1 ch each behavioral stage and to become fully
kindled (
8.63 +/- 0.92 vs. 16.17 +/- 1.37; Mean +/- S.E.
2 , a separate group of animals (Expt. 3) were
kindled accordingly and processed for c-Fos and basic fi
3 hrough cannulae in the LC ipsilateral to the
kindled amygdala in 6 kittens.
4 ae adjacent to stimulating electrodes in the
kindled amygdala, and evoked seizures were tested 10-12
5 and synaptic plasticity in naive and in MDA-
kindled anaesthetised rats.
6 ing patterns during AGS were compared in non-
kindled and AGS kindled GEPR-9s.
7 us burst frequency between slices taken from
kindled and control animals.
8 portions of sprouted mossy fiber synapses in
kindled and kainic acid-treated rats.
9 During AGS in both AGS
kindled and non-kindled GEPR-9s, vlPAG neurons displayed
10 as significantly longer in slices taken from
kindled animals as compared to those taken from implante
11 ng effects on spontaneous epileptogenesis in
kindled animals as well.
12 but not of p38, is significantly enhanced in
kindled animals compared with their naive counterparts i
13 indling as well as phenobarbital-treated non-
kindled animals recovered within 2 months after the lesi
14 f the JNK substrate paxillin was enhanced in
kindled animals, but not that of c-Jun.
15 f the generalised seizure threshold in fully
kindled animals.
16 f late paired-pulse depression recorded from
kindled animals.
17 shows for the first time that in control and
kindled basolateral amygdala neurons, two pharmacologica
18 However, the 'inflammatory fire'
kindled by the host response must be tightly controlled
19 leptic effects of 2DG were evaluated in rats
kindled from olfactory bulb or perforant path.
20 r was induced by AP7 (1 and 7.5 nmol) in AGS-
kindled GEPR-3s.
21 However, in AGS-
kindled GEPR-9s these responses showed a precipitous fir
22 isorders, on the CeA to vlPAG pathway in AGS-
kindled GEPR-9s was also evaluated.
23 ortant for its anticonvulsant effects in AGS-
kindled GEPR-9s, and this effect may contribute to gabap
24 During AGS in both AGS kindled and non-
kindled GEPR-9s, vlPAG neurons displayed tonic firing du
25 led levels and reversibly blocked PTC in AGS-
kindled GEPR-9s.
26 asing stimulus intensity, as compared to non-
kindled GEPR-9s.
27 lPAG neuronal firing change was seen only in
kindled GEPR-9s.
28 ing AGS were compared in non-kindled and AGS
kindled GEPR-9s.
29 etanus-induced afterdischarges that could be
kindled in the absence of NMDA receptor activation.
30 The data suggest that
kindled-
induced hyperexcitability of the amygdala and li
31 arity complex in Drosophila and vertebrates,
kindled intense interest in polarity pathways.
32 The findings suggest a 'critical period' in
kindled kittens for onset of spontaneous temporal lobe e
33 ontaneous seizure susceptibility in amygdala-
kindled kittens.
34 onal responses to CeA stimulation to pre-AGS-
kindled levels and reversibly blocked PTC in AGS-kindled
35 eorge Schwert in the biochemistry department
kindled my interest in biochemistry.
36 h EC50 values of 36 nM (control) and 1.2 nM (
kindled neurons).
37 EC50 values of 297 nM (control) and 10.8 nM (
kindled neurons).
38 Moreover, in
kindled neurons, agonists at either group II- or group I
39 The rats were
kindled once daily using afterdischarge-threshold electr
40 8 and each day thereafter until either fully
kindled or until day 20, the animals received daily trea
41 ivation was comparable between human TLE and
kindled rat granule cells.
42 tor 6 Hz (32 mA) (mice, ip), and hippocampal
kindled (
rat, ip) seizure tests providing excellent prot
43 Hippocampus-
kindled rats also displayed fear-potentiated startle, bu
44 voltage-clamp methods in granule cells from
kindled rats experiencing spontaneous seizures demonstra
45 ceptor agonist, DCG-IV, were tested in fully
kindled rats following daily electrical stimulation of t
46 Amygdala-
kindled rats had exaggerated fear-potentiated startle co
47 tion in the number of GAD67-cells in caudate-
kindled rats in the dentate gyrus and CA3 hippocampal su
48 Using hippocampal-
kindled rats to ensure maximal clinical expression, we d
49 ir cells in the dentate gyrus of hippocampal-
kindled rats, which suggests a possible reduction of syn
50 ed startle, but no greater than that of sham-
kindled rats.
51 ed fear-potentiated startle compared to sham-
kindled rats.
52 on at high stimulus intensities in naive and
kindled rats.
53 In brain slices obtained from epileptic (
kindled)
rats, the excitatory drive onto inhibitory inte
54 ched the values previously reported for TLE (
kindled)
rats.
55 The study confirms previous findings in
kindled rodents to show that CLON and IDA can have oppos
56 ffectively laden touch in humans has further
kindled scientific interest in the area.
57 This report describes amygdala-
kindled seizure development and the post-kindling course
58 have contrasting effects on evoked amygdala-
kindled seizure susceptibility.
59 indled seizures exhibited full-scale 5 stage-
kindled seizures 24 h after DBS.
60 tic neural networks are involved in amygdala-
kindled seizures and that DBS, if well timed with the on
61 d caused a twofold slowing in progression of
kindled seizures at both stimulation sites.
62 y 1 of the 10 rats for which DBS had blocked
kindled seizures exhibited full-scale 5 stage-kindled se
63 Our DBS protocol completely blocked
kindled seizures in 10 out of 23 (43.5%) rats studied.
64 line-3-carboxylate, picrotoxin, and amygdala-
kindled seizures in mice (ED50 values, 84-97 mg/kg).
65 R127303 delays the acquisition and decreases
kindled seizures in mice, indicating that the drug slows
66 Kindled seizures markedly elevated CRF and CRF-BP in the
67 idization to examine the effects of amygdala-
kindled seizures on the mRNA levels of CRF and CRF-BP.
68 When stage 5-
kindled seizures were achieved by daily amygdala kindlin
69 1 (0.25 mg/kg, i.p.), and the development of
kindled seizures, produced an increase in AD thresholds
70 ntia nigra pars reticulata (SNr) on amygdala-
kindled seizures.
71 networks that may prevent the recurrence of
kindled seizures.
72 ling and on fully developed stage 5 amygdala
kindled seizures.
73 ces from control rats and rats with amygdala-
kindled seizures.
74 mals is altered following the development of
kindled seizures.
75 nd are enhanced following the development of
kindled seizures.
76 In the dark recovery phase, the
kindled state relaxes thermally on a time scale of secon
77 te to the enduring changes that underlie the
kindled state.
78 er at different time points to check for the
kindled state.
79 The "
kindled"
state, wherein depressive episodes occur with l
80 Beginning 48 h after surgery, animals were
kindled to evoke Stage 0 or Stage 1 seizure activity dur
81 Rats were
kindled to stage 5 by afterdischarge-threshold electrost
82 h stage V seizures and to become permanently
kindled was significantly greater in TRH-NP-treated subj
83 has enhanced anticonvulsant effects in fully
kindled wild-type mice, but not delta-subunit knock-out