ライフサイエンスコーパス: kidney cell

1 f DNA replication in HEK293T human embryonic kidney cells.

2 cortex, hippocampus, and cultured embryonic kidney cells.

3 of FH loss in immortalized and primary mouse kidney cells.

4 vated simultaneously in large percentages of kidney cells.

5 lyl cyclase activity by edema toxin in human kidney cells.

6 wth in either immortalized or primary monkey kidney cells.

7 We validated this sensor in human embryonic kidney cells.

8 in vitro were transfected into baby hamster kidney cells.

9 ial cell-cell contacts in Madin-Darby canine kidney cells.

10 nced in CXCR7-transfected Madin-Darby canine kidney cells.

11 ential tropism for salivary gland cells over kidney cells.

12 r-suppressive potential in tumorigenic human kidney cells.

13 rom chickens and mice and Madin-Darby canine kidney cells.

14 ly degraded by proteasome in human embryonic kidney cells.

15 at the apical surface of Madin-Darby canine kidney cells.

16 nant in our preparation from human embryonic kidney cells.

17 in TLR2- or TLR4-transformed human embryonic kidney cells.

18 -sorting of E-cadherin in Madin-Darby canine kidney cells.

19 a characteristic cytopathic effect in feline kidney cells.

20 s in vivo and of transfected human embryonic kidney cells.

21 ) in induced inflammatory responses of human kidney cells.

22 well as hESC-CMs, but not in human embryonic kidney cells.

23 eral surface of polarized Madin-Darby canine kidney cells.

24 junctional actin ring in Madin-Darby canine kidney cells.

25 s lost in HGPRT-deficient Madin-Darby canine kidney cells.

26 eased ciliary PC2 expression levels in mouse kidney cells.

27 pid remodeling process in Madin-Darby canine kidney cells.

28 a synthetic gene circuit integrated in human kidney cells.

29 active oxygen species (ROS)/PKC signaling in kidney cells.

30 xpression at the gene transcription level in kidney cells.

31 surface of live polarized Madin-Darby canine kidney cells.

32 an epithelial monolayer, Madin-Darby canine kidney cells.

33 the expression of stably integrated genes in kidney cells.

34 cell contact formation in Madin-Darby canine kidney cells.

35 mal delivery in polarized Madin-Darby canine kidney cells.

36 and S202A) D(1) receptors in human embryonic kidney cells.

37 located on the plasma membrane of liver and kidney cells.

38 nd at apical junctions in Madin-Darby canine kidney cells.

39 uration of ABCB4-I541F in Madin-Darby canine kidney cells.

40 c cytoplasmic body of adenovirus-transformed kidney cells.

41 , inhibit ciliogenesis in Madin-Darby canine kidney cells.

42 rpin RNA (shRNA) plasmid-transfected opossum kidney cells.

43 ty in the apical domain in polarized opossum kidney cells.

44 tips of primary cilia in Madin-Darby canine kidney cells.

45 lizes with centrosomes in Madin-Darby canine kidney cells.

46 suggesting reduced oxygen consumption by the kidney cells.

47 spartate receptor-expressing human embryonic kidney cells.

48 ) and beta(2) subunits in Madin-Darby canine kidney cells.

49 e surface expression of CFTR on baby hamster kidney cells.

50 not have been studied using human embryonic kidney cells.

51 sma membrane localization in human embryonic kidney cells.

52 me PCR in BoHV-1 infected Madin-darby bovine kidney cells.

53 in cultured and primary VHL loss-of-function kidney cells.

54 ase I hypersensitivity sites (DHSs) in human kidney cells.

55 alance, particularly for mammalian brain and kidney cells.

56 obtain pure populations of specific types of kidney cells.

57 id cell line derived from rainbow trout head kidney cells.

58 ication machinery in HEK293T human embryonic kidney cells.

59 y of Sindbis virus particles in baby hamster kidney cells.

60 hil-attracting chemokines CXCL1 and CXCL5 in kidney cells.

61 ructures in combination with mouse embryonic kidney cells.

62 inst mammalian HepG2 cells and human primary kidney cells.

63 eys and can be cultivated in vitro in monkey kidney cells.

64 kidney, with much lower expression in other kidney cells.

65 y acquires properties of a sub-population of kidney cells.

66 odulation of cytokine production by resident kidney cells.

67 otein synthesis, uptake, and localization in kidney cells.

68 elial cell colonies using Madin-Darby canine kidney cells.

69 n green monkey kidney and Madin-Darby canine kidney cells.

70 ithin monolayers of MDCK (Madin Darby canine kidney) cells.

71 In opossum kidney cells, a clonal cell line in which the PTHR1 and

72 GRHL2 in tubulogenesis of Madin-Darby canine kidney cells, a process requiring transient, partial EMT

73 3D cell culture spheroids of primary murine kidney cells after exposure to CDK inhibitors.

74 When expressed in human embryonic kidney cells, all three new mutations resulted in a loss

75 knockdown of Mbnl1 in hepatoma and embryonic kidney cells altered the levels of exon 11 inclusion.

76 her, knockdown of NCX1 in Madin-Darby canine kidney cells alters epithelial morphology and characteri

77 2XB, P2XC, P2XD, and P2XE in human embryonic kidney cells and altered the ionic and proton environmen

78 in A549 cells as well as in human embryonic kidney cells and Chinese hamster ovary cells heterologou

79 t are capable of differentiation into mature kidney cells and have high potential for regenerative ki

80 ecreased transcriptional activity in patient kidney cells and impaired binding of the transcription f

81 t HSP90 client proteins in Pkd1(-/-) primary kidney cells and in vivo.

82 otein synthesis inhibition using Vero monkey kidney cells and inhibition of metabolic activity (reduc

83 sed the rat P2X7 receptor in human embryonic kidney cells and measured membrane currents before and a

84 curs primarily by killing of proximal tubule kidney cells and mechanosensory hair cells, though the m

85 AMP levels are increased in HNF-1beta mutant kidney cells and mice.

86 on two different cell lines: human embryonic kidney cells and mouse embryonic fibroblasts.

87 ophotonic assay performed in human embryonic kidney cells and Nicotiana benthamiana leaf cells.

88 ay in PKD, transactivated miR-21 promoter in kidney cells and promoted miR-21 expression in cystic ki

89 e recombinantly expressed in human embryonic kidney cells and purified by immobilized metal ion affin

90 ing of protein extracts from human embryonic kidney cells and rat organs reveals that regulatory subu

91 normalized the proliferation rate of primary kidney cells and significantly rescued the disease pheno

92 BK polyomavirus (BKPyV) infection in primary kidney cells and that the upregulated enzyme is active.

93 inylation techniques in both human embryonic kidney cells and the human microvascular endothelial cel

94 ds were allowed to adsorb on human embryonic kidney cells and then detached one by one.

95 esis using Tuba knockdown Madin-Darby canine kidney cells and tuba knockdown in zebrafish.

96 ally mutated TRP channels in human embryonic kidney cells and used calcium imaging or whole-cell patc

97 ee cell lines: parental HEK (human embryonic kidney) cells and transfected HEK cells that stably expr

98 Receptors were expressed in human embryonic kidney cells, and disulfide formation was assessed by ob

99 ction in both human salivary gland cells and kidney cells, and expressed viral DNA and Tag protein.

100 xpression systems, COS-7 cells, baby hamster kidney cells, and in VWF-deficient mice through hydrodyn

101 n a dual-luciferase assay in human embryonic kidney cells, and they strongly inhibit the infectivity

102 y delayed growth kinetics in primary porcine kidney cells, and they were significantly attenuated in

103 pharmacologically or genetically attenuated kidney cell apoptosis and tissue damage, preserving rena

104 available to diagnose the presence of HIV in kidney cells are complex, the rate of infection is certa

105 In human kidney cells, ARL15 regulated TRPM6-mediated currents.

106 ted with ABCB19 expressed in human embryonic kidney cells as measured by patch-clamp electrophysiolog

107 f cellular CEP290 in primary human and mouse kidney cells as well as in zebrafish embryos leads to en

108 observed when NCC was expressed in mammalian kidney cells, as the cotransporter was polyubiquitinated

109 glycine receptor-transfected human embryonic kidney cells at room temperature, and caused internaliza

110 f2 activators were tested in human embryonic kidney cells bearing the Swedish mutation of amyloid pre

111 g to contactin-1-transfected human embryonic kidney cells, binding to paranodes of murine teased fibr

112 ge gaps exist in the roles of these genes in kidney cell biology and renal diseases.

113 ation is present in normal mammary acini and kidney cells but absent in cancerous cells.

114 POL1) protein circulates and is localized in kidney cells, but the contribution of APOL1 location to

115 rent has been measured from primary cilia of kidney cells, but the responsible genes have not been id

116 orms, in the migration of Madin-Darby canine kidney cells by suppressing expression of their alpha su

117 study, we isolated primary cilia from mouse kidney cells by using a calcium-shock method and identif

118 The presence of HIV in kidney cells can manifest itself in multiple ways, rangi

119 vein endothelial cells, and human embryonic kidney cells (cell line HEK293).

120 otype 1 virus is severely inhibited in swine kidney cells compared to its translation in rhesus macaq

121 n of SGLT2 activity also occurred in opossum kidney cells cotransfected with SGLT2 and MAP17.

122 d, three-dimensional (3D) Madin-Darby canine kidney cell culture method.

123 In Madin-Darby canine kidney cell cultures overlaid with human or swine mucus,

124 genesis in Tuba knockdown Madin-Darby canine kidney cell cysts cultured in a collagen gel.

125 f mTORC2, was silenced in Madin-Darby canine kidney cell cysts grown in 3D cultures.

126 spindle misorientation in Madin-Darby canine kidney cell cysts.

127 term (5.5 days after surgery), Vhl-deficient kidney cells demonstrate both spindle misorientation and

128 tivity of DbpA and YAP in Madin-Darby canine kidney cells depleted either of ZO-1, or one of the rela

129 Madin-Darby canine kidney cell-derived and Vero cell-derived glycovariants

130 MAL depletion in Madin-Darby canine kidney cells did not affect, however, the apical sorting

131 ld-type proximal tubule cells and in opossum kidney cells, dopamine increased NHERF-1 phosphorylation

132 In Myo1c-depleted Madin-Darby canine kidney cells, E-cadherin localization was dis-organized

133 Using human embryonic kidney cells enabled the purification of the TBCD.ARL2.b

134 or formation by mammary epithelial cells and kidney cells engineered to express SV40 early region pro

135 ly higher activation of both human embryonic kidney cells engineered to express TLR2 (HEK-TLR2) and w

136 Expression of MT4-MMP in Madin-Darby canine kidney cells enhanced cell migration and invasion of Mat

137 Human kidney cells evidence increased EP4 and decreased Rap1GA

138 ats with streptozotocin-induced diabetes and kidney cells exposed to high glucose.

139 Here, Madin-Darby canine kidney cells expressing ABCB1-GFP were used as a model t

140 Madin-Darby canine kidney cells expressing connexin-32 (Cx32) and Cx43 were

141 brain, cultured neurons, and human embryonic kidney cells expressing GAD65, GAD67, alpha1-subunit of

142 Human embryonic kidney cells expressing P2X7 exhibited a more tumorigeni

143 brane currents in individual human embryonic kidney cells expressing rat P2X7 receptors.

144 Kidney cells expressing renin were genetically fate-mapp

145 ssing the receptor and in Madin-Darby canine kidney cells expressing the native receptor.

146 he basolateral surface of Madin-Darby canine kidney cells expressing the rat neonatal Fc receptor for

147 e-cell current recordings in human embryonic kidney cells expressing the wild-type or the mutant Kir2

148 1N1) were inoculated onto Madin-Darby canine kidney cells for virus isolation.

149 phosphorylated Smad3 and ERK1/2, in primary kidney cells from CDA1 knockout animals.

150 Compared with those from WT mice, primary kidney cells from Cep290-deficient mice exhibited supern

151 isease, emphasizing the importance of PC2 to kidney cell function.

152 During in vitro diabetes, human kidney cells had down-regulation of Tyro3 and Mer mRNA a

153 le galectins expressed in Madin-Darby canine kidney cells had no effect on p75 sorting, suggesting th

154 Mitochondrial dysfunction in kidney cells has been implicated in the pathogenesis of

155 g the actin cytoskeleton in human epithelial kidney cells (HEK 293) and rat vascular smooth muscle ce

156 strategy over an image data set of embryonic kidney cells (HEK 293T) from multiple experiments.

157 al melanoma cells (MEL 270), human embryonic kidney cells (HEK) and breast cancer cells (MCF7) we sho

158 n our setup, drug binding to human embryonic kidney cell (HEK293) homogenate was measured in a small-

159 Rings of human embryonic kidney cells (HEK293) and tracheal smooth muscle cells (

160 ium was detected in cultured human embryonic kidney cells (HEK293) expressing heterologous ZIP8-Ala39

161 expression of QKI mutants in human embryonic kidney cells (HEK293) significantly decreased the abunda

162 terminal mutant receptors in human embryonic kidney cells (HEK293) with respect to trafficking, stabi

163 from mitochondria in lysates human embryonic kidney cells HEK293T.

164 as not specific to the cell type (baby mouse kidney cells, hematopoietic) nor the apoptotic stimulus

165 n contrast to monolayered Madin-Darby canine kidney cells, hepatocytic epithelial cells, which typica

166 sed whole-cell recordings of human embryonic kidney cells heterologously expressing either wild-type

167 Human-pluripotent-stem-cell-derived kidney cells (hPSC-KCs) have important potential for dis

168 in situ in murine kidney slices and in 786-O kidney cells in culture as determined by reverse transcr

169 Upregulation of autophagy protected kidney cells in culture from oxidative stress and reduce

170 Kidney cells in HIV transgenic mice and HIV-infected pod

171 idney tumor cells give rise to nonneoplastic kidney cells in mice, proving that they have not undergo

172 g organotypic cultures of Madin-Darby canine kidney cells in reconstituted basement membrane, we show

173 as replenishable sources for replacement of kidney cells in the setting of human disease.

174 up-regulated inflammatory gene expression in kidney cells in vitro and when injected in vivo.

175 n the apical and basolateral side of Xenopus kidney cells increased both ENaC channel density and cha

176 les derived from transfected human embryonic kidney cells induced a significant neutrophil chemotacti

177 Knockdown of NCX1 in Madin-Darby canine kidney cells induced fibroblastic morphology, increased

178 DSTYK knockdown in human embryonic kidney cells inhibited FGF-stimulated phosphorylation of

179 Diabetes-induced kidney cell injury involves an increase in matrix protei

180 uces the malignant SN12C, but not benign HK2 kidney cell invasion.

181 Here we show that mTOR signalling in rat kidney cells is inhibited during initiation of autophagy

182 underwent additional testing against primary kidney cells isolated from human kidneys to better predi

183 alcium oxalate binding and toxicity in human kidney cells, it may provide a different therapeutic app

184 the lipidomes of the host Madin-Darby canine kidney cell, its apical membrane, and the IFV budding fr

185 Using primary kidney cells lacking a catalytic subunit of Cn (CnAalpha

186 sette transporter A1-expressing baby hamster kidney cells leads to formation of two populations of FC

187 r localization in MDCKII (Madin-Darby canine kidney (cell line)) cells and in mouse liver, and tested

188 R-based R2 relaxation measurements of monkey kidney cell line (CV1) fibroblasts that overexpress FHC,

189 loop deletion expressed in a human embryonic kidney cell line (HEK 293) revealed no effect of the fla

190 s well in the nontumorigenic human embryonic kidney cell line (HEK-293T), showing in some cases impor

191 CF7) and in a nontumorigenic human embryonic kidney cell line (HEK-293T).

192 y-based safety testing using human embryonic kidney cell line 293 cells expressing human ether-a-go-g

193 ology in Xenopus oocytes and human embryonic kidney cell line 293 cells in which we coexpressed rat C

194 ncer (TOV112D) and noncancer human embryonic kidney cell line 293 to 15.8 and 18.1 nM, respectively,

195 zation via endogenous M3R in human embryonic kidney cell line 293T (HEK293T) or mouse insulinoma (MIN

196 Utilizing the human embryonic kidney cell line 293T, it was possible to demonstrate th

197 ation and infectious viral burdens in a frog kidney cell line and in tadpoles.

198 n of human 293H cells, Crandall Renal Feline Kidney cell line and primary feline peripheral blood mon

199 show low cytotoxicity in the nontumorigenic kidney cell line BGM and therefore high selectivity fact

200 o be optically analyzed in CRL-2794, a human kidney cell line expressing an unstable green fluorescen

201 However, by utilizing the human embryonic kidney cell line HEK293T, it was possible to demonstrate

202 homeostasis of hMSH4 in the human embryonic kidney cell line HEK293T.

203 demonstrate that overexpression of OCT4 in a kidney cell line is sufficient for signal-dependent acti

204 Overexpression in a human kidney cell line showed that wild-type PCBD1 binds HNF1B

205 al receptor for FMDV, we transduced a bovine kidney cell line to stably express both the alphaV and b

206 from tubulin purified from a human embryonic kidney cell line with isoform composition characteristic

207 ype levels of adherence to a human embryonic kidney cell line, biofilm formation, and in vivo fitness

208 reactive to self-antigens and a lysate of a kidney cell line, hence revealing a pattern of polyreact

209 ted intracellular vesicle acidification in a kidney cell line, providing validation for the utility o

210 rated a stably transfected BHK (baby hamster kidney) cell line that expresses a moderate level of myc

211 e and 2 pM against HEK 293T (human embryonic kidney) cell line], and a set of valuable structure-acti

212 L, KB, BT-549, SK-OV-3) and two noncancerous kidney cell lines (LLC-PK1 and Vero).

213 esis, we generated stable Madin-Darby canine kidney cell lines depleted of both ZO-1 and -2.

214 ty studies were performed on human embryonic kidney cell lines expressing human P2X7R (HEK293-hP2X7R)

215 infections and syncytium formation in monkey kidney cell lines.

216 (FRET) in stably expressing human embryonic kidney cell lines.

217 romocytoma (PC12) and fibroblast (normal rat kidney) cell lines.

218 ecipitate in Lilly Laboratories cell porcine kidney cells (LLC-PK1) as well as in rat kidney medullae

219 on of NCX1 by KB-R7943 in Madin-Darby canine kidney cells, LLC-PK1, and human primary renal epithelia

220 lial cells (bEND.3), (ii) Madin Darby Canine Kidney Cells (MDCK-2), and mouse myoblast (C2C12) (all f

221 Lipidomic analysis of Madin-Darby canine kidney cell membranes and of the corresponding detergent

222 E3 closely associates with NHERF2 in opossum kidney cell microvilli.

223 this method to measure ECM accumulation in a kidney cell model, we demonstrated good agreement with e

224 Tension across Madin-Darby canine kidney cell monolayers was increased by a low level of u

225 nese Hamster Ovary cells and Human Embrionic Kidney cells on two polyelectrolytes that are widely use

226 ructures in combination with embryonic mouse kidney cells over a period of 18 d.

227 f selected compounds with Madin-Darby canine kidney cells over a period of 96 h revealed that they ex

228 y-derived virus had preferential tropism for kidney cells over salivary gland cells.

229 ylation arrays, we show that human embryonal kidney cells over-expressing WT1 acquire DNA methylation

230 D-Cav-1 mutant transduced in human embryonic kidney cells overexpressing eNOS and reduced Ca(2+)-indu

231 Expression of cyclin E1 in human embryonic kidney cells prevents Cdk5-mediated phosphorylation of K

232 insight into the role of FLCN in regulating kidney cell proliferation and facilitate the development

233 the function of wild-type FLCN in regulating kidney cell proliferation and, therefore, act as an onco

234 nd K508R missense mutations promote aberrant kidney cell proliferation leading to pathogenicity, we g

235 nd K508R missense mutations promote aberrant kidney cell proliferation, but to different degrees.

236 ypes observed in Lgr4KO intestines, impaired kidney cell proliferation, reduced epidermal thickness,

237 the role that V2R-stimulated ERK1/2 plays in kidney cell proliferation, this transactivation mechanis

238 ophils and 5-LOX-transfected human embryonic kidney cells, propofol attenuated the production of 5-LO

239 overexpression of viperin in human embryonic kidney cells reduces the intracellular rate of accumulat

240 1-stimulated collagen expression by cultured kidney cells requires integrin-dependent activation of f

241 Complementary in vitro studies in normal rat kidney cells showed that dh404 significantly upregulates

242 In human embryonic kidney cells stably expressing a Flag-tagged version of

243 Notably, Madin-Darby canine kidney cells stably expressing apically mistrafficked ER

244 When expressed in human embryonic kidney cells stably expressing Ca(v)2.3, beta(Sm) accele

245 Immunocytochemistry of Madin-Darby canine kidney cells stably expressing CPO showed localization t

246 In Madin-Darby canine kidney cells stably expressing epitope-tagged alphabetag

247 5 and basolateral VSVG in Madin-Darby canine kidney cells still undergo progressive sorting after the

248 linked to dysregulated signaling via PKC in kidney cells such as podocytes.

249 ed when ENaC was co-transfected with sgk1 in kidney cells, suggesting that ENaC brings sgk1 to the ce

250 , or the APOL1 G2 variant in human embryonic kidney cells (T-REx-293) using a tetracycline-mediated (

251 receptors were expressed in human embryonic kidney cells, taurine and AL34662, a non-specific 5-HT(2

252 s expressed recombinantly in human embryonic kidney cells; the effects of the receptor mutations were

253 ic growth of benign MDCK (Madin Darby Canine Kidney) cells through effects on the Rho-like GTPase cdc

254 bserve dynamic variations of human embryonic kidney cells, through a silicon substrate, in response t

255 we reduced the percentage of Pkd1-deficient kidney cells to 8%.

256 variants proliferated in Madin-Darby canine kidney cells to nearly the degree as WT PR8.

257 al preparations ranging from human embryonic kidney cells to neurons in culture, slices, and in vivo.

258 GF-4) and transfected them into baby hamster kidney cells together with normal alphav or alphaIIb.

259 tory sensory neurones and in human embryonic kidney cells, together with electrophysiological recordi

260 s in polarized epithelial Madin-Darby canine kidney cell transfectants.

261 results were confirmed in Madin-Darby canine kidney cells transfected with a reporter construct drive

262 alysis of RNA extracted from human embryonic kidney cells transfected with exon trapping constructs.

263 ed transport studies with Madin-Darby canine kidney cells transfected with human MDR1 gene (MDCK/MDR1

264 m a control subject, stained human embryonic kidney cells transfected with the TRPM1 gene, and Wester

265 Madin-Darby canine kidney cells transiently transfected with meprin alpha o

266 of PRDX2 is also observed in human embryonic kidney cells treated with TNF-alpha.

267 ional activation of proinflammatory genes in kidney cells treated with TNFalpha or infected by HIV.

268 xpansion, and differentiation of appropriate kidney cell types and the integration of these cells int

269 single-base resolution of Madin-Darby canine kidney cells undergoing EMT and translated the identifie

270 signaling in cilia of mouse fibroblasts and kidney cells upon chemical or mechanical stimulation wit

271 the global kinome of HEK293T human embryonic kidney cells upon treatment with methylglyoxal, a glycol

272 on) and nitric oxide-induced human embryonic kidney cell using 2 labeling reagents: the cysteine-reac

273 etion of luminal Ca(2+) in living normal rat kidney cells using cyclopiazonic acid (CPA) resulted in

274 dynamics directly in living human embryonic kidney cells using fluorescence fluctuation spectroscopy

275 regation domains, in live Madin-Darby canine kidney cells using spinning disk confocal microscopy.

276 ion of V-ATPase localization and activity in kidney cells via direct PKA-dependent phosphorylation of

277 e siRNA internalization into non-tumorigenic kidney cells was negligible with all fatty acyl-peptide

278 moregulation of the mouse Cln3 mRNA level in kidney cells was recently reported.

279 (1) subunits expressed in Madin-Darby canine kidney cells was used to assess beta(1)-beta(1) interact

280 term tracking of cultured Madin-Darby canine kidney cells we demonstrate that inhibition of cell divi

281 2R and the MC1R variants are present in head kidney cells, we hypothesized that MC2R activity is modu

282 fall protein in polarized Madin-Darby canine kidney cells, we show here that a cysteine-altering muta

283 rsor protein in vitro and in human embryonic kidney cells, we show that gamma-secretase is a very slo

284 tantly, using experiments in human embryonic kidney cells, we show that specific parameters of the sy

285 zation with GluN1-expressing human embryonic kidney cells were confirmed to be against the NMDAR.

286 Madin-Darby canine kidney cells were treated under isotonic or hypertonic c

287 o-go-related gene expressing human embryonic kidney cells were used as controls.

288 eral surface in polarized Madin-Darby canine kidney cells, whereas in double tyrosine mutant, it was

289 sents a novel function for these specialized kidney cells, which are best known for their role in mod

290 eight concatenated cDNAs in human embryonic kidney cells, which encoded three serially joined, epito

291 contrast, in primary cultures of human fetal kidney cells, which maintain WNT activation and more clo

292 Treatment of cultured embryonic kidney cells with HDACi recapitulated these gene express

293 es in cells with hyperactive mTORC1, such as kidney cells with mutations in the tumor suppressor gene

294 Madin-Darby canine kidney cells with Na,K-beta knockdown have reduced NCX1

295 Treatment of human kidney cells with nicotinic agonists, an NFkappaB inhibi

296 /2007 (H1N1) was grown in Madin-Darby canine kidney cells with or without escalating concentrations o

297 Human kidney cells with Polbeta knockout (KO) had higher endog

298 Surface NHE3 was reduced in opossum kidney cells with reduced expression of ClC-5, whereas t

299 processing, and activation of GC prodrugs by kidney cells would potentiate nephritis resolution, with

300 In kidney cells, Wt1 recruits Cbp and p300 as coactivators;