ライフサイエンスコーパス: keratosis

1 of this study are not limited to seborrheic keratosis.

2 racil (5-FU) as an immunotherapy for actinic keratosis.

3 t of the precancerous skin condition actinic keratosis.

4 itaria in addition to punctuate palmoplantar keratosis.

5 inoma (0 vs. 31 [26.1%]; P < .001), verrucal keratosis (0 vs. 79 [66.4%]; P < .001), and Grover disea

6 sis, followed by pterygium (36%) and actinic keratosis (19%).

7 carcinoma, 39 (8.5%) were lichen planus-like keratosis, 21 (4.6%) were melanomas, and 4 (0.9%) were n

8 4 [38.9%], respectively [P = .95]), verrucal keratosis (79 [66.4%] and 26 [72.2%], respectively [P =

9 --changes that resemble hyperplastic actinic keratosis, a commonly observed human precancerous epithe

10 ich is approved for the treatment of actinic keratosis, a precancerous skin condition.

11 in, PTEN levels are reduced in human actinic keratosis, a precancerous skin lesion caused by solar UV

12 0, 2013, and included the search terms solar keratosis, actinic keratosis, photodynamic therapy, and

13 Actinic keratosis (AK) is a skin growth induced by UV light expo

14 ime, 6 months [range, 0-18 months]), actinic keratosis (AK) of the same sun-exposed skin area in the

15 lignancies, ranging from a precursor actinic keratosis (AK) to squamous cell carcinoma (SCC) in situ

16 it and harm associated with treating actinic keratosis (AK) with the immune response modifier imiquim

17 Actinic keratosis (AK), a skin growth induced by ultraviolet lig

18 a distinct precancerous lesion, the actinic keratosis (AK).

19 us cell carcinoma (SCC; n = 28), and actinic keratosis [AK (a precursor to SCC); n = 38].

20 Keratosis also affects other sites such as elbows and kn

21 ly, we evaluated the Raman signal in actinic keratosis and basal cell carcinoma lesions and perilesio

22 es of epidermal carcinogenesis; late actinic keratosis and cSCC.

23 PV viral replication is associated with skin keratosis and cutaneous squamous cell carcinoma.

24 Epidermodysplasia verruciformis keratosis and in some cases actinic keratoses demonstrat

25 in PTEN levels in human premalignant actinic keratosis and malignant SCCs, supporting a key role for

26 The mice exhibited palmoplantar keratosis and progressive alopecia, leading to alopecia

27 expression is up-regulated in human actinic keratosis and squamous cell carcinoma.

28 nd show promise for the treatment of actinic keratosis and squamous cell carcinoma.

29 , XPC p53 mutant mice have more severe solar keratosis and suffer accelerated skin cancer compared wi

30 lesions resulting from sun exposure (actinic keratosis), and individual Neandertal alleles were signi

31 nt of a precancerous skin condition (actinic keratosis), and phorbol derivatives such as resiniferato

32 biopsies from 40 healthy donors, 13 actinic keratosis, and 74 cSCC patients.

33 c dysfunction or fibroadiposis, palmoplantar keratosis, and alopecia, resembling the human cardiocuta

34 ectious skin diseases such as genital warts, keratosis, and basal cell carcinoma.

35 ferative diseases such as psoriasis, actinic keratosis, and certain neoplasias.

36 Basal cell carcinoma, solar keratosis, and colorectal cancer were investigated as ne

37 ures include oral leukokeratosis, follicular keratosis, and cysts (steatocysts and pilosebaceous cyst

38 ac arrhythmias and dysfunction, palmoplanter keratosis, and hair abnormalities (cardiocutaneous syndr

39 , which results in skin thinning, wrinkling, keratosis, and malignancy.

40 ronan inhibits basal cell carcinoma, actinic keratosis, and murine colon-26 growth in vivo.

41 or eradicating basal cell carcinoma, actinic keratosis, and squamous cell carcinoma (SCC) in situ by

42 for the treatment of genital warts, actinic keratosis, and superficial basal cell carcinoma.

43 entrations in basal cell carcinomas, actinic keratosis, and their perilesional skin demonstrate a sig

44 osteoarthritis, osteoporosis, and seborrheic keratosis are strongly associated with aging, implying a

45 -induced skin lesions, in particular actinic keratosis, are generally considered as premalignant skin

46 eg, seborrheic keratosis, lichen planus-like keratosis, basal cell carcinomas) misclassified as melan

47 1.21, 95% CI 1.17-1.25, p < 0.001, for solar keratosis), but there was no increased risk for colorect

48 to treat (NNT) for one patient to have their keratosis completely cleared after 12-16 weeks was 2.2 (

49 any cancer-related end points or in actinic keratosis counts.

50 plus 5-FU for the field treatment of actinic keratosis in a randomized, double-blind clinical trial i

51 PC1 could potentially contribute to abnormal keratosis in DD.

52 y of a new topical field therapy for actinic keratosis, ingenol mebutate gel (0.015% for face and sca

53 Actinic keratosis is a common precursor to sun-related squamous-

54 Actinic keratosis is a precursor to cutaneous squamous cell carc

55 : Normal epidermis, solar elastosis, actinic keratosis KIN1-2, advanced actinic keratosis KIN3 and we

56 , actinic keratosis KIN1-2, advanced actinic keratosis KIN3 and well-differentiated cSCC.

57 eeth adjacent to the subject's unilateral ST keratosis lesion) to NST-site teeth (contralateral corre

58 Teeth present, ST keratosis lesion, plaque and gingival index, probing dep

59 users, with a unilateral mandibular oral ST keratosis lesion, were recruited.

60 a and nonmelanocytic lesions (eg, seborrheic keratosis, lichen planus-like keratosis, basal cell carc

61 g palmoplantar carcinoma (MSPC) and familial keratosis lichenoides chronica (FKLC).

62 Seborrheic keratosis-like melanomas can be dermoscopically challeng

63 Persons who were diagnosed with keratosis, melanosis, Bowen's disease, or squamous cell

64 Nagashima-type palmoplantar keratosis (NPPK) is an autosomal recessive skin disorder

65 ly in invasive SCC nests, but not in actinic keratosis or in situ SCC, compared with normal epidermis

66 Some patients have only palmoplantar keratosis or periodontitis, and in rare individuals the

67 1.7-2.8), and those with a history of solar keratosis (OR, 1.3; 95% CI, 1.1-1.6).

68 The presence of wild type p53 (seborrheic keratosis) or mutant p53 (cutaneous squamous cell carcin

69 ught clinically to be pingueculitis, actinic keratosis, or ocular surface squamous neoplasia (OSSN),

70 r, dermatophytosis, acne rosacea, seborrheic keratosis, or warts; 74.1% of the subjects responded to

71 Papillon-Lefevre syndrome, or keratosis palmoplantaris with periodontopathia (PLS, MIM

72 ed the search terms solar keratosis, actinic keratosis, photodynamic therapy, and photochemotherapy.

73 Keratosis pilaris (KP) is a common skin disorder of foll

74 Although keratosis pilaris (KP) is common, its etiopathogenesis r

75 inearity and in some cases fine-scale and/or keratosis pilaris, whereas homozygotes or compound heter

76 rised by dry skin, palmar hyperlinearity and keratosis pilaris.

77 he arms and legs, palmar hyperlinearity, and keratosis pilaris.

78 Over 80% had actinic keratosis present, with approximately 30% having 5 or mo

79 Melanomas that clinically mimic seborrheic keratosis (SK) can delay diagnosis and adequate treatmen

80 ified as malignant melanoma (MM), seborrheic keratosis (SK), and benign nevi by a consultant dermatol

81 roliferative skin diseases including actinic keratosis, squamous and basal cell carcinoma as well as

82 eel et al. have demonstrated that seborrheic keratosis, the most common of all skin tumors, is depend

83 Evaluation of actinic keratosis therapy depends on reliable measures of the le

84 epidermis, solar elastosis and early actinic keratosis to the 'late' stages of epidermal carcinogenes

85 have limited the efficacy of current actinic keratosis treatments.

86 Palmoplantar keratosis, varying from mild psoriasiform scaly skin to

87 xpression of CD200 in normal skin vs actinic keratosis vs SCC in situ vs invasive SCC.

88 alysis, there was strong evidence that solar keratosis was associated with future PDE5 inhibitor use

89 Actinic keratosis was the most common diagnosis associated with