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1 esponds to dietary manipulation such as the 'ketogenic diet'.
2 nd glycolysis), valproic acid (VPA), and the ketogenic diet.
3 cover possible therapeutic mechanisms of the ketogenic diet.
4 est in alternative therapies, especially the ketogenic diet.
5  mice consuming a high-fat, low-carbohydrate ketogenic diet.
6 ic action of a high fat and low carbohydrate ketogenic diet.
7 sant effect of the medium chain triglyceride ketogenic diet.
8 en fasted or fed a high-fat low-carbohydrate ketogenic diet.
9 nt antitumor mechanisms were launched by the ketogenic diet.
10 PHOS deficiency can be bypassed by feeding a ketogenic diet.
11 -intensity exercise, or the low-carbohydrate ketogenic diet.
12 to respond appropriately to consumption of a ketogenic diet.
13 tely reversed in GF mice by consumption of a ketogenic diet.
14  after C75 injection in rats maintained on a ketogenic diet.
15 LUT1-DS) who objected to or did not tolerate ketogenic diets.
16 olic and physiologic alterations provoked by ketogenic diets.
17  were 3-5 times those reported with high-fat ketogenic diets.
18  as the modified Atkins diet) other than the ketogenic diet, (2) the relatively frequent occurrence (
19 nged deprivation of food or consumption of a ketogenic diet activates the IRE1alpha-XBP1 pathway in m
20                                          The ketogenic diet also may facilitate mechanisms by which t
21 d mortality, whereas carbohydrate-restricted ketogenic diets ameliorate T2D and suppress breast cance
22 ldren were available for analysis: 54 on the ketogenic diet and 49 controls.
23             73 children were assigned to the ketogenic diet and 72 children to the control group.
24 ch for a 4-wk period-an LC (4% carbohydrate) ketogenic diet and an MC (35% carbohydrate) diet-randomi
25             Dietary therapies, including the ketogenic diet and its variations, have been available f
26 lating FGF21 in response to starvation and a ketogenic diet and that FGF21 is required for behavioral
27 he epigenome gives us an appreciation of the ketogenic diet and the potential for bioenergetic therap
28  expression were observed in mice that ate a ketogenic diet and when fasting, which suggests that FGF
29 gamma coactivator-1alpha pathway activation, ketogenic diets and other new strategies is required.
30 of GLUT1 deficiency has important treatment (ketogenic diet) and genetic counseling implications.
31 tabolic factors such as caloric restriction, ketogenic diet, and hyperglycemia influence the inflamma
32                             In addition, the ketogenic diet appears to favor the synthesis of glutami
33                                              Ketogenic diets are also now being increasingly studied
34                                              Ketogenic diets are very efficient to control epilepsy a
35 usions, general anesthesia, hypothermia, and ketogenic diet as treatment for uncontrolled status epil
36                            In vivo, BHB or a ketogenic diet attenuates caspase-1 activation and IL-1b
37                       A commonly used rodent ketogenic diet (Bio-Serv F3666) that is very high in fat
38 , not only as the mechanism of action of the ketogenic diet, but also as a potential target of future
39  how the well-recognized clinical effects of ketogenic diets can alter human behavior via the same ce
40 profiling demonstrates that maintenance on a ketogenic diet causes a 25% reduction of myocardial (13)
41                                            A ketogenic diet consisting of a high ratio of fat to carb
42 e, we have explored whether treatment with a ketogenic diet could lead to a similar rescue through in
43                                              Ketogenic diet, diazepam, valproate, potassium bromide a
44 Children were randomly assigned to receive a ketogenic diet, either immediately or after a 3-month de
45 ermia, magnesium, pyridoxine, immunotherapy, ketogenic diet, emergency neurosurgery, electroconvulsiv
46 he mechanisms through which low carbohydrate ketogenic diets exert their ameliorative effects still r
47                            Weaning mice to a ketogenic diet extends viability to 8.5 wk.
48 ncentrations are higher within the hearts of ketogenic diet-fed mice challenged with ketones compared
49 mpared with chow-fed C57BL/6 mice, fasted or ketogenic diet-fed mice displayed increased hepatic TG c
50 s induced by an overnight fast or short term ketogenic diet feeding.
51 esions in Eker rats (Tsc2+/-) subjected to a ketogenic diet for 4, 6 and 8 months.
52 by parents of some children treated with the ketogenic diet for epilepsy control, although this side
53                                   The use of ketogenic diets for weight loss is not warranted.
54                                          The ketogenic diet had no effect in these models.
55                                            A ketogenic diet has also been found to induce mitochondri
56                                          The ketogenic diet has been widely and successfully used to
57 creased availability of anticonvulsants, the ketogenic diet has re-emerged as a therapeutic option.
58  therapy for pediatric epilepsy known as the ketogenic diet has seen a revival in its clinical use du
59 n of the diet (the medium-chain triglyceride ketogenic diet), have been shown to directly inhibit AMP
60   High-fat, low-carbohydrate diets, known as ketogenic diets, have been used as a non-pharmacological
61 ion, pyridoxine, steroids and immunotherapy, ketogenic diet, hypothermia, emergency resective neurosu
62 f this study was to test the efficacy of the ketogenic diet in a randomised controlled trial.
63                       Here, we report that a ketogenic diet in Kmt2d(+/betaGeo) mice modulates H3ac a
64 onsible for the antiepileptic effects of the ketogenic diet in mice.
65 ning could in part explain the effect of the ketogenic diet in reducing epileptic seizures.
66 es that several mechanisms may exist for the ketogenic diet, including disruption of glutamatergic sy
67           Ketogenic conditions-starvation or ketogenic diet-increase K8(+/+) HMGCS2, whereas this res
68                                Strikingly, a ketogenic diet increased lysine acetylation in Cpt2M(-/-
69   Here we focus on a novel hypothesis that a ketogenic diet-induced change in energy metabolism incre
70 tion of a high fat, low carbohydrate (HF-LC) ketogenic diet induces hepatic fibroblast growth factor
71                                          The ketogenic diet is a high-fat, adequate protein, low carb
72                The medium chain triglyceride ketogenic diet is an established treatment for drug-resi
73 ingly, the use of therapeutic hypothermia or ketogenic diet is described as a strategy for super-refr
74                                            A ketogenic diet is used as anti-seizure therapy i.a. in t
75 er 80 years ago with the introduction of the ketogenic diet, is required.
76        The full anticonvulsant effect of the ketogenic diet (KD) can require weeks to develop in rats
77                  We asked whether a high-fat ketogenic diet (KD) increases UCP levels and activity in
78                                            A ketogenic diet (KD) is a high-fat, low-carbohydrate meta
79                                          The ketogenic diet (KD) is a high-fat, low-carbohydrate, and
80 gated whether an isocaloric low-carbohydrate ketogenic diet (KD) is associated with changes in EE, re
81 g or treatment with the high-fat antiseizure ketogenic diet (KD).
82  increases with fasting and consumption of a ketogenic diet (KD).
83                   High-fat, low-carbohydrate ketogenic diets (KDs) have shown beneficial effects in m
84                               Second, common ketogenic diets lead to a therapeutically counterintuiti
85 flammatory effects of caloric restriction or ketogenic diets may be linked to BHB-mediated inhibition
86 d be suppressed by a carbohydrate-restricted ketogenic diet, MEDICA treatment suppressed tumor growth
87 ll now turn to dietary therapies such as the ketogenic diet, medium-chain triglyceride diet, modified
88                The mechanisms underlying the ketogenic diet might also have roles in other disorders,
89 asting or by maintaining a low-carbohydrate (ketogenic) diet might reduce epileptic seizures and offe
90 o mediate the stimulatory effect of an HF-LC ketogenic diet on FGF21 expression.
91  information is available on the effect of a ketogenic diet on plasma lipoproteins in children with d
92  liver AQP9 levels were observed in rats fed ketogenic diets or high-protein diets, but AQP9 levels w
93 and had not been treated previously with the ketogenic diet participated in a randomised controlled t
94 ch as starvation, diabetic ketoacidosis, and ketogenic diets, play a potentially important role in re
95                                          The ketogenic diet produced good seizure control.
96                                   A high-fat ketogenic diet produced significant increases in the ath
97 bgroup of the 371 patients accepted into the ketogenic diet program between 1994 and 2001.
98                       Prolonged feeding of a ketogenic diet promotes the growth of renal tumors by re
99 e short term, high-protein, low-carbohydrate ketogenic diets reduce hunger and lower food intake sign
100                    These data suggest that a ketogenic diet-related bleeding tendency occurs in about
101             Anticonvulsant mechanisms of the ketogenic diet remain incompletely understood.
102                           A low-carbohydrate ketogenic diet remains one of the most effective (but my
103             All three patients who tried the ketogenic diet responded well to this treatment, and fut
104                    At 6 months, the high-fat ketogenic diet significantly increased the mean plasma l
105              Additional animals received the ketogenic diet (starting at 20 or 60 d old).
106  consideration of dietary choline content in ketogenic diet studies in rodents to limit hepatic mitoc
107 Analysing medium-chain fatty acids in future ketogenic diet studies will provide further insights int
108           The results from this trial of the ketogenic diet support its use in children with treatmen
109  as the perceived absolute requirement for a ketogenic diet, the assumed lack of structural brain def
110 R in mice suppressed the ability of an HF-LC ketogenic diet to induce hepatic FGF21 gene expression.
111          Depletion of glucose levels using a ketogenic diet to lower lactate production by glycolytic
112 mised cells points to the potential use of a ketogenic diet to treat patients with heteroplasmic mtDN
113 (24 h of fasting and a very low carbohydrate ketogenic diet) to demonstrate that heart muscle engages
114 y pharmacological stimulation of PDH or by a ketogenic diet, two treatments used for PDH deficiencies
115                                        A 4:1 ketogenic diet was maintained for 15 mo, and whole-body
116                        Only a decade ago the ketogenic diet was seen as a last resort; however, it ha
117 bohydrates are restricted in patients on the ketogenic diet, we evaluated the effects of limiting car
118 g opinions about the anticancer actions of a ketogenic diet, we have shown that this anti-seizure the
119 iciency on the pleiotropic effects of rodent ketogenic diets, we studied four custom diets that diffe
120        In 2009, two controlled trials of the ketogenic diet were published, as well as an Internation
121 and adults with G1D who were not receiving a ketogenic diet were selected on a first-come, first-enro
122 duals with epilepsy benefit from consuming a ketogenic diet, which is similar to the more commonly kn
123 at liver were dramatically up-regulated by a ketogenic diet, which lowered HNE basal level.
124 tant adjunct to pharmacologic therapy is the ketogenic diet, which often improves seizure control, ev
125  compared with littermate control mice fed a ketogenic diet, yet it did not improve cardiac hypertrop

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