ライフサイエンスコーパス: kyphosis

1 ld, short stature with long fingers and mild kyphosis.

2 hands, muscular hypotonia, and thoracolumbar kyphosis.

3 lular matrix, moderate chondrodysplasia, and kyphosis.

4 d its clinical manifestations, fractures and kyphosis.

5 sion of the mandible beyond the maxilla, and kyphosis.

6 These animals were also phenotyped for kyphosis.

7 include increased fracture rates and severe kyphosis.

8 counts while 16 regions were significant for kyphosis.

9 had moderate chondrodysplasia, and developed kyphosis.

10 rks of underdeveloped musculature, including kyphosis, 20% reduction in body mass, and 34% reduction

11 ite adipose tissue, reduced muscle strength, kyphosis, alopecia, hypothermia and shortened lifespan.

12 Eed deficiency in chondrocytes causes severe kyphosis and a growth defect with decreased chondrocyte

13 tended their longevity by threefold, reduced kyphosis and the development of muscle disease, and main

14 ephropathy, progressive edema, splenomegaly, kyphosis and urinary excretion of sialylated oligosaccha

15 TL regions and no region was associated with kyphosis and vertebra counts.

16 rolisthesis, osteophytic spurring, lordosis, kyphosis, and nerve root impingement.

17 -) mice exhibited severe growth retardation, kyphosis, and premature death, closely resembling the ph

18 ncreased anxiety-related behavior, seizures, kyphosis, and stereotypic forelimb motions.

19 The mean kyphosis angle (+/- SD) for this group was markedly abno

20 re events were confirmed by radiography, and kyphosis angles were measured by using the Cobb method.

21 spondylolisthesis, and surgical treatment of kyphosis are also reviewed.

22 ignated supermodel, that caused leanness and kyphosis associated with myopathy and adipocyte defects

23 veloped central nervous system and eyes, and kyphosis at exposures of 0.5 mg/L.

24 , reduced skin fat deposition, osteoporosis, kyphosis, cachexia, and premature death.

25 In age-adjusted models, each SD increase in kyphosis carried a 1.14-fold increased risk for death (9

26 Low bone density, fractures, and kyphosis complicate the lives of adults with cystic fibr

27 t involvement, bisphosphonate use, and local kyphosis did not confer increased risk.

28 A video that contrasts kyphosis, gait, joint contractures, and mobility in mdx/

29 tions, resulting in progressive dwarfism and kyphosis in mice.

30 followed by morphometric analysis revealed a kyphosis in mutant animals.

31 to bone loss and its sequelae, fractures and kyphosis, in patients with lung disease, many of whom al

32 Excessive kyphosis may be associated with earlier mortality, but p

33 elopment of ALS-related phenotypes including kyphosis, motor dysfunctions, muscle weakness/atrophy, m

34 ion and exercises, whereas patients who have kyphosis of up to 75 degrees and how have growth remaini

35 ators of senescence (alopecia, skin atrophy, kyphosis, osteoporosis, testicular atrophy, lipofuscin a

36 ave muscle hypotonia, delayed ambulation, or kyphosis, pointing to an underlying skeletal muscle defe

37 ound head, mid-face hypoplasia at birth, and kyphosis progression during postnatal development.

38 Skeletal defects include kyphosis, scoliosis, crooked tails and curvature and ove

39 h premature aging-like phenotypes, including kyphosis, severe muscle wasting, hypogonadism, osteopeni

40 muscle weakness, histological abnormalities, kyphosis (spinal curvature), and aberrant kinetics of mu

41 tractures, pronounced growth retardation and kyphosis, suggesting that dystrophin and utrophin play c

42 (-/-) mice display progressive scoliosis and kyphosis, tibial bowing and abnormalities in skull and a

43 s consistent with premature aging, including kyphosis, uncoordinated movement, hypogonadism, infertil

44 re known to severely reduce suckling-induced kyphosis (upright crouched nursing), which is necessary

45 Less genomic variation for kyphosis was described by QTL regions and no region was

46 female rats, the possible role of the PAG in kyphosis was investigated using c-fos immunocytochemistr

47 lent vertebral fractures, women with greater kyphosis were at increased risk for earlier death (relat

48 accelerated hypertrophic differentiation and kyphosis, whereas the overactivation of TGF-beta signall

49 patient progressively developed very severe kyphosis with resultant severe disability.

50 raniofacial dysplasia, chondrodysplasia, and kyphosis, with most mice dying by d 5 of age.