ライフサイエンスコーパス: laboratory mice

1 ructure of genetic variation among classical laboratory mice.

2 ng-term protective immunity in a majority of laboratory mice.

3 erences in the general learning abilities of laboratory mice.

4 igen of the HGE agent and was infectious for laboratory mice.

5 havior-->gene) approaches in both humans and laboratory mice.

6 responsible for the xenotropism of X-MLVs in laboratory mice.

7 d strain]), were studied in cell culture and laboratory mice.

8 ose previously identified using conventional laboratory mice.

9 activity, as well as the ability to colonize laboratory mice.

10 well as xenotropic MLV, which do not infect laboratory mice.

11 RNA virus is thus maintained indefinitely in laboratory mice.

12 al inoculation of the spleen homogenate into laboratory mice.

13 gulatory effect of TNF on T cells in healthy laboratory mice.

14 etween M. castaneus and MCF MuLV-susceptible laboratory mice.

15 al inflammation were not observed in Jackson Laboratory mice.

16 aced mating paradigm, in female but not male laboratory mice.

17 at establishes acute and latent infection in laboratory mice.

18 els after acute injury in several strains of laboratory mice.

19 els is widely used to initiate thrombosis in laboratory mice.

20 (-) mice, which include commonly used inbred laboratory mice.

21 mals except most of the classical strains of laboratory mice.

22 at establishes acute and latent infection in laboratory mice.

23 ria parasite Plasmodium chabaudi evolving in laboratory mice.

24 osed to BD and its potent carcinogenicity in laboratory mice.

25 VP establishes acute and latent infection in laboratory mice.

26 n minimize a widespread source of anxiety in laboratory mice.

27 pansions found in old specific pathogen-free laboratory mice.

28 ighly activated myeloid cells not present in laboratory mice.

29 to establish virus latency in the spleens of laboratory mice.

30 t a mean r(2) value of 0.98 in 2,073 outbred laboratory mice (0.15x sequencing coverage).

31 ne the rodent malaria Plasmodium chabaudi in laboratory mice, a parasite-host system in which virulen

32 re acquired by the wild mouse progenitors of laboratory mice about 1 million years ago.

33 ns that are inherited as Mendelian traits in laboratory mice affect susceptibility to spontaneous TGC

34 quenced erp loci of bacteria reisolated from laboratory mice after 1 year of infection and found them

35 se microbial experience, and were induced in laboratory mice after co-housing with pet store mice, su

36 f the range of germline mutations induced in laboratory mice after parental exposure to ionizing radi

37 Laboratory mice and 2 species of bats were exposed, thro

38 Immune responses differ between laboratory mice and humans.

39 hin populations of wild and laboratory fish, laboratory mice and humans.

40 ort genome sequences of 17 inbred strains of laboratory mice and identify almost ten times more varia

41 s) are found in the common inbred strains of laboratory mice and in the house mouse subspecies ofMus

42 However, studies on laboratory mice and nonhuman primates revealed ambiguous

43 onstrate that the low recombination rates in laboratory mice and rats reflect a more general reductio

44 esembles WS2 than comparable Mitf alleles in laboratory mice and rats, which are expressed as purely

45 y the onset of age-associated pathologies in laboratory mice and rats.

46 lived species, from unicellular organisms to laboratory mice and rats.

47 bits parental behaviors and social memory in laboratory mice and rats.

48 We utilize laboratory mice and their innate inter-individual differ

49 ly shown to contribute to Fv1 restriction in laboratory mice, and 3 codons in a 10-codon segment over

50 e seen in blood from pet store-raised versus laboratory mice, and adult versus cord blood in humans.

51 N) virus causes fatal meningoencephalitis in laboratory mice, and gammadelta T cells are involved in

52 1 resistance alleles have been identified in laboratory mice, and they target virus capsid genes to p

53 ites are common in humans, but are absent in laboratory mice, and thus represent potential contributo

54 eri and the spirochetemia of B. hispanica in laboratory mice, apolipoprotein E (apoE)-deficient and l

55 veral methods to model human Ph+ leukemia in laboratory mice are available, including propagation of

56 Together, our results demonstrate that laboratory mice are capable of exhibiting dynamic and ac

57 The alpha1-protease inhibitor proteins of laboratory mice are homologous in sequence and function

58 indicate that diverse learning abilities of laboratory mice are influenced by a common source of var

59 e data suggest that the cold stress to which laboratory mice are ubiquitously subjected profoundly af

60 nt research that calls into question the way laboratory mice are used to address questions in basic s

61 Although laboratory mice are usually highly susceptible to Yersin

62 Laboratory mice are valuable models of HGE agent infecti

63 Using rodent malaria in laboratory mice as a model system and the statistical fr

64 comprehensive basis for validating (or not) laboratory mice as a useful and relevant immunological m

65 family are not as numerous in the genomes of laboratory mice as are members of the older A and F subf

66 Laboratory mice bearing inactivating mutations in the ge

67 ne leukemia viruses cannot infect cells from laboratory mice because of the lack of a functional cell

68 signal transduction and for colonization of laboratory mice by C. rodentium.

69 The isolate was pathogenic for laboratory mice by the intracerebral and intramuscular r

70 Laboratory mice carry mouse leukemia viruses (MLVs) of t

71 fferentiation are derived from pathogen-free laboratory mice challenged with a single pathogen or vac

72 sters identified in humans, chimpanzees, and laboratory mice, characterized by differences in Bactero

73 estoring physiological microbial exposure in laboratory mice could provide a relevant tool for modell

74 The laboratory mice developed clinical signs and splenomegal

75 In the laboratory, mice dig vigorously in deep bedding such as

76 t may reside in the challenges, which normal laboratory mice do not encounter.

77 However, there is growing concern that laboratory mice do not reflect relevant aspects of the h

78 ion define three MLV host range subgroups in laboratory mice: ecotropic, polytropic, and xenotropic M

79 e report that the retinas of some strains of laboratory mice exhibit robust circadian rhythms of mela

80 oxyadenosine (1,N(6)-HMHP-dA), in tissues of laboratory mice exposed to 6.25-625 ppm BD.

81 arameters of wild mice and compare them with laboratory mice, finding that wild mouse cellular immune

82 odentium espB mutant also failed to colonize laboratory mice following experimental inoculation.

83 In recent years in silico analysis of common laboratory mice has been introduced and subsequently app

84 Research using laboratory mice has led to fundamental insights into the

85 s analysis of the recombination landscape in laboratory mice has revealed that the different subspeci

86 Studies on laboratory mice have demonstrated that host genetics can

87 Laboratory mice have longer telomeres relative to humans

88 Genetic crosses of laboratory mice have provided extensive information abou

89 Standard housing temperature for laboratory mice in research facilities is mandated to be

90 -thermoneutral housing temperatures used for laboratory mice in research institutes is sufficient to

91 permissive Xpr1(sxv) allele in 7 strains of laboratory mice, including a Bxv1-positive strain, F/St,

92 p. nov., which was isolated from colonies of laboratory mice independently by two laboratories.

93 se results suggest that unlike pathogen-free laboratory mice, infection or immunization of latently i

94 ptor for entry, and the unique resistance of laboratory mice is due to two mutations in different put

95 Standard Mus musculus laboratory mice lack a functional XPR1 receptor for XMRV

96 Laboratory mice--like newborn, but not adult, humans--la

97 Laboratory mice live in abnormally hygienic specific pat

98 In humans and certain strains of laboratory mice, male tissue is recognized as nonself an

99 , if not most, TCE in specific pathogen-free laboratory mice may be Ag-independent.

100 In laboratory mice (Mus musculus domesticus), alpha(1)-PI o

101 Laboratory mice (Mus musculus) have long telomeres, alth

102 in Western Europe, which is unable to infect laboratory mice (Mus sp.) without the aid of powerful im

103 -type Mx1 gene (Mx1+/+) differ from standard laboratory mice (Mx1-/-) in being highly resistant to in

104 ety of cell lines and is also able to infect laboratory mice, offering an ideal model with which to s

105 ablish acute and persistent infection within laboratory mice offers a unique opportunity to investiga

106 MOLF/EiJ mice, which diverged from classical laboratory mice over a million years ago.

107 art and was transferred to and maintained in laboratory mice over several generations.

108 tanding of immunology was largely defined in laboratory mice, partly because they are inbred and gene

109 Laboratory mice provide a ready source of diverse, high-

110 Laboratory mice reconstituted with natural microbiota ex

111 e function, but how well immune responses of laboratory mice reflect those of free-living animals is

112 ckcrossing of wild mice with knockout mutant laboratory mice retrieves behavioural traits exhibited e

113 metaanalysis of data from 54 experiments on laboratory mice reveals that basic ecological rules gove

114 All inbred strains and outbred stocks of laboratory mice studied to date have been found to be su

115 In laboratory mice, TGCTs arise from primordial germ cells

116 We further find that selection is weaker in laboratory mice than in humans and it does not affect th

117 imulated a search for IRG alleles unknown in laboratory mice that might confer resistance to virulent

118 of intense study of coat color mutations in laboratory mice, thereby creating an impressive list of

119 N) virus causes fatal meningoencephalitis in laboratory mice, thereby partially mimicking human disea

120 The 12 X-MLV ERVs predate the origins of laboratory mice; they were all traced to Japanese wild m

121 Exposure of laboratory mice to carbon nanotubes mimics exposure to a

122 ed to be a major cause for the resistance of laboratory mice to JUNV infection.

123 te the prospects for using dense SNP maps in laboratory mice to refine previous QTL regions and ident

124 at captures 90% of the known variation among laboratory mice, to identify the genetic loci controllin

125 ise quantitation of bis-N7G-BD in tissues of laboratory mice treated with low ppm and subppm concentr

126 Because laboratory mice vary widely in their proviral contents a

127 A yeast causing widespread infection of laboratory mice was identified from 26S rRNA gene sequen

128 ty of Bb(DeltaA66)-infected nymphs to infect laboratory mice was significantly impaired compared to t

129 However, laboratory mice we use for most biomedical studies are b

130 Using laboratory mice, we artificially selected for high maxim

131 tivation of the ribosomal protein S6 gene in laboratory mice, we found that reduced ribosomal protein

132 Applying EDGE to laboratory mice, we show that a loss-of-function mutatio

133 Transmission studies in laboratory mice were negative.

134 types are found in the classical strains of laboratory mice, which are genetic mosaics of 3 wild mou

135 single nucleotide polymorphisms (SNPs) from laboratory mice, which are largely genetic hybrids betwe

136 This effect was not observed in Jackson Laboratory mice, which are not colonized with SFB.

137 Laboratory mice, while paramount for understanding basic

138 tis virus (VEEV) is highly virulent in adult laboratory mice, while Sindbis virus (SINV) is avirulent

139 Hence, compared with laboratory mice, wild-derived mutant mice constitute an

140 Infection of laboratory mice with C. rodentium provides a useful in-v

141 Mutant laboratory mice with distinctive hair phenotypes are use

142 To test this, we sequentially infected laboratory mice with herpesviruses, influenza, and an in

143 ablish high-titer hepatotropic infections in laboratory mice with immunological features resembling t

144 Laboratory mice with over half a megabase of DNA upstrea

145 Infection of different strains of laboratory mice with the agent of Lyme disease, Borrelia

146 Given the difficulty of infecting laboratory mice with these diarrhea-causing pathogens, a