1 erase and butyrylcholinesterase using enzyme
kinetic analysis.
2 sembly process as constraints for subsequent
kinetic analysis.
3 etamide are rationalized by detailed in situ
kinetic analysis.
4 y extent, and can provide valuable real-time
kinetic analysis.
5 dynamics simulation, electrophysiology, and
kinetic analysis.
6 lecular dynamic simulations (BOMD) and micro-
kinetic analysis.
7 mproved precatalyst (thtAuBr3) to facilitate
kinetic analysis.
8 mplified methods by comparing them with full
kinetic analysis.
9 rebellum was used as the reference region in
kinetic analysis.
10 series of XAS spectra and opens the door to
kinetic analysis.
11 g regional nerve density with PET and tracer
kinetic analysis.
12 -resolved absorption spectroscopy and global
kinetic analysis.
13 cular interactions, making them suitable for
kinetic analysis.
14 ion was overturned on the basis of extensive
kinetic analysis.
15 ry excretion during a 24-h period and from a
kinetic analysis.
16 nce of the present intact cell-based binding
kinetics analysis.
17 Kinetic analysis allowed investigation of the reaction m
18 Rapid
kinetics analysis also shows that ring resetting of a se
19 Preliminary
kinetic analysis and density functional calculations sup
20 A combination of
kinetic analysis and DFT calculations reveals the comple
21 Kinetic analysis and DFT calculations suggest that the C
22 Pre-steady-state
kinetic analysis and elucidation of the crystal structur
23 echanism-based inhibitors that allow precise
kinetic analysis and faithfully mimic the transition sta
24 To simplify
kinetic analysis and handling, a variant PCM-F was gener
25 Quantitative
kinetic analysis and immunochemistry studies suggest tha
26 Mechanistic investigation through
kinetic analysis and isotopic labeling studies indicates
27 Reaction progress
kinetic analysis and kinetic isotope effects suggest tha
28 Steady-state
kinetic analysis and modeling independently support this
29 The
kinetic analysis and molecular dynamics of the A25.C20 d
30 Through
kinetic analysis and optimization, we report an improved
31 arious formats of assays for equilibrium and
kinetic analysis and rapid determination of degradation
32 Kinetic analysis and reaction profile fitting of both th
33 SCADS combines microscopy, single-cell
kinetic analysis,
and population/cluster analysis to dis
34 with that obtained from conventional Biacore
kinetic analysis,
and the stoichiometries for the result
35 By using reaction progress
kinetic analysis as an evaluation method for the obtaine
36 d enzymes were characterized by steady-state
kinetic analysis at temperatures from 0 to 25 degrees C
37 8alpha and allowed discovery of a predictive
kinetic analysis based on cooperativity to distinguish T
38 Kinetic analysis based on time-resolved fluorescence rev
39 pensive and simple alternative to do binding
kinetics analysis between small molecules in solution an
40 cessfully demonstrate binding specificity in
kinetic analysis biomechanics in peptide aptamers and GO
41 inhibitors that we have subjected to further
kinetic analysis by comparing k(off) constants determine
42 ve tissue are not amenable to structural and
kinetic analysis by conventional methods.
43 of multiple assembly pathways, and show how
kinetic analysis can be used to distinguish different as
44 f Ki, Kd, IC50, and/or EC50, a more thorough
kinetic analysis can provide useful information for the
45 An enzyme
kinetic analysis comparing ERBB2(YVMA) to wild type usin
46 Site-directed mutagenesis and
kinetic analysis confirm the critical catalytic role pla
47 Kinetic analysis confirmed the existence of an intermedi
48 The steady-state
kinetic analysis coupled with the normal (13)(V/K) kinet
49 mentary biophysical, structure-function, and
kinetic analysis define the features that facilitate the
50 Binding and
kinetic analysis demonstrate that the MUG-K68N substitut
51 Further
kinetic analysis demonstrated first-order kinetics with
52 -fluoro-2-deoxyglucose ((18)FDG) ligand with
kinetic analysis demonstrated increased mean lung parenc
53 Preliminary
kinetic analysis demonstrated that BPND values obtained
54 This massively parallel enzyme
kinetics analysis detailed the specificity of ADAMTS13 a
55 ks and guides users through the key steps of
kinetic analysis:
determination of constraints to be pla
56 The
kinetic analysis enabled by the platform provided a rapi
57 Surface plasmon resonance-based
kinetic analysis enabled the selection of mini-Abs with
58 Kinetic analysis establishes that the reaction rate is d
59 Steady state
kinetic analysis establishes the enzyme is specific for
60 The
kinetic analysis exhibited discontinuities in the Arrhen
61 Molecular modeling studies in tandem with
kinetic analysis exhibited that these hybrids target bot
62 spectroscopy/steady-state isotopic transient
kinetic analysis)
experiments demonstrates that the rate
63 Kinetic analysis for different E2 concentrations shows t
64 Kinetic analysis for different E2 concentrations shows t
65 A
kinetic analysis for TVBN, SSP, hardness, adhesiveness a
66 PL titrations, thermochemical cycles, and
kinetic analysis (
for the mcb compounds) provided self-c
67 Kinetic analysis further indicated that PPTases possess
68 Kinetic analysis further shows that W32 oxidation likely
69 Kinetic analysis further shows the antioxidant reactivit
70 via a Makosza-like interfacial process, and
kinetic analysis has shown that the reaction possesses a
71 In steady-state
kinetic analysis,
hpol eta preferred to incorporate dATP
72 Modeling and
kinetic analysis identified Pz-1 as a type II tyrosine k
73 Kinetics analysis identifies that the slow solid-state s
74 ce temperature and Z-value commonly used for
kinetic analysis in food microbiology.
75 Kinetic analysis indicated an overall 5,000-fold and 1,1
76 occupies the active site cavity of G9a, and
kinetic analysis indicates competitive inhibition of G9a
77 in general acid-base chemistry, whereas our
kinetic analysis indicates that thioredoxin is likely to
78 Kinetic analysis indicates that, in LB medium supplement
79 LTA4 concentrations during the steady-state
kinetics analysis,
indicating poor lipid substrate bindi
80 al evidence (spectroscopic characterization,
kinetic analysis,
intermolecular reactivity, and radical
81 his noninvasive dual-window acquisition with
kinetic analysis is recommended.
82 rovide good fits to the data, so blood-based
kinetic analysis is recommended.
83 Kinetic analysis,
labeling, competition, and nonlinear e
84 sion estimates is not affected by the tracer
kinetic analysis method used.
85 ecently reported variable-time normalization
kinetic analysis method was used to delineate the comple
86 pressure IR (HPIR) and the reaction progress
kinetic analysis methodology suggested two steps in the
87 Furthermore,
kinetic analysis monitoring FVIIIa inactivation by APC v
88 Kinetic analysis of (11)C-GSK215083 uptake in the human
89 Steady-state
kinetic analysis of 20 G. max ATP sulfurylase point muta
90 The 2-tissue-compartment
kinetic analysis of a 90-min dynamic scan with arterial
91 study, we present the first biochemical and
kinetic analysis of a peptidoglycan O-acetyltransferase
92 Herein we report the first steady-state
kinetic analysis of a PKS DH domain employing LC-MS/MS a
93 Here, we identify and provide a detailed
kinetic analysis of a transcription cycle analogous to a
94 ractions, and the results were compared with
kinetic analysis of active site mutants.
95 Here, we report the cloning, expression, and
kinetic analysis of ASNA from Leishmania donovani.
96 A detailed
kinetic analysis of cardiac myosin has shown that the dr
97 Kinetic analysis of channel gating revealed that AITC ac
98 pattern was confirmed through synthesis and
kinetic analysis of cleavage of a set of optimized pepti
99 Intriguingly, cell
kinetic analysis of clonal isolates derived from single
100 Here, the authors show an operando
kinetic analysis of CO2 hydrogenation over a palladium c
101 Reaction progress
kinetic analysis of data obtained through in situ FTIR s
102 Kinetic analysis of DNA cleavage suggests flexible tethe
103 These proteins allowed
kinetic analysis of DNA recognition and structural analy
104 This detailed
kinetic analysis of Drosophila myosin carrying the R759E
105 Kinetic analysis of extraction was performed.
106 Kinetic analysis of five different AD-causing mutations
107 Kinetic analysis of fluorescence signals indicate that P
108 Here we report a
kinetic analysis of fluorescent guanine nucleotides bind
109 In contrast, the
kinetic analysis of GDP-ManPP was only possible with thr
110 This allowed detailed
kinetic analysis of global and stoichiometric phosphoryl
111 Kinetic analysis of GoxA revealed allosteric cooperativi
112 Moreover,
kinetic analysis of human carbonic anhydrase refolding s
113 sing these techniques, we describe the first
kinetic analysis of LD growth and secretion at peak lact
114 Kinetic analysis of lipid and content transfer showed th
115 A general differential approach for the
kinetic analysis of living polymerizations with fast pro
116 Most interestingly, results of
kinetic analysis of LPS bioactivity, using modified limu
117 ssay was developed that allowed steady-state
kinetic analysis of LsrK to be undertaken with the subst
118 Kinetic analysis of M. tuberculosis growth in the lungs
119 Kinetic analysis of miniature EPSCs revealed quantal rel
120 A detailed steady-state
kinetic analysis of MtNadD suggests that ATP must first
121 Steady-state
kinetic analysis of multiple point mutants of the lipid-
122 s likely to be bendable in one direction and
kinetic analysis of mutant DNA sequences with biolayer i
123 e subject of controversy; we report that the
kinetic analysis of ndSQR is consistent with glutathione
124 Kinetic analysis of NF-kappaB levels following loss of s
125 Kinetic analysis of NiCl(CCl2CCl3)(CNAr(Mes2))2 decompos
126 Here we report a detailed
kinetic analysis of nucleotide incorporation and exonucl
127 Kinetic analysis of peptide substrate phosphorylation an
128 Kinetic analysis of peptide-membrane interactions genera
129 Kinetic analysis of percentage and yield of preplasma an
130 -dependent Stokes and anti-Stokes SERS, with
kinetic analysis of photocatalytic reactions in an Ag na
131 ar approach can be potentially developed for
kinetic analysis of protein-small molecule binding by ot
132 ation by multiple catalytic inhibitors using
kinetic analysis of purified BRAF(V600E) and RAF(WT) enz
133 Kinetic analysis of purified oligo-ulvans incubated with
134 We describe a novel method of
kinetic analysis of radioligand binding to neuroreceptor
135 Enzyme
kinetic analysis of recombinant AtXTH31 confirmed this p
136 Kinetic analysis of ribozymes with systematically altere
137 Here we develop Single Molecule
Kinetic Analysis of RNA Transient Structure (SiM-KARTS)
138 Kinetic analysis of single-channel recordings made with
139 The x-ray structures and
kinetic analysis of site-directed mutants are consistent
140 Kinetic analysis of specific peptide generation reveals
141 It provides accurate
kinetic analysis of strand assimilation in high-throughp
142 We report a pre-steady-state
kinetic analysis of structural rearrangements of the DNA
143 Kinetic analysis of sugar translocation obtained from si
144 s article, we complement our two-dimensional
kinetic analysis of TCR-pMHC-CD8 interaction with concur
145 Kinetic analysis of the activation reaction according to
146 presents a detailed computational study and
kinetic analysis of the aminolysis of dithioates, dithio
147 Steady-state and transient
kinetic analysis of the ATPase cycle shows that the ADP
148 presents a detailed computational study and
kinetic analysis of the aza-Michael addition of primary
149 Here, by means of a systematic
kinetic analysis of the Bi-Te system reacting to Bi2Te3,
150 We have performed a
kinetic analysis of the blocking mechanism of the protot
151 le quantitative results were obtained from a
kinetic analysis of the changes in FRET.
152 Kinetic analysis of the cysteine:SufE sulfurtransferase
153 Kinetic analysis of the DNA unwinding and translocation
154 We undertook a detailed
kinetic analysis of the drug responses of K13 wild-type
155 Critically, pre-steady-state
kinetic analysis of the E3 rRNase(IDP)-Im3 complex demon
156 Further
kinetic analysis of the ECEC path revealed that base inc
157 Kinetic analysis of the effect of B6 on co-polymerizatio
158 er indirect flight muscle S1, we performed a
kinetic analysis of the effect of mutations in the conve
159 Kinetic analysis of the electrochemical response with ti
160 rode: (i) allows time-resolved detection and
kinetic analysis of the electrode response (the underlyi
161 Kinetic analysis of the encapsulation-isomerization even
162 Kinetic analysis of the enzymatic activities toward 3-ox
163 he sliding clamp and clamp loader mechanism,
kinetic analysis of the events following beta.gamma comp
164 Kinetic analysis of the formation of the 75 kDa iPLA2bet
165 tions with a comprehensive thermodynamic and
kinetic analysis of the growth process, which explains t
166 Kinetic analysis of the HIV-1 Tat (transactivator of tra
167 Interestingly, the steady-state
kinetic analysis of the initial rates determined at vary
168 The
kinetic analysis of the ligations using model peptides s
169 e report here the first detailed biochemical
kinetic analysis of the motor domain of the human beta-c
170 Site-directed mutagenesis and
kinetic analysis of the mutant enzymes, in conjunction w
171 Steady-state
kinetic analysis of the new mechanism is consistent with
172 By X-ray absorption spectroscopy and
kinetic analysis of the oxygen evolution reaction, we sh
173 Kinetic analysis of the pancreas was performed using a 1
174 We present here the first complete in situ
kinetic analysis of the PD-1/PD-ligands/B7-1 system.
175 da N protein (lambdaN) to initiate the first
kinetic analysis of the proteolytic mechanism of this en
176 This was corroborated by
kinetic analysis of the purified enantiomers, which show
177 ITC and
kinetic analysis of the R93A mutant also showed a comple
178 Kinetic analysis of the reaction between the respective
179 Steady state and time-resolved
kinetic analysis of the reaction catalyzed by the bacter
180 Kinetic analysis of the reaction is presented showing a
181 Mechanistic studies through in situ
kinetic analysis of the reaction reveal key differences
182 Here we present a thorough
kinetic analysis of the relationship between C-extein co
183 centration, with no need for model-dependent
kinetic analysis of the signal used for detection or the
184 Here, we report detailed
kinetic analysis of the Steap4 cell surface metalloreduc
185 s in continuous assays, and pre-steady-state
kinetic analysis of the target enzymes.
186 A transient
kinetic analysis of the ternary complex formation aided
187 Kinetic analysis of these effects confirm this observati
188 Steady-state
kinetic analysis of these substances demonstrates that t
189 To date,
kinetic analysis of this complex process has been achiev
190 Our
kinetic analysis of this response shows that cells have
191 ion by Trm10, we performed a biochemical and
kinetic analysis of Trm10 and variants with alterations
192 Here we present a thorough
kinetic analysis of Trypanosoma brucei TryS in a newly d
193 The assay was used to conduct a comparative
kinetic analysis of two LanM enzymes (HalM2 and ProcM) t
194 Kinetic analysis of wild-type UGDH and the inducible hex
195 Kinetic analysis of WT ADAMTS13 revealed approximately 2
196 It is shown from
kinetics analysis of both the enzyme catalytic responses
197 Here we demonstrate a quantitative binding
kinetics analysis of drug-target interactions to investi
198 Kinetics analysis of SV release supports that the proxim
199 fied conserved transitions between them, and
kinetic analysis paralleled these observations.
200 Kinetic experiments using reaction progress
kinetic analysis protocols demonstrate that inhibition o
201 Kinetic analysis provided an activation energy (E(a)) of
202 Kinetic analysis provides evidence that the turnover lim
203 to parameters derived from full quantitative
kinetic analysis (
R(2) < 0.34).
204 es obtained from tail currents together with
kinetics analysis reveal that the fast and slow gates of
205 Kinetic analysis revealed a larger turnover number for r
206 Kinetic analysis revealed apparent Km values of 20 and 9
207 Biochemical and
kinetic analysis revealed Lys(147) to be an intramolecul
208 Kinetic analysis revealed position 596 also plays a role
209 nalysis using Logan plots and full nonlinear
kinetic analysis revealed significant inhibition for bot
210 A detailed
kinetic analysis revealed that (E)-4-amino-3-methylbut-2
211 Enzyme
kinetic analysis revealed that BA inhibited tyrosinase a
212 Steady-state
kinetic analysis revealed that Dpo4 catalytic efficiency
213 Kinetic analysis revealed that most target mutation occu
214 In contrast, our
kinetic analysis revealed the presence of abundant CD25(
215 Kinetic analysis revealed the rate-limiting step of inac
216 Kinetic analysis reveals co-occupancy of the allosteric
217 Our steady-state
kinetic analysis reveals that A3A discriminates against
218 Our
kinetic analysis reveals that aggregation proceeds via m
219 Kinetic analysis reveals that perovskite films with less
220 Simple
kinetic analysis reveals that photo-oxidation of PhPyr b
221 Quantitative
kinetic analysis reveals that the CP-mDia1 antagonism th
222 Furthermore, multiple-turnover
kinetic analysis reveals that the rate-determining step
223 Steady-state
kinetic analysis reveals that WRN improves hpol kappa-ca
224 The force-dependent
kinetics analysis reveals a mechanism that requires DNA
225 tunnel and LPA in the pocket, together with
kinetic analysis,
reveals that bile salts act as partial
226 -depth kinetic study using reaction progress
kinetic analysis (
RPKA) has been performed to probe the
227 The results of
kinetic analysis show that among the closely related adu
228 EPR studies in tandem with
kinetic analysis show that the 490 nm chromophore of 2 i
229 Kinetics analysis show that the HYH gene is indeed late
230 Overall,
kinetic analysis showed an apparent zero-order model fit
231 Kinetic analysis showed brain uptake to be relatively hi
232 Kinetic analysis showed splicing rates with the selected
233 Coimmunoprecipitation and
kinetic analysis showed that E299V and wild-type isoform
234 Kinetic analysis showed that long-chain (C14-C18) substr
235 Our prior
kinetic analysis showed that nonnucleoside inhibitors bi
236 hat NaOt-Bu was necessary for catalysis, but
kinetic analysis showed that the base is not involved in
237 Kinetic analysis showed that the compounds function as r
238 Kinetic analysis showed that the predominant effect was
239 Kinetic analysis showed that these two domains contribut
240 Kinetic analysis showed that XXT5 has a 7-fold higher Km
241 The
kinetic analysis showed the absorption efficiency was hi
242 Kinetic analysis shows a minor role for substrate bindin
243 A steady-state
kinetic analysis shows that interaction with SSB stimula
244 ctors are required for optimal activity, and
kinetic analysis shows that MMCoA is incorporated first,
245 Our
kinetic analysis shows that NNI2 do not significantly bl
246 Our growth
kinetic analysis shows that the calcined materials have
247 Dynamic images were processed with
kinetic analysis software using a 1-tissue-compartment m
248 Results from
kinetic analysis,
stoichiometric reactions of isolated c
249 The structure and
kinetic analysis suggest that ATP sulfurylase overcomes
250 Kinetic analysis suggested a minor and variable contribu
251 Molecular
kinetic analysis suggested that intravascular taste sens
252 In depth
kinetic analysis suggested that loss of catalytic activi
253 Kinetic analysis suggests that binding of AM-8138 to the
254 Kinetic analysis suggests that hFACT decreases the lifet
255 The
kinetic analysis suggests that prolyl cis --> trans isom
256 Molecular modeling studies combined with
kinetic analysis supported favorable interaction with th
257 The most successful binding
kinetics analysis systems at this moment include surface
258 This prevents successful application of
kinetic analysis techniques and causes semiquantitative
259 AUC) provides a better correlation with full
kinetic analysis than does standard SUV.
260 By using the micro-
kinetic analysis,
the CO oxidation following the tri-mol
261 irected mutagenesis followed by steady state
kinetic analysis to ascertain their catalytic functions
262 e key microscopic steps by applying a global
kinetic analysis to both the decrease in the concentrati
263 L-nucleotides and performed pre-steady-state
kinetic analysis to determine the D-stereoselectivity me
264 ition of highly reproducible data and global
kinetic analysis to determine the mechanistic influence
265 erial species were subjected to steady-state
kinetic analysis to determine their specificities toward
266 We carried out pre-steady-state
kinetic analysis to elucidate the kinetic mechanism of t
267 e performed a detailed non-linear regression
kinetic analysis to simultaneously fit families of subst
268 we therefore extend contemporary statistical
kinetic analysis to study collective transport phenomena
269 Here we used a combination of fine
kinetic analysis under specific conditions (pH, PN conce
270 Kinetic analysis underscored the importance of motif 1a
271 to evaluate (18)F-AV-1451 binding with full
kinetic analysis using a metabolite-corrected arterial i
272 -site affinity label, together with detailed
kinetic analysis using a variety of well defined oligosa
273 isition times were tested comparatively to a
kinetic analysis using MRTM2.
274 Here, we demonstrate by
kinetic analysis using physically tethered DNA substrate
275 In vitro
kinetic analysis using purified protein demonstrated tha
276 uptake (BPND), was measured in subjects with
kinetic analysis using the arterial input function both
277 ntration (VT) were measured in subjects with
kinetic analysis using the arterial input function.
278 tal cortical subdivisions) was measured with
kinetic analysis using the arterial input function.
279 Ferricyanide reduction
kinetic analysis (
variation of ferricyanide absorption w
280 ding potential (BPND) obtained from the full
kinetic analysis was compared with the SUVR and with non
281 Kinetic analysis was conducted to evaluate the dose-resp
282 residue impacts the L-glutaminase property,
kinetic analysis was coupled with crystal structure dete
283 In this article, the reliability of the
kinetic analysis was improved by obtaining steady-state
284 Third, a preliminary
kinetic analysis was performed using the radiometabolite
285 Reaction progress
kinetic analysis was performed, shedding light on a poss
286 egistered magnetic resonance images and full
kinetic analysis was performed.
287 KIF3A and KIF3B stepping, a presteady-state
kinetic analysis was pursued.
288 glucose uptake with (18)F-FDG PET and Patlak
kinetic analysis was systematically assessed using the v
289 A sigmoidal response
kinetic analysis was used to calculate both the diffusio
290 Stopped flow
kinetic analysis was used to confirm this prediction.
291 Reaction progress
kinetic analysis was used to obtain insight into the mec
292 Using real-time
kinetic analysis we show that mcm(5)-modified tRNA(Lys)
293 From the structural and
kinetic analysis,
we can state that five residues at pos
294 ingle-molecule fluorescence measurements and
kinetic analysis,
we find that the reaction in solution
295 ents, chemical shift perturbation and enzyme
kinetic analysis,
we provide structural insights into th
296 Through the use of mutagenesis and
kinetic analysis,
we show that the active site of ssNAT
297 The results of
kinetic analysis were confirmed by chronocoulometry meth
298 rast, decreasing time-activity curves in the
kinetic analysis were highly prognostic for shorter prog
299 Site-directed mutagenesis and
kinetic analysis with substrate analogs revealed the rol
300 ction and to compare parameters derived from
kinetic analysis with SUV ratio (SUVR) calculated over d