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1 on of major Na(+) transporters all along the kidney tubule.
2 determinants of Na(+) reabsorption along the kidney tubule.
3 nsor, detecting changes in fluid flow in the kidney tubule.
4  cells induced the acquisition of functional kidney tubules.
5 nes PKD1 or PKD2 induces cyst formation from kidney tubules.
6  organ ducts such as the digestive tract and kidney tubules.
7 r that is characterized by cyst formation in kidney tubules.
8 efect in oriented cell division in precystic kidney tubules.
9 usters of interstitial cells surrounding the kidney tubules.
10 its differentiation of these precursors into kidney tubules.
11 ns at concentrations present in seawater and kidney tubules.
12   Histology revealed a subsequent absence of kidney tubules, an enlarged cardinal vein and expansion
13 potassium channel (ROMK) is expressed in the kidney tubule and critically regulates sodium and potass
14 all protein), is exclusively produced by the kidney tubule and has specific biochemical properties th
15 it active NaK transport by as much as 50% in kidney tubule and other tissues.
16 e considered tantamount to the injury of the kidney tubule and the epithelial cells thereof (AKI).
17 r plexiform layers of the retina, and in the kidney tubules and collecting ducts.
18 s in the developing skin, as well as dilated kidney tubules and ectatic Bowmans spaces.
19 es, tendon, and the connective tissue around kidney tubules and lung alveoli, which all contain fibri
20           The SUR2B is also expressed in rat kidney tubules and may combine with Kir.1 to form renal
21 c disease characterized by cyst formation in kidney tubules and other ductular epithelia.
22 in mice induced cilium elongation defects in kidney tubules and predisposed animals to cyst developme
23  EMT in epithelial cells from mammary gland, kidney tubules, and epidermis.
24 stinal tract, the thick straight segments of kidney tubules, and the marrow stromal cells in adults.
25 stimulating factor-1 (CSF-1) is expressed by kidney tubules at the onset of LN, increases with diseas
26 s angiotensin-converting enzyme (ACE) in the kidney tubules but not in other tissues.
27 nd on epithelial cells, such as those of the kidney tubule, but also on nonepithelial cells, such as
28 ssessed multiple large cysts in the proximal kidney tubules, but even in tensin null mice with normal
29 t establishes and maintains the structure of kidney tubules, but the role of this pathway in the path
30 mmals has been attributed to the blockage of kidney tubules by insoluble complexes of melamine with c
31 has been used to determine whether embryonic kidney tubules can be stimulated by cAMP to form cysts.
32                                              Kidney tubule cells (KTC) are targets of T lymphocyte in
33 kyrin and Fas was confirmed in vivo in mouse kidney tubule cells by coimmunoprecipitation and colocal
34  by fluorescence intensity in primary canine kidney tubule cells, Madin-Darby canine kidney cells, an
35 o paracrine profibrotic signaling in injured kidney tubule cells.
36 king complex is required for ciliogenesis in kidney tubule cells.
37 on the underlying proliferative potential of kidney tubule cells.
38 zed and secreted into the medium by cultured kidney tubules derived from cystic C57BL/6J-cpk mice.
39  Signaling from the primary cilium regulates kidney tubule development and cyst formation.
40 inate from normal and nondilated ADPKD human kidney tubules display normal ciliary expression of the
41 irectional cell growth, including defects in kidney tubule elongation that lead to formation of kidne
42                                    Along rat kidney tubules, endolyn is variously localized to the ap
43 ls of Kid1 mRNA correlate with maturation of kidney tubule epithelia in rat post-natal kidney develop
44                              Irradiating rat kidney tubule epithelial cells (NRK52E) with 1-20 Gy gam
45 -sensorial function of the primary cilium in kidney tubule epithelial cells and (ii) the distinct thr
46 lia, first cervical vertebra, thyroid gland, kidney tubules, esophagus, stomach, and intestines.
47 uding zebrafish posterior axis formation and kidney tubule formation.
48                        Moreover, PT cells in kidney tubules from TRACK mice exhibit increased genomic
49 t zebrafish organs shows sdf-1 expression in kidney tubules, gills, and skin.
50 macrophages and T cells accumulate at sites (kidney tubules, glomeruli, pulmonary bronchioli, lymph n
51  colleagues found that deleting Nedd4-2 from kidney tubules in adult mice led to ENaC accumulation, b
52 tudy, we have observed that WNV infection of kidney tubules in mice coincides with the loss of expres
53     We examined ErbB4 function in developing kidney tubules in vivo with Pax8-Cre-mediated conditiona
54 ressing epithelial cells after injury in rat kidney tubules in vivo.
55 outperformed either SCr or BUN for detecting kidney tubule injury and TFF3 augmented the potential of
56 epatocytes in the liver, infiltration of the kidney tubule interstitial by chronic inflammatory cells
57 eported Stx binding sites were identified in kidney tubules, intestinal lymphoid aggregates, sinusoid
58 hese results demonstrate that mTOR regulates kidney tubule ion handling and suggest that mTOR regulat
59   Hormone regulation of ion transport in the kidney tubules is essential for fluid and electrolyte ho
60           Inactivation of HNF-1beta in mouse kidney tubules leads to early-onset cyst formation and p
61 the target nor the site of ROS production in kidney tubule mitochondria in short-term diabetes.
62 P components in cytoskeletal assembly during kidney tubule morphogenesis in Xenopus laevis and zebraf
63 d the effect of selective mTOR inhibitors on kidney tubule Na+ and K+ transport in WT and Sgk1-/- mic
64 n a wide range of epithelial tissues such as kidney tubules or breast acini, cells organize into bidi
65 t in the early stage of acute pyelonephritis kidney tubules participate actively in the local host re
66 fen-induced expression of Kim-1 in zebrafish kidney tubules resulted in loss of the tubule brush bord
67 cohort (n = 95) of normal and fibrotic human kidney tubule samples followed by systems and network an
68 ximal tubule and decreased it in more distal kidney tubule segments.
69     After ischemia-reperfusion injury (IRI), kidney tubules show activated transforming growth factor
70                               We report that kidney tubule-specific inactivation of Inpp5b on a globa
71 of 1-3 microM, without affecting other major kidney tubule transporters.
72                                       In rat kidneys, tubules undergoing atrophy late after IRI but n
73 AnkG190, the isoform of ankyrin expressed in kidney tubules, was used as bait in a yeast two-hybrid s
74 divisions maintain the diameter of postnatal kidney tubules, we find that cell divisions are randomly
75 y were found in the brush border of proximal kidney tubules where megalin is localized.
76 A class are expressed in epithelial cells in kidney tubules, where they are required for the formatio
77 ng saccharides in the epithelium of proximal kidney tubules, whereas scattered glomeruli appeared col
78 s disruption of key cytoskeletal elements in kidney tubules, which contributes causally to the injury
79 in development and uptake of proteins by the kidney tubule, yolk sac, and thyroid.

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