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1 mat, and cell surface receptors, such as the killer immunoglobulin-like receptors.
2 pecifically downregulates disease-associated killer immunoglobulin-like receptors.
3 els of the activating receptor NKp46 and the killer immunoglobulin-like receptors 2DL1/S1 and 3DL1, r
5 CD117(+)M-CSFR(+)) showed more expression of killer immunoglobulin-like receptors, a fraction of kill
6 tural killer (NK) cells and their activating killer immunoglobulin-like receptors (aKIRs) influence t
7 rtain the association of natural killer cell killer immunoglobulin-like receptors and human leukocyte
8 ike receptors, mannose-binding lectin, CD14, killer immunoglobulin-like receptors, and matrix metallo
9 e-binding positions 9, 99, 116, and 156, and killer immunoglobulin-like receptor binding position 77
10 sion is closely monitored by NK cells, whose killer immunoglobulin-like receptors encode MHC-I-specif
11 ng patients (n=222) and controls (n=191) for killer immunoglobulin-like receptor gene polymorphisms d
12 g/m(2)/d on days -6 through -2), followed by killer immunoglobulin-like receptor-human leukocyte anti
13 regard, NK cells that express an inhibitory killer immunoglobulin-like receptor (iKIR) for which the
14 ating natural killer (NK) cell receptor, the killer immunoglobulin-like receptor (KIR) 3DS1, and its
15 ells expressing CD94: NKG2A but no change in killer immunoglobulin-like receptor (KIR) expression.
17 in reaction (PCR) assays to compare NKG2 and killer immunoglobulin-like receptor (KIR) gene expressio
18 -cells played by the inherited repertoire of killer immunoglobulin-like receptor (KIR) genes therefor
20 atural killer (NK) receptors, in particular, killer immunoglobulin-like receptor (KIR) KIR3DL2, a rec
21 ent MHC class I ligands for donor inhibitory killer immunoglobulin-like receptor (KIR) receptors, as
24 " In humans, interactions between inhibitory killer immunoglobulin-like receptors (KIR) and human MHC
27 een HLA-B27 and immune receptors such as the killer immunoglobulin-like receptors (KIR) found on a ra
28 are transmitted by NK inhibitory receptors (killer immunoglobulin-like receptors, KIR) at the site o
30 ct to two such families of NK receptors, the killer immunoglobulin-like receptors (KIRs) and the kill
32 I molecules as ligands to NK cell inhibitory killer immunoglobulin-like receptors (KIRs) as a means o
34 city is attenuated by ligation of inhibitory killer immunoglobulin-like receptors (KIRs) by HLA class
35 portant role of NK cells expressing specific killer immunoglobulin-like receptors (KIRs) in the contr
40 argely been attributed to mismatches between killer immunoglobulin-like receptors (KIRs) on NK cells
44 ansplantation, produce cytokines and express killer immunoglobulin-like receptors (KIRs) that regulat
45 h increasing age, T cells gain expression of killer immunoglobulin-like receptors (KIRs) that transmi
46 Instead, CD4(+)CD28(null) T cells express killer immunoglobulin-like receptors (KIRs) with a prefe
47 ors, including the activating and inhibitory killer immunoglobulin-like receptors (KIRs), and human l
48 tural killer (NK) cells possess little or no killer immunoglobulin-like receptors (KIRs), high interf
53 ic factors for improved outcome were missing killer immunoglobulin-like receptor ligand (PFS and OS),
54 ng CBRM1/5-positive granulocytes and missing killer immunoglobulin-like receptor ligand as positive i
55 When the model for OS also included missing killer immunoglobulin-like receptor ligand, human antimo
58 immunoglobulin-like receptors, a fraction of killer immunoglobulin-like receptor-positive-expressing
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