ライフサイエンスコーパス: kidney dysfunction

1 200 mg/m(2), with dose reduction for severe kidney dysfunction.

2 of increased serum retinol in the context of kidney dysfunction.

3 o identify inflammatory processes related to kidney dysfunction.

4 atterning and neonatal death consistent with kidney dysfunction.

5 A miRNA panel was associated with human kidney dysfunction.

6 and venous thromboembolism in patients with kidney dysfunction.

7 and 85 (9.0%) had both retinopathy and early kidney dysfunction.

8 deposition but neither mesangial injury nor kidney dysfunction.

9 rdial edema, phenotypes typically induced by kidney dysfunction.

10 F508 homozygous patients do not present with kidney dysfunction.

11 ions in CFTR result in little or no apparent kidney dysfunction.

12 n E (apoE) might independently contribute to kidney dysfunction.

13 oporosis, 9.6% [95% CI, 8.0%-11.5%]), renal (kidney dysfunction, 5.0% [95% CI, 4.0%-6.3%]), and hemat

14 ut retain NK cells, showed similar levels of kidney dysfunction 65 days after transplantation (creati

15 fied renal failure and reported disorders of kidney dysfunction after adjustment for biological donor

16 ced MRA after a nondiagnostic ultrasound for kidney dysfunction after transplantation.

17 ccinylacetone which induces severe liver and kidney dysfunction along with mutagenic changes and hepa

18 sent in treated HIV infection contributes to kidney dysfunction among HIV-infected men receiving high

19 optosis in acute folate nephropathy and less kidney dysfunction and a lower mortality rate in cisplat

20 to cryptic antigens as novel accelerators of kidney dysfunction and acute or chronic allograft reject

21 iency of either PD-1 ligand also exacerbated kidney dysfunction and acute tubular necrosis after subt

22 tients with ESRD, cancer risk is affected by kidney dysfunction and by immunosuppression after transp

23 ain the association between mild to moderate kidney dysfunction and cardiovascular mortality.

24 Early kidney dysfunction and clinical pancreatitis were higher

25 inflammatory markers while decreasing early kidney dysfunction and clinical posttransplant pancreati

26 s a microvascular complication that leads to kidney dysfunction and ESRD, but the underlying mechanis

27 eruli within 4 days, leading to irreversible kidney dysfunction and failure to thrive.

28 Kidney dysfunction and high C-reactive protein (CRP) lev

29 e data indicate strong short-term effects of kidney dysfunction and immunosuppression on cancer incid

30 Kidney dysfunction and inflammation commonly accompany T

31 or a zoonotic disease known to induce severe kidney dysfunction and inflammation.

32 o offer a new pathway to noninvasively image kidney dysfunction and local injuries at the anatomical

33 Little is known about the association of kidney dysfunction and outcome in acute severe hypertens

34 ow that Kim-1-deficient mice had more severe kidney dysfunction and tissue damage after bilateral ren

35 systems as a noninvasive means of monitoring kidney dysfunction and treatment.

36 heir TCR repertoire, have significantly less kidney dysfunction and tubular injury after renal IRI co

37 need to understand how brain death leads to kidney dysfunction and, hence, how this can be prevented

38 sozymes and they exhibit low blood pressure, kidney dysfunctions, and male infertility.

39 Kidney dysfunction, as assessed by cystatin C, is associ

40 is, acute hypertension, vascular injury, and kidney dysfunction associated with pathophysiology drive

41 79 years) and the prevalence of preadmission kidney dysfunction (baseline eGFR <60 ml/min per 1.73 m(

42 olipids, Krebs and urea cycles, and revealed kidney dysfunction biomarkers.

43 e patients may have reversible components of kidney dysfunction, both HF and renal parameters improvi

44 e antiretroviral therapy, is associated with kidney dysfunction, but the magnitude of the effect and

45 Tenofovir exposure increased the risk of kidney dysfunction by 63% (HR, 1.63; 95% confidence inte

46 0/yr) was strongly associated with prevalent kidney dysfunction by cystatin C >1.29 g/dl (adjusted OR

47 In unadjusted analysis, kidney dysfunction by either measure was strongly associ

48 Kidney dysfunction causes a myriad of adverse influences

49 may partially explain their higher burden of kidney dysfunction compared with uninfected men.

50 jury markers, reduced apoptosis and improved kidney dysfunction, concomitant with mitigated histologi

51 ios (OR) for the association between TDF and kidney dysfunction (defined as eGFR <90 mL/min/1.73 m(2)

52 scores, incidence of jaw osteonecrosis, and kidney dysfunction did not differ significantly between

53 blood cells and schistocytes) and transplant kidney dysfunction during the first 2 weeks after transp

54 Finally, 47 of 131 patients experienced kidney dysfunction during the median 15-year follow-up p

55 HD), periventricular leukomalacia (PVL), and kidney dysfunction; Fer-1 inhibited lipid peroxidation,

56 plant or altered in response to detection of kidney dysfunction, histologic evidence of allograft dam

57 sociation between long-term TDF exposure and kidney dysfunction in a cohort of 1043 human immunodefic

58 vascular hemodynamics recovery, and limiting kidney dysfunction in a vasopressinergic-dependent manne

59 entional biomarkers in indicating hepatic or kidney dysfunction in different animal models.

60 Rather, kidney dysfunction in elderly African Americans seems mo

61 This study aimed to assess kidney dysfunction in general surgical patients and exam

62 (BUN) are the primary options for monitoring kidney dysfunction in humans.

63 osuppressant-related factors associated with kidney dysfunction in islet transplant recipients.

64 eNOS deficiency on BP, atherosclerosis, and kidney dysfunction in nnee mice.

65 g recognition of the incidence and impact of kidney dysfunction in recipients of nonrenal solid organ

66 e activation of AMPK to a potential risk for kidney dysfunction in the context of an HFD.

67 ent aly/aly (MAP3K14(aly/aly)) mice had less kidney dysfunction, inflammation, and apoptosis in acute

68 In conclusion, kidney dysfunction is associated with an increased odds

69 Kidney dysfunction is associated with bone loss, and pat

70 patients with ESRD, but whether less severe kidney dysfunction is associated with CAC is uncertain.

71 Whether kidney dysfunction is associated with coronary artery ca

72 Whether early kidney dysfunction is associated with small and large ar

73 The aim of this study was to examine whether kidney dysfunction is associated with the type of clinic

74 readily differentiate between the stages of kidney dysfunction is highly desired for improving our f

75 Kidney dysfunction is known to decrease life expectancy

76 The development of kidney dysfunction is one of the most important after li

77 Early kidney dysfunction is significantly associated with decr

78 taracts and glaucoma, mental retardation and kidney dysfunction, is caused by mutations in the OCRL g

79 injury (IRI) as assessed by tubular injury, kidney dysfunction, necrosis, apoptosis and inflammatory

80 Studies reporting health assessments, kidney dysfunction, neurological disorders and symptoms,

81 and many disease states, including liver and kidney dysfunction, neurological disorders, and cancer.

82 health care, but the effects of less severe kidney dysfunction on these outcomes are less well defin

83 s of retinopathy only, 250 (26.5%) had early kidney dysfunction only, and 85 (9.0%) had both retinopa

84 can ancestral group were not associated with kidney dysfunction or kidney disease progression.

85 uartile vs the remaining 3 quartiles), early kidney dysfunction (OR 1.56, 95% CI 1.06-2.28, per stand

86 the longitudinal, noninvasive monitoring of kidney dysfunction progression in preclinical research.

87 gold nanoparticles, can noninvasively detect kidney dysfunction, report on the dysfunctional stages,

88 ver, the prognostic biomarker used to report kidney dysfunction (serum creatinine concentration) has

89 ity), incidence of osteonecrosis of the jaw, kidney dysfunction, skeletal morbidity rate (mean number

90 0.80-1.18) and did not significantly vary by kidney dysfunction stage for either of these primary end

91 CD2AP is important in kidney dysfunction that is accompanied by inflammation.

92 C seems to identify a "preclinical" state of kidney dysfunction that is not detected with serum creat

93 l trials in patients with moderate-to-severe kidney dysfunction to further refine their optimal manag

94 Those with a history of kidney dysfunction, tobacco abuse, and higher ejection f

95 both severity and recovery of postoperative kidney dysfunction using the pattern of longitudinal cha

96 mild nonproliferative retinopathy) and early kidney dysfunction was defined as AER >/=7.5 mug/min.