ライフサイエンスコーパス: ketosis

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1 s inhibiting fat synthesis during periods of ketosis.

2 diabetic ketoacidosis or hyperglycemia with ketosis.

3 of ChREBP to the cytoplasm during periods of ketosis.

4 lism by increasing gluconeogenesis (GNG) and ketosis.

5 ges associated with starvation, which create ketosis.

6 uction in beta-cell mass with progression to ketosis.

7 Higher HbA1c levels were correlated with ketosis.

8 and specificity of HbA1c as an indicator for ketosis.

9 less restrictive, therapy that also creates ketosis and can lower the number of seizures.

10 act on serum lipids without the accompanying ketosis and have the potential to provide a nutritionall

11 ing of 10% kcal protein also caused moderate ketosis and hepatic fat accumulation, which were again a

12 elation (r = 0.36, P < 0.05; n = 34) between ketosis and oxidative stress as well as between oxidativ

13 line restricted (~300 mg/kg) provokes robust ketosis and weight loss in mice, but through unknown mec

14 These diets induce a moderate level of ketosis and, in some studies, have been shown to improve

15 ic program, including glycolysis, lipolysis, ketosis, and nucleotide breakdown.

16 ve study consisted of 253 T2DM patients with ketosis at Shanghai 10th People's Hospital during a peri

17 with uDM, hyperglucagonemia is required for ketosis but not for increased HGP or hyperglycemia.

18 Under ketosis, C75 was also less efficient at stimulating mTOR

19 glucose production (HGP), hyperglycemia, and ketosis characteristic of uncontrolled insulin-deficient

20 t mice has hyperlipidemia, hypoglycemia, and ketosis exacerbated by fasting.

21 l body weight for height and maximal urinary ketosis for seizure control.

22 overnight fastings of 11 +/- 1 h and during ketosis-hyperglycemia events for the minors.

23 l process of defective energy production and ketosis in A(-)beta(+) KPD.

24 HbA1c is a potential screening tool for ketosis in patients with T2DM.

25 Ketosis in patients with type 2 diabetes mellitus (T2DM)

26 moglobin A1c (HbA1c) as a screening tool for ketosis in T2DM patients.

27 This study examined the hypothesis that ketosis increases the TNF-alpha secretion, both in a cel

28 We conclude that both hyperglycemia and ketosis independently cause reductions in cerebral high-

29 Ketosis is much more likely with HbA1c values at >/=10.1

30 y was undertaken to test the hypothesis that ketosis might increase lipid peroxidation and lower glut

31 the independent effects of hyperglycemia and ketosis on cerebral metabolism, blood flow, and water di

32 ere are few data on carbohydrate content and ketosis on motivation to eat and ad libitum intake.

33 Ketosis-prone atypical diabetes is mostly recorded in pe

34 Ketosis-prone diabetes (KPD) is a rare form of type 2 di

35 A(-)beta(+) ketosis-prone diabetes (KPD) is an emerging syndrome of

36 ying mechanisms for beta-cell dysfunction in ketosis-prone type 2 diabetes are not known; however, pr

37 group consisted of 221 T2DM patients without ketosis randomly selected from inpatients during the sam

38 a key biomarker for diagnosis of subclinical ketosis (SCK), and provides information on the health st

39 f adult mice led to lethal hyperglycemia and ketosis that were attributed to loss of beta cell functi

40 n is metabolized to glutamine and because in ketosis there is increased consumption of acetate, which

41 he response of the minors to the presence of ketosis was consistent but unique for each individual.

42 Body weight was measured daily, and ketosis was monitored by analysis of plasma and urine sa

43 ately 10 days their severe hyperglycemia and ketosis were corrected.

44 The LC diet induced ketosis with mean 3-hydroxybutyrate concentrations of 1.

45 ed serious adverse events (hyperglycaemia or ketosis without acidosis) resulting in hospital admissio

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