ライフサイエンスコーパス: koseri

コーパス検索結果 (１語後でソート)

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1 ting microglial activation in response to C. koseri.

2 r regulation of one such gene in Citrobacter koseri.

3 wth of Escherichia coli K1/r and Citrobacter koseri.

4 in UTI89 and by enteric bacteria Citrobacter koseri and Salmonella enterica serovar typhimurium.

5 nce was 97.8% similar to that of Citrobacter koseri but 97.0% similar to that of Enterobacter cloacae

6 In this study, we showed that C. koseri causes meningitis and brain abscesses in the neon

7 pneumoniae, Klebsiella oxytoca, Citrobacter koseri, Citrobacter freundii group, Enterobacter spp., a

8 f a novel bacterial homolog from Citrobacter koseri, CLC-ck2, has yielded surprising discoveries abou

9 4 mutant and MyD88 KO microglia following C. koseri exposure, indicating a contribution of TLR4- and

10 Gut-associated Citrobacter koseri genomes harbored 47 polymorphic sites that we use

11 brain parenchyma, microglial responses to C. koseri have not yet been examined.

12 n macrophages in vitro and that uptake of C. koseri increases in the presence of human pooled serum i

13 ng exposure to either live or heat-killed C. koseri, indicating a critical role for both TLR4- and My

14 ns) were the primary target for long-term C. koseri infection.

15 marily facilitates the entry of opsonized C. koseri into macrophages.

16 Citrobacter koseri is a Gram-negative bacterium that can cause a hig

17 A unique feature of Citrobacter koseri is the extremely high propensity to initiate brai

18 n and that more than 90% of intracellular C. koseri organisms are colocalized within phagolysosomes.

19 To better understand C. koseri pathogenesis, we have characterized the interacti

20 These lesions were caused by Citrobacter koseri septicaemia, identified by transfontanelle ultras

21 n macrophages may be a mechanism by which C. koseri subverts the host response and elicits chronic in

22 We found that C. koseri survives and replicates within macrophages in vit

23 fluorescence microscopy demonstrates that C. koseri survives phagolysosomal fusion and that more than

24 The ability of C. koseri to survive phagolysosome fusion and replicate wit

25 udies lend support to the hypothesis that C. koseri uses morphologically different methods of uptake

26 Interestingly, C. koseri was capable of surviving intracellularly in both

27 entration [IC(50)], approximately 10 nM), C. koseri was intermediate (IC(50), approximately 1,000 nM)

28 Salmonella typhimurium LT2, and Citrobacter koseri were able to cross-seed in vitro.

29 we have characterized the interactions of C. koseri with human macrophages.

30 lts demonstrate that microglia respond to C. koseri with the robust expression of proinflammatory mol

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