1 The most common initial drug was
labetalol (
48%), followed by nicardipine (15%), hydralaz
2 adrenergic receptor blockers (phentolamine,
labetalol),
a calcium channel blocker (nifedipine), and
3 Labetalol,
a combined alpha1, beta1, and beta2 adrenocep
4 surements were repeated 10 and 30 mins after
labetalol administration.
5 wering of blood pressure with lisinopril and
labetalol after acute stroke seems to be a promising app
6 Labetalol and lisinopril are effective antihypertensive
7 phase, the separation of the beta-blockers,
labetalol and sotalol, and the binaphthyl derivatives, 1
8 Prior administration of phentolamine,
labetalol,
and nitroglycerine prevented the phenylephrin
9 re treated with phenylephrine, phentolamine,
labetalol,
and nitroglycerine.
10 he mixed alpha-/beta-adrenoceptor antagonist
labetalol,
and the alpha1-adrenoceptor antagonist prazos
11 f mixed adrenoceptor blockers carvedilol and
labetalol,
and the atypical antipsychotic clozapine, in
12 e outcomes were found between flecainide and
labetalol antiarrhythmic effects in vitro and the clinic
13 a mediator, while no significant removal of
labetalol can be achieved in the absence of ABTS.
14 Our experiments indicate that
labetalol can be effectively transformed by laccase-cata
15 he widespread occurrence of the beta-blocker
labetalol causes environmental health concern.
16 This indicates that
labetalol enhances GABAergic synaptic transmission by a
17 clude nitroprusside, diazoxide, hydralazine,
labetalol,
esmolol, nicardipine, nifedipine, enalaprilat
18 splenic, and pulmonary blood pools, whereas
labetalol increased only the pulmonary blood pool.
19 Based on these data, we postulate that
labetalol-
induced analgesia is at least in part ascribed
20 In the presence of metoprolol,
labetalol-
induced increase in sIPSC frequency was signif
21 These data indicate that
labetalol-
induced inhibition of PAG cell firing is attri
22 ternet central randomisation to receive oral
labetalol,
lisinopril, or placebo if they were non-dysph
23 anted cell entry in liver sinusoids, whereas
labetalol,
nifedipine, CGRP, and glucagon were ineffecti
24 mental information for laccase-ABTS mediated
labetalol reactions and the effect of graphene, which co
25 Administration of
labetalol resulted in a decrease in MAP (mm Hg+/-SEM) in
26 Using patch clamp techniques, we found that
labetalol reversibly increases the frequency of sIPSCs w
27 We further showed that
labetalol reversibly reduced the firing rate of PAG neur
28 These results suggest that
labetalol shares the same pathway as metoprolol in enhan
29 o if they were non-dysphagic, or intravenous
labetalol,
sublingual lisinopril, or placebo if they had
30 Intravenous
labetalol was administered 90 mins after administration
31 rately inhibiting skin vasoconstriction, and
labetalol was ineffective.
32 Dobutamine and
labetalol were titrated to vary VO2 (range 204 to 584 mL
33 which has a much greater reactivity towards
labetalol when graphene is present.
34 Furthermore,
Labetalol,
which is marketed for hypertension as a nonse