ライフサイエンスコーパス: keratopathy

1 care unit are at increased risk of exposure keratopathy.

2 nduced lipidosis and development of a vortex keratopathy.

3 gnificant role in the progression of bullous keratopathy.

4 f corneal damage as assessed by the grade of keratopathy.

5 wing to nocturnal lagophthalmos and exposure keratopathy.

6 ease, exposure keratopathy, and neurotrophic keratopathy.

7 t survival in eyes with pseudophakic bullous keratopathy.

8 or Fuchs' dystrophy and pseudophakic bullous keratopathy.

9 apeutic option for the treatment of diabetic keratopathy.

10 ry corneal edema due to pseudophakic bullous keratopathy.

11 f corneal sensation, leading to neurotrophic keratopathy.

12 ongest in patients with pseudophakic bullous keratopathy.

13 chs endothelial dystrophy and 5 with bullous keratopathy.

14 ital thermal injuries and resultant exposure keratopathy.

15 ndothelial dystrophy or pseudophakic bullous keratopathy.

16 lp to elucidate the pathogenesis of aniridic keratopathy.

17 d to understand the pathogenesis of diabetic keratopathy.

18 lar adhesion molecules is altered in bullous keratopathy.

19 dominant features of contemporaneous bullous keratopathy.

20 romising potential therapeutics for diabetic keratopathy.

21 may be a potential therapeutic for diabetic keratopathy.

22 t of post-PRK dry eye and other neurotrophic keratopathies.

23 endothelial dystrophy (260 eyes) or bullous keratopathy (15 eyes).

24 ases were performed for pseudophakic bullous keratopathy (2 cases, 1 in each cohort), and the remaini

25 Sixteen patients demonstrated dendritiform keratopathy after exposure to the preservative polyquate

26 Three patients developed severe toxic keratopathy after orofacial surgery on the left side wit

27 eal densitometry in patients with amiodarone keratopathy (AK).

28 and/or tear film may contribute to diabetic keratopathy and delayed epithelial wound healing in diab

29 by tear lipocalin from corneas with bullous keratopathy and dry eye.

30 on eight cases each of pseudophakic bullous keratopathy and healthy corneas.

31 t with silicone oil tamponade developed band keratopathy and phthisis bulbi.

32 halmic socket syndrome, and one had exposure keratopathy and traumatic dilated pupil.

33 rneal findings, such as superficial punctate keratopathy, and abnormal results of dry eye tests, such

34 ective in symptomatic improvement of bullous keratopathy, and infectious keratitis but further studie

35 om human corneas with chronic edema, bullous keratopathy, and keratoconus and from normal corneas wer

36 tion, postherpetic keratitis scarring, lipid keratopathy, and limbal stem cell deficiency.

37 ferentiated ocular surface disease, exposure keratopathy, and neurotrophic keratopathy.

38 unctal plugs were more effective in limiting keratopathy, and their use, particularly of bandage cont

39 ca, and South America), pseudophakic bullous keratopathy/aphakic bullous keratopathy (North America),

40 t common indication for PTK is still bullous keratopathy, as PTK can be successfully used while waiti

41 thelium differentially and may contribute to keratopathy associated with severe ocular allergy.

42 ndothelial dystrophy or pseudophakic bullous keratopathy at a single tertiary center.

43 surgical indication was pseudophakic bullous keratopathy at a single tertiary center.

44 of all patients diagnosed with dendritiform keratopathy between 1999 and 2014 who had documented exp

45 Fuchs endothelial dystrophy (FED) or bullous keratopathy (BK) in Asian eyes.

46 l corneal dystrophy (FECD; n = 314), bullous keratopathy (BK; n = 31), and failed previous endothelia

47 In early-stage aniridic keratopathy, BSCVA and tear BUT were reduced relative to

48 vels of ephrin-A1 may contribute to diabetic keratopathies by persistently engaging EphA2 and prohibi

49 th application of simple protocols, exposure keratopathy can be prevented, thus improving patient car

50 hesis that epithelial alterations in bullous keratopathy compromise the surface of the cornea and its

51 age was not detected in pseudophakic bullous keratopathy corneas, whereas it colocalized with termina

52 Diabetic keratopathy decreases corneal sensation and tear secreti

53 dothelial dystrophy and pseudophakic bullous keratopathy, EK achieved better average best-corrected a

54 cluded corneal melts resulting from exposure keratopathy, endophthalmitis, and infectious keratitis o

55 In our study of eyes with bullous keratopathy, endothelial keratoplasty under a previously

56 In contrast, none of the bullous keratopathy eyes showed any improvement throughout the f

57 ndothelial dystrophy or pseudophakic bullous keratopathy from January 2006 through December 2011.

58 p 2 (n = 2), patients with progressive lipid keratopathy; group 3 (n = 4), post keratoplasty patients

59 Band keratopathy, >/= 3+ vitreous cells, exudative retinal de

60 Patients in group 2 with lipid keratopathy had 100% obliteration of vessels with stabil

61 ith normal controls and pseudophakic bullous keratopathy (iatrogenic CE cell loss) specimens.

62 dothelial dystrophy and pseudophakic bullous keratopathy in patients without other vision-limiting oc

63 thylamiodarone correlates with the extent of keratopathy in the anterior layer, whereas chronic chang

64 The grade of keratopathy in the ocular lubricant group increased sign

65 ery in the treatment of pseudophakic bullous keratopathy in the presence of sf-IOL and if-IOL can suc

66 the first report of pressure-induced stromal keratopathy in this context.

67 Pathogenesis of diabetic keratopathy involves multiple tissues and/or cell types

68 d with Fuchs' endothelial dystrophy, bullous keratopathy, iridocorneal endothelial syndrome or a fail

69 Early-stage aniridic keratopathy is characterized by the development of focal

70 associated with nonocular surgery, but toxic keratopathy is rare.

71 The clinical presentation of exposure keratopathy is reviewed with tips for recognition for th

72 defects in this series included neurotrophic keratopathy, lattice and Avellino dystrophy, Stevens-Joh

73 Pseudophakic bullous keratopathy manifests an abnormal corneal ocular surface

74 n line, Acanthamoeba keratitis, mucus plaque keratopathy, medication-related keratopathy, or limbal s

75 elial corneal dystrophy (n = 28) and bullous keratopathy (n = 11).

76 ft failure (n = 3), and pseudophakic bullous keratopathy (n = 2).

77 = 127; 90%) followed by pseudophakic bullous keratopathy (n = 4; 4%) and regrafts (n = 9; 6.4%).

78 Neurotrophic keratopathy (NK) is a corneal degeneration associated wi

79 SS) aqueous tear deficiency, or neurotrophic keratopathy (NK), and 17 asymptomatic control subjects w

80 dophakic bullous keratopathy/aphakic bullous keratopathy (North America), and keratitis (Asia).

81 Exposure keratopathy occurred after severe periorbital thermal in

82 mucus plaque keratopathy, medication-related keratopathy, or limbal stem cell deficiency characterize

83 penetrating grafts for pseudophakic bullous keratopathy (P <0.001).

84 o had Fuchs dystrophy or pseudophakic bullus keratopathy (PBK) and underwent DSAEK or combined DSAEK

85 ndothelial dystrophy or pseudophakic bullous keratopathy (PBK) at a single institution.

86 FED and pseudophakic bullous keratopathy (PBK) corneal buttons were removed during tr

87 mal autopsy corneas and pseudophakic bullous keratopathy (PBK) corneas.

88 strophy and 9 eyes with pseudophakic bullous keratopathy (PBK) that underwent DSAEK, and 17 eyes with

89 helial dystrophy (FED), pseudophakic bullous keratopathy (PBK), infection and other indications.

90 helial dystrophy (FED), pseudophakic bullous keratopathy (PBK), or keratoconus who had undergone a pe

91 cemet membrane disorder), but not in bullous keratopathy (primarily an endothelial depletion).

92 ongest in patients with pseudophakic bullous keratopathy (r = -0.62 [P = .01]).

93 n of the corneal surface in aniridia-related keratopathy relates to both a deficiency within the limb

94 ed to advancing age and caused a progressive keratopathy, resulting in a dense vascularized corneal p

95 ed by 88 (21%) and mild superficial punctate keratopathy seen in 7 (1.7%).

96 uperficial cell layers were lower in bullous keratopathy specimens (1.6 vs. 2.0; P < 0.0001) than in

97 ent in sections was increased in the bullous keratopathy specimens compared with controls (0.36 vs. 0

98 ith antibodies to MUC16 was lower in bullous keratopathy specimens than in controls (0.5 vs. 1.2; P <

99 mer test I, presence of superficial punctate keratopathy (SPK), LG volume, and molecular analysis of

100 coexisting potential causes for dendritiform keratopathy, such as prior herpes simplex keratitis, var

101 from the literature regarding central toxic keratopathy syndrome (CTK).

102 ning polyquaternium-1 may cause dendritiform keratopathy that may be confused with infections of the

103 In eyes with pseudophakic bullous keratopathy, the EndoGlide group had a superior graft su

104 antiseptic solutions may cause severe toxic keratopathy; this possibility should be considered in or

105 dothelial dystrophy and pseudophakic bullous keratopathy undergoing DSAEK surgery were included and r

106 ed in a pre-clinical rabbit model of bullous keratopathy using a tissue-engineered endothelial kerato

107 ated with them, such as pseudophakic bullous keratopathy, uveitis-glaucoma-hyphema syndrome, and chro

108 EK failure in pseudophakic bullous keratopathy was associated with center experience of few

109 duction of layers expressing MUC1 in bullous keratopathy was not statistically significant.

110 hthalmic examination, significant healing of keratopathy was noted in the lens group (p = 0.02 and 0.

111 for Fuchs' dystrophy or pseudophakic bullous keratopathy was poorer than survival of penetrating graf

112 To understand the cause of diabetic keratopathy, we investigated innervation and its correla

113 ocular surface protection, or resolution of keratopathy) were achieved in all but 2 of these subject

114 ngle, together with pressure-induced stromal keratopathy with a fluid interface between the corneal s

115 nd Afghanistan and were treated for exposure keratopathy with the BOSP, a Food and Drug Administratio

116 in the lubrication group developed exposure keratopathy, with a summary odds ratio of 0.208 (95% con