ライフサイエンスコーパス: killing of

1 AR23 greatly augments myeloid cell-dependent killing of a collection of hematopoietic and nonhematopo

2 Here, I report facultative matricide (worker killing of a colony's queen) in the social wasp Dolichov

3 innate immunity, mediating CXCR3-independent killing of a diverse array of pathogenic microorganisms.

4 y contrasting mechanisms and efficiencies of killing of a range of tumor cell lines.

5 e effective than AMX at enhancing phagocytic killing of A. actinomycetemcomitans by neutrophils.

6 ve transport system for AZM that may enhance killing of A. actinomycetemcomitans.

7 ation increased conidial uptake and enhanced killing of A. fumigatus by MDMs and neutrophils.

8 IRAK4 inhibition promoted killing of ABC DLBCL lines harboring MYD88 L265P, by dow

9 inflammatory mediators coincided with rapid killing of actively dividing bacilli, whereas slower del

10 persistent microfilaridermia and in enhanced killing of adult worms after repeated standard IVM treat

11 ith neutrophils, enhancing the intracellular killing of ALF-exposed M. tuberculosis and up-regulating

12 CD3xCD123 DART also induces a dose-dependent killing of AML cell lines and primary AML blasts in vitr

13 /CDK6 as an effective means to enhance Ara-C killing of AML cells at reduced doses, which has implica

14 itor (CHK1i) GDC-0575 enhances AraC-mediated killing of AML cells both in vitro and in vivo, thus abr

15 Using this antibody, we achieved selective killing of AML MNC, CD34+CD38+, and CD34+CD38- cells.

16 -dependent cellular cytotoxicity (ADCC), the killing of an antibody-coated virus-infected cell by cyt

17 y been shown to enhance complement-dependent killing of, and facilitate bacterial clearance in, anima

18 ar trap (NET) formation, are involved in the killing of Aspergillus fumigatus conidia and hyphae, usi

19 ctions to limit and prevent autoimmunity via killing of autologous immune cells.

20 antigens and found these cells could mediate killing of autologous lymphoma cells.

21 s and were equally capable of MHC-restricted killing of autologous target cells.

22 iciently internalized via hCD22 resulting in killing of B-cell lymphoma cells.

23 showed inhibitory 2B4 function and defective killing of B-EBV cells.

24 observed in healthy individuals and improved killing of B. pseudomallei in vitro.

25 eneity arises, in part, through differential killing of bacteria after the onset of adaptive immunity

26 g proteins (GBPs); this led to intracellular killing of bacteria and DNA release.

27 to reduced reactive oxygen species-mediated killing of bacteria by alveolar macrophages.

28 X) mediated signaling enhanced intracellular killing of bacteria by neutrophils.

29 echanism of reactive oxygen species-mediated killing of bacteria by neutrophils.

30 hemical processes including, in our opinion, killing of bacteria by protozoa.

31 clearance, and iv) increased phagocytic cell killing of bacteria compared with tail trauma.

32 f bacteria throughout the colony exceeds the killing of bacteria on the surface and pinpoints how the

33 le for the opsonization and subsequent lytic killing of bacteria, work over the last century firmly e

34 tibodies that can induce complement-mediated killing of bacteria.

35 suggested no permanent cell injury or direct killing of bacteria.

36 hils, and correspondingly, there was limited killing of bacteria.

37 Killing of bacterial pathogens by granulocytes is a satu

38 er cell lines, explaining its broad spectrum killing of BCa lines, which did not occur with its compo

39 Moreover, neutrophil-mediated killing of biofilm bacteria correlated with the evident

40 Impaired killing of biofilm parallels the decrease in NET product

41 ation of action and failure to induce direct killing of Bordetella pertussis, a synthetic scheme was

42 oprotection was proliferation-independent as killing of both 5-ethynyl-2'-deoxyuridine-positive (or K

43 vents that culminate in Perforin-2-dependent killing of both intracellular and extracellular, cell-ad

44 se to the cytosol, enhanced PDT-induced cell killing of both resistant and sensitive cells.

45 s to radiation, and mediate synthetic lethal killing of BRCA2-deficient cancer cells, effects that we

46 4-targeted conjugate demonstrated acute cell killing of breast cancer cells with high CD44 expression

47 ed by inhibition of biofilm formation and by killing of C. albicans in mature biofilms.

48 (2003-2008) provided fecal samples to assess killing of C. difficile and changes to components of the

49 oring phage03 showed, on average, 17% higher killing of C. elegans (P = 4.4e(-6)).

50 out the cellular recognition, ingestion, and killing of C. neoformans and discuss the unique and rema

51 ial role of C5a-C5aR signaling in neutrophil killing of C. neoformans in real time.

52 nd CCL5/RANTES and more efficient uptake and killing of C. neoformans These findings suggest that DAP

53 ophage lysosome damage and enabled increased killing of C. neoformans.

54 ired neutrophil migration and its subsequent killing of C. neoformans.

55 resistance to chloramphenicol, and increased killing of Caenorhabditis elegans compared to the inocul

56 intervention that could increase immunotoxin killing of cancer cells and enhance our understanding of

57 nical cancer therapy, due to their selective killing of cancer cells and high safety characteristics.

58 herapy can enhance T cell-dependent targeted killing of cancer cells both by stimulating the hematopo

59 e metabolism results in dramatic synergistic killing of cancer cells both in vitro and in vivo.

60 Effective immunotherapy promotes the killing of cancer cells by cytotoxic T cells.

61 o CD95-mediated apoptosis and DICE, and that killing of cancer cells can be maximized by concomitant

62 is and immunogenic cell death contributed to killing of cancer cells in DBC-UiO-induced PDT.

63 CD8(+) T cell-dependent killing of cancer cells requires efficient presentation

64 bility that could be leveraged for selective killing of cancer cells through targeting of an unlinked

65 ossibly transcription), leading to selective killing of cancer cells with BRCA1/2 mutations.

66 circuits triggered selective T cell-mediated killing of cancer cells, but not of normal cells, in vit

67 ntratumoral vessels as a result of increased killing of cancer cells, setting up a positive feedback

68 D-28 had a high specificity in the selective killing of cancer cells, without any apparent effects on

69 ibition in sensitizing anticancer agents for killing of cancer cells.

70 e pharmacologically exploited to enhance the killing of cancer cells.

71 vation-induced apoptosis and T cell-mediated killing of cancer cells.

72 f AB307.30 but failed to increase phagocytic killing of capsule-positive strains.

73 HIV killing of CD4 T cells that replicate HIV can involve HI

74 ls, which inhibited IFN-gamma production and killing of CEF-pulsed monocytes.

75 cific receptors, such as Ly49A, that inhibit killing of cells expressing self-MHC-I.

76 for selective and efficient light-controlled killing of cells which are positive for epidermal growth

77 ranzyme B production in vitro and by in vivo killing of CFSE-labeled targets.

78 They also effectively mediated killing of chronically and acutely HIV-1 infected T cell

79 he armed virus was associated with efficient killing of CICs, reduced expression of ascitic CXCL12 an

80 ead compounds with a high selectivity toward killing of clinically important multidrug-resistant E. c

81 Concomitant plasma membrane damage increased killing of CLL cells by DEX.

82 igate the role of p53 dynamics in fractional killing of colon cancer cells in response to chemotherap

83 be broad spectrum, leading to indiscriminate killing of commensal bacteria and accelerated evolution

84 rovide an example of pathogen T6SS-dependent killing of commensal bacteria as a mechanism to successf

85 This activation results in T6SS-mediated killing of competing bacteria but renders A. baumannii s

86 dent bacterial weapon that allows for direct killing of competitors through the translocation of prot

87 terial competition through contact-dependent killing of competitors.

88 plicated an NKG2D-dependent mechanism for NK killing of CSCs.

89 teins is a prerequisite for efficient serial killing of CTLs and identify key events in this process.

90 Here we report a natural cancer killing of different types of cancer cells by sera from

91 Rather, rapid killing of donor lymphocytes by host alloreactive NK cel

92 l genetic perturbations that can enhance the killing of drug-resistant bacteria with antibiotic treat

93 was reduced, consistent with their impaired killing of EBV-infected cells.

94 ed activation by, rather than effects during killing of, EBV-transformed B cells.

95 ongoing autoimmune inflammation by selective killing of effector autoreactive T cells.

96 fic opsonic antibodies in mice, resulting in killing of encapsulated bacteria by phagocytic activity.

97 ically enhanced natural killer cell-mediated killing of eosinophils from healthy and eosinophilic don

98 These results demonstrate the killing of Escherichia coli and Salmonella typhimurium,

99 s factor and interleukin 6 and intracellular killing of Escherichia coli by monocytes and peritoneal

100 This is the first report of killing of established adult schistosome worms by a vacc

101 ralize laboratory-adapted HIV-1, and mediate killing of field-isolate HIV-1-infected CD4(+) T cells.

102 tudies have shown that NOX2 is essential for killing of G. bethesdensis by neutrophils and monocytes

103 luding collagen binding, HCAEC invasion, and killing of G. mellonella.

104 unized fish were preincubated with CC41 mAb, killing of G14D-CCV targets was reduced by approximately

105 artery endothelial cells (HCAEC) and in the killing of Galleria mellonella.

106 genes, which are essential for the enhanced killing of ganetespib treated melanoma cells by T cells.

107 ctin as a key functional arm of NET-mediated killing of gonococci.

108 ent structural features are relevant for the killing of Gram-negative and Gram-positive bacteria.

109 ented insecurity and violence, including the killing of health workers and the onset of a state of em

110 The security challenge has led to the killing of health workers, destruction of health facilit

111 ng a robust response and avoiding off-target killing of healthy tissues, which is difficult to accomp

112 g drug storage, drug release (via heat), and killing of HeLa cells in culture is investigated.

113 cific antibody-drug conjugates for selective killing of HER2-positive breast cancer cells.

114 e combinations via phagemids to increase the killing of highly drug-resistant E. coli harboring New D

115 s but the estimates differ considerably, and killing of HIV-1-infected cells was unexpectedly low.

116 ll maturation and improved effector-mediated killing of HIV-infected CD4 T cells by the HIV envelope-

117 stimates into perspective and shows that CTL killing of HIV-infected cells may not be atypically low.

118 ary CD4 T cells, venetoclax causes selective killing of HIV-infected cells, resulting in decreased nu

119 nce of PIs led to variations in CTL-mediated killing of HIV-infected cells.

120 ells underwent clonal expansion and mediated killing of HIV-infected cells.

121 the BsAbs were able to efficiently redirect killing of HLA-DR(+) leukemic cells by human CD5(+) cyto

122 Direct killing of host cells by the amoebae is likely to be the

123 and M-CSF-Mphi prevent the IFN-gamma-induced killing of HSCs.

124 ulation is associated with cytokine-mediated killing of human beta-cells, a process partially prevent

125 tissues, which is probably driven by direct killing of human cells.

126 The killing of human leukemia lymphoblasts by NK cells depen

127 ical for Staphylococcus aureus targeting and killing of human neutrophils ex vivo and is produced in

128 IL-15 withdrawal led to FasL-dependent killing of IL-2Ralpha(hi) NK cells by more educated IL-2

129 PD-L1-specific CTL may boost immunity by the killing of immunosuppressive tumor cells as well as regu

130 EBV miRNAs strongly inhibit recognition and killing of infected B cells by EBV-specific CD8(+) T cel

131 xic EBV-specific CD4(+) effector T cells and killing of infected B cells.

132 ified T cells will hopefully provide durable killing of infected cells and sustained virus suppressio

133 n mediated by CD8(+) T cells reflects direct killing of infected cells as opposed to indirect, noncyt

134 rly Gag epitope presentation, allowing early killing of infected cells before Nef-mediated downregula

135 We estimated the rate of killing of infected cells by CD8+ T cells in two distinc

136 uction of HIV-1 transcription and subsequent killing of infected cells by cytolytic T lymphocytes (CT

137 It promotes killing of infected cells by Fc-mediated antibody-depend

138 cytotoxic T lymphocyte (CTL) recognition and killing of infected cells.

139 ntrol intracellular infection leading to the killing of infected cells.

140 ed with strategies to induce immune-mediated killing of infected hepatocytes.

141 ages from female mice and humans show better killing of ingested bacteria.

142 to complement activation, phagocytosis, and killing of ingested fungi.

143 Type 1 diabetes mellitus is caused by the killing of insulin-producing beta cells by CD8+T cells.

144 ole for neutrophil calprotectin in uptake or killing of intracellular A. fumigatus conidia either in

145 erichia coli or apoptotic HEp2 cells and (2) killing of intracellular E coli.

146 share structural semblances and both induce killing of intracellular Leishmania, we surmised that pl

147 ctivation, and, most importantly, macrophage killing of intracellular Mtb.

148 al was required for phagosome maturation and killing of intracellular Mycobacterium tuberculosis (Mtb

149 antituberculosis treatment would enhance the killing of intracerebral Mycobacterium tuberculosis orga

150 healthy donors, we assessed phagocytosis and killing of L-ficolin-opsonized live A. fumigatus conidia

151 S, but not nitric oxide, also contributed to killing of L. braziliensis by IFN-gamma activated monocy

152 production, and 3) lower miltefosine-induced killing of L. donovani.

153 trol innate responses that contribute to the killing of L. major.

154 c Leukaemia (RBL-2H3) MCs led to significant killing of L. tropica and to a lesser extent of L. donov

155 ediated cell depletion, therapeutic antibody-killing of LCMV infected cells and human CD20-expressing

156 Role of protein kinase R in the killing of Leishmania major by macrophages in response t

157 show that this system can achieve effective killing of leukemic cells.

158 We thus hypothesized that the killing of liver-stage malarial parasites by IFN-gamma i

159 ls (LSCs) with D4476 showed highly selective killing of LSCs over normal HSPCs.

160 We also observed effective killing of lymphoid tumour cell lines in vitro following

161 combined with ibrutinib induced synergistic killing of lymphoma cell lines, primary human lymphoma s

162 Moreover, COMP inhibits phagocytic killing of M. catarrhalis by human neutrophils.

163 The enhanced killing of M. tuberculosis in macrophages in vivo by CD4

164 ruit and activate myeloid cells for enhanced killing of mAb-opsonized tumors.

165 The rapid killing of macrophages induced by Y. pestis, dependent u

166 both males and females despite the one-sided killing of male reproductives by larval female soldiers

167 and increased cell apoptosis, leading to the killing of MDA-MB-231 cells.

168 recruitment of NK cells and TRAIL-dependent killing of melanoma cells by macrophages.

169 Ab is required for complement-dependent killing of meningococci.

170 Blocking NKG2D in vitro resulted in less killing of mESC by allogeneic NK cells, indicating NKG2D

171 NKG2D is a likely mechanism for NK-mediated killing of mESC.

172 ored degranulation, IFNgamma production, and killing of MHC class I negative hematopoietic grafts.

173 revent killing of self-cells, while enabling killing of MHC I-deficient cells.

174 ecies oral biofilms in vitro led to enhanced killing of microorganisms, whereas a single exposure of

175 rimary MM, at low E:T ratios (56.2% +/- 3.9% killing of MM.1s at 48 h, E:T ratio 1:32; P < .01) and o

176 EC = 0.2-12.5 muM), as well as the effective killing of MRSA persister cells in non-biofilm cultures.

177 urn lead to increased susceptibility to host killing of MRSA.

178 teine to isoniazid treatment potentiated the killing of Mtb Furthermore, we demonstrate that the addi

179 ns of anisomycin induced selective apoptotic killing of Mtb-infected human macrophages, which was com

180 Finally, selective killing of Mtb-infected macrophages and subsequent bacte

181 in-induced autophagy increased intracellular killing of Mtb.

182 argeting the ETC can be exploited to enhance killing of Mtb.

183 only polyrhamnose promoted opsonophagocytic killing of multiple GAS serotypes and protected against

184 gized with rifampin to promote intracellular killing of mycobacteria.

185 ls (DCs) that impairs T-cell recognition and killing of myeloma cells.

186 azepine dimers demonstrated potent bystander killing of neighboring CD30(-) cells.

187 anti-Rmp Abs) can block complement-dependent killing of Neisseria gonorrhoeae by otherwise bactericid

188 oid of toxin and SpaA pili exhibited delayed killing of nematodes with similar kinetics.

189 ulated process to allow for effective serial killing of NK cells.

190 nd pyridoxine did not only cause synergistic killing of NSCLC cells but also elicited signs of immuno

191 Phagocytosis and killing of NTHi by macrophages were evaluated by an in v

192 The second mechanism for the killing of opsonized C albicans was strictly dependent o

193 These aggregates can drive contact-dependent killing of other organisms, or Caulobacter cells not pro

194 Pentobra demonstrated potent and selective killing of P. acnes but not against human skin cells in

195 Patients with impaired serum-mediated killing of P. aeruginosa by IgG2 have poorer respiratory

196 ted bacterial phagocytosis and intracellular killing of P. aeruginosa by targeting Rheb (Ras homolog

197 1 function, we further demonstrate selective killing of p53-deficient cells with camptothecin while s

198 ) showed enhanced phagocytosis and bacterial killing of PAO1.

199 in these mice, demonstrating that bystander killing of parasites does not occur during the CD8(+) T

200 teristic of candidate antimalarials is rapid killing of parasites.

201 chanism of host defense, responsible for the killing of pathogens and the production of potent anaphy

202 e specialized pathways that ensure efficient killing of pathogens in phagosomes.

203 re a new generation of PS for light-mediated killing of pathogens with outstanding singlet oxygen qua

204 ar traps (NETs) facilitate the extracellular killing of pathogens.

205 o the peritoneum, or improve phagocytic cell killing of pathogens.

206 P90 with ganetespib enhances T-cell-mediated killing of patient-derived human melanoma cells by their

207 of the stages involved in the detection and killing of peptide-pulsed targets in vitro revealed that

208 pproximately 40% compared to that of HC, and killing of periodontal pathogens, including Porphyromona

209 le nitric oxide synthase (iNOS) and enhanced killing of persistent pathogens, suggesting that inhibit

210 the lifetime of iNOS and thereby enhance the killing of persistent pathogens.

211 pendent complement deposition and phagocytic killing of pneumococci expressing ST11A but not those ex

212 ation plays a vital role in opsonophagocytic killing of pneumococci in blood.

213 e plus AC significantly reduced in vitro AMo killing of pneumococci, relative to other conditions, in

214 elates with the efficacy of opsonophagocytic killing of pneumococci.

215 Moreover, direct recognition and killing of primary autologous lymphoma cells by activate

216 an expansion of cytotoxic effector cells and killing of productively infected CD4 T cells.

217 This damage occurs primarily by killing of proximal tubule kidney cells and mechanosenso

218 Similarly, hypothiocyanite-dependent killing of Pseudomonas aeruginosa was inhibited by urate

219 Killing of Pseudomonas aeruginosa was significantly incr

220 Hypercapnia also reduced macrophage killing of Pseudomonas aeruginosa.

221 An acidic pH also attenuated LL-37 killing of Pseudomonas aeruginosa.

222 om the public good provided by ExoU-mediated killing of recruited innate immune cells.

223 e presence of renal MECs, and induced direct killing of renal MECs in vitro.

224 Further, biofilm disruption fostered killing of resident bacteria by previously ineffective a

225 RT elicits effects that go beyond the direct killing of residual tumor cells.

226 oration with treatment significantly enabled killing of S aureus both ex vivo and in vivo.

227 agonist pioglitazone, and phagocyte ROS and killing of S aureus were investigated.

228 efense against S. aureus both through direct killing of S. aureus and enhancing the antimicrobial fun

229 ntibodies mediated in vitro opsonophagocytic killing of S. aureus expressing CP5 or CP8, whereas CP5-

230 ce macrophage phagocytosis and intracellular killing of S. aureus In this study we report evidence in

231 associated with enhanced neutrophil-mediated killing of S. aureus, increased C5a release, and modulat

232 We concluded that killing of S. mutans by ME promotes effective reminerali

233 G preparation that promoted opsonophagocytic killing of S. pyogenes in vitro and provided passive imm

234 mediated and cell-dependent opsonophagocytic killing of S. Typhimurium in vitro.

235 ent isotypes that exert in vitro and in vivo killing of S. Typhimurium.

236 logous O-antigen, which mediated significant killing of Salmonella Choleraesuis and provided full pro

237 lf-MHC class I (MHC I) molecules and prevent killing of self-cells, while enabling killing of MHC I-d

238 ed serotype 6A and 6B antibodies mediate the killing of serotype 6E pneumococci.

239 d serotype 6B antibodies were able to elicit killing of serotype 6E pneumococci.

240 Furthermore, PRELP enhances phagocytic killing of serum-opsonized M. catarrhalis by human neutr

241 pecific antibodies, which trigger phagocytic killing of staphylococci and protect mice against lethal

242 immunity are not yet known, opsonophagocytic killing of staphylococci by phagocytic cells offers oppo

243 uld enhance phagocyte oxidant production and killing of Staphylococcus aureus, a significant pathogen

244 n-induced signals in stellate cells and thus killing of stellate cells by bile acids might have impor

245 ages enhances lysosome-based proteolysis and killing of subsequently phagocytosed E. coli compared to

246 troy target cells while avoiding nonspecific killing of surrounding healthy cells.

247 at oxidative processes are involved in TLF-1 killing of T. brucei brucei.

248 ULK1 and Beclin 1 to stimulate autophagy and killing of T. gondii.

249 type 1 envelope (Env)-specific ADCC-mediated killing of target cells (P<.001) was observed after 96 w

250 nyl leukotrienes (CystLTs), allowing for the killing of target cells in a T cell receptor-independent

251 ified CMV-specific T cells retained specific killing of target cells pulsed with the CMV peptide NLV

252 However, the killing of target cells sometimes requires multiple enga

253 Killing of target cells was mediated by different cytoto

254 r function (e.g., production of IFNgamma and killing of target cells) in NK cells, whereas C-HIV and

255 characterize the functional response of CTL killing of target cells, and find that the total killing

256 between T cells and APCs, and for cytotoxic killing of target cells.

257 -mediated signaling and by increasing serial killing of target cells.

258 for inflammatory cytokine production and the killing of target cells; however, much less is known abo

259 cytotoxic machinery of Ly49s(+) NK cells in killing of target T cells.

260 Selective killing of tetraploid cells was observed for a series of

261 A14_46880 strains showed increased cytotoxic killing of the airway cells independent of the total num

262 This resulted in weak killing of the bacteria by human neutrophils and a corre

263 NAI-107 treatment, which could contribute to killing of the bacterial cell.

264 LSCs and significantly enhanced TKI-mediated killing of the cells.

265 hance bacterial phagocytosis but did enhance killing of the few bacteria ingested by neutrophils.

266 Despite lack of killing of the Gal-Gal-Glc-HepI mutants, mAb 2C7 deposit

267 use of venetoclax, which causes preferential killing of the HIV-expressing cells.

268 he increase in viral production will lead to killing of the infected cell either by the virus itself

269 to respond upon infection and mediate either killing of the infected cell or induce killing mechanism

270 . Typhimurium to effect in vitro and in vivo killing of the invasive African S. Typhimurium strain D2

271 almost completely abolished phagocytosis and killing of the organisms by neutrophils.

272 lting in optimal phagocytosis and subsequent killing of the organisms.

273 x compartments with lysosomes and subsequent killing of the pathogen.

274 r of these showed significant dose-dependent killing of the schistosome larvae and markedly impaired

275 antiserum mediated in vitro opsonophagocytic killing of the strain harboring the pIP501 plasmid and a

276 cate that S. aureus uses hemolysin-dependent killing of these cells as an immune evasion strategy.

277 s production (26 to 71% increase), bacterial killing of these periodontal pathogens (22 to 38% reduct

278 layed by dendritic cells (DCs) in the direct killing of this bacterium.

279 rtant stimulator of autophagy and autophagic killing of Toxoplasma gondii in host cells.

280 CD40-mediated autophagic killing of Toxoplasma gondii is known to require TNF-alp

281 trate that natural killer (NK) cell-mediated killing of tumor cells coated with the Fc-optimized CD33

282 -1) disruption augmented CAR T cell mediated killing of tumor cells in vitro and enhanced clearance o

283 T-cell-directed killing of tumor cells using bispecific antibodies is a

284 s with coumarin dye, we demonstrate targeted killing of tumor cells with doxorubicin.

285 ec-E-deficient mice showed increased in vivo killing of tumor cells, and this effect was reversed by

286 For selective killing of tumor cells, there are many novel and promisi

287 oach that potentiates human NK cell-mediated killing of tumor cells.

288 scription was previously shown to potentiate killing of tumor cells.

289 suppress their activation and inhibit immune killing of tumors, a role for myeloid cell A2ARs in supp

290 ly studied with regard to NK recognition and killing of tumors.

291 denosine receptor blockers to enhance immune killing of tumors.

292 on-dependent NK cell survival advantages and killing of uneducated NK cells result in the maintenance

293 BCL-2 antagonist venetoclax induced minimal killing of uninfected CD4 T cells but markedly increased

294 The first mechanism for the killing of unopsonized C albicans was found to be depend

295 It was found that killing of V583 resulted from lethal cross-talk between

296 we have shown that natural killer (NK) cell killing of various tumors is inhibited in the presence o

297 ells have been defined by nonspecific innate killing of virus-infected and tumor cells.

298 Using an assay designed to measure the killing of virus-infected cells by antibody-dependent ce

299 l for neutrophil phagocytosis and subsequent killing of yeast cells.

300 onocytes, generation of TipDC, and phagocyte killing of yeast.