ライフサイエンスコーパス: kidney cortex

1 KRD11, and TNRC18 map to active enhancers in kidney cortex.

2 rtex and isolated CCDs from mouse and rabbit kidney cortex.

3 downstream events in mRNA translation in the kidney cortex.

4 d show concordant DNA methylation changes in kidney cortex.

5 oduct of ARID1A, than did the matched normal kidney cortex.

6 in G30 fetus, G45 liver, and adult liver and kidney cortex.

7 ho-tuberin and fibronectin expression in the kidney cortex.

8 iabetic rats had more apoptotic cells in the kidney cortex.

9 ortex was significantly reduced in OVE26 RKO kidney cortex.

10 antly lower in OVE26 RKO compared with OVE26 kidney cortex.

11 itochondrial Nox4 expression is increased in kidney cortex.

12 hat show expression changing with age in the kidney cortex.

13 n embryo day 14.5, in adult hepatocytes, and kidney cortex.

14 ons from noninfected LLC-PK(1) cells and rat kidney cortex.

15 NA isolated from LLC-PK1 cells, and from pig kidney cortex.

16 rcalated cells identified in sections of rat kidney cortex.

17 ipids has been recently purified from rabbit kidney cortex.

18 st protein liquid chromatography from rabbit kidney cortex.

19 alcium-independent PLA2 activity from rabbit kidney cortex.

20 antly lower levels of IR mRNA and protein in kidney cortex (49-56% of the WT) and medulla (32-47%) ho

21 Immunoblot analysis of rat kidney cortex and brain identified immunoreactive protei

22 oducts from RNA isolated from rat and rabbit kidney cortex and cerebellum and rabbit S(2) segments we

23 -salt diet led to lower levels of EGF in the kidney cortex and enhanced the expression and activity o

24 ssion in the streptozotocin-induced diabetic kidney cortex and glomeruli, concomitant with activation

25 argets under physiological conditions in the kidney cortex and in primary renal cell cultures.

26 expressed in the nephrogenic zone of newborn kidney cortex and in the medulla.

27 cinic acid ((99m)Tc-DMSA) accumulates in the kidney cortex and is widely used for imaging of the rena

28 ression of mRNA encoding SDF1 (or CXCL12) in kidney cortex and isolated CCDs from mouse and rabbit ki

29 ntitation of the drug and its metabolites in kidney cortex and liver tissues.

30 -1 versus -2 was 2- and 7-fold higher in the kidney cortex and liver, respectively, whereas expressio

31 expressed at sites of ADMA metabolism in the kidney cortex and liver, whereas EDRF/NO is regulated pr

32 brane-enriched fraction of brain, liver, and kidney cortex and medulla by radiochemical assay.

33 regionally based surviving fractions for the kidney cortex and medulla in terms of their concentratio

34 Expression microarray data from the kidney cortex and medulla, liver, lungs, and spleen were

35 cellular (EC) and copper/zinc (CuZn) SOD, in kidney cortex and medulla.

36 s as well as markers of oxidative stress, in kidney cortex and medulla.

37 cultured rat mesangial cells (MC) as well as kidney cortex and microdissected glomeruli were examined

38 brary and sequencing revealed that the human kidney cortex and neuronal beta(3) subunits were identic

39 te that cortactin is highly expressed in the kidney cortex and polarized epithelial cells, and is loc

40 -PCR revealed expression of Gi but not Go in kidney cortex and preglomerular vessels.

41 otide and amino acid composition, and rabbit kidney cortex and rabbit neuronal GABA(A) receptor beta(

42 he kidney PCR products revealed that the rat kidney cortex and rat neuronal GABA(A) receptor beta(2)

43 he kidney PCR products revealed that the rat kidney cortex and rat neuronal GABA(A) receptor beta(3)

44 ression of SLC26A7 remained unchanged in the kidney cortex and stomach in water deprivation, indicati

45 ract separate factors that correspond to the kidney cortex and the kidney pelvis.

46 protocol, 68 ablations were performed in the kidneys (cortex and medulla) and livers of 27 adult pigs

47 marked increase in non-heme iron content of kidney cortex, and a marked increase in the non-heme iro

48 chment of pathways involving NFkappaB in the kidney cortex, and a targeted data mining approach ident

49 pathways and reduces metabolism in the fetal kidney cortex, and ketamine blocks or ameliorates this r

50 al RNAs were isolated from both duodenum and kidney cortex, and the VDR and calbindin mRNA levels wer

51 duced DC numbers in the healthy and inflamed kidney cortex, and to a lesser degree in the kidney medu

52 At PTPN6/PHB2 cg19942083, methylation in kidney cortex associates with lower renal PTPN6 expressi

53 tribution was highly organ specific with the kidney cortex being the major target organ, followed by

54 nding protein in membranes obtained from the kidney cortex but not in membranes from oocytes.

55 lial cells from ADPKD cysts and normal human kidney cortex cells (HKC) were cultured, and cAMP levels

56 significant polyol pathway activation in the kidney cortex characterized by high levels of aldose red

57 Gene expression in fetal kidney cortex collected 24 h after the onset of hypoxia

58 protein expression was increased in diabetic kidney cortex compared with non-diabetic controls and wa

59 Immunoblot analysis of human kidney cortex demonstrated that hOAT1 is an 80- to 90-ki

60 liver, bladder, sublinguinal salivary gland, kidney cortex, dermis, and synovial membrane were comple

61 lternative splicing of the DNase I mRNA, rat kidney cortex DNA complementary to RNA library was scree

62 g sequences derived from the purified rabbit kidney cortex enzyme.

63 rsed the up-regulation of mRNA expression of kidney cortex fibrosis genes (CCN2, Col1a2, TGF-beta1, a

64 By immunoblotting, kidney cortex homogenate from patients treated with angi

65 cs and phosphoproteomics of freshly isolated kidney cortex identified either reduced expression or lo

66 Northern blot analysis of kidney cortex in both mice revealed nearly equivalent me

67 We observed GPR4 expression in the kidney cortex, in the outer and inner medulla, in isolat

68 g the plasma membrane of L2 cells and in the kidney cortex is expressed in glomerular and proximal tu

69 In the kidney cortex, kidney medulla, and lungs, the TH-associa

70 By screening a rat kidney cortex library with a CaT1 probe, we isolated a c

71 ere reduced selectively by 35% to 85% in the kidney cortex, liver, and MRVs 72 hours following the co

72 elaxation times were 21% longer (P <.05) for kidney cortex, liver, and spleen and T2 relaxation times

73 Angiotensinogen mRNA and protein levels in kidney cortex, measured by real-time reverse transcripta

74 e presence of rOCT1A mRNA transcripts in rat kidney cortex, medulla, and intestine.

75 Replenishment of the kidney cortex microvasculature requires 1-5 seconds comp

76 liver of rodents, but in both the liver and kidney cortex of humans and pigs; therefore, the pig was

77 MP, whereas the cAMP effect was abolished in kidney cortex of NHERF1(-/-) mice.

78 was isolated mitochondria obtained from the kidney cortex of rats treated with IFO or IFO + AGM.

79 In the kidney cortex of TGF-beta transgenic mice, the significa

80 Although studies in BB isolated from kidney cortex of wild type and NHERF1(-/-) mice have sho

81 ding of intravenously injected F-Dapa in the kidney cortexes of rats and wild-type and Sglt1-knockout

82 For kidney cortex, ovoid zones of 1.5 cm +/- 0.1 x 0.5 cm +/

83 In the kidney cortex, perturbation of homeostasis by a TLR7-dep

84 In kidney cortex, preparations consisting mainly of proxima

85 Primarily CX3CR1-dependent DCs in the kidney cortex processed antigen for the intrarenal stimu

86 s expression system, and the purified rabbit kidney cortex protein exhibit both CaIPLA2 and lysophosp

87 ound that they were enriched 4.5-fold in the kidney cortex relative to nonspecific control T cells 24

88 Immunofluorescence studies in human kidney cortex revealed that ATP6N1B localizes almost exc

89 lot analysis in cultured mesangial cells and kidney cortex revealed that Nox4 is present in crude mit

90 ate gel analysis of extracts from normal rat kidney cortex revealed the presence of a DNase with an a

91 analysis on 404 ccRCC tumors and 167 normal kidney cortex samples using publicly available databases

92 imes (mean +/- SD) at 3.0 T are reported for kidney cortex (T1, 1,142 msec +/- 154; T2, 76 msec +/- 7

93 spite a 50% lower AIF protein content in the kidney cortex, there was no loss of complex I activity o

94 tion of this PLA2 to homogeneity from rabbit kidney cortex through sequential column chromatography i

95 Conversely, synthesis of NAADP in kidney cortex was almost undetectable.

96 Microsomes from human kidney cortex were found to convert AA to a single major

97 ild type and NHERF1(-/-) ileum and wild type kidney cortex were inhibited by cAMP, whereas the cAMP e

98 2 transcripts are very abundant in liver and kidney cortex, whereas the expression is significantly l