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1 the conversion of the glutamic acid to alpha-ketoglutaric acid.
2 es of the receptor with glutaric acid, alpha-ketoglutaric acid, adipic acid, pimelic acid, suberic ac
3 acid, citric acid, isocitric acid, and alpha-ketoglutaric acid (all members of the citric acid cycle)
4 H and R132C mutant proteins can reduce alpha-ketoglutaric acid (alpha-KG) to d-2-hydroxyglutaric acid
5 yeast extract medium supplemented with alpha-ketoglutaric acid (BCYEalpha) were compared to those of
6 ppuric acid, malic acid, fumaric acid, and 2-ketoglutaric acid between the sedentary and fatigued mic
8 ted ptaA mutant selectively lacked the alpha-ketoglutaric acid form of pyoverdine, and recombinant Pt
10 uvic acid, acetoin, methylglyoxal, and alpha-ketoglutaric acid in wine with only a small addition of
11 e the reaction by reducing the product alpha-ketoglutaric acid is explained by the very high reductio
12 tion of the conversion of glutamate to alpha-ketoglutaric acid prevented the production of 2-hydroxyg
13 ntification of eight alpha-keto acids (alpha-ketoglutaric acid, pyruvic acid, 4-hydroxyphenylpyruvic
15 id; (S)-alpha-hydroxyglutaric acid and alpha-ketoglutaric acid; (S)-lactic acid and pyruvic acid; and
16 alyzed the NADH-dependent reduction of alpha-ketoglutaric acid to (S)-hydroxyglutaric acid, a compone
17 tant enzymes catalyze the reduction of alpha-ketoglutaric acid to d-2-hydroxyglutaric acid (D2HG), le
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