ライフサイエンスコーパス: ketoprofen

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1 inflammatory drugs: ibuprofen, naproxen, and ketoprofen.

2 profen, oxaprozin, fenoprofen, naproxen, and ketoprofen.

3 5700 days for carbamazepine to <1-2 days for ketoprofen.

4 after IH-1 by systemic NSAID administration (ketoprofen; 55 +/- 9%; p < 0.001) or spinal p38 MAP kina

5 s been applied to the rapid synthesis of (S)-ketoprofen, a commercially successful oral and topical a

6 ed, whereas the recoveries for acidic drugs (ketoprofen and ibuprofen) were in the range of 76%-86%.

7 Pain-related acid effects were blocked by ketoprofen and morphine but not by norBNI.

8 The desired separation for ketoprofen and papaverine was established based on a sin

9 artificial analytes and experimental data of ketoprofen and papaverine were used to test the proposed

10 sics (the nonsteroidal antiinflammatory drug ketoprofen and the mu-opioid agonist morphine) or by the

11 ry drugs) naproxen, ibuprofen, flurbiprofen, ketoprofen, and fenoprofen.

12 93 effects were blocked by norBNI but not by ketoprofen, and were only attenuated by morphine.

13 roimidazolone formation at Arg-410 inhibited ketoprofen binding and esterase activity; correspondingl

14 ution of CYP450 in further transformation of ketoprofen byproducts is also reported.

15 Five organic acids (sulindac, ibuprofen, ketoprofen, diclofenac, and norfloxacin) were infused in

16 nically used NSAIDs (indomethacin, sulindac, ketoprofen, ibuprofen, diclofenac, ketorolac, etc., cycl

17 orrespondingly, glycation in the presence of ketoprofen inhibited Arg-410 modification and loss of es

18 nti-inflammatory drugs, spantide II (SP) and ketoprofen (KP) on the skin permeation.

19 Spantide II (SP) and ketoprofen (KP) were used as model drugs for combined de

20 ntitation of the (R)- and (S)-enantiomers of ketoprofen (kt), a potent nonsteroidal, anti-inflammator

21 he pharmacokinetics and safety of the NSAID, ketoprofen (KTP), in gel formulations.

22 ic acid, diclofenac, gemfibrozil, ibuprofen, ketoprofen, naproxen, sulfamethoxazole, and sildenafil).

23 zation of intracellular transformation using ketoprofen showed that CYP450 is not the sole intracellu

24 with either orally or topically administered ketoprofen were generated using both pcSFC and LC as the

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