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1 t a time corresponding to duplication of the kinetoplast DNA.
2 oci, and uniparental retention of maxicircle kinetoplast DNA.
3 ce-based genomic library for cells that lose kinetoplast DNA.
4 necropsy material, using probes specific for kinetoplast DNA.
5 mitochondrion, above and below the condensed kinetoplast DNA.
6 c properties that target the minor groove of kinetoplast DNA.
8 terference (RNAi) of p38 resulted in loss of kinetoplast DNA and accumulation of a novel free minicir
9 than wild type topo II alpha in decatenating kinetoplast DNA and also exhibits a 2-4-fold decrease in
10 approximately equal distribution of parental kinetoplast DNA between daughter kinetoplasts resulted i
13 ocalized in the antipodal sites flanking the kinetoplast DNA disk, as previously shown in C. fascicul
14 A time-course of DNA synthesis (nuclear and kinetoplast DNA), duplication of organelles (basal body,
15 ctivity as judged by an inability to convert kinetoplast DNA from the catenated to the decatenated fo
19 ondrial genome of Trypanosoma brucei, called kinetoplast DNA, is a network of topologically interlock
22 ically amplified whole linearized minicircle kinetoplast DNA (kDNA) of the Leishmania subgenus Vianni
23 sitivity of OligoC-TesT with those of nested kinetoplast DNA (kDNA) PCR, nested internal transcribed
25 mitochondrial genome of trypanosomes, termed kinetoplast DNA (kDNA), contains thousands of minicircle
30 otype upon induction of RNAi was the loss of kinetoplast DNA (kDNA), the cell's catenated mitochondri
43 fit the working hypothesis that the loss of kinetoplast DNA leads to a respiratory defect which then
47 mania parasite contains approximately 10,000 kinetoplast DNA minicircles, which are unequally distrib
48 ing the 75 C-terminal amino acids can rescue kinetoplast DNA missegregation but not the lack of ATOM
49 ultimately led to shrinkage and loss of the kinetoplast DNA network and cessation of growth of the c
51 ed guide RNAs upon segregation of the single kinetoplast DNA network into daughter cells at cell divi
52 tions with respect to the mitochondrial DNA (kinetoplast DNA network) in this organism are strikingly
54 The final disappearance of the stainable kinetoplast DNA occurred at a cell division in which all
55 ween the complex restriction patterns of the kinetoplast DNA of any of the parasites from Timargara c
59 these results suggest a point of control for kinetoplast DNA replication through the regulation of th
60 tor with genes involved in processes such as kinetoplast DNA replication, mitochondrial mRNA synthesi
71 ed by proteolysis of a helicase; the complex kinetoplast DNA system yields a clear view of how mitoch
72 We evaluated the serum levels of T. cruzi kinetoplast DNA (TckDNA), T. cruzi 18S ribosomal DNA (Tc
73 have an unusual mitochondrial genome, called kinetoplast DNA, that is a giant network containing thou
78 g region of the TOP2 gene, which encodes the kinetoplast DNA topoisomerase, and have carried out dele
79 two kinetoplast ribosomal proteins with the kinetoplast DNA was observed by immunofluorescence, sugg
80 division in which all the remaining visible kinetoplast DNA was retained by one of the daughter cell
81 parasitic heterocyclic dications can have on kinetoplast DNA, we have constructed ligation ladders, w
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