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1 on of major Na(+) transporters all along the kidney tubule.
2 determinants of Na(+) reabsorption along the kidney tubule.
3 nsor, detecting changes in fluid flow in the kidney tubule.
4 cells induced the acquisition of functional kidney tubules.
5 nes PKD1 or PKD2 induces cyst formation from kidney tubules.
6 organ ducts such as the digestive tract and kidney tubules.
7 r that is characterized by cyst formation in kidney tubules.
8 efect in oriented cell division in precystic kidney tubules.
9 usters of interstitial cells surrounding the kidney tubules.
10 its differentiation of these precursors into kidney tubules.
11 ns at concentrations present in seawater and kidney tubules.
12 Histology revealed a subsequent absence of kidney tubules, an enlarged cardinal vein and expansion
13 potassium channel (ROMK) is expressed in the kidney tubule and critically regulates sodium and potass
14 all protein), is exclusively produced by the kidney tubule and has specific biochemical properties th
16 e considered tantamount to the injury of the kidney tubule and the epithelial cells thereof (AKI).
19 es, tendon, and the connective tissue around kidney tubules and lung alveoli, which all contain fibri
22 in mice induced cilium elongation defects in kidney tubules and predisposed animals to cyst developme
24 stinal tract, the thick straight segments of kidney tubules, and the marrow stromal cells in adults.
25 stimulating factor-1 (CSF-1) is expressed by kidney tubules at the onset of LN, increases with diseas
27 nd on epithelial cells, such as those of the kidney tubule, but also on nonepithelial cells, such as
28 ssessed multiple large cysts in the proximal kidney tubules, but even in tensin null mice with normal
29 t establishes and maintains the structure of kidney tubules, but the role of this pathway in the path
30 mmals has been attributed to the blockage of kidney tubules by insoluble complexes of melamine with c
31 has been used to determine whether embryonic kidney tubules can be stimulated by cAMP to form cysts.
33 kyrin and Fas was confirmed in vivo in mouse kidney tubule cells by coimmunoprecipitation and colocal
34 by fluorescence intensity in primary canine kidney tubule cells, Madin-Darby canine kidney cells, an
38 zed and secreted into the medium by cultured kidney tubules derived from cystic C57BL/6J-cpk mice.
40 inate from normal and nondilated ADPKD human kidney tubules display normal ciliary expression of the
41 irectional cell growth, including defects in kidney tubule elongation that lead to formation of kidne
43 ls of Kid1 mRNA correlate with maturation of kidney tubule epithelia in rat post-natal kidney develop
45 -sensorial function of the primary cilium in kidney tubule epithelial cells and (ii) the distinct thr
50 macrophages and T cells accumulate at sites (kidney tubules, glomeruli, pulmonary bronchioli, lymph n
51 colleagues found that deleting Nedd4-2 from kidney tubules in adult mice led to ENaC accumulation, b
52 tudy, we have observed that WNV infection of kidney tubules in mice coincides with the loss of expres
53 We examined ErbB4 function in developing kidney tubules in vivo with Pax8-Cre-mediated conditiona
55 outperformed either SCr or BUN for detecting kidney tubule injury and TFF3 augmented the potential of
56 epatocytes in the liver, infiltration of the kidney tubule interstitial by chronic inflammatory cells
57 eported Stx binding sites were identified in kidney tubules, intestinal lymphoid aggregates, sinusoid
58 hese results demonstrate that mTOR regulates kidney tubule ion handling and suggest that mTOR regulat
59 Hormone regulation of ion transport in the kidney tubules is essential for fluid and electrolyte ho
62 P components in cytoskeletal assembly during kidney tubule morphogenesis in Xenopus laevis and zebraf
63 d the effect of selective mTOR inhibitors on kidney tubule Na+ and K+ transport in WT and Sgk1-/- mic
64 n a wide range of epithelial tissues such as kidney tubules or breast acini, cells organize into bidi
65 t in the early stage of acute pyelonephritis kidney tubules participate actively in the local host re
66 fen-induced expression of Kim-1 in zebrafish kidney tubules resulted in loss of the tubule brush bord
67 cohort (n = 95) of normal and fibrotic human kidney tubule samples followed by systems and network an
69 After ischemia-reperfusion injury (IRI), kidney tubules show activated transforming growth factor
73 AnkG190, the isoform of ankyrin expressed in kidney tubules, was used as bait in a yeast two-hybrid s
74 divisions maintain the diameter of postnatal kidney tubules, we find that cell divisions are randomly
76 A class are expressed in epithelial cells in kidney tubules, where they are required for the formatio
77 ng saccharides in the epithelium of proximal kidney tubules, whereas scattered glomeruli appeared col
78 s disruption of key cytoskeletal elements in kidney tubules, which contributes causally to the injury
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