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1 extensive ligand-receptor interactions with killer-cell immunoglobulin-like receptors.
2 stocompatibility complex class I-recognizing killer-cell immunoglobulin-like receptors.
3 he crystal structure of the inhibitory human killer cell immunoglobulin-like receptor 2DL1 (KIR2DL1)
4 in immunity, but how HLA class I (HLA-I) and killer cell immunoglobulin-like receptor 3DL1 (KIR3DL1)
5 upregulated NKp44 expression, and remarkable killer cell immunoglobulin-like receptor acquisition.
6 costimulatory molecules (such as stimulatory killer cell immunoglobulin-like receptors and NKG2D) app
7 NKG2C(+) NK cells predominately expressed killer cell immunoglobulin-like receptor, and self-kille
9 entified perforin, CD161, and members of the killer-cell immunoglobulin-like receptors as being diffe
10 ype, and more frequently expressed educating killer cell immunoglobulin-like receptors compared with
12 n complex is important for NK cell function, killer cell immunoglobulin-like receptor inhibitory sign
13 : IPD-KIR, contains the allelic sequences of killer-cell immunoglobulin-like receptors, IPD-MHC, a da
14 : IPD-KIR, contains the allelic sequences of Killer-cell Immunoglobulin-like Receptors, IPD-MHC, is a
15 : IPD-KIR, contains the allelic sequences of Killer-cell Immunoglobulin-like Receptors; IPD-MHC, a da
16 s of hybridization on Southern blotting with killer cell immunoglobulin-like receptor (KIR) cDNA prob
17 Here we show that the number of activating killer cell immunoglobulin-like receptor (KIR) copies in
18 single locus in the centromeric motif of the killer cell immunoglobulin-like receptor (KIR) gene fami
20 of several human leukocyte antigen (HLA) and killer cell immunoglobulin-like receptor (KIR) loci.
22 ize, that NK cell alloreactivity mediated by killer cell immunoglobulin-like receptor (KIR)-HLA inter
23 , transplantations before 2004, patient C2C2 killer cell immunoglobulin-like receptor (KIR)-ligand ph
25 le of activating and inhibitory forms of the killer cell immunoglobulin-like receptors (KIR) in natur
28 r (NK) cell function can be modulated by the killer cell immunoglobulin-like receptors (KIR) which in
29 ortant in innate immunity as ligands for the killer cell immunoglobulin-like receptors (KIR), which m
38 maternal natural killer (NK) cells that use killer-cell immunoglobulin-like receptor (KIR) to recogn
42 ype is partially mediated through binding of killer-cell immunoglobulin-like receptors (KIR) with HLA
43 ) related to human leukocyte antigens (HLA), killer-cell immunoglobulin-like receptors (KIR), major h
44 city, whereas knockdown of its receptor, the killer cell immunoglobulin-like receptor KIR3DL2, on hum
46 n requires an interaction between inhibitory killer cell immunoglobulin-like receptors (KIRs) and HLA
48 ibility complex (MHC) class I, including the killer cell immunoglobulin-like receptors (KIRs) and the
49 ler (NK) cells are functionally regulated by killer cell immunoglobulin-like receptors (KIRs) and the
52 ion depend upon diverse interactions between killer cell immunoglobulin-like receptors (KIRs) and the
57 human immunity and reproduction are diverse killer cell immunoglobulin-like receptors (KIRs) that re
60 tients with vasculitis were found to express killer cell immunoglobulin-like receptors (KIRs) with th
61 ins by diverse receptor families such as the killer cell immunoglobulin-like receptors (KIRs), and (i
63 y interest are a group of genes encoding the killer cell immunoglobulin-like receptors (KIRs), which
64 NK)-cell alloreactivity, determined by donor killer-cell immunoglobulin-like receptors (KIRs) and rec
65 wo highly polymorphic sets of molecules: the killer-cell immunoglobulin-like receptors (KIRs) and the
67 gs underscore the crucial role of activating killer-cell immunoglobulin-like receptors (KIRs) in NK c
68 ate GVL effect by careful mismatching on the killer-cell immunoglobulin-like receptors (KIRs) ligand.
70 ic regions such as human leukocyte antigens, killer-cell immunoglobulin-like receptors, major histoco
71 ells versus healthy twin PCs included RAD51, killer cell immunoglobulin-like receptor protein, and ap
72 CSF2); interferon gamma receptor 2 (IFNGR2); killer cell immunoglobulin-like receptor, three domain,
73 cell immunoglobulin-like receptor, and self-killer cell immunoglobulin-like receptors were required
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