinocytes and in different mouse models of skin infl

63 of decreased photocarcinogenesis in in vitro keratinocytes and in well-characterized mouse models in

64 h inhibiting the terminal differentiation of keratinocytes and inducing antimicrobial peptides and se

65 nes but was also a feature of nontransformed keratinocytes and lung fibroblasts.

66 We show that keratinocytes and microvascular endothelial cells show g

67 epidermal-related proteins and cytokines in keratinocytes and on skin architecture.

68 th CTSB expression in normal differentiating keratinocytes and other cell lines, but not with FDFT1 o

69 normalized PPARgamma mRNA levels in VDR(-/-) keratinocytes and restored anagen responsiveness in vivo

70 the skin, HAT-L4 expression was abundant in keratinocytes and sebaceous glands.

71 positive for Lgr6 expression in immortalized keratinocytes and show that their frequency increases in

72 romatin architecture, and gene expression in keratinocytes and suggest nuclear envelope-associated ge

73 that B. pseudomallei invades fibroblasts and keratinocytes and survives inside these cells as well as

74 te proteoglycan 4) was detected in epidermal keratinocytes and the cardiac conduction system.

75 Our findings highlight a role for keratinocytes and their interplay with myeloid cells in

76 e dedifferentiation program of tumor-forming keratinocytes and to molecular cascades leading to tumor

77 y allowed for the distinction between local (keratinocyte) and systemic GC activity.

78 as knockdown of Dstyk in mouse melanocytes, keratinocytes, and fibroblasts, were associated with inc

79 l-like receptor 2 and UVB treatment in lupus keratinocytes, and neutralization of IFN-kappa decreased

80 ls fail to transdifferentiate into epidermal keratinocytes, and there was no improvement in the clini

81 -kappa) increased IL-6 production by control keratinocytes, and type I IFN blockade decreased IL-6 se

82 -lox-mediated novel, inducible, fibroblast-, keratinocyte-, and wound-specific STAT3-knockdown postna

83 orated the H2O2-dependent increase in matrix keratinocyte apoptosis and reversed the reactive oxygen

84 Thus, lupus keratinocytes are primed for IL-6 hyperproduction in a t

85 Strikingly, when keratinocytes are stretched or hypo-osmotically shocked,

86 Here, we sought to examine the keratinocyte as an important source of IL-6 and define t

87 erates with cGAS during DNA sensing in human keratinocytes, as both cGAS and IFI16 are required for t

88 2D (NKG2D) ligand expression in the lesional keratinocytes associated with a marked CD4+ T cell infil

89 h-affinity interaction with the cognate CTLR keratinocyte-associated C-type lectin (KACL) selectively

90 ta show that the innate response of infected keratinocytes attracts virus-permissive myeloid cells th

91 Using a keratinocyte-based activity assay in conjunction with sm

92 ively inhibits differentiation and growth of keratinocytes both ex vivo and in IL-23-injected ears of

93 cts barrier gene expression in primary human keratinocytes; both of these processes are likely to be

94 Suppression of mRNA for these KLKs in keratinocytes by targeted small interfering RNA silencin

95 Keratinocyte cancer puts a large burden on health care s

96 Risk of developing malignancies after keratinocyte cancer was assessed among 118,440 Caucasian

97 Keratinocyte cancers (KCs), including basal cell carcino

98 In the general population, keratinocyte cancers are associated with increased risks

99 et al. report on the increasing incidence of keratinocyte cancers in Germany.

100 the principal environmental risk factor for keratinocyte cancers, but other carcinogens have also be

101 ecipients have increased risk for developing keratinocyte cancers, including cutaneous squamous cell

102 of these new patients will develop multiple keratinocyte cancers.

103 The Veterans Affairs Keratinocyte Carcinoma Chemoprevention Trial was a rando

104 ta genus human papillomaviruses (betaPV) and keratinocyte carcinoma in OTRs.

105 screening may improve melanoma outcomes and keratinocyte carcinoma morbidity, but little is known ab

106 Keratinocyte carcinomas (KCs), consisting of squamous ce

107 veterans with a recent history of 2 or more keratinocyte carcinomas performed from September 30, 201

108 wo critical binary classification use cases: keratinocyte carcinomas versus benign seborrheic keratos

109 In keratinocyte cell cultures RAPTOR upregulation or AKT1 s

110 d against staphylococcal alpha-toxin-induced keratinocyte cell death.

111 noma cell line (SKMEL- 147) and immortalized keratinocyte cells (HaCaT).

112 Finally, using vaspin-overexpressing keratinocyte cells, we show that a significant part of s

113 Proinflammatory cytokines, keratinocyte chemoattractant (KC), and interleukin 6 (IL

114 Administration of L. reuteri suppressed keratinocyte chemoattractant (KC), Il22, Il6, Tnf, and I

115 Plasma leakage and keratinocyte chemoattractant production in the tibiotars

116 roinflammatory, cancer-associated cytokines, keratinocyte chemoattractant, IL-22, and IL-6, in plasma

117 ss and renewal capacity of the proliferating keratinocyte compartment.

118 pithelial barrier function of differentiated keratinocytes, comprising keratinocyte immune function a

119 al analyses of IL-17C+ and IL-17C+KO primary keratinocytes confirmed increased expression of proinfla

120 ol uptake in normal but not in AQP3-knockout keratinocytes, confirming that the expressed AQP3 was fu

121 of clusters of melanoma cells surrounded by keratinocytes constraining their proliferation.

122 therapies that target IFN-gamma signaling in keratinocytes could be safe and effective new treatments

123 Exposing a stratified keratinocyte culture to a reduced-hydration environment

124 conditioned medium collected from stratified keratinocyte culture under reduced-hydration conditions

125 h the physical epithelial barrier, including keratinocyte cytoskeleton, intercellular junctions, and

126 IP2 may alter both epidermal homeostasis and keratinocyte defense responses to influence psoriasis ri

127 nce activated cell sorting method to isolate keratinocytes, dendritic cells, CD4+ T effector cells, a

128 In vitro, primary keratinocytes derived from IL1r(-/-) mice were more resi

129 matory immunomediators in kidney showed that keratinocyte-derived chemokine, macrophage inflammatory

130 f FBXO17 inhibits agonist-induced release of keratinocyte-derived cytokine (KC) and interleukin-6 (IL

131 or epidermal knockout mice demonstrated that keratinocyte-derived GC synthesis protected skin from to

132 Blocking keratinocyte-derived IL-1beta alone reduced infection of

133 , with increased expression of IL-17-induced keratinocyte-derived inflammatory mediators, epidermal h

134 Loss of keratinocyte-derived MIF leads to a loss of control of e

135 veral genes, including those associated with keratinocyte differentiation and antioxidative responses

136 complexes to regulate mammary epithelium and keratinocyte differentiation and proliferation during em

137 meobox transcription regulator DLX3 disrupts keratinocyte differentiation and results in an IL-17-lin

138 to improve calcium mobilization and improve keratinocyte differentiation and wound healing.

139 ory skin disease, is driven by both terminal keratinocyte differentiation defects and strong type 2 i

140 matin remodeling in the context of epidermal keratinocyte differentiation in the skin.

141 Unchecked inflammation, impaired keratinocyte differentiation, and heightened host defens

142 al regulation has proven to be important for keratinocyte differentiation, little is known about the

143 entiation-specific protein expression during keratinocyte differentiation.

144 ced expression of TSLP and reverses impaired keratinocyte differentiation.

145 amellae in the stratum corneum, and aberrant keratinocyte differentiation.

146 During CD44H cell generation, transformed keratinocytes display evidence of mitophagy, including m

147 s converted into increased proliferation and keratinocytes displaying genomic instability are maintai

148 3,000 genes were differentially expressed in keratinocytes due to HPV16 infection.

149 SCC cell lines compared with normal primary keratinocytes due to increased protein stability.

150 In cultured keratinocytes, Edaradd rescues DNA damage, cell survival

151 signaling events involving cytokines between keratinocytes, endothelial cells, T cells, macrophages a

152 Keratinocytes exposed to S. aureus showed enhanced degra

153 8, tumor necrosis factor, and IL-1B, whereas keratinocytes exposed to several L. major isolates did n

154 and upstream transcription factors in human keratinocytes exposed to ssUVR.

155 Terminally differentiating epidermal keratinocytes express a large number of structural and a

156 Subcellular fractionation of keratinocytes expressing E2(Y131A) showed a marked chang

157 Induction of keratinocyte expression of the antimicrobial/host defens

158 yrosine kinases that are highly expressed in keratinocytes for binding to BPV-1 E2.

159 g stress-enduring cells into fibroblasts and keratinocytes for skin reconstitution.

160 Human keratinocytes from a single donor treated with or withou

161 Keratinocytes from an affected individual showed loss of

162 in a 3-dimensional skin model using primary keratinocytes from LCE3B/C-del genotyped donors.

163 /b-influenced genes was detected in cultured keratinocytes from miR-146a(-/-) and skin fibroblasts fr

164 odels showed that Pak1 confers protection to keratinocytes from UV-B-induced apoptosis and DNA damage

165 nown regarding the direct effects of IL-4 on keratinocyte function.

166 Next, to elucidate the role of keratinocyte gene expression in late events during the v

167 Keratinocyte gene expression is critically shaped by IL-

168 We found that mitogenic stimulation with keratinocyte growth factor (KGF) markedly accelerated mo

169 l, and human lung injury models suggest that keratinocyte growth factor (KGF) might be beneficial in

170 cal trials in the transplant setting include keratinocyte growth factor, cytokines (IL-7 and IL-22),

171 Human keratinocyte (HaCaT) cell line was employed for the bio-

172 ed using q-PCR and western blotting in human keratinocytes (HaCaT) and endothelial cells (HECV).

173 L. major-exposed keratinocytes had no comparable effect.

174 teomic data from disaggregated primary human keratinocytes held in suspension to induce differentiati

175 we demonstrated that human limbal epithelial keratinocytes (HLEKs) engineered to overexpress miR-184

176 role of the atypical E2Fs, E2F7 and E2F8, in keratinocyte homeostasis, regeneration and tumorigenesis

177 cathepsin B, a cysteine protease involved in keratinocyte homeostasis.

178 explored the role of GM3 in regulating human keratinocyte IGF1R signaling.

179 PSMalpha induces the release of keratinocyte IL-1alpha and IL-36alpha, and signaling via

180 In human keratinocytes, IL-36alpha mRNA was induced by HSV-1, whi

181 of differentiated keratinocytes, comprising keratinocyte immune function and cellular structure, was

182 e and secretion of AD-related cytokines from keratinocytes in a PAR2 independent manner.

183 ass I genes is downregulated in HPV-positive keratinocytes in an E7-dependent manner.

184 end, we investigated possible involvement of keratinocytes in connective tissue healing.

185 beta signaling is operative in mouse primary keratinocytes in conventional cultures as determined by

186 s, loss of Ash1l leads to more proliferative keratinocytes in disturbed differentiation stages.

187 Given that keratinocytes in geriatric skin display reduced activati

188 A key role for keratinocytes in this complex process has yet to be esta

189 cs are strongly suppressed in differentiated keratinocytes in two distinct steps due to alterations i

190 the processing of laminin-gamma2 in cultured keratinocytes in vitro and in wound epidermis in vivo.

191 ng UVB exposure, here we used cultured human keratinocytes in vitro and skin explants ex vivo to exam

192 miR-200c expression in both human and mouse keratinocytes in vitro revealed inhibitory effects of mi

193 les, suggesting that in the absence of IL-6, keratinocytes increase production of numerous additional

194 Keratinocytes incubated with L. infantum significantly i

195 We propose that keratinocytes initiate or withhold a proinflammatory res

196 stroma is sufficient to reprogram wild-type keratinocytes into nipple-like epidermis.

197 How p63 regulates metabolism in epidermal keratinocytes is incompletely understood, and it is unkn

198 A key target of Mi-2beta in keratinocytes is the pro-inflammatory cytokine thymic st

199 epidermal equivalents (HEEs) generated with keratinocytes isolated from nonlesional skin of patients

200 Keratinocyte (KC) hyper-proliferation and epidermal thic

201 in close contact with surrounding epidermal keratinocytes (KCs).

202 is attenuated in female mice by deletion of keratinocyte lathosterol 5-desaturase, then UVR accelera

203 onstrate that LCE3B/C-del leads to increased keratinocyte LCE3A expression.

204 1, alpha9beta1, or both integrins as well as keratinocyte lines expressing these integrin combination

205 nd DNA methylation using normal immortalized keratinocyte lines, NIKS, NIKS-16, NIKS-18, and NIKS-16E

206 ts seeded with turtle keratinocytes and (ii) keratinocyte maturation induced by raising raft or biops

207 We utilised isogenic keratinocytes, microvascular endothelial cells, melanocy

208 cluding by exposure to high glucose, inhibit keratinocyte migration and IGF-1-induced chemotaxis in a

209 favor of FSTL1 upon wounding, which enhances keratinocyte migration and promotes re-epithelialization

210 Keratinocyte migration is a key aspect of re-epitheliali

211 Wound closure requires the activation of keratinocyte migration via a dual-state molecular switch

212 1 (IGF-1) receptor (IGF1R) signaling induces keratinocyte migration, but little is known about its re

213 Thus, keratinocyte Myd88 signaling in response to S. aureus PS

214 ell lines (n = 8) and normal human epidermal keratinocytes (n = 11) with real-time quantitative PCR a

215 ations of normal, spontaneously immortalized keratinocytes (NIKS) and NIKS stably transfected with HP

216 E6 and E7 transfected normal oral keratinocytes (NOK) were used to induce hypermethylation

217 The cornification of keratinocytes on the surface of skin and oral epithelia

218 D K5 mutants into KtyII-lacking (KtyII(-/-)) keratinocytes prevents keratin network formation altoget

219 Keratinocytes produced IL-17c during HSV-2 reactivation,

220 Moreover, E2f7/8-deficient primary keratinocytes proliferate more efficiently under stress

221 and altered AP1 factor function can perturb keratinocyte proliferation and differentiation.

222 trate that gene silencing of KIND1 decreased keratinocyte proliferation and increased apoptosis in vi

223 iR-146a/b suppressed and inhibition enhanced keratinocyte proliferation and the expression of psorias

224 to delayed re-epithlialization but increased keratinocyte proliferation at the wound edge.

225 ndicate that KIND1 is important not only for keratinocyte proliferation but also for the suppression

226 of modulation of inflammatory responses and keratinocyte proliferation in psoriatic skin.

227 HF keratinocyte proliferation was evident within the Lrig1+

228 ion, with studies showing its involvement in keratinocyte proliferation, differentiation, and migrati

229 siology, including sebocyte differentiation, keratinocyte proliferation, epithelial stem cell surviva

230 ompared with irhom2(+/+)mice, due to reduced keratinocyte proliferation.

231 aka and Mutlu propose that p63 regulates the keratinocyte proliferation/differentiation switch by aff

232 or elucidate which proteolysis products from keratinocytes promote skin inflammation.

233 ctor, we demonstrate for the first time that keratinocytes regulate proliferation of fibroblasts and

234 We investigated the role of Act1 in keratinocyte responses to IL-17 using a tetracycline ind

235 Overexpression of GLI2DeltaN in human keratinocytes resulted in cytoplasmic accumulation and n

236 iation-mitosis checkpoint in human epidermal keratinocytes, resulting in impaired cell division and s

237 (RXR)-alpha and RXR-beta in mouse epidermal keratinocytes (RXR-alphabeta(ep-/-)) or a topical applic

238 Ultraviolet (UV)-irradiated keratinocytes secrete the lipid mediator of inflammation

239 th the gammac-ligand IL-15, gammac-deficient keratinocytes show significantly impaired secretion of s

240 Confirming this hypothesis, Zc3h12a(-/-) keratinocytes showed increased responsiveness to IL-17A

241 nt mice and CTSH short hairpin RNA knockdown keratinocytes showed reduced filaggrin processing, and t

242 ngiogenic roles for alpha3beta1, alpha3-null keratinocytes showed reduced paracrine stimulation of en

243 differentiated uninfected and HPV16-positive keratinocytes showed that almost 3,000 genes were differ

244 Knockdown experiments in primary human keratinocytes showed that decreased claudin-1 expression

245 confirm that UVR-induced production of D3 in keratinocytes significantly restrains murine BCC tumorig

246 In keratinocytes, SIRT1 knockdown inhibited EMT, cell migra

247 Surprisingly, keratinocyte-specific deletion of E2F7 and E2F8 in mice

248 MCs were also evaluated in mice with keratinocyte-specific deletion of Scf.

249 anscriptional activity and relocation of the keratinocyte-specific gene loci away from the sites of a

250 Secretion of keratinocyte-specific IFN-kappa was significantly increa

251 Global or keratinocyte-specific Pnpla1-deficient neonates die due

252 Studies in mice with keratinocyte-specific PPARgamma haploinsufficiency were

253 phogenic hair cycles as a result of impaired keratinocyte stem cell (KSC) function.

254 proliferation was evident within the Lrig1+ keratinocyte stem cell population (69 vs. 55%, P < 0.01,

255 = 7), and not in the CD34+, LGR5+, and LGR6+ keratinocyte stem cell populations.

256 To determine the role of HF keratinocyte stem cells in beta-HPV-induced skin carcino

257 fection within long-lived hair follicle (HF) keratinocyte stem cells.

258 8 expression in differentiated but not basal keratinocytes suggesting that CGI-58 is essential for li

259 Interestingly, alpha9beta1 in keratinocytes suppressed alpha3beta1-mediated stimulatio

260 e determinants of autoantibody formation and keratinocyte susceptibility in pemphigus were discussed.

261 ciency ectopically induces the expression of keratinocyte terminal differentiation markers in the duc

262 skin, whereas IL-27 leads to suppression of keratinocyte terminal differentiation.

263 n of more than 1000 genes expressed in human keratinocytes that are involved in a broad spectrum of n

264 anscriptional differences in mouse and human keratinocytes that converges on major metabolic processe

265 es a "feed forward" inflammatory response in keratinocytes that is self-amplifying and drives the dev

266 We show, using immortalized and primary keratinocytes, that S. aureus protease SspA/V8 is the do

267 were significantly lower in Mdm2(SNP309G/G) keratinocytes, the cell-type susceptible to squamous cel

268 We evaluated the effects of HSV infection on keratinocytes, the initial target of HSV replication, to

269 ng terminal differentiation of the epidermal keratinocytes, the nucleus undergoes programmed transfor

270 Here we show that keratinocytes, the only cell type directly infected by h

271 AND Embryoid bodies were derived from human keratinocytes, their action potential characteristics de

272 n of Arf in tumor stromal cells, as in tumor keratinocytes themselves, contributes to suppression of

273 idermis is orchestrated by interactions with keratinocytes through multiple mechanisms.

274 opose that loss of cell contact in epidermal keratinocytes through reactive oxygen species-mediated d

275 In addition, KIND1 loss sensitized keratinocytes to cytokine and UV-induced NF-kappaB and c

276 udy, we show that S. aureus stimulates human keratinocytes to increase their endogenous protease acti

277 EAP1/4 knockdown also reduced the ability of keratinocytes to induce neutrophil chemotaxis.

278 ypothesized that Leishmania infection causes keratinocytes to produce immunomodulatory factors that i

279 Mechanistically, IL-27 feeds back on keratinocytes to stimulate cell proliferation and re-epi

280 dendritic projections and squeezing between keratinocytes to survey the tissue for pathogens.

281 les for bioimaging of fixed and living human keratinocytes, to localize hyaluronan and sialylation si

282 d by miRNAs along the malignant evolution of keratinocytes towards cSCC.

283 five stages along the malignant evolution of keratinocytes towards cSCC: Normal epidermis, solar elas

284 PV infection manipulates the differentiating keratinocyte transcriptome to create an environment cond

285 To examine HPV-associated changes in the keratinocyte transcriptome, RNAs isolated from undiffere

286 ethal ultraviolet B (UVB) irradiation, human keratinocytes transiently block progression of the cell

287 We show that primary mouse keratinocytes transiently exposed to the SASP exhibit in

288 s ectopically overexpressed and inhibited in keratinocytes treated with IL-17.

289 mal initiating signals produced by DLX3-null keratinocytes, we performed acute deletion of DLX3 in ad

290 Human HaCaT keratinocytes were exposed to either a single dose or 5

291 To investigate this, HaCaT keratinocytes were studied in vitro with regard to cell

292 fibroblasts, and lymphatic endothelium, but keratinocytes were the earliest targets of infection and

293 sses and in proliferation/differentiation of keratinocytes were under-expressed.

294 Primary polymorphonuclear leukocytes and keratinocytes were used to examine phagocytosis and bact

295 rmosensitive ion channel widely expressed in keratinocytes, where together with epidermal growth fact

296 a and IL-17A was reduced in Trim32-deficient keratinocytes, whereas CC chemokine ligand 5 induction b

297 the skin microbiome drives SCF production in keratinocytes, which triggers the differentiation of der

298 Skin aging processes are modeled by treating keratinocytes with H2O2.

299 Incubation of primary or immortalized human keratinocytes with Leishmania infantum or Leishmania maj

300 of fibronectin critically impaired the human keratinocyte wound response.